Information processing in adolescents with bipolar I disorder / Jane WHITNEY in Journal of Child Psychology and Psychiatry, 53-9 (September 2012)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 53-9 (September 2012) . - p.937-45 Titre : Information processing in adolescents with bipolar I disorder Type de document : Texte imprimé et/ou numérique Auteurs : Jane WHITNEY, Auteur ; Jutta JOORMANN, Auteur ; Ian H. GOTLIB, Auteur ; Ryan G. KELLEY, Auteur ; Tenah ACQUAYE, Auteur ; Meghan HOWE, Auteur ; Kiki D. CHANG, Auteur ; Manpreet K. SINGH, Auteur Année de publication : 2012 Article en page(s) : p.937-45 Langues : Anglais (eng) Mots-clés : Adolescence  bipolar disorder  information-processing  memory bias  longitudinal Index. décimale : PER Périodiques Résumé : Background:  Cognitive models of bipolar I disorder (BD) may aid in identification of children who are especially vulnerable to chronic mood dysregulation. Information-processing biases related to memory and attention likely play a role in the development and persistence of BD among adolescents; however, these biases have not been extensively studied in youth with BD. Methods:  We administered the self-referent encoding task and the dot-probe task to adolescents with bipolar I disorder (BD, n = 35) and a demographically similar healthy comparison group (HC, n = 25) at baseline, and at a 1-year follow-up in a subset of this cohort (n = 22 per group). Results:  At both baseline and 1-year follow-up, there were significant interactions of group (BD, HC) and valence of stimulus (positive, negative adjective) on endorsement and recall of self-referent adjectives. HC adolescents endorsed and recalled more positive self-referent adjectives at baseline and follow-up while adolescents with BD endorsed and recalled more negative self-referent adjectives at baseline but not follow-up. Over time, depression symptomatology was associated with impaired memory for positive self-referent adjectives. There were no group differences in attentional bias at either time points. Conclusions:  Adolescents with BD exhibit bias away from endorsement and recall of positive adjectives, which remained stable over time and independent of mood state. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2012.02543.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=179 [article] Information processing in adolescents with bipolar I disorder [Texte imprimé et/ou numérique] / Jane WHITNEY, Auteur ; Jutta JOORMANN, Auteur ; Ian H. GOTLIB, Auteur ; Ryan G. KELLEY, Auteur ; Tenah ACQUAYE, Auteur ; Meghan HOWE, Auteur ; Kiki D. CHANG, Auteur ; Manpreet K. SINGH, Auteur . - 2012 . - p.937-45. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 53-9 (September 2012) . - p.937-45 Mots-clés : Adolescence  bipolar disorder  information-processing  memory bias  longitudinal Index. décimale : PER Périodiques Résumé : Background:  Cognitive models of bipolar I disorder (BD) may aid in identification of children who are especially vulnerable to chronic mood dysregulation. Information-processing biases related to memory and attention likely play a role in the development and persistence of BD among adolescents; however, these biases have not been extensively studied in youth with BD. Methods:  We administered the self-referent encoding task and the dot-probe task to adolescents with bipolar I disorder (BD, n = 35) and a demographically similar healthy comparison group (HC, n = 25) at baseline, and at a 1-year follow-up in a subset of this cohort (n = 22 per group). Results:  At both baseline and 1-year follow-up, there were significant interactions of group (BD, HC) and valence of stimulus (positive, negative adjective) on endorsement and recall of self-referent adjectives. HC adolescents endorsed and recalled more positive self-referent adjectives at baseline and follow-up while adolescents with BD endorsed and recalled more negative self-referent adjectives at baseline but not follow-up. Over time, depression symptomatology was associated with impaired memory for positive self-referent adjectives. There were no group differences in attentional bias at either time points. Conclusions:  Adolescents with BD exhibit bias away from endorsement and recall of positive adjectives, which remained stable over time and independent of mood state. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2012.02543.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=179

Prevention of bipolar disorder in at-risk children: Theoretical assumptions and empirical foundations / David J. MIKLOWITZ in Development and Psychopathology, 20-3 (Summer 2008)  

Public ISBD [article]  inDevelopment and Psychopathology > 20-3 (Summer 2008) . - p.881-897 Titre : Prevention of bipolar disorder in at-risk children: Theoretical assumptions and empirical foundations Type de document : Texte imprimé et/ou numérique Auteurs : David J. MIKLOWITZ, Auteur ; Kiki D. CHANG, Auteur Année de publication : 2008 Article en page(s) : p.881-897 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This article examines how bipolar symptoms emerge during development, and the potential role of psychosocial and pharmacological interventions in the prevention of the onset of the disorder. Early signs of bipolarity can be observed among children of bipolar parents and often take the form of subsyndromal presentations (e.g., mood lability, episodic elation or irritability, depression, inattention, and psychosocial impairment). However, many of these early presentations are diagnostically nonspecific. The few studies that have followed at-risk youth into adulthood find developmental discontinuities from childhood to adulthood. Biological markers (e.g., amygdalar volume) may ultimately increase our accuracy in identifying children who later develop bipolar I disorder, but few such markers have been identified. Stress, in the form of childhood adversity or highly conflictual families, is not a diagnostically specific causal agent but does place genetically and biologically vulnerable individuals at risk for a more pernicious course of illness. A preventative family-focused treatment for children with (a) at least one first-degree relative with bipolar disorder and (b) subsyndromal signs of bipolar disorder is described. This model attempts to address the multiple interactions of psychosocial and biological risk factors in the onset and course of bipolar disorder. En ligne : http://dx.doi.org/10.1017/s0954579408000424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=543 [article] Prevention of bipolar disorder in at-risk children: Theoretical assumptions and empirical foundations [Texte imprimé et/ou numérique] / David J. MIKLOWITZ, Auteur ; Kiki D. CHANG, Auteur . - 2008 . - p.881-897. Langues : Anglais (eng) in Development and Psychopathology > 20-3 (Summer 2008) . - p.881-897 Index. décimale : PER Périodiques Résumé : This article examines how bipolar symptoms emerge during development, and the potential role of psychosocial and pharmacological interventions in the prevention of the onset of the disorder. Early signs of bipolarity can be observed among children of bipolar parents and often take the form of subsyndromal presentations (e.g., mood lability, episodic elation or irritability, depression, inattention, and psychosocial impairment). However, many of these early presentations are diagnostically nonspecific. The few studies that have followed at-risk youth into adulthood find developmental discontinuities from childhood to adulthood. Biological markers (e.g., amygdalar volume) may ultimately increase our accuracy in identifying children who later develop bipolar I disorder, but few such markers have been identified. Stress, in the form of childhood adversity or highly conflictual families, is not a diagnostically specific causal agent but does place genetically and biologically vulnerable individuals at risk for a more pernicious course of illness. A preventative family-focused treatment for children with (a) at least one first-degree relative with bipolar disorder and (b) subsyndromal signs of bipolar disorder is described. This model attempts to address the multiple interactions of psychosocial and biological risk factors in the onset and course of bipolar disorder. En ligne : http://dx.doi.org/10.1017/s0954579408000424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=543

The role of the amygdala in bipolar disorder development / Amy GARRETT in Development and Psychopathology, 20-4 (Fall 2008)  

Public ISBD [article]  inDevelopment and Psychopathology > 20-4 (Fall 2008) . - p.1285-1296 Titre : The role of the amygdala in bipolar disorder development Type de document : Texte imprimé et/ou numérique Auteurs : Amy GARRETT, Auteur ; Kiki D. CHANG, Auteur Année de publication : 2008 Article en page(s) : p.1285-1296 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The amygdala has received great interest as a possible neurophysiological substrate of bipolar disorder (BD). This review summarizes information about the structure and function of the amygdala with attention to its role in experienced emotion and mood. We review the evidence for amygdala pathology in psychiatric conditions and discuss the role of the amygdala in BD during development. There appear to be consistent findings in the neuroimaging literature that suggest an etiological model for BD that involves abnormalities in the structure and function of the amygdala, but also depends on the failure of prefrontal cortical regions to modulate amygdala activity. In addition, evidence is accumulating to suggest that this model has flexible outcomes, depending on factors intrinsic and extrinsic to BD, and may follow several possible paths across the course of maturational development. En ligne : http://dx.doi.org/10.1017/s0954579408000618 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=603 [article] The role of the amygdala in bipolar disorder development [Texte imprimé et/ou numérique] / Amy GARRETT, Auteur ; Kiki D. CHANG, Auteur . - 2008 . - p.1285-1296. Langues : Anglais (eng) in Development and Psychopathology > 20-4 (Fall 2008) . - p.1285-1296 Index. décimale : PER Périodiques Résumé : The amygdala has received great interest as a possible neurophysiological substrate of bipolar disorder (BD). This review summarizes information about the structure and function of the amygdala with attention to its role in experienced emotion and mood. We review the evidence for amygdala pathology in psychiatric conditions and discuss the role of the amygdala in BD during development. There appear to be consistent findings in the neuroimaging literature that suggest an etiological model for BD that involves abnormalities in the structure and function of the amygdala, but also depends on the failure of prefrontal cortical regions to modulate amygdala activity. In addition, evidence is accumulating to suggest that this model has flexible outcomes, depending on factors intrinsic and extrinsic to BD, and may follow several possible paths across the course of maturational development. En ligne : http://dx.doi.org/10.1017/s0954579408000618 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=603

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