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Auteur Ahra KO |
Documents disponibles écrits par cet auteur (1)
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Stress, Psychological Resources, and HPA and Inflammatory Reactivity During Late Adolescence / Jessica J. CHIANG in Development and Psychopathology, 31-2 (May 2019)
[article]
Titre : Stress, Psychological Resources, and HPA and Inflammatory Reactivity During Late Adolescence Type de document : Texte imprimé et/ou numérique Auteurs : Jessica J. CHIANG, Auteur ; Ahra KO, Auteur ; Julienne E. BOWER, Auteur ; Shelley E. TAYLOR, Auteur ; Michael R. IRWIN, Auteur ; Andrew J. FULIGNI, Auteur Article en page(s) : p.699-712 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Psychosocial stress during childhood and adolescence is associated with alterations in the hypothalamic–pituitary–adrenal (HPA) axis and with heightened inflammation, both of which are implicated in poor health; however, factors that may protect against these effects relatively early in life are not well understood. Thus, we examined whether psychosocial resources protect against stress-related alterations in the HPA axis and heightened inflammation in a sample of 91 late adolescents. Participants completed measures of various stressors (major life events, daily interpersonal stress, early adversity), and psychosocial resources (mastery, optimism, self-esteem, and positive reappraisal). They also completed the Trier Social Stress Test and provided saliva and blood samples for the assessment of cortisol and interleukin-6 reactivity. Each of the stressors was associated with lower cortisol reactivity. Additionally, associations with major life events and daily stress were moderated by psychological resources, such that more life events and daily stress were associated with decreased HPA reactivity among adolescents with lower levels of psychological resources, but not among those with higher levels of psychological resources. This pattern of findings was observed only for cortisol reactivity and not for interleukin-6 reactivity. Findings suggest that psychological resources may counteract the effects of certain adversity-related decreases in cortisol reactivity. En ligne : http://dx.doi.org/10.1017/S0954579418000287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=393
in Development and Psychopathology > 31-2 (May 2019) . - p.699-712[article] Stress, Psychological Resources, and HPA and Inflammatory Reactivity During Late Adolescence [Texte imprimé et/ou numérique] / Jessica J. CHIANG, Auteur ; Ahra KO, Auteur ; Julienne E. BOWER, Auteur ; Shelley E. TAYLOR, Auteur ; Michael R. IRWIN, Auteur ; Andrew J. FULIGNI, Auteur . - p.699-712.
Langues : Anglais (eng)
in Development and Psychopathology > 31-2 (May 2019) . - p.699-712
Index. décimale : PER Périodiques Résumé : Psychosocial stress during childhood and adolescence is associated with alterations in the hypothalamic–pituitary–adrenal (HPA) axis and with heightened inflammation, both of which are implicated in poor health; however, factors that may protect against these effects relatively early in life are not well understood. Thus, we examined whether psychosocial resources protect against stress-related alterations in the HPA axis and heightened inflammation in a sample of 91 late adolescents. Participants completed measures of various stressors (major life events, daily interpersonal stress, early adversity), and psychosocial resources (mastery, optimism, self-esteem, and positive reappraisal). They also completed the Trier Social Stress Test and provided saliva and blood samples for the assessment of cortisol and interleukin-6 reactivity. Each of the stressors was associated with lower cortisol reactivity. Additionally, associations with major life events and daily stress were moderated by psychological resources, such that more life events and daily stress were associated with decreased HPA reactivity among adolescents with lower levels of psychological resources, but not among those with higher levels of psychological resources. This pattern of findings was observed only for cortisol reactivity and not for interleukin-6 reactivity. Findings suggest that psychological resources may counteract the effects of certain adversity-related decreases in cortisol reactivity. En ligne : http://dx.doi.org/10.1017/S0954579418000287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=393