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Auteur Thalia C. ELEY |
Documents disponibles écrits par cet auteur (18)
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I think, therefore I am: a twin study of attributional style in adolescents / Jennifer Y.F. LAU in Journal of Child Psychology and Psychiatry, 47-7 (July 2006)
[article]
Titre : I think, therefore I am: a twin study of attributional style in adolescents Type de document : Texte imprimé et/ou numérique Auteurs : Jennifer Y.F. LAU, Auteur ; Thalia C. ELEY, Auteur ; Frühling V. RIJSDIJK, Auteur Année de publication : 2006 Article en page(s) : p.696–703 Langues : Anglais (eng) Mots-clés : Attributional-style depression parental-discipline adolescence genetics Index. décimale : PER Périodiques Résumé : Background: Parenting factors may be important to the development of attributional style in adolescence, which in turn relates to depression symptoms. These relationships have mainly been considered in terms of social risk mechanisms, and little is known about the role of genetic influences.
Method: Self-reported measures of attributional style, depression symptoms and parental disciplinary styles were administered to over 1300 adolescent twin and sibling pairs. Model-fitting techniques were used to examine the role of genetic and environmental influences.
Results: Moderate genetic influences on attributional style were demonstrated, and furthermore, its association with depression reflected considerable genetic effects. Familial factors were implicated in the association between attributional style and punitive parenting, although genetic from shared environmental causes could not be distinguished.
Conclusions: Our results demonstrate that attributional style is influenced by genetic, as well as social factors. Implications for aetiological pathways integrating cognitive, genetic and social factors on adolescent depression are discussed.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2005.01532.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=754
in Journal of Child Psychology and Psychiatry > 47-7 (July 2006) . - p.696–703[article] I think, therefore I am: a twin study of attributional style in adolescents [Texte imprimé et/ou numérique] / Jennifer Y.F. LAU, Auteur ; Thalia C. ELEY, Auteur ; Frühling V. RIJSDIJK, Auteur . - 2006 . - p.696–703.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 47-7 (July 2006) . - p.696–703
Mots-clés : Attributional-style depression parental-discipline adolescence genetics Index. décimale : PER Périodiques Résumé : Background: Parenting factors may be important to the development of attributional style in adolescence, which in turn relates to depression symptoms. These relationships have mainly been considered in terms of social risk mechanisms, and little is known about the role of genetic influences.
Method: Self-reported measures of attributional style, depression symptoms and parental disciplinary styles were administered to over 1300 adolescent twin and sibling pairs. Model-fitting techniques were used to examine the role of genetic and environmental influences.
Results: Moderate genetic influences on attributional style were demonstrated, and furthermore, its association with depression reflected considerable genetic effects. Familial factors were implicated in the association between attributional style and punitive parenting, although genetic from shared environmental causes could not be distinguished.
Conclusions: Our results demonstrate that attributional style is influenced by genetic, as well as social factors. Implications for aetiological pathways integrating cognitive, genetic and social factors on adolescent depression are discussed.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2005.01532.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=754 Interpersonal cognitive biases as genetic markers for pediatric depressive symptoms: Twin data from the Emotions, Cognitions, Heredity and Outcome (ECHO) study / Jennifer Y. F. LAU in Development and Psychopathology, 26-4 (Part 2) (November 2014)
[article]
Titre : Interpersonal cognitive biases as genetic markers for pediatric depressive symptoms: Twin data from the Emotions, Cognitions, Heredity and Outcome (ECHO) study Type de document : Texte imprimé et/ou numérique Auteurs : Jennifer Y. F. LAU, Auteur ; Stefano R. BELLI, Auteur ; Alice M. GREGORY, Auteur ; Thalia C. ELEY, Auteur Année de publication : 2014 Article en page(s) : p.1267-1276 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Childhood depressive symptoms may arise from genetic and environmental risks, which act to bias the ways in which children process emotional information. Previous studies show that several “cognitive biases” are heritable and share genetic and environmental risks with depressive symptoms. Past research suggests that many cognitive biases only reflect genetic risks for depressive symptoms from adolescence. The present study sought to identify (a) when interpersonal cognitions mature as risk factors for depressive symptoms by examining whether these factors are stable and predict symptoms across time in childhood, and (b) the extent to which interpersonal cognitions reflect inherited/environmental risks on children's depressive symptoms. Results showed that there was some stability for interpersonal cognitive biases from age 8 to 10 years (rs = .32–.43). Only the absence of positive self/other perceptions, and negative peer and mother expectations at age 8 predicted depressive symptoms at age 10 (after controlling for depressive symptoms at age 8). The absence of positive self/other perceptions shared genetic influences with depressive symptoms within and across time. Across middle to late childhood, interpersonal cognitions begin to operate as vulnerability-trait factors for depressive symptoms, gradually reflecting distal genetic risks on symptoms. En ligne : http://dx.doi.org/10.1017/S0954579414001011 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=245
in Development and Psychopathology > 26-4 (Part 2) (November 2014) . - p.1267-1276[article] Interpersonal cognitive biases as genetic markers for pediatric depressive symptoms: Twin data from the Emotions, Cognitions, Heredity and Outcome (ECHO) study [Texte imprimé et/ou numérique] / Jennifer Y. F. LAU, Auteur ; Stefano R. BELLI, Auteur ; Alice M. GREGORY, Auteur ; Thalia C. ELEY, Auteur . - 2014 . - p.1267-1276.
Langues : Anglais (eng)
in Development and Psychopathology > 26-4 (Part 2) (November 2014) . - p.1267-1276
Index. décimale : PER Périodiques Résumé : Childhood depressive symptoms may arise from genetic and environmental risks, which act to bias the ways in which children process emotional information. Previous studies show that several “cognitive biases” are heritable and share genetic and environmental risks with depressive symptoms. Past research suggests that many cognitive biases only reflect genetic risks for depressive symptoms from adolescence. The present study sought to identify (a) when interpersonal cognitions mature as risk factors for depressive symptoms by examining whether these factors are stable and predict symptoms across time in childhood, and (b) the extent to which interpersonal cognitions reflect inherited/environmental risks on children's depressive symptoms. Results showed that there was some stability for interpersonal cognitive biases from age 8 to 10 years (rs = .32–.43). Only the absence of positive self/other perceptions, and negative peer and mother expectations at age 8 predicted depressive symptoms at age 10 (after controlling for depressive symptoms at age 8). The absence of positive self/other perceptions shared genetic influences with depressive symptoms within and across time. Across middle to late childhood, interpersonal cognitions begin to operate as vulnerability-trait factors for depressive symptoms, gradually reflecting distal genetic risks on symptoms. En ligne : http://dx.doi.org/10.1017/S0954579414001011 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=245 A multivariate genetic analysis of anxiety sensitivity, environmental sensitivity and reported life events in adolescents / Alicia J. PEEL in Journal of Child Psychology and Psychiatry, 64-2 (February 2023)
[article]
Titre : A multivariate genetic analysis of anxiety sensitivity, environmental sensitivity and reported life events in adolescents Type de document : Texte imprimé et/ou numérique Auteurs : Alicia J. PEEL, Auteur ; Olakunle OGINNI, Auteur ; Elham ASSARY, Auteur ; Georgina KREBS, Auteur ; Celestine LOCKHART, Auteur ; Thomas MCGREGOR, Auteur ; Elisavet PALAIOLOGOU, Auteur ; Angelica RONALD, Auteur ; Andrea DANESE, Auteur ; Thalia C. ELEY, Auteur Article en page(s) : p.289-298 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Background Despite being considered a measure of environmental risk, reported life events are partly heritable. One mechanism that may contribute to this heritability is genetic influences on sensitivity, relating to how individuals process and interpret internal and external signals. The aim of this study was to explore the genetic and environmental overlap between self-reported life events and measures of sensitivity. Methods At age 17, 2,939 individuals from the Twins Early Development Study (TEDS) completed measures of anxiety sensitivity (Children's Anxiety Sensitivity Index), environmental sensitivity (Highly Sensitive Child Scale) and reported their experience of 20 recent life events. Using multivariate Cholesky decomposition models, we investigated the shared genetic and environmental influences on the associations between these measures of sensitivity and the number of reported life events, as well as both negative and positive ratings of life events. Results The majority of the associations between anxiety sensitivity, environmental sensitivity and reported life events were explained by shared genetic influences (60%-75%), with the remainder explained by nonshared environmental influences (25%-40%). Environmental sensitivity showed comparable genetic correlations with both negative and positive ratings of life events (rAÂ = .21 and .15), anxiety sensitivity only showed a significant genetic correlation with negative ratings of life events (rAÂ = .33). Approximately 10% of the genetic influences on reported life events were accounted for by influences shared with anxiety sensitivity and environmental sensitivity. Conclusion Differences in how individuals process the contextual aspects of the environment or interpret their own physical and emotional response to environmental stimuli may be one mechanism through which genetic liability influences the subjective experience of life events. En ligne : https://doi.org/10.1111/jcpp.13725 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492
in Journal of Child Psychology and Psychiatry > 64-2 (February 2023) . - p.289-298[article] A multivariate genetic analysis of anxiety sensitivity, environmental sensitivity and reported life events in adolescents [Texte imprimé et/ou numérique] / Alicia J. PEEL, Auteur ; Olakunle OGINNI, Auteur ; Elham ASSARY, Auteur ; Georgina KREBS, Auteur ; Celestine LOCKHART, Auteur ; Thomas MCGREGOR, Auteur ; Elisavet PALAIOLOGOU, Auteur ; Angelica RONALD, Auteur ; Andrea DANESE, Auteur ; Thalia C. ELEY, Auteur . - p.289-298.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 64-2 (February 2023) . - p.289-298
Index. décimale : PER Périodiques Résumé : Background Despite being considered a measure of environmental risk, reported life events are partly heritable. One mechanism that may contribute to this heritability is genetic influences on sensitivity, relating to how individuals process and interpret internal and external signals. The aim of this study was to explore the genetic and environmental overlap between self-reported life events and measures of sensitivity. Methods At age 17, 2,939 individuals from the Twins Early Development Study (TEDS) completed measures of anxiety sensitivity (Children's Anxiety Sensitivity Index), environmental sensitivity (Highly Sensitive Child Scale) and reported their experience of 20 recent life events. Using multivariate Cholesky decomposition models, we investigated the shared genetic and environmental influences on the associations between these measures of sensitivity and the number of reported life events, as well as both negative and positive ratings of life events. Results The majority of the associations between anxiety sensitivity, environmental sensitivity and reported life events were explained by shared genetic influences (60%-75%), with the remainder explained by nonshared environmental influences (25%-40%). Environmental sensitivity showed comparable genetic correlations with both negative and positive ratings of life events (rAÂ = .21 and .15), anxiety sensitivity only showed a significant genetic correlation with negative ratings of life events (rAÂ = .33). Approximately 10% of the genetic influences on reported life events were accounted for by influences shared with anxiety sensitivity and environmental sensitivity. Conclusion Differences in how individuals process the contextual aspects of the environment or interpret their own physical and emotional response to environmental stimuli may be one mechanism through which genetic liability influences the subjective experience of life events. En ligne : https://doi.org/10.1111/jcpp.13725 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492 Normative childhood repetitive routines and obsessive compulsive symptomatology in 6-year-old twins / Derek BOLTON in Journal of Child Psychology and Psychiatry, 50-9 (September 2009)
[article]
Titre : Normative childhood repetitive routines and obsessive compulsive symptomatology in 6-year-old twins Type de document : Texte imprimé et/ou numérique Auteurs : Derek BOLTON, Auteur ; Thomas G. O'CONNOR, Auteur ; Thalia C. ELEY, Auteur ; Jacqueline A. BRISKMAN, Auteur ; Sean PERRIN, Auteur ; Frühling V. RIJSDIJK, Auteur Année de publication : 2009 Article en page(s) : p.1139-1146 Langues : Anglais (eng) Mots-clés : Obsessive-compulsive-disorder Childhood-Routines-Inventory children twins genetics Index. décimale : PER Périodiques Résumé : Background: To investigate the association between normative repetitive routines of childhood and paediatric obsessive compulsive symptom syndrome (OCSS) and the extent to which it is genetically mediated.
Methods: In a two-phase design a community sample of 4,662 6-year-old twin-pairs were sampled and 854 pairs were assessed in the second phase for normative repetitive routines using the Childhood Routines Inventory (CRI) and for OCSS by maternal-informant diagnostic interview. The OCSS phenotype was defined using standard diagnostic criteria for obsessive compulsive disorder, though regardless of impairment.
Results: In the bivariate model, correlation between the CRI defined phenotype and the OCSS phenotype was estimated to be .40 (95% CI .27–.50), and this correlation was attributable wholly to additive genetic effects. The bivariate model also provided estimates of heritability of the two phenotypes separately: 55% (95% CI 80–89%) for the OCSS phenotype, with the remaining variance attributable mainly to non-shared environment, and 50% (95% CI 39–62%) for CRI assessed normative repetitive routines of childhood, with 36% of the remaining variance attributable to shared environment and 14% to non-shared.
Conclusions: The moderate correlation between normative childhood repetitive routines and obsessive compulsive symptomatology, attributable to genetic factors, is consistent with the hypothesis that high levels of this trait in young children constitute a risk factor for the development of obsessive compulsive symptoms.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02094.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=829
in Journal of Child Psychology and Psychiatry > 50-9 (September 2009) . - p.1139-1146[article] Normative childhood repetitive routines and obsessive compulsive symptomatology in 6-year-old twins [Texte imprimé et/ou numérique] / Derek BOLTON, Auteur ; Thomas G. O'CONNOR, Auteur ; Thalia C. ELEY, Auteur ; Jacqueline A. BRISKMAN, Auteur ; Sean PERRIN, Auteur ; Frühling V. RIJSDIJK, Auteur . - 2009 . - p.1139-1146.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 50-9 (September 2009) . - p.1139-1146
Mots-clés : Obsessive-compulsive-disorder Childhood-Routines-Inventory children twins genetics Index. décimale : PER Périodiques Résumé : Background: To investigate the association between normative repetitive routines of childhood and paediatric obsessive compulsive symptom syndrome (OCSS) and the extent to which it is genetically mediated.
Methods: In a two-phase design a community sample of 4,662 6-year-old twin-pairs were sampled and 854 pairs were assessed in the second phase for normative repetitive routines using the Childhood Routines Inventory (CRI) and for OCSS by maternal-informant diagnostic interview. The OCSS phenotype was defined using standard diagnostic criteria for obsessive compulsive disorder, though regardless of impairment.
Results: In the bivariate model, correlation between the CRI defined phenotype and the OCSS phenotype was estimated to be .40 (95% CI .27–.50), and this correlation was attributable wholly to additive genetic effects. The bivariate model also provided estimates of heritability of the two phenotypes separately: 55% (95% CI 80–89%) for the OCSS phenotype, with the remaining variance attributable mainly to non-shared environment, and 50% (95% CI 39–62%) for CRI assessed normative repetitive routines of childhood, with 36% of the remaining variance attributable to shared environment and 14% to non-shared.
Conclusions: The moderate correlation between normative childhood repetitive routines and obsessive compulsive symptomatology, attributable to genetic factors, is consistent with the hypothesis that high levels of this trait in young children constitute a risk factor for the development of obsessive compulsive symptoms.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02094.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=829 Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence / Yasmin I. AHMADZADEH in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)
[article]
Titre : Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence Type de document : Texte imprimé et/ou numérique Auteurs : Yasmin I. AHMADZADEH, Auteur ; Thalia C. ELEY, Auteur ; Laurie HANNIGAN, Auteur ; Cathy CRESWELL, Auteur ; Paul LICHTENSTEIN, Auteur ; Erica SPOTTS, Auteur ; Jody GANIBAN, Auteur ; Jenae M. NEIDERHISER, Auteur ; Frühling V. RIJSDIJK, Auteur ; Tom A. MCADAMS, Auteur Article en page(s) : p.599-607 Langues : Anglais (eng) Mots-clés : Adolescent Humans Parents Phenotype Sweden Twins/genetics/psychology Parenting genetics intergenerational psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Parental criticism is correlated with internalising symptoms in adolescent offspring. This correlation could in part reflect their genetic relatedness, if the same genes influence behaviours in both parents and offspring. We use a Children-of-Twins design to assess whether parent-reported criticism and offspring internalising symptoms remain associated after controlling for shared genes. To aid interpretation of our results and those of previous Children-of-Twins studies, we examine statistical power for the detection of genetic effects and explore the direction of possible causal effects between generations. METHODS: Data were drawn from two Swedish twin samples, comprising 876 adult twin pairs with adolescent offspring and 1,030 adolescent twin pairs with parents. Parent reports of criticism towards their offspring were collected concurrently with parent and offspring reports of adolescent internalising symptoms. Children-of-Twins structural equation models were used to control for genetic influence on the intergenerational association between parental criticism and adolescent internalising. RESULTS: Parental criticism was associated with adolescent internalising symptoms after controlling for genetic influence. No significant role was found for shared genes influencing phenotypes in both generations, although power analyses suggested that some genetic effects may have gone undetected. Models could not distinguish directionality for nongenetic, causal effects between generations. CONCLUSIONS: Parental criticism may be involved in psychosocial family processes in the context of adolescent internalising. Future studies should seek to identify these processes and provide clarity on the direction of potential causal effects. En ligne : http://dx.doi.org/10.1111/jcpp.13498 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=476
in Journal of Child Psychology and Psychiatry > 63-5 (May 2022) . - p.599-607[article] Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence [Texte imprimé et/ou numérique] / Yasmin I. AHMADZADEH, Auteur ; Thalia C. ELEY, Auteur ; Laurie HANNIGAN, Auteur ; Cathy CRESWELL, Auteur ; Paul LICHTENSTEIN, Auteur ; Erica SPOTTS, Auteur ; Jody GANIBAN, Auteur ; Jenae M. NEIDERHISER, Auteur ; Frühling V. RIJSDIJK, Auteur ; Tom A. MCADAMS, Auteur . - p.599-607.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 63-5 (May 2022) . - p.599-607
Mots-clés : Adolescent Humans Parents Phenotype Sweden Twins/genetics/psychology Parenting genetics intergenerational psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Parental criticism is correlated with internalising symptoms in adolescent offspring. This correlation could in part reflect their genetic relatedness, if the same genes influence behaviours in both parents and offspring. We use a Children-of-Twins design to assess whether parent-reported criticism and offspring internalising symptoms remain associated after controlling for shared genes. To aid interpretation of our results and those of previous Children-of-Twins studies, we examine statistical power for the detection of genetic effects and explore the direction of possible causal effects between generations. METHODS: Data were drawn from two Swedish twin samples, comprising 876 adult twin pairs with adolescent offspring and 1,030 adolescent twin pairs with parents. Parent reports of criticism towards their offspring were collected concurrently with parent and offspring reports of adolescent internalising symptoms. Children-of-Twins structural equation models were used to control for genetic influence on the intergenerational association between parental criticism and adolescent internalising. RESULTS: Parental criticism was associated with adolescent internalising symptoms after controlling for genetic influence. No significant role was found for shared genes influencing phenotypes in both generations, although power analyses suggested that some genetic effects may have gone undetected. Models could not distinguish directionality for nongenetic, causal effects between generations. CONCLUSIONS: Parental criticism may be involved in psychosocial family processes in the context of adolescent internalising. Future studies should seek to identify these processes and provide clarity on the direction of potential causal effects. En ligne : http://dx.doi.org/10.1111/jcpp.13498 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=476 Practitioner Review: Differential susceptibility theory: might it help in understanding and treating mental health problems in youth? / Elham ASSARY in Journal of Child Psychology and Psychiatry, 64-8 (August 2023)
PermalinkPredicting outcomes following cognitive behaviour therapy in child anxiety disorders: the influence of genetic, demographic and clinical information / Jennifer L. HUDSON in Journal of Child Psychology and Psychiatry, 54-10 (October 2013)
PermalinkThe role of gene–environment correlations and interactions in middle childhood depressive symptoms / Paul O. WILKINSON in Development and Psychopathology, 25-1 (February 2013)
PermalinkUsing DNA to predict behaviour problems from preschool to adulthood / Agnieszka GIDZIELA in Journal of Child Psychology and Psychiatry, 63-7 (July 2022)
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