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Détail de l'auteur
Auteur J. HARRIS |
Documents disponibles écrits par cet auteur (2)
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Titre : Autism Spectrum Disorders in adults living in households throughout England : Report from the Adult Psychiatric Morbidity Survey 2007 Type de document : Texte imprimé et/ou numérique Auteurs : T. S. BRUGHA, Auteur ; S. MCMANUS, Auteur ; Howard MELTZER, Auteur ; J. SMITH, Auteur ; Fiona J. SCOTT, Auteur ; S. PURDON, Auteur ; J. HARRIS, Auteur ; J. BANKART, Auteur Editeur : Londres [Angleterre] : National Health Service - NHS Année de publication : 2009 Importance : 70 p. Format : 21cm x 29,7cm x 0,4cm ISBN/ISSN/EAN : 978-1-84636-338-2 Note générale : Bibliogr., Glossaire Langues : Anglais (eng) Index. décimale : ADU-D ADU-D - Adultes - Vie en Institution Résumé : This report presents data on the presence of Autism Spectrum Disorders (ASD), based on the data collected at phases one and two of the Adult Psychiatric Morbidity Survey (APMS) 2007. These findings were not included in the initial survey report. Estimated disorder prevalence is presented by age, sex, ethnic group, marital status, highest educational qualification, equivalised household income, economic activity status, receipt of benefits, housing tenure, area level deprivation and predicted verbal IQ. The level and nature of treatment and service use is considered, although the sample size means that this cannot be explored in detail. En ligne : http://www.ic.nhs.uk/webfiles/publications/mental%20health/mental%20health%20sur [...] Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=131 Autism Spectrum Disorders in adults living in households throughout England : Report from the Adult Psychiatric Morbidity Survey 2007 [Texte imprimé et/ou numérique] / T. S. BRUGHA, Auteur ; S. MCMANUS, Auteur ; Howard MELTZER, Auteur ; J. SMITH, Auteur ; Fiona J. SCOTT, Auteur ; S. PURDON, Auteur ; J. HARRIS, Auteur ; J. BANKART, Auteur . - Londres [Angleterre] : National Health Service - NHS, 2009 . - 70 p. ; 21cm x 29,7cm x 0,4cm.
ISBN : 978-1-84636-338-2
Bibliogr., Glossaire
Langues : Anglais (eng)
Index. décimale : ADU-D ADU-D - Adultes - Vie en Institution Résumé : This report presents data on the presence of Autism Spectrum Disorders (ASD), based on the data collected at phases one and two of the Adult Psychiatric Morbidity Survey (APMS) 2007. These findings were not included in the initial survey report. Estimated disorder prevalence is presented by age, sex, ethnic group, marital status, highest educational qualification, equivalised household income, economic activity status, receipt of benefits, housing tenure, area level deprivation and predicted verbal IQ. The level and nature of treatment and service use is considered, although the sample size means that this cannot be explored in detail. En ligne : http://www.ic.nhs.uk/webfiles/publications/mental%20health/mental%20health%20sur [...] Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=131 Exemplaires (1)
Code-barres Cote Support Localisation Section Disponibilité DOC0001914 ADU-D BRU Livre Centre d'Information et de Documentation du CRA Rhône-Alpes ADU - Autisme à l'âge adulte Disponible Pre-pulse inhibition and antisaccade performance indicate impaired attention modulation of cognitive inhibition in 22q11.2 deletion syndrome (22q11DS) / K. L. MCCABE in Journal of Neurodevelopmental Disorders, 6-1 (December 2014)
[article]
Titre : Pre-pulse inhibition and antisaccade performance indicate impaired attention modulation of cognitive inhibition in 22q11.2 deletion syndrome (22q11DS) Type de document : Texte imprimé et/ou numérique Auteurs : K. L. MCCABE, Auteur ; R. J. ATKINSON, Auteur ; Gavin COOPER, Auteur ; J. L. MELVILLE, Auteur ; J. HARRIS, Auteur ; U. SCHALL, Auteur ; C. M. LOUGHLAND, Auteur ; R. THIENEL, Auteur ; Linda E. CAMPBELL, Auteur Article en page(s) : p.38 Langues : Anglais (eng) Mots-clés : Antisaccade Neurocognition Ppf Ppi Startle modification Index. décimale : PER Périodiques Résumé : BACKGROUND: 22q11.2 deletion syndrome (22q11DS) is associated with a number of physical anomalies and neuropsychological deficits including impairments in executive and sensorimotor function. It is estimated that 25% of children with 22q11DS will develop schizophrenia and other psychotic disorders later in life. Evidence of genetic transmission of information processing deficits in schizophrenia suggests performance in 22q11DS individuals will enhance understanding of the neurobiological and genetic substrates associated with information processing. In this report, we examine information processing in 22q11DS using measures of startle eyeblink modification and antisaccade inhibition to explore similarities with schizophrenia and associations with neurocognitive performance. METHODS: Startle modification (passive and active tasks; 120- and 480-ms pre-pulse intervals) and antisaccade inhibition were measured in 25 individuals with genetically confirmed 22q11DS and 30 healthy control subjects. RESULTS: Individuals with 22q11DS exhibited increased antisaccade error as well as some evidence (trend-level effect) of impaired sensorimotor gating during the active condition, suggesting a dysfunction in controlled attentional processing, rather than a pre-attentive dysfunction using this paradigm. CONCLUSIONS: The findings from the present study show similarities with previous studies in clinical populations associated with 22q11DS such as schizophrenia that may indicate shared dysfunction of inhibition pathways in these groups. En ligne : http://dx.doi.org/10.1186/1866-1955-6-38 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=346
in Journal of Neurodevelopmental Disorders > 6-1 (December 2014) . - p.38[article] Pre-pulse inhibition and antisaccade performance indicate impaired attention modulation of cognitive inhibition in 22q11.2 deletion syndrome (22q11DS) [Texte imprimé et/ou numérique] / K. L. MCCABE, Auteur ; R. J. ATKINSON, Auteur ; Gavin COOPER, Auteur ; J. L. MELVILLE, Auteur ; J. HARRIS, Auteur ; U. SCHALL, Auteur ; C. M. LOUGHLAND, Auteur ; R. THIENEL, Auteur ; Linda E. CAMPBELL, Auteur . - p.38.
Langues : Anglais (eng)
in Journal of Neurodevelopmental Disorders > 6-1 (December 2014) . - p.38
Mots-clés : Antisaccade Neurocognition Ppf Ppi Startle modification Index. décimale : PER Périodiques Résumé : BACKGROUND: 22q11.2 deletion syndrome (22q11DS) is associated with a number of physical anomalies and neuropsychological deficits including impairments in executive and sensorimotor function. It is estimated that 25% of children with 22q11DS will develop schizophrenia and other psychotic disorders later in life. Evidence of genetic transmission of information processing deficits in schizophrenia suggests performance in 22q11DS individuals will enhance understanding of the neurobiological and genetic substrates associated with information processing. In this report, we examine information processing in 22q11DS using measures of startle eyeblink modification and antisaccade inhibition to explore similarities with schizophrenia and associations with neurocognitive performance. METHODS: Startle modification (passive and active tasks; 120- and 480-ms pre-pulse intervals) and antisaccade inhibition were measured in 25 individuals with genetically confirmed 22q11DS and 30 healthy control subjects. RESULTS: Individuals with 22q11DS exhibited increased antisaccade error as well as some evidence (trend-level effect) of impaired sensorimotor gating during the active condition, suggesting a dysfunction in controlled attentional processing, rather than a pre-attentive dysfunction using this paradigm. CONCLUSIONS: The findings from the present study show similarities with previous studies in clinical populations associated with 22q11DS such as schizophrenia that may indicate shared dysfunction of inhibition pathways in these groups. En ligne : http://dx.doi.org/10.1186/1866-1955-6-38 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=346