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Auteur Jan K. BUITELAAR |
Documents disponibles écrits par cet auteur (107)
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Insecure and Disorganised Attachment in Children with a Pervasive Developmental Disorder: Relationship with Social Interaction and Heart Rate / Sophie H. N. SWINKELS in Journal of Child Psychology and Psychiatry, 41-6 (September 2000)
[article]
Titre : Insecure and Disorganised Attachment in Children with a Pervasive Developmental Disorder: Relationship with Social Interaction and Heart Rate Type de document : Texte imprimé et/ou numérique Auteurs : Sophie H. N. SWINKELS, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur ; Jan K. BUITELAAR, Auteur ; Marinus H. VAN IJZENDOORN, Auteur ; Herman VAN ENGELAND, Auteur Année de publication : 2000 Article en page(s) : p.759-767 Langues : Anglais (eng) Mots-clés : Pervasive Developmental Disorder attachment autistic disorder child development ethology behavioural measures Index. décimale : PER Périodiques Résumé : This study on children with a Pervasive Developmental Disorder (PDD; N= 32), children with developmental language disorder (N= 22), and normally developing children (N= 28) sought to answer questions concerning attachment and autistic behaviour. We could replicate the finding that children with a PDD are able to develop secure attachment relationships to their primary caregiver. Children withPDDwho had an insecure attachment showed fewer social initiatives and responses than children with PDD who had a secure attachment, even when the insecurely and securely attached PDD children were matched on chronological and mental age. Children with both a PDD and mental retardation were more often classified as disorganised. Three findings suggested that a disorganised attachment does not merely reflect the presence of “autistic” behaviour: (1) children with PDD did not reveal higher rates of a disorganised attachment than matched comparison children; (2) having a PDD diagnosis and having a disorganised attachment were found to be associated with opposite effects on an ethological measure of level of behavioural organisation; and (3) a disorganised attachment but not a PDD diagnosis was associated with an increase in heart rate during parting with the caregiver and a decrease in heart rate during reunion. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=125
in Journal of Child Psychology and Psychiatry > 41-6 (September 2000) . - p.759-767[article] Insecure and Disorganised Attachment in Children with a Pervasive Developmental Disorder: Relationship with Social Interaction and Heart Rate [Texte imprimé et/ou numérique] / Sophie H. N. SWINKELS, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur ; Jan K. BUITELAAR, Auteur ; Marinus H. VAN IJZENDOORN, Auteur ; Herman VAN ENGELAND, Auteur . - 2000 . - p.759-767.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 41-6 (September 2000) . - p.759-767
Mots-clés : Pervasive Developmental Disorder attachment autistic disorder child development ethology behavioural measures Index. décimale : PER Périodiques Résumé : This study on children with a Pervasive Developmental Disorder (PDD; N= 32), children with developmental language disorder (N= 22), and normally developing children (N= 28) sought to answer questions concerning attachment and autistic behaviour. We could replicate the finding that children with a PDD are able to develop secure attachment relationships to their primary caregiver. Children withPDDwho had an insecure attachment showed fewer social initiatives and responses than children with PDD who had a secure attachment, even when the insecurely and securely attached PDD children were matched on chronological and mental age. Children with both a PDD and mental retardation were more often classified as disorganised. Three findings suggested that a disorganised attachment does not merely reflect the presence of “autistic” behaviour: (1) children with PDD did not reveal higher rates of a disorganised attachment than matched comparison children; (2) having a PDD diagnosis and having a disorganised attachment were found to be associated with opposite effects on an ethological measure of level of behavioural organisation; and (3) a disorganised attachment but not a PDD diagnosis was associated with an increase in heart rate during parting with the caregiver and a decrease in heart rate during reunion. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=125 Intact Spectral but Abnormal Temporal Processing of Auditory Stimuli in Autism / Wouter B. GROEN in Journal of Autism and Developmental Disorders, 39-5 (May 2009)
[article]
Titre : Intact Spectral but Abnormal Temporal Processing of Auditory Stimuli in Autism Type de document : Texte imprimé et/ou numérique Auteurs : Wouter B. GROEN, Auteur ; Sophie H. N. SWINKELS, Auteur ; Rutger Jan VAN DER GAAG, Auteur ; Jan K. BUITELAAR, Auteur ; Linda VAN ORSOUW, Auteur ; Marcel ZWIERS, Auteur ; Niels ter HUURNE, Auteur Année de publication : 2009 Article en page(s) : p.742-750 Langues : Anglais (eng) Mots-clés : Autism Connectivity Auditory Perception Speech-in-noise Index. décimale : PER Périodiques Résumé : The perceptual pattern in autism has been related to either a specific localized processing deficit or a pathway-independent, complexity-specific anomaly. We examined auditory perception in autism using an auditory disembedding task that required spectral and temporal integration. 23 children with high-functioning-autism and 23 matched controls participated. Participants were presented with two-syllable words embedded in various auditory backgrounds (pink noise, moving ripple, amplitude-modulated pink noise, amplitude-modulated moving ripple) to assess speech-in-noise-reception thresholds. The gain in signal perception of pink noise with temporal dips relative to pink noise without temporal dips was smaller in children with autism (p = 0.008). Thus, the autism group was less able to integrate auditory information present in temporal dips in background sound, supporting the complexity-specific perceptual account. En ligne : http://dx.doi.org/10.1007/s10803-008-0682-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=732
in Journal of Autism and Developmental Disorders > 39-5 (May 2009) . - p.742-750[article] Intact Spectral but Abnormal Temporal Processing of Auditory Stimuli in Autism [Texte imprimé et/ou numérique] / Wouter B. GROEN, Auteur ; Sophie H. N. SWINKELS, Auteur ; Rutger Jan VAN DER GAAG, Auteur ; Jan K. BUITELAAR, Auteur ; Linda VAN ORSOUW, Auteur ; Marcel ZWIERS, Auteur ; Niels ter HUURNE, Auteur . - 2009 . - p.742-750.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 39-5 (May 2009) . - p.742-750
Mots-clés : Autism Connectivity Auditory Perception Speech-in-noise Index. décimale : PER Périodiques Résumé : The perceptual pattern in autism has been related to either a specific localized processing deficit or a pathway-independent, complexity-specific anomaly. We examined auditory perception in autism using an auditory disembedding task that required spectral and temporal integration. 23 children with high-functioning-autism and 23 matched controls participated. Participants were presented with two-syllable words embedded in various auditory backgrounds (pink noise, moving ripple, amplitude-modulated pink noise, amplitude-modulated moving ripple) to assess speech-in-noise-reception thresholds. The gain in signal perception of pink noise with temporal dips relative to pink noise without temporal dips was smaller in children with autism (p = 0.008). Thus, the autism group was less able to integrate auditory information present in temporal dips in background sound, supporting the complexity-specific perceptual account. En ligne : http://dx.doi.org/10.1007/s10803-008-0682-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=732 Internal model deficits impair joint action in children and adolescents with autism spectrum disorders / Astrid M.B. STOIT in Research in Autism Spectrum Disorders, 5-4 (October-December 2011)
[article]
Titre : Internal model deficits impair joint action in children and adolescents with autism spectrum disorders Type de document : Texte imprimé et/ou numérique Auteurs : Astrid M.B. STOIT, Auteur ; Hein T. VAN SCHIE, Auteur ; Madelon RIEM, Auteur ; Ruud G.J. MEULENBROEK, Auteur ; Roger D. NEWMAN-NORLUND, Auteur ; Dorine I.E. SLAATS–WILLEMSE, Auteur ; Harold BEKKERING, Auteur ; Jan K. BUITELAAR, Auteur Année de publication : 2011 Article en page(s) : p.1526-1537 Langues : Anglais (eng) Mots-clés : Autism Joint action Internal models Agency Self-other differentiation Index. décimale : PER Périodiques Résumé : Qualitative differences in social interaction and communication are diagnostic hallmarks in autism spectrum disorders (ASD). The present study investigated the hypothesis that impaired social interaction in ASD reflects a deficit to internally model the behavior of a co-actor. Children and adolescents with ASD and matched controls performed a computerized bar-balancing task in a solo condition (S), and together with another individual in two joint action conditions (J2 and J4) in which they used either two or four hands to control the bar lift. Consistent with predictions derived from the ‘internal modelling hypothesis’, results from the J2 task indicated that ASD dyads were impaired in predicting the occurrence of their partner's response and failed to coordinate their actions in time. Furthermore, results from the J4 task showed that ASD participants used an adaptive strategy to disambiguate their responses from their partner's by regulating opposite sides of the bar during lifting. These findings provide empirical support of theories positing the existence of an internal modelling deficit in ASD. In addition, our findings suggest that impaired social reciprocal behavior and joint cooperative play exhibited by individuals with ASD may reflect behavioral adaptations to evade conflicting or ambiguous information in social settings. En ligne : http://dx.doi.org/10.1016/j.rasd.2011.02.016 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=126
in Research in Autism Spectrum Disorders > 5-4 (October-December 2011) . - p.1526-1537[article] Internal model deficits impair joint action in children and adolescents with autism spectrum disorders [Texte imprimé et/ou numérique] / Astrid M.B. STOIT, Auteur ; Hein T. VAN SCHIE, Auteur ; Madelon RIEM, Auteur ; Ruud G.J. MEULENBROEK, Auteur ; Roger D. NEWMAN-NORLUND, Auteur ; Dorine I.E. SLAATS–WILLEMSE, Auteur ; Harold BEKKERING, Auteur ; Jan K. BUITELAAR, Auteur . - 2011 . - p.1526-1537.
Langues : Anglais (eng)
in Research in Autism Spectrum Disorders > 5-4 (October-December 2011) . - p.1526-1537
Mots-clés : Autism Joint action Internal models Agency Self-other differentiation Index. décimale : PER Périodiques Résumé : Qualitative differences in social interaction and communication are diagnostic hallmarks in autism spectrum disorders (ASD). The present study investigated the hypothesis that impaired social interaction in ASD reflects a deficit to internally model the behavior of a co-actor. Children and adolescents with ASD and matched controls performed a computerized bar-balancing task in a solo condition (S), and together with another individual in two joint action conditions (J2 and J4) in which they used either two or four hands to control the bar lift. Consistent with predictions derived from the ‘internal modelling hypothesis’, results from the J2 task indicated that ASD dyads were impaired in predicting the occurrence of their partner's response and failed to coordinate their actions in time. Furthermore, results from the J4 task showed that ASD participants used an adaptive strategy to disambiguate their responses from their partner's by regulating opposite sides of the bar during lifting. These findings provide empirical support of theories positing the existence of an internal modelling deficit in ASD. In addition, our findings suggest that impaired social reciprocal behavior and joint cooperative play exhibited by individuals with ASD may reflect behavioral adaptations to evade conflicting or ambiguous information in social settings. En ligne : http://dx.doi.org/10.1016/j.rasd.2011.02.016 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=126 Investigating the factors underlying adaptive functioning in autism in the EU-AIMS Longitudinal European Autism Project / J. TILLMANN in Autism Research, 12-4 (April 2019)
[article]
Titre : Investigating the factors underlying adaptive functioning in autism in the EU-AIMS Longitudinal European Autism Project Type de document : Texte imprimé et/ou numérique Auteurs : J. TILLMANN, Auteur ; Antonia SAN JOSE CACERES, Auteur ; Christopher H. CHATHAM, Auteur ; D. CRAWLEY, Auteur ; R. HOLT, Auteur ; B. OAKLEY, Auteur ; Tobias BANASCHEWSKI, Auteur ; Simon BARON-COHEN, Auteur ; Sven BÖLTE, Auteur ; Jan K. BUITELAAR, Auteur ; S. DURSTON, Auteur ; L. HAM, Auteur ; E. LOTH, Auteur ; E. SIMONOFF, Auteur ; W. SPOOREN, Auteur ; D. G. MURPHY, Auteur ; Tony CHARMAN, Auteur Article en page(s) : p.645-657 Langues : Anglais (eng) Mots-clés : adaptive functioning autism spectrum disorder intellectual functioning psychiatric symptoms symptom severity Index. décimale : PER Périodiques Résumé : Individuals with autism spectrum disorder (ASD) exhibit significant impairments in adaptive functioning that impact on their ability to meet the demands of everyday life. A recurrent finding is that there is a pronounced discrepancy between level of cognitive ability and adaptive functioning, and this is particularly prominent among higher-ability individuals. However, the key clinical and demographic associations of these discrepancies remain unclear. This study included a sample of 417 children, adolescents, and adults with ASD as part of the EU-AIMS LEAP cohort. We examined how age, sex, IQ, levels of ASD symptom and autistic trait severity and psychiatric symptomatology are associated with adaptive functioning as measured by the Vineland Adaptive Behavior Scales-Second Edition and IQ-adaptive functioning discrepancies. Older age, lower IQ and higher social-communication symptoms were associated with lower adaptive functioning. Results also demonstrate that older age, higher IQ and higher social-communication symptoms are associated with greater IQ-adaptive functioning discrepancy scores. By contrast, sensory ASD symptoms, repetitive and restricted behaviors, as well as symptoms of attention deficit/hyperactivity disorder (ADHD), anxiety and depression, were not associated with adaptive functioning or IQ-adaptive functioning discrepancy scores. These findings suggest that it is the core social communication problems that define ASD that contribute to adaptive function impairments that people with ASD experience. They show for the first time that sensory symptoms, repetitive behavior and associated psychiatric symptoms do not independently contribute to adaptive function impairments. Individuals with ASD require supportive interventions across the lifespan that take account of social-communicative ASD symptom severity. Autism Res 2019, 12: 645-657. (c) 2019 The Authors. Autism Research published by International Society for Autism Research published by Wiley Periodicals, Inc. LAY SUMMARY: This study investigated key clinical and demographic associations of adaptive functioning impairments in individuals with autism. We found that older age, lower IQ and more severe social-communicative symptoms, but not sensory or repetitive symptoms or co-occurring psychiatric symptoms, are associated with lower adaptive functioning and greater ability-adaptive function discrepancies. This suggests that interventions targeting adaptive skills acquisition should be flexible in their timing and intensity across developmental periods, levels of cognitive ability and take account of social-communicative ASD symptom severity. En ligne : https://dx.doi.org/10.1002/aur.2081 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=389
in Autism Research > 12-4 (April 2019) . - p.645-657[article] Investigating the factors underlying adaptive functioning in autism in the EU-AIMS Longitudinal European Autism Project [Texte imprimé et/ou numérique] / J. TILLMANN, Auteur ; Antonia SAN JOSE CACERES, Auteur ; Christopher H. CHATHAM, Auteur ; D. CRAWLEY, Auteur ; R. HOLT, Auteur ; B. OAKLEY, Auteur ; Tobias BANASCHEWSKI, Auteur ; Simon BARON-COHEN, Auteur ; Sven BÖLTE, Auteur ; Jan K. BUITELAAR, Auteur ; S. DURSTON, Auteur ; L. HAM, Auteur ; E. LOTH, Auteur ; E. SIMONOFF, Auteur ; W. SPOOREN, Auteur ; D. G. MURPHY, Auteur ; Tony CHARMAN, Auteur . - p.645-657.
Langues : Anglais (eng)
in Autism Research > 12-4 (April 2019) . - p.645-657
Mots-clés : adaptive functioning autism spectrum disorder intellectual functioning psychiatric symptoms symptom severity Index. décimale : PER Périodiques Résumé : Individuals with autism spectrum disorder (ASD) exhibit significant impairments in adaptive functioning that impact on their ability to meet the demands of everyday life. A recurrent finding is that there is a pronounced discrepancy between level of cognitive ability and adaptive functioning, and this is particularly prominent among higher-ability individuals. However, the key clinical and demographic associations of these discrepancies remain unclear. This study included a sample of 417 children, adolescents, and adults with ASD as part of the EU-AIMS LEAP cohort. We examined how age, sex, IQ, levels of ASD symptom and autistic trait severity and psychiatric symptomatology are associated with adaptive functioning as measured by the Vineland Adaptive Behavior Scales-Second Edition and IQ-adaptive functioning discrepancies. Older age, lower IQ and higher social-communication symptoms were associated with lower adaptive functioning. Results also demonstrate that older age, higher IQ and higher social-communication symptoms are associated with greater IQ-adaptive functioning discrepancy scores. By contrast, sensory ASD symptoms, repetitive and restricted behaviors, as well as symptoms of attention deficit/hyperactivity disorder (ADHD), anxiety and depression, were not associated with adaptive functioning or IQ-adaptive functioning discrepancy scores. These findings suggest that it is the core social communication problems that define ASD that contribute to adaptive function impairments that people with ASD experience. They show for the first time that sensory symptoms, repetitive behavior and associated psychiatric symptoms do not independently contribute to adaptive function impairments. Individuals with ASD require supportive interventions across the lifespan that take account of social-communicative ASD symptom severity. Autism Res 2019, 12: 645-657. (c) 2019 The Authors. Autism Research published by International Society for Autism Research published by Wiley Periodicals, Inc. LAY SUMMARY: This study investigated key clinical and demographic associations of adaptive functioning impairments in individuals with autism. We found that older age, lower IQ and more severe social-communicative symptoms, but not sensory or repetitive symptoms or co-occurring psychiatric symptoms, are associated with lower adaptive functioning and greater ability-adaptive function discrepancies. This suggests that interventions targeting adaptive skills acquisition should be flexible in their timing and intensity across developmental periods, levels of cognitive ability and take account of social-communicative ASD symptom severity. En ligne : https://dx.doi.org/10.1002/aur.2081 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=389 Lack of replication of previous autism spectrum disorder GWAS hits in European populations / Bàrbara TORRICO in Autism Research, 10-2 (February 2017)
[article]
Titre : Lack of replication of previous autism spectrum disorder GWAS hits in European populations Type de document : Texte imprimé et/ou numérique Auteurs : Bàrbara TORRICO, Auteur ; Andreas G. CHIOCCHETTI, Auteur ; Elena BACCHELLI, Auteur ; Elisabetta TRABETTI, Auteur ; Amaia HERVAS, Auteur ; Barbara FRANKE, Auteur ; Jan K. BUITELAAR, Auteur ; Nanda N. ROMMELSE, Auteur ; Afsheen YOUSAF, Auteur ; Eftichia DUKETIS, Auteur ; Christine M. FREITAG, Auteur ; Rafaela CABALLERO-ANDALUZ, Auteur ; Amalia MARTINEZ-MIR, Auteur ; Francisco G. SCHOLL, Auteur ; Marta RIBASES, Auteur ; ITAN, Auteur ; Agatino BATTAGLIA, Auteur ; Giovanni MALERBA, Auteur ; Richard DELORME, Auteur ; Marion BENABOU, Auteur ; Elena MAESTRINI, Auteur ; Thomas BOURGERON, Auteur ; Bru CORMAND, Auteur ; Claudio TOMA, Auteur Article en page(s) : p.202-211 Langues : Anglais (eng) Mots-clés : genome-wide association study replication autism spectrum disorder European populations MACROD2 SEMA5A MSNP1 Index. décimale : PER Périodiques Résumé : Common variants contribute significantly to the genetics of autism spectrum disorder (ASD), although the identification of individual risk polymorphisms remains still elusive due to their small effect sizes and limited sample sizes available for association studies. During the last decade several genome-wide association studies (GWAS) have enabled the detection of a few plausible risk variants. The three main studies are family-based and pointed at SEMA5A (rs10513025), MACROD2 (rs4141463) and MSNP1 (rs4307059). In our study we attempted to replicate these GWAS hits using a case-control association study in five European populations of ASD patients and gender-matched controls, all Caucasians. Results showed no association of individual variants with ASD in any of the population groups considered or in the combined European sample. We performed a meta-analysis study across five European populations for rs10513025 (1,904 ASD cases and 2,674 controls), seven European populations for rs4141463 (2,855 ASD cases and 36,177 controls) and five European populations for rs4307059 (2,347 ASD cases and 2,764 controls). The results showed an odds ratio (OR) of 1.05 (95% CI?=?0.84–1.32) for rs10513025, 1.0002 (95% CI?=?0.93–1.08) for rs4141463 and 1.01 (95% CI?=?0.92–1.1) for rs4307059, with no significant P-values (rs10513025, P?=?0.73; rs4141463, P?=?0.95; rs4307059, P?=?0.9). No association was found when we considered either only high functioning autism (HFA), genders separately or only multiplex families. Ongoing GWAS projects with larger ASD cohorts will contribute to clarify the role of common variation in the disorder and will likely identify risk variants of modest effect not detected previously. En ligne : http://dx.doi.org/10.1002/aur.1662 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=303
in Autism Research > 10-2 (February 2017) . - p.202-211[article] Lack of replication of previous autism spectrum disorder GWAS hits in European populations [Texte imprimé et/ou numérique] / Bàrbara TORRICO, Auteur ; Andreas G. CHIOCCHETTI, Auteur ; Elena BACCHELLI, Auteur ; Elisabetta TRABETTI, Auteur ; Amaia HERVAS, Auteur ; Barbara FRANKE, Auteur ; Jan K. BUITELAAR, Auteur ; Nanda N. ROMMELSE, Auteur ; Afsheen YOUSAF, Auteur ; Eftichia DUKETIS, Auteur ; Christine M. FREITAG, Auteur ; Rafaela CABALLERO-ANDALUZ, Auteur ; Amalia MARTINEZ-MIR, Auteur ; Francisco G. SCHOLL, Auteur ; Marta RIBASES, Auteur ; ITAN, Auteur ; Agatino BATTAGLIA, Auteur ; Giovanni MALERBA, Auteur ; Richard DELORME, Auteur ; Marion BENABOU, Auteur ; Elena MAESTRINI, Auteur ; Thomas BOURGERON, Auteur ; Bru CORMAND, Auteur ; Claudio TOMA, Auteur . - p.202-211.
Langues : Anglais (eng)
in Autism Research > 10-2 (February 2017) . - p.202-211
Mots-clés : genome-wide association study replication autism spectrum disorder European populations MACROD2 SEMA5A MSNP1 Index. décimale : PER Périodiques Résumé : Common variants contribute significantly to the genetics of autism spectrum disorder (ASD), although the identification of individual risk polymorphisms remains still elusive due to their small effect sizes and limited sample sizes available for association studies. During the last decade several genome-wide association studies (GWAS) have enabled the detection of a few plausible risk variants. The three main studies are family-based and pointed at SEMA5A (rs10513025), MACROD2 (rs4141463) and MSNP1 (rs4307059). In our study we attempted to replicate these GWAS hits using a case-control association study in five European populations of ASD patients and gender-matched controls, all Caucasians. Results showed no association of individual variants with ASD in any of the population groups considered or in the combined European sample. We performed a meta-analysis study across five European populations for rs10513025 (1,904 ASD cases and 2,674 controls), seven European populations for rs4141463 (2,855 ASD cases and 36,177 controls) and five European populations for rs4307059 (2,347 ASD cases and 2,764 controls). The results showed an odds ratio (OR) of 1.05 (95% CI?=?0.84–1.32) for rs10513025, 1.0002 (95% CI?=?0.93–1.08) for rs4141463 and 1.01 (95% CI?=?0.92–1.1) for rs4307059, with no significant P-values (rs10513025, P?=?0.73; rs4141463, P?=?0.95; rs4307059, P?=?0.9). No association was found when we considered either only high functioning autism (HFA), genders separately or only multiplex families. Ongoing GWAS projects with larger ASD cohorts will contribute to clarify the role of common variation in the disorder and will likely identify risk variants of modest effect not detected previously. En ligne : http://dx.doi.org/10.1002/aur.1662 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=303 Latent trajectories of adaptive behaviour in infants at high and low familial risk for autism spectrum disorder / G. BUSSU in Molecular Autism, 10 (2019)
PermalinkLinking functional and structural brain organisation with behaviour in autism: a multimodal EU-AIMS Longitudinal European Autism Project (LEAP) study / Alberto LLERA ; Ting MEI ; Koen HAAK ; Christina ISAKOGLOU ; Dorothea L. FLORIS ; Sarah DURSTON ; Carolin MOESSNANG ; Tobias BANASCHEWSKI ; Simon BARON-COHEN ; Eva LOTH ; Flavio DELL'ACQUA ; Tony CHARMAN ; Declan G. M. MURPHY ; Christine ECKER ; Jan K. BUITELAAR ; Christian F. BECKMANN in Molecular Autism, 14 (2023)
PermalinkLong-term effects of neurofeedback treatment in autism / Mirjam E.J. KOUIJZER in Research in Autism Spectrum Disorders, 3-2 (April-june 2009)
PermalinkMatch or Mismatch? Influence of Parental and Offspring ASD and ADHD Symptoms on the Parent–Child Relationship / Daphne J. STEIJN in Journal of Autism and Developmental Disorders, 43-8 (August 2013)
PermalinkMotor control in children with ADHD and non-affected siblings: deficits most pronounced using the left hand / Nanda N. ROMMELSE in Journal of Child Psychology and Psychiatry, 48-11 (November 2007)
PermalinkNarrowly Versus Broadly Defined Autism Spectrum Disorders: Differences in Pre- and Perinatal Risk Factors / Janne C. VISSER in Journal of Autism and Developmental Disorders, 43-7 (July 2013)
PermalinkNetwork Structure of Autism Spectrum Disorder Behaviors and Its Evolution in Preschool Children: Insights from a New Longitudinal Network Analysis Method / Farhad MONTAZERI in Journal of Autism and Developmental Disorders, 53-11 (November 2023)
PermalinkNeurocognitive markers of late-onset ADHD: a 6-year longitudinal study / Shahrzad ILBEGI in Journal of Child Psychology and Psychiatry, 62-2 (February 2021)
PermalinkNeurocognitive predictors of substance use disorders and nicotine dependence in ADHD probands, their unaffected siblings, and controls: a 4-year prospective follow-up / Annabeth P. GROENMAN in Journal of Child Psychology and Psychiatry, 56-5 (May 2015)
PermalinkNeurofeedback treatment in autism. Preliminary findings in behavioral, cognitive, and neurophysiological functioning / Mirjam E.J. KOUIJZER in Research in Autism Spectrum Disorders, 4-3 (July-September 2010)
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