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Auteur Paul LICHTENSTEIN |
Documents disponibles écrits par cet auteur (32)
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Machine-Learning prediction of comorbid substance use disorders in ADHD youth using Swedish registry data / Yanli ZHANG-JAMES in Journal of Child Psychology and Psychiatry, 61-12 (December 2020)
[article]
Titre : Machine-Learning prediction of comorbid substance use disorders in ADHD youth using Swedish registry data Type de document : Texte imprimé et/ou numérique Auteurs : Yanli ZHANG-JAMES, Auteur ; Qi CHEN, Auteur ; Ralf KUJA-HALKOLA, Auteur ; Paul LICHTENSTEIN, Auteur ; Henrik LARSSON, Auteur ; Stephen V. FARAONE, Auteur Article en page(s) : p.1370-1379 Langues : Anglais (eng) Mots-clés : Machine learning attention-deficit hyperactive disorder comorbidity risk factor substance use disorder Index. décimale : PER Périodiques Résumé : BACKGROUND: Children with attention-deficit/hyperactivity disorder (ADHD) have a high risk for substance use disorders (SUDs). Early identification of at-risk youth would help allocate scarce resources for prevention programs. METHODS: Psychiatric and somatic diagnoses, family history of these disorders, measures of socioeconomic distress, and information about birth complications were obtained from the national registers in Sweden for 19,787 children with ADHD born between 1989 and 1993. We trained (a) a cross-sectional random forest (RF) model using data available by age 17 to predict SUD diagnosis between ages 18 and 19; and (b) a longitudinal recurrent neural network (RNN) model with the Long Short-Term Memory (LSTM) architecture to predict new diagnoses at each age. RESULTS: The area under the receiver operating characteristic curve (AUC) was 0.73(95%CI 0.70-0.76) for the random forest model (RF). Removing prior diagnosis from the predictors, the RF model was still able to achieve significant AUCs when predicting all SUD diagnoses (0.69, 95%CI 0.66-0.72) or new diagnoses (0.67, 95%CI: 0.64, 0.71) during age 18-19. For the model predicting new diagnoses, model calibration was good with a low Brier score of 0.086. Longitudinal LSTM model was able to predict later SUD risks at as early as 2 years age, 10 years before the earliest diagnosis. The average AUC from longitudinal models predicting new diagnoses 1, 2, 5 and 10 years in the future was 0.63. CONCLUSIONS: Population registry data can be used to predict at-risk comorbid SUDs in individuals with ADHD. Such predictions can be made many years prior to age of the onset, and their SUD risks can be monitored using longitudinal models over years during child development. Nevertheless, more work is needed to create prediction models based on electronic health records or linked population registers that are sufficiently accurate for use in the clinic. En ligne : http://dx.doi.org/10.1111/jcpp.13226 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434
in Journal of Child Psychology and Psychiatry > 61-12 (December 2020) . - p.1370-1379[article] Machine-Learning prediction of comorbid substance use disorders in ADHD youth using Swedish registry data [Texte imprimé et/ou numérique] / Yanli ZHANG-JAMES, Auteur ; Qi CHEN, Auteur ; Ralf KUJA-HALKOLA, Auteur ; Paul LICHTENSTEIN, Auteur ; Henrik LARSSON, Auteur ; Stephen V. FARAONE, Auteur . - p.1370-1379.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 61-12 (December 2020) . - p.1370-1379
Mots-clés : Machine learning attention-deficit hyperactive disorder comorbidity risk factor substance use disorder Index. décimale : PER Périodiques Résumé : BACKGROUND: Children with attention-deficit/hyperactivity disorder (ADHD) have a high risk for substance use disorders (SUDs). Early identification of at-risk youth would help allocate scarce resources for prevention programs. METHODS: Psychiatric and somatic diagnoses, family history of these disorders, measures of socioeconomic distress, and information about birth complications were obtained from the national registers in Sweden for 19,787 children with ADHD born between 1989 and 1993. We trained (a) a cross-sectional random forest (RF) model using data available by age 17 to predict SUD diagnosis between ages 18 and 19; and (b) a longitudinal recurrent neural network (RNN) model with the Long Short-Term Memory (LSTM) architecture to predict new diagnoses at each age. RESULTS: The area under the receiver operating characteristic curve (AUC) was 0.73(95%CI 0.70-0.76) for the random forest model (RF). Removing prior diagnosis from the predictors, the RF model was still able to achieve significant AUCs when predicting all SUD diagnoses (0.69, 95%CI 0.66-0.72) or new diagnoses (0.67, 95%CI: 0.64, 0.71) during age 18-19. For the model predicting new diagnoses, model calibration was good with a low Brier score of 0.086. Longitudinal LSTM model was able to predict later SUD risks at as early as 2 years age, 10 years before the earliest diagnosis. The average AUC from longitudinal models predicting new diagnoses 1, 2, 5 and 10 years in the future was 0.63. CONCLUSIONS: Population registry data can be used to predict at-risk comorbid SUDs in individuals with ADHD. Such predictions can be made many years prior to age of the onset, and their SUD risks can be monitored using longitudinal models over years during child development. Nevertheless, more work is needed to create prediction models based on electronic health records or linked population registers that are sufficiently accurate for use in the clinic. En ligne : http://dx.doi.org/10.1111/jcpp.13226 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 Maltreatment-associated neurodevelopmental disorders: a co-twin control analysis / Lisa DINKLER in Journal of Child Psychology and Psychiatry, 58-6 (June 2017)
[article]
Titre : Maltreatment-associated neurodevelopmental disorders: a co-twin control analysis Type de document : Texte imprimé et/ou numérique Auteurs : Lisa DINKLER, Auteur ; Sebastian LUNDSTROM, Auteur ; Ruchika GAJWANI, Auteur ; Paul LICHTENSTEIN, Auteur ; Christopher GILLBERG, Auteur ; Helen MINNIS, Auteur Article en page(s) : p.691-701 Langues : Anglais (eng) Mots-clés : Child maltreatment child abuse neurodevelopmental disorders behavior genetics co-twin control design Index. décimale : PER Périodiques Résumé : Background Childhood maltreatment (CM) is strongly associated with psychiatric disorders in childhood and adulthood. Previous findings suggest that the association between CM and psychiatric disorders is partly causal and partly due to familial confounding, but few studies have investigated the mechanisms behind the association between CM and neurodevelopmental disorders (NDDs). Our objective was to determine whether maltreated children have an elevated number of NDDs and whether CM is a risk factor for an increased NDD ‘load’ and increased NDD symptoms when controlling for familial effects. Methods We used a cross-sectional sample from a population-representative Swedish twin study, comprising 8,192 nine-year-old twins born in Sweden between 1997 and 2005. CM was defined as parent-reported exposure to emotional abuse/neglect, physical neglect, physical abuse, and/or sexual abuse. Four NDDs were measured with the Autism–Tics, AD/HD, and other comorbidities inventory. Results Maltreated children had a greater mean number of NDDs than nonmaltreated children. In a co-twin control design, CM-discordant monozygotic twins did not differ significantly for their number of NDDs, suggesting that CM is not associated with an increased load of NDDs when genetic and shared environmental factors are taken into account. However, CM was associated with a small increase in symptoms of attention-deficit/hyperactivity disorder and autism spectrum disorder in CM-discordant MZ twins, although most of the covariance of CM with NDD symptoms was explained by common genetic effects. Conclusions Maltreated children are at higher risk of having multiple NDDs. Our findings are, however, not consistent with the notion that CM causes the increased NDD load in maltreated children. Maltreated children should receive a full neurodevelopmental assessment, and clinicians should be aware that children with multiple NDDs are at higher risk of maltreatment. En ligne : http://dx.doi.org/10.1111/jcpp.12682 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=308
in Journal of Child Psychology and Psychiatry > 58-6 (June 2017) . - p.691-701[article] Maltreatment-associated neurodevelopmental disorders: a co-twin control analysis [Texte imprimé et/ou numérique] / Lisa DINKLER, Auteur ; Sebastian LUNDSTROM, Auteur ; Ruchika GAJWANI, Auteur ; Paul LICHTENSTEIN, Auteur ; Christopher GILLBERG, Auteur ; Helen MINNIS, Auteur . - p.691-701.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 58-6 (June 2017) . - p.691-701
Mots-clés : Child maltreatment child abuse neurodevelopmental disorders behavior genetics co-twin control design Index. décimale : PER Périodiques Résumé : Background Childhood maltreatment (CM) is strongly associated with psychiatric disorders in childhood and adulthood. Previous findings suggest that the association between CM and psychiatric disorders is partly causal and partly due to familial confounding, but few studies have investigated the mechanisms behind the association between CM and neurodevelopmental disorders (NDDs). Our objective was to determine whether maltreated children have an elevated number of NDDs and whether CM is a risk factor for an increased NDD ‘load’ and increased NDD symptoms when controlling for familial effects. Methods We used a cross-sectional sample from a population-representative Swedish twin study, comprising 8,192 nine-year-old twins born in Sweden between 1997 and 2005. CM was defined as parent-reported exposure to emotional abuse/neglect, physical neglect, physical abuse, and/or sexual abuse. Four NDDs were measured with the Autism–Tics, AD/HD, and other comorbidities inventory. Results Maltreated children had a greater mean number of NDDs than nonmaltreated children. In a co-twin control design, CM-discordant monozygotic twins did not differ significantly for their number of NDDs, suggesting that CM is not associated with an increased load of NDDs when genetic and shared environmental factors are taken into account. However, CM was associated with a small increase in symptoms of attention-deficit/hyperactivity disorder and autism spectrum disorder in CM-discordant MZ twins, although most of the covariance of CM with NDD symptoms was explained by common genetic effects. Conclusions Maltreated children are at higher risk of having multiple NDDs. Our findings are, however, not consistent with the notion that CM causes the increased NDD load in maltreated children. Maltreated children should receive a full neurodevelopmental assessment, and clinicians should be aware that children with multiple NDDs are at higher risk of maltreatment. En ligne : http://dx.doi.org/10.1111/jcpp.12682 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=308 Maternal age at first birth and offspring criminality: Using the children of twins design to test causal hypotheses / Claire A. COYNE in Development and Psychopathology, 25-1 (February 2013)
[article]
Titre : Maternal age at first birth and offspring criminality: Using the children of twins design to test causal hypotheses Type de document : Texte imprimé et/ou numérique Auteurs : Claire A. COYNE, Auteur ; Niklas LANGSTROM, Auteur ; Martin E. RICKERT, Auteur ; Paul LICHTENSTEIN, Auteur ; Brian M. D'ONOFRIO, Auteur Article en page(s) : p.17-35 Index. décimale : PER Périodiques Résumé : Teenage childbirth is a risk factor for poor offspring outcomes, particularly offspring antisocial behavior. It is not clear, however, if maternal age at first birth (MAFB) is causally associated with offspring antisocial behavior or if this association is due to selection factors that influence both the likelihood that a young woman gives birth early and that her offspring engage in antisocial behavior. The current study addresses the limitations of previous research by using longitudinal data from Swedish national registries and children of siblings and children of twins comparisons to identify the extent to which the association between MAFB and offspring criminal convictions is consistent with a causal influence and confounded by genetic or environmental factors that make cousins similar. We found offspring born to mothers who began childbearing earlier were more likely to be convicted of a crime than offspring born to mothers who delayed childbearing. The results from comparisons of differentially exposed cousins, especially born to discordant monozygotic twin sisters, provide support for a causal association between MAFB and offspring criminal convictions. The analyses also found little evidence for genetic confounding due to passive gene–environment correlation. Future studies are needed to replicate these findings and to identify environmental risk factors that mediate this causal association. En ligne : http://dx.doi.org/10.1017/S0954579412000879 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=190
in Development and Psychopathology > 25-1 (February 2013) . - p.17-35[article] Maternal age at first birth and offspring criminality: Using the children of twins design to test causal hypotheses [Texte imprimé et/ou numérique] / Claire A. COYNE, Auteur ; Niklas LANGSTROM, Auteur ; Martin E. RICKERT, Auteur ; Paul LICHTENSTEIN, Auteur ; Brian M. D'ONOFRIO, Auteur . - p.17-35.
in Development and Psychopathology > 25-1 (February 2013) . - p.17-35
Index. décimale : PER Périodiques Résumé : Teenage childbirth is a risk factor for poor offspring outcomes, particularly offspring antisocial behavior. It is not clear, however, if maternal age at first birth (MAFB) is causally associated with offspring antisocial behavior or if this association is due to selection factors that influence both the likelihood that a young woman gives birth early and that her offspring engage in antisocial behavior. The current study addresses the limitations of previous research by using longitudinal data from Swedish national registries and children of siblings and children of twins comparisons to identify the extent to which the association between MAFB and offspring criminal convictions is consistent with a causal influence and confounded by genetic or environmental factors that make cousins similar. We found offspring born to mothers who began childbearing earlier were more likely to be convicted of a crime than offspring born to mothers who delayed childbearing. The results from comparisons of differentially exposed cousins, especially born to discordant monozygotic twin sisters, provide support for a causal association between MAFB and offspring criminal convictions. The analyses also found little evidence for genetic confounding due to passive gene–environment correlation. Future studies are needed to replicate these findings and to identify environmental risk factors that mediate this causal association. En ligne : http://dx.doi.org/10.1017/S0954579412000879 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=190 Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence / Yasmin I. AHMADZADEH in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)
[article]
Titre : Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence Type de document : Texte imprimé et/ou numérique Auteurs : Yasmin I. AHMADZADEH, Auteur ; Thalia C. ELEY, Auteur ; Laurie HANNIGAN, Auteur ; Cathy CRESWELL, Auteur ; Paul LICHTENSTEIN, Auteur ; Erica SPOTTS, Auteur ; Jody GANIBAN, Auteur ; Jenae M. NEIDERHISER, Auteur ; Frühling V. RIJSDIJK, Auteur ; Tom A. MCADAMS, Auteur Article en page(s) : p.599-607 Langues : Anglais (eng) Mots-clés : Adolescent Humans Parents Phenotype Sweden Twins/genetics/psychology Parenting genetics intergenerational psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Parental criticism is correlated with internalising symptoms in adolescent offspring. This correlation could in part reflect their genetic relatedness, if the same genes influence behaviours in both parents and offspring. We use a Children-of-Twins design to assess whether parent-reported criticism and offspring internalising symptoms remain associated after controlling for shared genes. To aid interpretation of our results and those of previous Children-of-Twins studies, we examine statistical power for the detection of genetic effects and explore the direction of possible causal effects between generations. METHODS: Data were drawn from two Swedish twin samples, comprising 876 adult twin pairs with adolescent offspring and 1,030 adolescent twin pairs with parents. Parent reports of criticism towards their offspring were collected concurrently with parent and offspring reports of adolescent internalising symptoms. Children-of-Twins structural equation models were used to control for genetic influence on the intergenerational association between parental criticism and adolescent internalising. RESULTS: Parental criticism was associated with adolescent internalising symptoms after controlling for genetic influence. No significant role was found for shared genes influencing phenotypes in both generations, although power analyses suggested that some genetic effects may have gone undetected. Models could not distinguish directionality for nongenetic, causal effects between generations. CONCLUSIONS: Parental criticism may be involved in psychosocial family processes in the context of adolescent internalising. Future studies should seek to identify these processes and provide clarity on the direction of potential causal effects. En ligne : http://dx.doi.org/10.1111/jcpp.13498 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=476
in Journal of Child Psychology and Psychiatry > 63-5 (May 2022) . - p.599-607[article] Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence [Texte imprimé et/ou numérique] / Yasmin I. AHMADZADEH, Auteur ; Thalia C. ELEY, Auteur ; Laurie HANNIGAN, Auteur ; Cathy CRESWELL, Auteur ; Paul LICHTENSTEIN, Auteur ; Erica SPOTTS, Auteur ; Jody GANIBAN, Auteur ; Jenae M. NEIDERHISER, Auteur ; Frühling V. RIJSDIJK, Auteur ; Tom A. MCADAMS, Auteur . - p.599-607.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 63-5 (May 2022) . - p.599-607
Mots-clés : Adolescent Humans Parents Phenotype Sweden Twins/genetics/psychology Parenting genetics intergenerational psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Parental criticism is correlated with internalising symptoms in adolescent offspring. This correlation could in part reflect their genetic relatedness, if the same genes influence behaviours in both parents and offspring. We use a Children-of-Twins design to assess whether parent-reported criticism and offspring internalising symptoms remain associated after controlling for shared genes. To aid interpretation of our results and those of previous Children-of-Twins studies, we examine statistical power for the detection of genetic effects and explore the direction of possible causal effects between generations. METHODS: Data were drawn from two Swedish twin samples, comprising 876 adult twin pairs with adolescent offspring and 1,030 adolescent twin pairs with parents. Parent reports of criticism towards their offspring were collected concurrently with parent and offspring reports of adolescent internalising symptoms. Children-of-Twins structural equation models were used to control for genetic influence on the intergenerational association between parental criticism and adolescent internalising. RESULTS: Parental criticism was associated with adolescent internalising symptoms after controlling for genetic influence. No significant role was found for shared genes influencing phenotypes in both generations, although power analyses suggested that some genetic effects may have gone undetected. Models could not distinguish directionality for nongenetic, causal effects between generations. CONCLUSIONS: Parental criticism may be involved in psychosocial family processes in the context of adolescent internalising. Future studies should seek to identify these processes and provide clarity on the direction of potential causal effects. En ligne : http://dx.doi.org/10.1111/jcpp.13498 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=476 Parental knowledge is an environmental influence on adolescent externalizing / Kristine MARCEAU in Journal of Child Psychology and Psychiatry, 56-2 (February 2015)
[article]
Titre : Parental knowledge is an environmental influence on adolescent externalizing Type de document : Texte imprimé et/ou numérique Auteurs : Kristine MARCEAU, Auteur ; Jurgita NARUSYTE, Auteur ; Paul LICHTENSTEIN, Auteur ; Jody M. GANIBAN, Auteur ; Erica L. SPOTTS, Auteur ; David REISS, Auteur ; Jenae M. NEIDERHISER, Auteur Article en page(s) : p.130-137 Langues : Anglais (eng) Mots-clés : Gene–environment correlation adolescent externalizing parental monitoring parental knowledge extended children of twins Index. décimale : PER Périodiques Résumé : Background There is evidence both that parental monitoring is an environmental influence serving to diminish adolescent externalizing problems and that this association may be driven by adolescents' characteristics via genetic and/or environmental mechanisms, such that adolescents with fewer problems tell their parents more, and therefore appear to be better monitored. Without information on how parents' and children's genes and environments influence correlated parent and child behaviors, it is impossible to clarify the mechanisms underlying this association. Method The present study used the Extended Children of Twins model to distinguish types of gene–environment correlation and direct environmental effects underlying associations between parental knowledge and adolescent (age 11–22 years) externalizing behavior with a Swedish sample of 909 twin parents and their adolescent offspring and a US-based sample of 405 White adolescent siblings and their parents. Results Results suggest that more parental knowledge is associated with less adolescent externalizing via a direct environmental influence independent of any genetic influences. There was no evidence of a child-driven explanation of the association between parental knowledge and adolescent externalizing problems. Conclusions In this sample of adolescents, parental knowledge exerted an environmental influence on adolescent externalizing after accounting for genetic influences of parents and adolescents. Because the association between parenting and child development originates in the parent, treatment for adolescent externalizing must not only include parents but should also focus on altering their parental style. Thus, findings suggest that teaching parents better knowledge-related monitoring strategies is likely to help reduce externalizing problems in adolescents. En ligne : http://dx.doi.org/10.1111/jcpp.12288 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.130-137[article] Parental knowledge is an environmental influence on adolescent externalizing [Texte imprimé et/ou numérique] / Kristine MARCEAU, Auteur ; Jurgita NARUSYTE, Auteur ; Paul LICHTENSTEIN, Auteur ; Jody M. GANIBAN, Auteur ; Erica L. SPOTTS, Auteur ; David REISS, Auteur ; Jenae M. NEIDERHISER, Auteur . - p.130-137.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.130-137
Mots-clés : Gene–environment correlation adolescent externalizing parental monitoring parental knowledge extended children of twins Index. décimale : PER Périodiques Résumé : Background There is evidence both that parental monitoring is an environmental influence serving to diminish adolescent externalizing problems and that this association may be driven by adolescents' characteristics via genetic and/or environmental mechanisms, such that adolescents with fewer problems tell their parents more, and therefore appear to be better monitored. Without information on how parents' and children's genes and environments influence correlated parent and child behaviors, it is impossible to clarify the mechanisms underlying this association. Method The present study used the Extended Children of Twins model to distinguish types of gene–environment correlation and direct environmental effects underlying associations between parental knowledge and adolescent (age 11–22 years) externalizing behavior with a Swedish sample of 909 twin parents and their adolescent offspring and a US-based sample of 405 White adolescent siblings and their parents. Results Results suggest that more parental knowledge is associated with less adolescent externalizing via a direct environmental influence independent of any genetic influences. There was no evidence of a child-driven explanation of the association between parental knowledge and adolescent externalizing problems. Conclusions In this sample of adolescents, parental knowledge exerted an environmental influence on adolescent externalizing after accounting for genetic influences of parents and adolescents. Because the association between parenting and child development originates in the parent, treatment for adolescent externalizing must not only include parents but should also focus on altering their parental style. Thus, findings suggest that teaching parents better knowledge-related monitoring strategies is likely to help reduce externalizing problems in adolescents. En ligne : http://dx.doi.org/10.1111/jcpp.12288 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259 Perceived child impairment and the 'autism epidemic' / Sebastian LUNDSTROM in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)
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