Dépouillements

Editorial: Troubled trajectories - new insights on risk pathways and developmental phenotypes of ADHD and externalizing problems / S. Alexandra BURT in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1033-1035 Titre : Editorial: Troubled trajectories - new insights on risk pathways and developmental phenotypes of ADHD and externalizing problems Type de document : Texte imprimé et/ou numérique Auteurs : S. Alexandra BURT, Auteur ; J. M. HALPERIN, Auteur ; A. J. OLDEHINKEL, Auteur Article en page(s) : p.1033-1035 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : We are pleased to present this special issue of JCPP, which brings together a collection of cutting-edge empirical studies that focuses on developmental trajectories and pathways associated with ADHD, conduct problems and other externalizing behaviors. JCPP has had a long-standing focus on theoretically strong prospective longitudinal studies that elucidate behavioral change and outcomes over time, including in the areas of ADHD and externalizing disorders (Kuja-Halkola, Lichtenstein, D'Onofrio & Larsson, 2015; Sonuga-Barke & Halperin, 2010). Keeping with that tradition, all of the papers in this special issue employ a prospective longitudinal design and a focus on changes that unfold over development. En ligne : http://dx.doi.org/10.1111/jcpp.12976 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Editorial: Troubled trajectories - new insights on risk pathways and developmental phenotypes of ADHD and externalizing problems [Texte imprimé et/ou numérique] / S. Alexandra BURT, Auteur ; J. M. HALPERIN, Auteur ; A. J. OLDEHINKEL, Auteur . - p.1033-1035. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1033-1035 Index. décimale : PER Périodiques Résumé : We are pleased to present this special issue of JCPP, which brings together a collection of cutting-edge empirical studies that focuses on developmental trajectories and pathways associated with ADHD, conduct problems and other externalizing behaviors. JCPP has had a long-standing focus on theoretically strong prospective longitudinal studies that elucidate behavioral change and outcomes over time, including in the areas of ADHD and externalizing disorders (Kuja-Halkola, Lichtenstein, D'Onofrio & Larsson, 2015; Sonuga-Barke & Halperin, 2010). Keeping with that tradition, all of the papers in this special issue employ a prospective longitudinal design and a focus on changes that unfold over development. En ligne : http://dx.doi.org/10.1111/jcpp.12976 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

The role of birth weight on the causal pathway to child and adolescent ADHD symptomatology: a population-based twin differences longitudinal design / K. X. LIM in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1036-1043 Titre : The role of birth weight on the causal pathway to child and adolescent ADHD symptomatology: a population-based twin differences longitudinal design Type de document : Texte imprimé et/ou numérique Auteurs : K. X. LIM, Auteur ; C. Y. LIU, Auteur ; T. SCHOELER, Auteur ; Charlotte A. M. CECIL, Auteur ; Edward D. BARKER, Auteur ; E. VIDING, Auteur ; C. U. GREVEN, Auteur ; J. B. PINGAULT, Auteur Article en page(s) : p.1036-1043 Langues : Anglais (eng) Mots-clés : Attention-deficit/hyperactivity disorder birth weight hyperactivity/impulsivity inattention twin differences Index. décimale : PER Périodiques Résumé : BACKGROUND: Available evidence points towards lower birth weight as a risk factor for the development of attention deficit/hyperactivity disorder (ADHD) symptoms. We probed the causal nature of this putative effect of birth weight on ADHD symptoms using the twin differences design, which accounts for genetic and shared environmental confounds. METHOD: In a large population-based twin sample - 3,499 monozygotic (MZ) and 6,698 dizygotic (DZ) pairs - parents, teachers or twins rated the twins' ADHD symptoms at nine assessment waves (2-16 years). We implemented the twin differences design, which completely accounts for shared environmental and genetic confounding in MZ twins. We tested whether: (a) the lighter-born twins had elevated ADHD symptoms compared to the heavier-born twins, by regressing within-pair differences of ADHD symptoms on within-pair differences of birth weight among MZ twins; (b) the effect of birth weight on ADHD was moderated by gender, gestational age and low birth weight; (c) this effect changed with age at ADHD assessment using adapted latent growth curve models; and (d) results differed for inattention and hyperactivity/impulsivity. RESULTS: Birth weight significantly predicted ADHD symptoms from early childhood to late adolescence. The lighter-born twin had more ADHD symptoms than the heavier-born cotwin among MZ twins across assessment waves and raters. No moderation effect was detected. The magnitude of the effect of birth weight decreased significantly across time for hyperactivity/impulsivity, but the decrease failed to reach significance for inattention. Estimates for inattention were significantly larger than for hyperactivity/impulsivity at each time point, implying stronger effect of birth weight on inattention symptoms. CONCLUSIONS: Our findings provide stringent evidence for environmental effect of lower birth weight on the causal pathway to elevated ADHD symptoms. Effect of birth weight persists across a 14-year period from childhood into late adolescence, in particular for inattention symptoms. En ligne : http://dx.doi.org/10.1111/jcpp.12949 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] The role of birth weight on the causal pathway to child and adolescent ADHD symptomatology: a population-based twin differences longitudinal design [Texte imprimé et/ou numérique] / K. X. LIM, Auteur ; C. Y. LIU, Auteur ; T. SCHOELER, Auteur ; Charlotte A. M. CECIL, Auteur ; Edward D. BARKER, Auteur ; E. VIDING, Auteur ; C. U. GREVEN, Auteur ; J. B. PINGAULT, Auteur . - p.1036-1043. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1036-1043 Mots-clés : Attention-deficit/hyperactivity disorder birth weight hyperactivity/impulsivity inattention twin differences Index. décimale : PER Périodiques Résumé : BACKGROUND: Available evidence points towards lower birth weight as a risk factor for the development of attention deficit/hyperactivity disorder (ADHD) symptoms. We probed the causal nature of this putative effect of birth weight on ADHD symptoms using the twin differences design, which accounts for genetic and shared environmental confounds. METHOD: In a large population-based twin sample - 3,499 monozygotic (MZ) and 6,698 dizygotic (DZ) pairs - parents, teachers or twins rated the twins' ADHD symptoms at nine assessment waves (2-16 years). We implemented the twin differences design, which completely accounts for shared environmental and genetic confounding in MZ twins. We tested whether: (a) the lighter-born twins had elevated ADHD symptoms compared to the heavier-born twins, by regressing within-pair differences of ADHD symptoms on within-pair differences of birth weight among MZ twins; (b) the effect of birth weight on ADHD was moderated by gender, gestational age and low birth weight; (c) this effect changed with age at ADHD assessment using adapted latent growth curve models; and (d) results differed for inattention and hyperactivity/impulsivity. RESULTS: Birth weight significantly predicted ADHD symptoms from early childhood to late adolescence. The lighter-born twin had more ADHD symptoms than the heavier-born cotwin among MZ twins across assessment waves and raters. No moderation effect was detected. The magnitude of the effect of birth weight decreased significantly across time for hyperactivity/impulsivity, but the decrease failed to reach significance for inattention. Estimates for inattention were significantly larger than for hyperactivity/impulsivity at each time point, implying stronger effect of birth weight on inattention symptoms. CONCLUSIONS: Our findings provide stringent evidence for environmental effect of lower birth weight on the causal pathway to elevated ADHD symptoms. Effect of birth weight persists across a 14-year period from childhood into late adolescence, in particular for inattention symptoms. En ligne : http://dx.doi.org/10.1111/jcpp.12949 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

A longitudinal, within-person investigation of the association between the P3 ERP component and externalizing behavior problems in young children / I. T. PETERSEN in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1044-1051 Titre : A longitudinal, within-person investigation of the association between the P3 ERP component and externalizing behavior problems in young children Type de document : Texte imprimé et/ou numérique Auteurs : I. T. PETERSEN, Auteur ; C. P. HOYNIAK, Auteur ; John E. BATES, Auteur ; A. D. STAPLES, Auteur ; D. L. MOLFESE, Auteur Article en page(s) : p.1044-1051 Langues : Anglais (eng) Mots-clés : P3 erp aggression attention-deficit/hyperactivity disorder early childhood externalizing behavior problems Index. décimale : PER Périodiques Résumé : BACKGROUND: Externalizing problems, including aggression and conduct problems, are thought to involve impaired attentional capacities. Previous research suggests that the P3 event-related potential (ERP) component is an index of attentional processing, and diminished P3 amplitudes to infrequent stimuli have been shown to be associated with externalizing problems and attention-deficit/hyperactivity disorder (ADHD). However, the vast majority of this prior work has been cross-sectional and has not examined young children. The present study is the first investigation of whether within-individual changes in P3 amplitude predict changes in externalizing problems, providing a stronger test of developmental process. METHOD: Participants included a community sample of children (N = 153) followed longitudinally at 30, 36, and 42 months of age. Children completed an oddball task while ERP data were recorded. Parents rated their children's aggression and ADHD symptoms. RESULTS: Children's within-individual changes in the P3 amplitude predicted concomitant within-child changes in their aggression such that smaller P3 amplitudes (relative to a child's own mean) were associated with more aggression symptoms. However, changes in P3 amplitudes were not significantly associated with ADHD symptoms. CONCLUSIONS: Findings suggest that the P3 may play a role in development of aggression, but do not support the notion that the P3 plays a role in development of early ADHD symptoms. En ligne : http://dx.doi.org/10.1111/jcpp.12975 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] A longitudinal, within-person investigation of the association between the P3 ERP component and externalizing behavior problems in young children [Texte imprimé et/ou numérique] / I. T. PETERSEN, Auteur ; C. P. HOYNIAK, Auteur ; John E. BATES, Auteur ; A. D. STAPLES, Auteur ; D. L. MOLFESE, Auteur . - p.1044-1051. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1044-1051 Mots-clés : P3 erp aggression attention-deficit/hyperactivity disorder early childhood externalizing behavior problems Index. décimale : PER Périodiques Résumé : BACKGROUND: Externalizing problems, including aggression and conduct problems, are thought to involve impaired attentional capacities. Previous research suggests that the P3 event-related potential (ERP) component is an index of attentional processing, and diminished P3 amplitudes to infrequent stimuli have been shown to be associated with externalizing problems and attention-deficit/hyperactivity disorder (ADHD). However, the vast majority of this prior work has been cross-sectional and has not examined young children. The present study is the first investigation of whether within-individual changes in P3 amplitude predict changes in externalizing problems, providing a stronger test of developmental process. METHOD: Participants included a community sample of children (N = 153) followed longitudinally at 30, 36, and 42 months of age. Children completed an oddball task while ERP data were recorded. Parents rated their children's aggression and ADHD symptoms. RESULTS: Children's within-individual changes in the P3 amplitude predicted concomitant within-child changes in their aggression such that smaller P3 amplitudes (relative to a child's own mean) were associated with more aggression symptoms. However, changes in P3 amplitudes were not significantly associated with ADHD symptoms. CONCLUSIONS: Findings suggest that the P3 may play a role in development of aggression, but do not support the notion that the P3 plays a role in development of early ADHD symptoms. En ligne : http://dx.doi.org/10.1111/jcpp.12975 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Elementary school children's associations of antisocial behaviour with risk-taking across 7-11 years / J. M. TIESKENS in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1052-1060 Titre : Elementary school children's associations of antisocial behaviour with risk-taking across 7-11 years Type de document : Texte imprimé et/ou numérique Auteurs : J. M. TIESKENS, Auteur ; Joanne Marieke BUIL, Auteur ; S. KOOT, Auteur ; L. KRABBENDAM, Auteur ; P. A. C. VAN LIER, Auteur Article en page(s) : p.1052-1060 Langues : Anglais (eng) Mots-clés : Aggression children covert antisocial behaviour oppositional defiant behaviour risk-taking Index. décimale : PER Périodiques Résumé : BACKGROUND: The impact of childhood antisocial behaviour on future maladaptation has been acknowledged. Risk-taking has been associated with antisocial behaviour in adolescents and adults, but its association with childhood antisocial behaviour is understudied. In this study, we explored the association of children's risk-taking with antisocial behaviour in mainstream elementary schoolchildren studied longitudinally across 7-11 years. METHODS: One thousand and eighty-six children (51% boys) were assessed in three annual waves. Antisocial behaviours (aggressive, covert antisocial and oppositional defiant behaviour) were assessed using teacher- and peer-reports. Risk-taking was measured using the Balloon Analogue Risk Task (BART). The association of antisocial behaviour with risk-taking was analysed using parallel growth models. RESULTS: Children with higher levels of risk-taking at age 7 showed increased growth in peer-reported aggression from age 7 to 11. Risk-taking, that is increased levels at age 7 in boys and increased growth in girls, predicted increased growth in peer-reported oppositional defiant behaviour. Associations of risk-taking with teacher-reported aggression and covert antisocial behaviour were at trend level. CONCLUSIONS: Results indicated that already in childhood, among typically developing children, risk-taking is associated with the development of antisocial behaviour. Future research focused on antisocial behaviour, but also school mental health workers and clinicians should take into account that already in childhood, risk-taking might affect antisocial behaviour development. En ligne : http://dx.doi.org/10.1111/jcpp.12943 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Elementary school children's associations of antisocial behaviour with risk-taking across 7-11 years [Texte imprimé et/ou numérique] / J. M. TIESKENS, Auteur ; Joanne Marieke BUIL, Auteur ; S. KOOT, Auteur ; L. KRABBENDAM, Auteur ; P. A. C. VAN LIER, Auteur . - p.1052-1060. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1052-1060 Mots-clés : Aggression children covert antisocial behaviour oppositional defiant behaviour risk-taking Index. décimale : PER Périodiques Résumé : BACKGROUND: The impact of childhood antisocial behaviour on future maladaptation has been acknowledged. Risk-taking has been associated with antisocial behaviour in adolescents and adults, but its association with childhood antisocial behaviour is understudied. In this study, we explored the association of children's risk-taking with antisocial behaviour in mainstream elementary schoolchildren studied longitudinally across 7-11 years. METHODS: One thousand and eighty-six children (51% boys) were assessed in three annual waves. Antisocial behaviours (aggressive, covert antisocial and oppositional defiant behaviour) were assessed using teacher- and peer-reports. Risk-taking was measured using the Balloon Analogue Risk Task (BART). The association of antisocial behaviour with risk-taking was analysed using parallel growth models. RESULTS: Children with higher levels of risk-taking at age 7 showed increased growth in peer-reported aggression from age 7 to 11. Risk-taking, that is increased levels at age 7 in boys and increased growth in girls, predicted increased growth in peer-reported oppositional defiant behaviour. Associations of risk-taking with teacher-reported aggression and covert antisocial behaviour were at trend level. CONCLUSIONS: Results indicated that already in childhood, among typically developing children, risk-taking is associated with the development of antisocial behaviour. Future research focused on antisocial behaviour, but also school mental health workers and clinicians should take into account that already in childhood, risk-taking might affect antisocial behaviour development. En ligne : http://dx.doi.org/10.1111/jcpp.12943 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Longitudinal structural brain development and externalizing behavior in adolescence / Marieke G. N. BOS in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1061-1072 Titre : Longitudinal structural brain development and externalizing behavior in adolescence Type de document : Texte imprimé et/ou numérique Auteurs : Marieke G. N. BOS, Auteur ; L. M. WIERENGA, Auteur ; N. E. BLANKENSTEIN, Auteur ; E. SCHREUDERS, Auteur ; C. K. TAMNES, Auteur ; E. A. CRONE, Auteur Article en page(s) : p.1061-1072 Langues : Anglais (eng) Mots-clés : Externalizing behavior adolescence aggression longitudinal design structural MRI Index. décimale : PER Périodiques Résumé : BACKGROUND: Cross-sectional studies report relations between externalizing behavior and structural abnormalities in cortical thickness of prefrontal regions and volume reductions in subcortical regions. To understand how these associations emerge and develop, longitudinal designs are pivotal. METHOD: In the current longitudinal study, a community sample of children, adolescents and young adults (N = 271) underwent magnetic resonance imaging (MRI) in three biennial waves (680 scans). At each wave, aspects of externalizing behavior were assessed with parent-reported aggression and rule-breaking scores (Child Behavior Checklist), and self-reported aggression scores (Buss-Perry Aggression Questionnaire). Regions of interest (ROIs) were selected based on prior research: dorsolateral prefrontal (dlPFC), orbitofrontal (OFC), anterior cingulate cortex (ACC), insula, and parahippocampal cortex, as well as subcortical regions. Linear mixed models were used to assess the longitudinal relation between externalizing behavior and structural brain development. Structural covariance analyses were employed to identify whether longitudinal relations between ROIs (maturational coupling) were associated with externalizing behavior. RESULTS: Linear mixed model analyses showed a negative relation between parent-reported aggression and right hippocampal volume. Moreover, this longitudinal relation was driven by change in hippocampal volume and not initial volume of hippocampus at time point 1. Exploratory analyses showed that stronger maturational coupling between prefrontal regions, the limbic system, and striatum was associated with both low and high externalizing behavior. CONCLUSIONS: Together, these findings reinforce the hypothesis that altered structural brain development coincides with development of more externalizing behavior. These findings may guide future research on normative and deviant development of externalizing behavior. En ligne : http://dx.doi.org/10.1111/jcpp.12972 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Longitudinal structural brain development and externalizing behavior in adolescence [Texte imprimé et/ou numérique] / Marieke G. N. BOS, Auteur ; L. M. WIERENGA, Auteur ; N. E. BLANKENSTEIN, Auteur ; E. SCHREUDERS, Auteur ; C. K. TAMNES, Auteur ; E. A. CRONE, Auteur . - p.1061-1072. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1061-1072 Mots-clés : Externalizing behavior adolescence aggression longitudinal design structural MRI Index. décimale : PER Périodiques Résumé : BACKGROUND: Cross-sectional studies report relations between externalizing behavior and structural abnormalities in cortical thickness of prefrontal regions and volume reductions in subcortical regions. To understand how these associations emerge and develop, longitudinal designs are pivotal. METHOD: In the current longitudinal study, a community sample of children, adolescents and young adults (N = 271) underwent magnetic resonance imaging (MRI) in three biennial waves (680 scans). At each wave, aspects of externalizing behavior were assessed with parent-reported aggression and rule-breaking scores (Child Behavior Checklist), and self-reported aggression scores (Buss-Perry Aggression Questionnaire). Regions of interest (ROIs) were selected based on prior research: dorsolateral prefrontal (dlPFC), orbitofrontal (OFC), anterior cingulate cortex (ACC), insula, and parahippocampal cortex, as well as subcortical regions. Linear mixed models were used to assess the longitudinal relation between externalizing behavior and structural brain development. Structural covariance analyses were employed to identify whether longitudinal relations between ROIs (maturational coupling) were associated with externalizing behavior. RESULTS: Linear mixed model analyses showed a negative relation between parent-reported aggression and right hippocampal volume. Moreover, this longitudinal relation was driven by change in hippocampal volume and not initial volume of hippocampus at time point 1. Exploratory analyses showed that stronger maturational coupling between prefrontal regions, the limbic system, and striatum was associated with both low and high externalizing behavior. CONCLUSIONS: Together, these findings reinforce the hypothesis that altered structural brain development coincides with development of more externalizing behavior. These findings may guide future research on normative and deviant development of externalizing behavior. En ligne : http://dx.doi.org/10.1111/jcpp.12972 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood / L. W. HYDE in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Titre : Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood Type de document : Texte imprimé et/ou numérique Auteurs : L. W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; L. MURRAY, Auteur ; E. E. FORBES, Auteur Article en page(s) : p.1073-1082 Langues : Anglais (eng) Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood [Texte imprimé et/ou numérique] / L. W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; L. MURRAY, Auteur ; E. E. FORBES, Auteur . - p.1073-1082. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Mediating pathways from childhood ADHD to adolescent tobacco and marijuana problems: roles of peer impairment, internalizing, adolescent ADHD symptoms, and gender / I. J. ELKINS in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1083-1093 Titre : Mediating pathways from childhood ADHD to adolescent tobacco and marijuana problems: roles of peer impairment, internalizing, adolescent ADHD symptoms, and gender Type de document : Texte imprimé et/ou numérique Auteurs : I. J. ELKINS, Auteur ; Gretchen R. B. SAUNDERS, Auteur ; S. M. MALONE, Auteur ; S. WILSON, Auteur ; M. MCGUE, Auteur ; W. G. IACONO, Auteur Article en page(s) : p.1083-1093 Langues : Anglais (eng) Mots-clés : Adhd gender difference longitudinal studies substance use twins Index. décimale : PER Périodiques Résumé : BACKGROUND: We examined whether increased risk for adolescent tobacco and marijuana problems associated with childhood ADHD is explained by key intermediary influences during adolescence and differs by gender. METHODS: Longitudinal structural equation models examined mediating effects on problems with both substances (or each substance separately) through age-14 peer impairment, internalizing, and adolescent ADHD symptoms in two twin samples, prospectively assessed since age 11 (N = 2,164). Whether these mediators contributed beyond mediating effects of early-adolescent substance use was also considered. Twin difference analyses further illuminated which mediators might be potentially causal. RESULTS: Direct effects of childhood ADHD on age-17 tobacco and marijuana problems (i.e., independent of included mediators) as well as effects of adolescent ADHD symptoms were significant only for females. By contrast, mediation by peer impairment, evident particularly for marijuana, was relatively stronger for males than females. Depression and anxiety were not prospectively associated with age-17 substance problems when earlier substance problems were considered. Consistent with causal influence of early substance use on later problems, monozygotic twins with more severe tobacco or marijuana problems at age 14 than their co-twins were also more likely to have substance problems later in adolescence. CONCLUSIONS: Mediation through peer impairment, continued presence of ADHD symptoms, and early substance use may alter development so that childhood ADHD indirectly contributes to problems with tobacco and marijuana. Targeting gender-sensitive interventions prior to mid-adolescence, before these patterns become established, is essential. En ligne : http://dx.doi.org/10.1111/jcpp.12977 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Mediating pathways from childhood ADHD to adolescent tobacco and marijuana problems: roles of peer impairment, internalizing, adolescent ADHD symptoms, and gender [Texte imprimé et/ou numérique] / I. J. ELKINS, Auteur ; Gretchen R. B. SAUNDERS, Auteur ; S. M. MALONE, Auteur ; S. WILSON, Auteur ; M. MCGUE, Auteur ; W. G. IACONO, Auteur . - p.1083-1093. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1083-1093 Mots-clés : Adhd gender difference longitudinal studies substance use twins Index. décimale : PER Périodiques Résumé : BACKGROUND: We examined whether increased risk for adolescent tobacco and marijuana problems associated with childhood ADHD is explained by key intermediary influences during adolescence and differs by gender. METHODS: Longitudinal structural equation models examined mediating effects on problems with both substances (or each substance separately) through age-14 peer impairment, internalizing, and adolescent ADHD symptoms in two twin samples, prospectively assessed since age 11 (N = 2,164). Whether these mediators contributed beyond mediating effects of early-adolescent substance use was also considered. Twin difference analyses further illuminated which mediators might be potentially causal. RESULTS: Direct effects of childhood ADHD on age-17 tobacco and marijuana problems (i.e., independent of included mediators) as well as effects of adolescent ADHD symptoms were significant only for females. By contrast, mediation by peer impairment, evident particularly for marijuana, was relatively stronger for males than females. Depression and anxiety were not prospectively associated with age-17 substance problems when earlier substance problems were considered. Consistent with causal influence of early substance use on later problems, monozygotic twins with more severe tobacco or marijuana problems at age 14 than their co-twins were also more likely to have substance problems later in adolescence. CONCLUSIONS: Mediation through peer impairment, continued presence of ADHD symptoms, and early substance use may alter development so that childhood ADHD indirectly contributes to problems with tobacco and marijuana. Targeting gender-sensitive interventions prior to mid-adolescence, before these patterns become established, is essential. En ligne : http://dx.doi.org/10.1111/jcpp.12977 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Differential impact of trait sluggish cognitive tempo and ADHD inattention in early childhood on adolescent functioning / Stephen P. BECKER in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1094-1104 Titre : Differential impact of trait sluggish cognitive tempo and ADHD inattention in early childhood on adolescent functioning Type de document : Texte imprimé et/ou numérique Auteurs : Stephen P. BECKER, Auteur ; G. Leonard BURNS, Auteur ; D. R. LEOPOLD, Auteur ; R. K. OLSON, Auteur ; E. G. WILLCUTT, Auteur Article en page(s) : p.1094-1104 Langues : Anglais (eng) Mots-clés : Adhd academic achievement attention-deficit/hyperactivity disorder comorbidity processing speed sluggish cognitive tempo working memory Index. décimale : PER Périodiques Résumé : BACKGROUND: Sluggish cognitive tempo (SCT) is distinct from attention-deficit/hyperactivity disorder inattention (ADHD-IN) and concurrently associated with a range of impairment domains. However, few longitudinal studies have examined SCT as a longitudinal predictor of adjustment. Studies to date have all used a relatively short longitudinal time span (6 months to 2 years) and only rating scale measures of adjustment. Using a prospective, multi-method design, this study examined whether SCT and ADHD-IN were differentially associated with functioning over a 10-year period between preschool and the end of ninth grade. METHODS: Latent state-trait modeling determined the trait variance (i.e. consistency across occasions) of SCT and ADHD-IN across four measurement points (preschool and the end of kindergarten, first grade, and second grade) in a large population-based longitudinal sample (N = 976). Regression analyses were used to examine trait SCT and ADHD-IN factors in early childhood as predictors of functioning at the end of ninth grade (i.e. parent ratings of psychopathology and social/academic functioning, reading and mathematics academic achievement scores, processing speed and working memory). RESULTS: Both SCT and ADHD-IN contained more trait variance (Ms = 65% and 61%, respectively) than occasion-specific variance (Ms = 35% and 39%) in early childhood, with trait variance increasing as children progressed from preschool through early elementary school. In regression analyses: (a) SCT significantly predicted greater withdrawal and anxiety/depression whereas ADHD-IN did not uniquely predict these internalizing domains; (b) ADHD-IN uniquely predicted more externalizing behaviors whereas SCT uniquely predicted fewer externalizing behaviors; (c) SCT uniquely predicted shyness whereas both SCT and ADHD-IN uniquely predicted global social difficulties; and (d) ADHD-IN uniquely predicted poorer math achievement and slower processing speed whereas SCT more consistently predicted poorer reading achievement. CONCLUSIONS: Findings of this study - from the longest prospective sample to date - provide the clearest evidence yet that SCT and ADHD-IN often differ when it comes to the functional outcomes they predict. En ligne : http://dx.doi.org/10.1111/jcpp.12946 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Differential impact of trait sluggish cognitive tempo and ADHD inattention in early childhood on adolescent functioning [Texte imprimé et/ou numérique] / Stephen P. BECKER, Auteur ; G. Leonard BURNS, Auteur ; D. R. LEOPOLD, Auteur ; R. K. OLSON, Auteur ; E. G. WILLCUTT, Auteur . - p.1094-1104. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1094-1104 Mots-clés : Adhd academic achievement attention-deficit/hyperactivity disorder comorbidity processing speed sluggish cognitive tempo working memory Index. décimale : PER Périodiques Résumé : BACKGROUND: Sluggish cognitive tempo (SCT) is distinct from attention-deficit/hyperactivity disorder inattention (ADHD-IN) and concurrently associated with a range of impairment domains. However, few longitudinal studies have examined SCT as a longitudinal predictor of adjustment. Studies to date have all used a relatively short longitudinal time span (6 months to 2 years) and only rating scale measures of adjustment. Using a prospective, multi-method design, this study examined whether SCT and ADHD-IN were differentially associated with functioning over a 10-year period between preschool and the end of ninth grade. METHODS: Latent state-trait modeling determined the trait variance (i.e. consistency across occasions) of SCT and ADHD-IN across four measurement points (preschool and the end of kindergarten, first grade, and second grade) in a large population-based longitudinal sample (N = 976). Regression analyses were used to examine trait SCT and ADHD-IN factors in early childhood as predictors of functioning at the end of ninth grade (i.e. parent ratings of psychopathology and social/academic functioning, reading and mathematics academic achievement scores, processing speed and working memory). RESULTS: Both SCT and ADHD-IN contained more trait variance (Ms = 65% and 61%, respectively) than occasion-specific variance (Ms = 35% and 39%) in early childhood, with trait variance increasing as children progressed from preschool through early elementary school. In regression analyses: (a) SCT significantly predicted greater withdrawal and anxiety/depression whereas ADHD-IN did not uniquely predict these internalizing domains; (b) ADHD-IN uniquely predicted more externalizing behaviors whereas SCT uniquely predicted fewer externalizing behaviors; (c) SCT uniquely predicted shyness whereas both SCT and ADHD-IN uniquely predicted global social difficulties; and (d) ADHD-IN uniquely predicted poorer math achievement and slower processing speed whereas SCT more consistently predicted poorer reading achievement. CONCLUSIONS: Findings of this study - from the longest prospective sample to date - provide the clearest evidence yet that SCT and ADHD-IN often differ when it comes to the functional outcomes they predict. En ligne : http://dx.doi.org/10.1111/jcpp.12946 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Investigating late-onset ADHD: a population cohort investigation in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1105-1113 Titre : Investigating late-onset ADHD: a population cohort investigation Type de document : Texte imprimé et/ou numérique Article en page(s) : p.1105-1113 Langues : Anglais (eng) Mots-clés : Avon Longitudinal Study of Parents and Children Late-onset ADHD Strengths and Difficulties Questionnaire neurodevelopment Index. décimale : PER Périodiques Résumé : BACKGROUND: Adult ADHD has been assumed to be a continuation of childhood-onset ADHD. However, recent studies have identified individuals with ADHD in adulthood who have not had ADHD in childhood. Whether or not these individuals have a 'typical' neurodevelopmental profile is not clear. METHODS: We tested two explanations for the emergence of apparent late-onset ADHD symptomatology using the ALSPAC epidemiological cohort, by grouping individuals according to their scores on the Strengths and Difficulties Questionnaire (SDQ) hyperactivity subscale at ages 12 and 17 years. First, we tested whether some of those with apparent late-onset ADHD symptoms had been potentially misclassified on the basis of earlier SDQ hyperactivity scores (ages 7, 8 and 9 years) or of subthreshold symptoms at age 12 years. Second, we investigated the possibility that those with 'genuine' late-onset ADHD symptoms had a delayed manifestation of the same liability that underlies childhood-onset symptoms, by investigating whether they had a similar profile of neurodevelopmental impairments (in the domains of autistic symptomatology, language, reading, spelling, executive functioning and IQ) as those with typical childhood-onset ADHD. RESULTS: N = 56/75 (75%) of those with apparent late-onset ADHD had had high ADHD scores at least one point in childhood, suggesting that they may have been misclassified on the basis of their score at age 12 years. The remaining 19 individuals (25%) with genuine late-onset ADHD symptoms did not show a profile of neurodevelopmental impairment typically seen in ADHD, instead showing similar levels of autistic symptoms, language skills, executive functioning ability and IQ to those without ADHD symptoms. The only exceptions were that this group showed reading and spelling problems at age 9 years. CONCLUSIONS: Our work suggests that this small number of individuals with genuine late-onset symptoms may not be most appropriately considered as having a typical neurodevelopmental disorder. En ligne : http://dx.doi.org/10.1111/jcpp.12911 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Investigating late-onset ADHD: a population cohort investigation [Texte imprimé et/ou numérique] . - p.1105-1113. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1105-1113 Mots-clés : Avon Longitudinal Study of Parents and Children Late-onset ADHD Strengths and Difficulties Questionnaire neurodevelopment Index. décimale : PER Périodiques Résumé : BACKGROUND: Adult ADHD has been assumed to be a continuation of childhood-onset ADHD. However, recent studies have identified individuals with ADHD in adulthood who have not had ADHD in childhood. Whether or not these individuals have a 'typical' neurodevelopmental profile is not clear. METHODS: We tested two explanations for the emergence of apparent late-onset ADHD symptomatology using the ALSPAC epidemiological cohort, by grouping individuals according to their scores on the Strengths and Difficulties Questionnaire (SDQ) hyperactivity subscale at ages 12 and 17 years. First, we tested whether some of those with apparent late-onset ADHD symptoms had been potentially misclassified on the basis of earlier SDQ hyperactivity scores (ages 7, 8 and 9 years) or of subthreshold symptoms at age 12 years. Second, we investigated the possibility that those with 'genuine' late-onset ADHD symptoms had a delayed manifestation of the same liability that underlies childhood-onset symptoms, by investigating whether they had a similar profile of neurodevelopmental impairments (in the domains of autistic symptomatology, language, reading, spelling, executive functioning and IQ) as those with typical childhood-onset ADHD. RESULTS: N = 56/75 (75%) of those with apparent late-onset ADHD had had high ADHD scores at least one point in childhood, suggesting that they may have been misclassified on the basis of their score at age 12 years. The remaining 19 individuals (25%) with genuine late-onset ADHD symptoms did not show a profile of neurodevelopmental impairment typically seen in ADHD, instead showing similar levels of autistic symptoms, language skills, executive functioning ability and IQ to those without ADHD symptoms. The only exceptions were that this group showed reading and spelling problems at age 9 years. CONCLUSIONS: Our work suggests that this small number of individuals with genuine late-onset symptoms may not be most appropriately considered as having a typical neurodevelopmental disorder. En ligne : http://dx.doi.org/10.1111/jcpp.12911 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

A multicohort, longitudinal study of cerebellar development in attention deficit hyperactivity disorder / P. SHAW in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1114-1123 Titre : A multicohort, longitudinal study of cerebellar development in attention deficit hyperactivity disorder Type de document : Texte imprimé et/ou numérique Auteurs : P. SHAW, Auteur ; A. ISHII-TAKAHASHI, Auteur ; M. T. PARK, Auteur ; G. A. DEVENYI, Auteur ; C. ZIBMAN, Auteur ; S. KASPAREK, Auteur ; G. SUDRE, Auteur ; A. MANGALMURTI, Auteur ; M. HOOGMAN, Auteur ; H. TIEMEIER, Auteur ; Georg G. VON POLIER, Auteur ; D. SHOOK, Auteur ; R. MUETZEL, Auteur ; M. M. CHAKRAVARTY, Auteur ; K. KONRAD, Auteur ; S. DURSTON, Auteur ; T. WHITE, Auteur Article en page(s) : p.1114-1123 Langues : Anglais (eng) Mots-clés : Cerebellum attention deficit hyperactivity disorder growth meta-analysis neuroanatomy white matter Index. décimale : PER Périodiques Résumé : BACKGROUND: The cerebellum supports many cognitive functions disrupted in attention deficit hyperactivity disorder (ADHD). Prior neuroanatomic studies have been often limited by small sample sizes, inconsistent findings, and a reliance on cross-sectional data, limiting inferences about cerebellar development. Here, we conduct a multicohort study using longitudinal data, to characterize cerebellar development. METHODS: Growth trajectories of the cerebellar vermis, hemispheres and white matter were estimated using piecewise linear regression from 1,656 youth; of whom 63% had longitudinal data, totaling 2,914 scans. Four cohorts participated, all contained childhood data (age 4-12 years); two had adolescent data (12-25 years). Growth parameters were combined using random-effects meta-analysis. RESULTS: Diagnostic differences in growth were confined to the corpus medullare (cerebellar white matter). Here, the ADHD group showed slower growth in early childhood compared to the typically developing group (left corpus medullare z = 2.49, p = .01; right z = 2.03, p = .04). This reversed in late childhood, with faster growth in ADHD in the left corpus medullare (z = 2.06, p = .04). Findings held when gender, intelligence, comorbidity, and psychostimulant medication were considered. DISCUSSION: Across four independent cohorts, containing predominately longitudinal data, we found diagnostic differences in the growth of cerebellar white matter. In ADHD, slower white matter growth in early childhood was followed by faster growth in late childhood. The findings are consistent with the concept of ADHD as a disorder of the brain's structural connections, formed partly by developing cortico-cerebellar white matter tracts. En ligne : http://dx.doi.org/10.1111/jcpp.12920 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] A multicohort, longitudinal study of cerebellar development in attention deficit hyperactivity disorder [Texte imprimé et/ou numérique] / P. SHAW, Auteur ; A. ISHII-TAKAHASHI, Auteur ; M. T. PARK, Auteur ; G. A. DEVENYI, Auteur ; C. ZIBMAN, Auteur ; S. KASPAREK, Auteur ; G. SUDRE, Auteur ; A. MANGALMURTI, Auteur ; M. HOOGMAN, Auteur ; H. TIEMEIER, Auteur ; Georg G. VON POLIER, Auteur ; D. SHOOK, Auteur ; R. MUETZEL, Auteur ; M. M. CHAKRAVARTY, Auteur ; K. KONRAD, Auteur ; S. DURSTON, Auteur ; T. WHITE, Auteur . - p.1114-1123. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1114-1123 Mots-clés : Cerebellum attention deficit hyperactivity disorder growth meta-analysis neuroanatomy white matter Index. décimale : PER Périodiques Résumé : BACKGROUND: The cerebellum supports many cognitive functions disrupted in attention deficit hyperactivity disorder (ADHD). Prior neuroanatomic studies have been often limited by small sample sizes, inconsistent findings, and a reliance on cross-sectional data, limiting inferences about cerebellar development. Here, we conduct a multicohort study using longitudinal data, to characterize cerebellar development. METHODS: Growth trajectories of the cerebellar vermis, hemispheres and white matter were estimated using piecewise linear regression from 1,656 youth; of whom 63% had longitudinal data, totaling 2,914 scans. Four cohorts participated, all contained childhood data (age 4-12 years); two had adolescent data (12-25 years). Growth parameters were combined using random-effects meta-analysis. RESULTS: Diagnostic differences in growth were confined to the corpus medullare (cerebellar white matter). Here, the ADHD group showed slower growth in early childhood compared to the typically developing group (left corpus medullare z = 2.49, p = .01; right z = 2.03, p = .04). This reversed in late childhood, with faster growth in ADHD in the left corpus medullare (z = 2.06, p = .04). Findings held when gender, intelligence, comorbidity, and psychostimulant medication were considered. DISCUSSION: Across four independent cohorts, containing predominately longitudinal data, we found diagnostic differences in the growth of cerebellar white matter. In ADHD, slower white matter growth in early childhood was followed by faster growth in late childhood. The findings are consistent with the concept of ADHD as a disorder of the brain's structural connections, formed partly by developing cortico-cerebellar white matter tracts. En ligne : http://dx.doi.org/10.1111/jcpp.12920 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369