11 recherche sur le mot-clé 'Slow-wave'

Abnormal sleep physiology in children with 15q11.2-13.1 duplication (Dup15q) syndrome / Vidya SARAVANAPANDIAN in Molecular Autism, 12 (2021)  

Public ISBD [article]  inMolecular Autism > 12 (2021) . - 54 p. Titre : Abnormal sleep physiology in children with 15q11.2-13.1 duplication (Dup15q) syndrome Type de document : texte imprimé Auteurs : Vidya SARAVANAPANDIAN, Auteur ; Divya NADKARNI, Auteur ; Sheng-Hsiou HSU, Auteur ; Shaun A. HUSSAIN, Auteur ; Kiran MASKI, Auteur ; Peyman GOLSHANI, Auteur ; Christopher S. COLWELL, Auteur ; Saravanavel BALASUBRAMANIAN, Auteur ; Amos DIXON, Auteur ; Daniel H. GESCHWIND, Auteur ; Shafali S. JESTE, Auteur Article en page(s) : 54 p. Langues : Anglais (eng) Mots-clés : Autism  Biomarkers  Dup15q syndrome  Eeg  Gabaar  Sleep  Slow wave sleep  Spindles  UBE3A  Hoffmann-La Roche Ltd. and Yamo Pharmaceuticals. All the other authors declare that  they have no competing interests. Index. décimale : PER Périodiques Résumé : BACKGROUND: Sleep disturbances in autism spectrum disorder (ASD) represent a common and vexing comorbidity. Clinical heterogeneity amongst these warrants studies of the mechanisms associated with specific genetic etiologies. Duplications of 15q11.2-13.1 (Dup15q syndrome) are highly penetrant for neurodevelopmental disorders (NDDs) such as intellectual disability and ASD, as well as sleep disturbances. Genes in the 15q region, particularly UBE3A and a cluster of GABA(A) receptor genes, are critical for neural development, synaptic protein synthesis and degradation, and inhibitory neurotransmission. During awake electroencephalography (EEG), children with Dup15q syndrome demonstrate increased beta band oscillations (12-30 Hz) that likely reflect aberrant GABAergic neurotransmission. Healthy sleep rhythms, necessary for robust cognitive development, are also highly dependent on GABAergic neurotransmission. We therefore hypothesized that sleep physiology would be abnormal in children with Dup15q syndrome. METHODS: To test the hypothesis that elevated beta oscillations persist in sleep in Dup15q syndrome and that NREM sleep rhythms would be disrupted, we computed: (1) beta power, (2) spindle density, and (3) percentage of slow-wave sleep (SWS) in overnight sleep EEG recordings from a cohort of children with Dup15q syndrome (n = 15) and compared them to age-matched neurotypical children (n = 12). RESULTS: Children with Dup15q syndrome showed abnormal sleep physiology with elevated beta power, reduced spindle density, and reduced or absent SWS compared to age-matched neurotypical controls. LIMITATIONS: This study relied on clinical EEG where sleep staging was not available. However, considering that clinical polysomnograms are challenging to collect in this population, the ability to quantify these biomarkers on clinical EEG-routinely ordered for epilepsy monitoring-opens the door for larger-scale studies. While comparable to other human studies in rare genetic disorders, a larger sample would allow for examination of the role of seizure severity, medications, and developmental age that may impact sleep physiology. CONCLUSIONS: We have identified three quantitative EEG biomarkers of sleep disruption in Dup15q syndrome, a genetic condition highly penetrant for ASD. Insights from this study not only promote a greater mechanistic understanding of the pathophysiology defining Dup15q syndrome, but also lay the foundation for studies that investigate the association between sleep and cognition. Abnormal sleep physiology may undermine healthy cognitive development and may serve as a quantifiable and modifiable target for behavioral and pharmacological interventions. En ligne : http://dx.doi.org/10.1186/s13229-021-00460-8 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459 [article] Abnormal sleep physiology in children with 15q11.2-13.1 duplication (Dup15q) syndrome [texte imprimé] / Vidya SARAVANAPANDIAN, Auteur ; Divya NADKARNI, Auteur ; Sheng-Hsiou HSU, Auteur ; Shaun A. HUSSAIN, Auteur ; Kiran MASKI, Auteur ; Peyman GOLSHANI, Auteur ; Christopher S. COLWELL, Auteur ; Saravanavel BALASUBRAMANIAN, Auteur ; Amos DIXON, Auteur ; Daniel H. GESCHWIND, Auteur ; Shafali S. JESTE, Auteur . - 54 p. Langues : Anglais (eng) in Molecular Autism > 12 (2021) . - 54 p. Mots-clés : Autism  Biomarkers  Dup15q syndrome  Eeg  Gabaar  Sleep  Slow wave sleep  Spindles  UBE3A  Hoffmann-La Roche Ltd. and Yamo Pharmaceuticals. All the other authors declare that  they have no competing interests. Index. décimale : PER Périodiques Résumé : BACKGROUND: Sleep disturbances in autism spectrum disorder (ASD) represent a common and vexing comorbidity. Clinical heterogeneity amongst these warrants studies of the mechanisms associated with specific genetic etiologies. Duplications of 15q11.2-13.1 (Dup15q syndrome) are highly penetrant for neurodevelopmental disorders (NDDs) such as intellectual disability and ASD, as well as sleep disturbances. Genes in the 15q region, particularly UBE3A and a cluster of GABA(A) receptor genes, are critical for neural development, synaptic protein synthesis and degradation, and inhibitory neurotransmission. During awake electroencephalography (EEG), children with Dup15q syndrome demonstrate increased beta band oscillations (12-30 Hz) that likely reflect aberrant GABAergic neurotransmission. Healthy sleep rhythms, necessary for robust cognitive development, are also highly dependent on GABAergic neurotransmission. We therefore hypothesized that sleep physiology would be abnormal in children with Dup15q syndrome. METHODS: To test the hypothesis that elevated beta oscillations persist in sleep in Dup15q syndrome and that NREM sleep rhythms would be disrupted, we computed: (1) beta power, (2) spindle density, and (3) percentage of slow-wave sleep (SWS) in overnight sleep EEG recordings from a cohort of children with Dup15q syndrome (n = 15) and compared them to age-matched neurotypical children (n = 12). RESULTS: Children with Dup15q syndrome showed abnormal sleep physiology with elevated beta power, reduced spindle density, and reduced or absent SWS compared to age-matched neurotypical controls. LIMITATIONS: This study relied on clinical EEG where sleep staging was not available. However, considering that clinical polysomnograms are challenging to collect in this population, the ability to quantify these biomarkers on clinical EEG-routinely ordered for epilepsy monitoring-opens the door for larger-scale studies. While comparable to other human studies in rare genetic disorders, a larger sample would allow for examination of the role of seizure severity, medications, and developmental age that may impact sleep physiology. CONCLUSIONS: We have identified three quantitative EEG biomarkers of sleep disruption in Dup15q syndrome, a genetic condition highly penetrant for ASD. Insights from this study not only promote a greater mechanistic understanding of the pathophysiology defining Dup15q syndrome, but also lay the foundation for studies that investigate the association between sleep and cognition. Abnormal sleep physiology may undermine healthy cognitive development and may serve as a quantifiable and modifiable target for behavioral and pharmacological interventions. En ligne : http://dx.doi.org/10.1186/s13229-021-00460-8 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459

Distinctions and similarities among working memory processes: an event-related potential study / Daniel S. RUCHKIN in Cognitive Brain Research, 1-1 (June 1992)

Public ISBD [article]  inCognitive Brain Research > 1-1 (June 1992) . - p.53-66 Titre : Distinctions and similarities among working memory processes: an event-related potential study Type de document : texte imprimé Auteurs : Daniel S. RUCHKIN, Auteur ; Ray Jr JOHNSON, Auteur ; Jordan GRAFMAN, Auteur ; Howard CANOUNE, Auteur ; Walter RITTER, Auteur Année de publication : 1992 Article en page(s) : p.53-66 Langues : Anglais (eng) Mots-clés : Event-related-potential Slow-wave Working-memory Short-term-memory Visuo-spatial-sketch-pad Articulatory-rehearsal Index. décimale : PER Périodiques Résumé : Working memory has been conceptualized as consiting of a number of components, such as an articulatory loop for rehearsing verbal material, a visuo-spatial sketch pad for maintaining visual images and a central executive that controls which information is made available for conscious processing. We recorded event-related brain potentials (ERPs) from normal human subjects while they maintained either visuo-spatial or phonological material in short-term memory for a 5-s interval. The results indicated that specialized brain systems for short-term storage of phonological and visuo-spatial information could be identified on the basis of marked differences between the topographies and morphologies of the ERP components elicited during these two types of short-term memory. The differences emerged during early encoding stages and continued through later retention stages. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=780 [article] Distinctions and similarities among working memory processes: an event-related potential study [texte imprimé] / Daniel S. RUCHKIN, Auteur ; Ray Jr JOHNSON, Auteur ; Jordan GRAFMAN, Auteur ; Howard CANOUNE, Auteur ; Walter RITTER, Auteur . - 1992 . - p.53-66. Langues : Anglais (eng) in Cognitive Brain Research > 1-1 (June 1992) . - p.53-66 Mots-clés : Event-related-potential Slow-wave Working-memory Short-term-memory Visuo-spatial-sketch-pad Articulatory-rehearsal Index. décimale : PER Périodiques Résumé : Working memory has been conceptualized as consiting of a number of components, such as an articulatory loop for rehearsing verbal material, a visuo-spatial sketch pad for maintaining visual images and a central executive that controls which information is made available for conscious processing. We recorded event-related brain potentials (ERPs) from normal human subjects while they maintained either visuo-spatial or phonological material in short-term memory for a 5-s interval. The results indicated that specialized brain systems for short-term storage of phonological and visuo-spatial information could be identified on the basis of marked differences between the topographies and morphologies of the ERP components elicited during these two types of short-term memory. The differences emerged during early encoding stages and continued through later retention stages. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=780

Neuroligin-2 shapes individual slow waves during slow-wave sleep and the response to sleep deprivation in mice / Tanya LEDUC in Molecular Autism, 15 (2024)  

Public ISBD [article]  inMolecular Autism > 15 (2024) . - 13p. Titre : Neuroligin-2 shapes individual slow waves during slow-wave sleep and the response to sleep deprivation in mice Type de document : texte imprimé Auteurs : Tanya LEDUC, Auteur ; Hiba EL ALAMI, Auteur ; Khadija BOUGADIR, Auteur ; Erika BÉLANGER-NELSON, Auteur ; Valérie MONGRAIN, Auteur Article en page(s) : 13p. Langues : Anglais (eng) Mots-clés : Animals  Humans  Mice  Electroencephalography  Neuroligins  Quality of Life  Sleep/physiology  Sleep Deprivation/metabolism  Sleep, Slow-Wave  Cerebral cortex  GABAergic neurotransmission  Gene expression  Sleep deprivation  Sleep-wake regulation  Slow waves  Synaptic adhesion molecules Index. décimale : PER Périodiques Résumé : BACKGROUND: Sleep disturbances are a common comorbidity to most neurodevelopmental disorders and tend to worsen disease symptomatology. It is thus crucial to understand mechanisms underlying sleep disturbances to improve patients' quality of life. Neuroligin-2 (NLGN2) is a synaptic adhesion protein regulating GABAergic transmission. It has been linked to autism spectrum disorders and schizophrenia in humans, and deregulations of its expression were shown to cause epileptic-like hypersynchronized cerebral activity in rodents. Importantly, the absence of Nlgn2 (knockout: KO) was previously shown to alter sleep-wake duration and quality in mice, notably increasing slow-wave sleep (SWS) delta activity (1-4 Hz) and altering its 24-h dynamics. This type of brain oscillation is involved in memory consolidation, and is also a marker of homeostatic sleep pressure. Sleep deprivation (SD) is notably known to impair cognition and the physiological response to sleep loss involves GABAergic transmission. METHODS: Using electrocorticographic (ECoG) recordings, we here first aimed to verify how individual slow wave (SW; 0.5-4 Hz) density and properties (e.g., amplitude, slope, frequency) contribute to the higher SWS delta activity and altered 24-h dynamics observed in Nlgn2 KO mice. We further investigated the response of these animals to SD. Finally, we tested whether sleep loss affects the gene expression of Nlgn2 and related GABAergic transcripts in the cerebral cortex of wild-type mice using RNA sequencing. RESULTS: Our results show that Nlgn2 KO mice have both greater SW amplitude and density, and that SW density is the main property contributing to the altered 24-h dynamics. We also found the absence of Nlgn2 to accelerate paradoxical sleep recovery following SD, together with profound alterations in ECoG activity across vigilance states. Sleep loss, however, did not modify the 24-h distribution of the hypersynchronized ECoG events observed in these mice. Finally, RNA sequencing confirmed an overall decrease in cortical expression of Nlgn2 and related GABAergic transcripts following SD in wild-type mice. CONCLUSIONS: This work brings further insight into potential mechanisms of sleep duration and quality deregulation in neurodevelopmental disorders, notably involving NLGN2 and GABAergic neurotransmission. En ligne : https://dx.doi.org/10.1186/s13229-024-00594-5 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=538 [article] Neuroligin-2 shapes individual slow waves during slow-wave sleep and the response to sleep deprivation in mice [texte imprimé] / Tanya LEDUC, Auteur ; Hiba EL ALAMI, Auteur ; Khadija BOUGADIR, Auteur ; Erika BÉLANGER-NELSON, Auteur ; Valérie MONGRAIN, Auteur . - 13p. Langues : Anglais (eng) in Molecular Autism > 15 (2024) . - 13p. Mots-clés : Animals  Humans  Mice  Electroencephalography  Neuroligins  Quality of Life  Sleep/physiology  Sleep Deprivation/metabolism  Sleep, Slow-Wave  Cerebral cortex  GABAergic neurotransmission  Gene expression  Sleep deprivation  Sleep-wake regulation  Slow waves  Synaptic adhesion molecules Index. décimale : PER Périodiques Résumé : BACKGROUND: Sleep disturbances are a common comorbidity to most neurodevelopmental disorders and tend to worsen disease symptomatology. It is thus crucial to understand mechanisms underlying sleep disturbances to improve patients' quality of life. Neuroligin-2 (NLGN2) is a synaptic adhesion protein regulating GABAergic transmission. It has been linked to autism spectrum disorders and schizophrenia in humans, and deregulations of its expression were shown to cause epileptic-like hypersynchronized cerebral activity in rodents. Importantly, the absence of Nlgn2 (knockout: KO) was previously shown to alter sleep-wake duration and quality in mice, notably increasing slow-wave sleep (SWS) delta activity (1-4 Hz) and altering its 24-h dynamics. This type of brain oscillation is involved in memory consolidation, and is also a marker of homeostatic sleep pressure. Sleep deprivation (SD) is notably known to impair cognition and the physiological response to sleep loss involves GABAergic transmission. METHODS: Using electrocorticographic (ECoG) recordings, we here first aimed to verify how individual slow wave (SW; 0.5-4 Hz) density and properties (e.g., amplitude, slope, frequency) contribute to the higher SWS delta activity and altered 24-h dynamics observed in Nlgn2 KO mice. We further investigated the response of these animals to SD. Finally, we tested whether sleep loss affects the gene expression of Nlgn2 and related GABAergic transcripts in the cerebral cortex of wild-type mice using RNA sequencing. RESULTS: Our results show that Nlgn2 KO mice have both greater SW amplitude and density, and that SW density is the main property contributing to the altered 24-h dynamics. We also found the absence of Nlgn2 to accelerate paradoxical sleep recovery following SD, together with profound alterations in ECoG activity across vigilance states. Sleep loss, however, did not modify the 24-h distribution of the hypersynchronized ECoG events observed in these mice. Finally, RNA sequencing confirmed an overall decrease in cortical expression of Nlgn2 and related GABAergic transcripts following SD in wild-type mice. CONCLUSIONS: This work brings further insight into potential mechanisms of sleep duration and quality deregulation in neurodevelopmental disorders, notably involving NLGN2 and GABAergic neurotransmission. En ligne : https://dx.doi.org/10.1186/s13229-024-00594-5 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=538

Topographic differences of slow event-related brain potentials in blind and sighted adult human subjects during haptic mental rotation / Frank ROSLER in Cognitive Brain Research, 1-3 (October 1993)

Public ISBD [article]  inCognitive Brain Research > 1-3 (October 1993) . - p.145-159 Titre : Topographic differences of slow event-related brain potentials in blind and sighted adult human subjects during haptic mental rotation Type de document : texte imprimé Auteurs : Frank ROSLER, Auteur ; Brigitte RODER, Auteur ; Martin HEIL, Auteur ; Erwin HENNIGHAUSEN, Auteur Année de publication : 1993 Article en page(s) : p.145-159 Langues : Anglais (eng) Mots-clés : Blindness Electroencephalogram Slow-wave Functional-organization-of-the-cortex Cortical-plasticity Haptic-mental-rotation Cognition Index. décimale : PER Périodiques Résumé : Twelve blindfolded sighted, nine congenitally blind, and seven adventitiously blind subjects were tested in a haptic mental rotation task while slow event-related potentials in the EEG were recorded from 17 scalp locations. The overall topography of the slow wave pattern which prevailed during the task differed for sighted and for blind, but not for congenitally and adventitiously blind subjects. While the tactile stimuli were encoded, the blind showed a pronounced occipital and the sighted a pronounced frontal activation. The task-specific amplitude increment of a negative slow wave which can be understood as a manifestation of the process of mental rotation proper, showed a different topography for sighted and for blind subjects too. It had its maximum over central to parietal cortical areas in both groups, but it extended more towards occipital regions in the blind. In both groups, the effects were very similar to those observed in former studies with visual versions of the mental rotation task, i.e. the slow wave amplitude over central to parietal areas increased monotonously with an increasing angular disparity of the two stimuli to be compared. These results are discussed with respect to the question of whether visual deprivation in the blind can cause a reorganization of cortical representational maps. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=781 [article] Topographic differences of slow event-related brain potentials in blind and sighted adult human subjects during haptic mental rotation [texte imprimé] / Frank ROSLER, Auteur ; Brigitte RODER, Auteur ; Martin HEIL, Auteur ; Erwin HENNIGHAUSEN, Auteur . - 1993 . - p.145-159. Langues : Anglais (eng) in Cognitive Brain Research > 1-3 (October 1993) . - p.145-159 Mots-clés : Blindness Electroencephalogram Slow-wave Functional-organization-of-the-cortex Cortical-plasticity Haptic-mental-rotation Cognition Index. décimale : PER Périodiques Résumé : Twelve blindfolded sighted, nine congenitally blind, and seven adventitiously blind subjects were tested in a haptic mental rotation task while slow event-related potentials in the EEG were recorded from 17 scalp locations. The overall topography of the slow wave pattern which prevailed during the task differed for sighted and for blind, but not for congenitally and adventitiously blind subjects. While the tactile stimuli were encoded, the blind showed a pronounced occipital and the sighted a pronounced frontal activation. The task-specific amplitude increment of a negative slow wave which can be understood as a manifestation of the process of mental rotation proper, showed a different topography for sighted and for blind subjects too. It had its maximum over central to parietal cortical areas in both groups, but it extended more towards occipital regions in the blind. In both groups, the effects were very similar to those observed in former studies with visual versions of the mental rotation task, i.e. the slow wave amplitude over central to parietal areas increased monotonously with an increasing angular disparity of the two stimuli to be compared. These results are discussed with respect to the question of whether visual deprivation in the blind can cause a reorganization of cortical representational maps. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=781

Annotation: Neurofeedback – train your brain to train behaviour / Hartmut HEINRICH in Journal of Child Psychology and Psychiatry, 48-1 (January 2007)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 48-1 (January 2007) . - p.3–16 Titre : Annotation: Neurofeedback – train your brain to train behaviour Type de document : texte imprimé Auteurs : Hartmut HEINRICH, Auteur ; Holger GEVENSLEBEN, Auteur ; Ute STREHL, Auteur Année de publication : 2007 Article en page(s) : p.3–16 Langues : Anglais (eng) Mots-clés : Neurofeedback electroencephalogram-(EEG) frequency-bands slow-cortical-potentials-(SCPs) attention-deficit-hyperactivity-disorder-(ADHD) epilepsy self-regulation Index. décimale : PER Périodiques Résumé : Neurofeedback (NF) is a form of behavioural training aimed at developing skills for self-regulation of brain activity. Within the past decade, several NF studies have been published that tend to overcome the methodological shortcomings of earlier studies. This annotation describes the methodical basis of NF and reviews the evidence base for its clinical efficacy and effectiveness in neuropsychiatric disorders. Methods: In NF training, self-regulation of specific aspects of electrical brain activity is acquired by means of immediate feedback and positive reinforcement. In frequency training, activity in different EEG frequency bands has to be decreased or increased. Training of slow cortical potentials (SCPs) addresses the regulation of cortical excitability. Results: NF studies revealed paradigm-specific effects on, e.g., attention and memory processes and performance improvements in real-life conditions, in healthy subjects as well as in patients. In several studies it was shown that children with attention-deficit hyperactivity disorder (ADHD) improved behavioural and cognitive variables after frequency (e.g., theta/beta) training or SCP training. Neurophysiological effects could also be measured. However, specific and unspecific training effects could not be disentangled in these studies. For drug-resistant patients with epilepsy, significant and long-lasting decreases of seizure frequency and intensity through SCP training were documented in a series of studies. For other child psychiatric disorders (e.g., tic disorders, anxiety, and autism) only preliminary investigations are available. Conclusions: There is growing evidence for NF as a valuable treatment module in neuropsychiatric disorders. Further, controlled studies are necessary to establish clinical efficacy and effectiveness and to learn more about the mechanisms underlying successful training. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01665.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=925 [article] Annotation: Neurofeedback – train your brain to train behaviour [texte imprimé] / Hartmut HEINRICH, Auteur ; Holger GEVENSLEBEN, Auteur ; Ute STREHL, Auteur . - 2007 . - p.3–16. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 48-1 (January 2007) . - p.3–16 Mots-clés : Neurofeedback electroencephalogram-(EEG) frequency-bands slow-cortical-potentials-(SCPs) attention-deficit-hyperactivity-disorder-(ADHD) epilepsy self-regulation Index. décimale : PER Périodiques Résumé : Neurofeedback (NF) is a form of behavioural training aimed at developing skills for self-regulation of brain activity. Within the past decade, several NF studies have been published that tend to overcome the methodological shortcomings of earlier studies. This annotation describes the methodical basis of NF and reviews the evidence base for its clinical efficacy and effectiveness in neuropsychiatric disorders. Methods: In NF training, self-regulation of specific aspects of electrical brain activity is acquired by means of immediate feedback and positive reinforcement. In frequency training, activity in different EEG frequency bands has to be decreased or increased. Training of slow cortical potentials (SCPs) addresses the regulation of cortical excitability. Results: NF studies revealed paradigm-specific effects on, e.g., attention and memory processes and performance improvements in real-life conditions, in healthy subjects as well as in patients. In several studies it was shown that children with attention-deficit hyperactivity disorder (ADHD) improved behavioural and cognitive variables after frequency (e.g., theta/beta) training or SCP training. Neurophysiological effects could also be measured. However, specific and unspecific training effects could not be disentangled in these studies. For drug-resistant patients with epilepsy, significant and long-lasting decreases of seizure frequency and intensity through SCP training were documented in a series of studies. For other child psychiatric disorders (e.g., tic disorders, anxiety, and autism) only preliminary investigations are available. Conclusions: There is growing evidence for NF as a valuable treatment module in neuropsychiatric disorders. Further, controlled studies are necessary to establish clinical efficacy and effectiveness and to learn more about the mechanisms underlying successful training. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01665.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=925

Attachment security, environmental adversity, and fast life history behavioral profiles in human adolescents / Hui Jing LU in Development and Psychopathology, 37-3 (August 2025)  

Permalink

Camera Movement Impacts on Mu-Wave Activity During Action Observation in Adults With Autism Spectrum Disorders Without Intellectual Disabilities / Renata DEL GIUDICE ; Francesca SERIO ; Giovanni BOIDO ; Gianmarco INGROSSO ; Francesco LOMBARDI ; Claudio SANGUINETI ; Valeria CASULA ; Adelaide BACCARA ; Elia CHIUDINELLI ; Francesca VAIRANO ; Federica Maria PANZERI ; Mauro GIORI ; Paolo Maria INGHILLERI DI VILLADAURO ; Raffaella FAGGIOLI ; Orsola GAMBINI ; Tomaso SUBINI ; Benedetta DEMARTINI in Autism Research, 18-4 (April 2025)  

Permalink

Child attachment in adjusting the species-general contingency between environmental adversities and fast life history strategies / Hui Jing LU in Development and Psychopathology, 34-2 (May 2022)  

Permalink

Electroretinography in adults with high-functioning autism spectrum disorder / Evelyne FRIEDEL in Autism Research, 15-11 (November 2022)  

Permalink

Is neurofeedback an efficacious treatment for ADHD? A randomised controlled clinical trial / Holger GEVENSLEBEN in Journal of Child Psychology and Psychiatry, 50-7 (July 2009)  

Permalink