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Association between MTHFR gene polymorphism and susceptibility to autism spectrum disorders: Systematic review and meta-analysis / Bahman RAZI in Research in Autism Spectrum Disorders, 70 (February 2020)
[article]
Titre : Association between MTHFR gene polymorphism and susceptibility to autism spectrum disorders: Systematic review and meta-analysis Type de document : Texte imprimé et/ou numérique Auteurs : Bahman RAZI, Auteur ; Danyal IMANI, Auteur ; Masoud HASSANZADEH MAKOUI, Auteur ; Ramazan REZAEI, Auteur ; Saeed ASLANI, Auteur Article en page(s) : p.101473 Langues : Anglais (eng) Mots-clés : Methylenetetrahydrofolate reductase Autism spectrum disorder Meta-analysis Polymorphism Index. décimale : PER Périodiques Résumé : Objective A growing number of studies have highlighted the potential link between MTHFR gene polymorphisms and autism spectrum disorder (ASD), however, the results are conflictive. Therefore, this meta-analysis was conducted to find a persuasive answer for possible association or lack of association between MTHFR gene polymorphism and ASD risk. Methods Systematic search was conducted in PubMed, Web of Science, and Scopus considering the association between MTHFR gene (C677?T and A1298C) polymorphisms and the risk of ASD prior to Jun 2019. Results A total of 17 studies for C677T and 8 studies for A1298C MTHFR gene polymorphism recognized eligible. The pooled results suggested a significant association between C677T SNP and ASD risk under dominant model (OR?=?1.47, 95%CI?=?1.13–2.93, REM), allelic model (OR?=?1.37, 95% CI?=?1.08–1.74, REM), and CT vs CC model (OR?=?1.45, 95% CI?=?1.13–1.85, REM). Moreover, the pooled results of subgroup analysis confirmed increased risk of ASD in Caucasians across all genetic models except recessive model. Besides, in spite of no significant association between A1298C SNP and ASD in overall population, subgroup analysis revealed that presence of A1298C SNP decrease risk of ASD in Caucasians. Conclusion This meta-analysis suggested a significant association between MTHFR gene polymorphism (C677T and A1298C) and ASD risk. En ligne : https://doi.org/10.1016/j.rasd.2019.101473 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=414
in Research in Autism Spectrum Disorders > 70 (February 2020) . - p.101473[article] Association between MTHFR gene polymorphism and susceptibility to autism spectrum disorders: Systematic review and meta-analysis [Texte imprimé et/ou numérique] / Bahman RAZI, Auteur ; Danyal IMANI, Auteur ; Masoud HASSANZADEH MAKOUI, Auteur ; Ramazan REZAEI, Auteur ; Saeed ASLANI, Auteur . - p.101473.
Langues : Anglais (eng)
in Research in Autism Spectrum Disorders > 70 (February 2020) . - p.101473
Mots-clés : Methylenetetrahydrofolate reductase Autism spectrum disorder Meta-analysis Polymorphism Index. décimale : PER Périodiques Résumé : Objective A growing number of studies have highlighted the potential link between MTHFR gene polymorphisms and autism spectrum disorder (ASD), however, the results are conflictive. Therefore, this meta-analysis was conducted to find a persuasive answer for possible association or lack of association between MTHFR gene polymorphism and ASD risk. Methods Systematic search was conducted in PubMed, Web of Science, and Scopus considering the association between MTHFR gene (C677?T and A1298C) polymorphisms and the risk of ASD prior to Jun 2019. Results A total of 17 studies for C677T and 8 studies for A1298C MTHFR gene polymorphism recognized eligible. The pooled results suggested a significant association between C677T SNP and ASD risk under dominant model (OR?=?1.47, 95%CI?=?1.13–2.93, REM), allelic model (OR?=?1.37, 95% CI?=?1.08–1.74, REM), and CT vs CC model (OR?=?1.45, 95% CI?=?1.13–1.85, REM). Moreover, the pooled results of subgroup analysis confirmed increased risk of ASD in Caucasians across all genetic models except recessive model. Besides, in spite of no significant association between A1298C SNP and ASD in overall population, subgroup analysis revealed that presence of A1298C SNP decrease risk of ASD in Caucasians. Conclusion This meta-analysis suggested a significant association between MTHFR gene polymorphism (C677T and A1298C) and ASD risk. En ligne : https://doi.org/10.1016/j.rasd.2019.101473 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=414 Association between MTHFR Gene Polymorphisms and the Risk of Autism Spectrum Disorders: A Meta-Analysis / Danhua PU in Autism Research, 6-5 (October 2013)
[article]
Titre : Association between MTHFR Gene Polymorphisms and the Risk of Autism Spectrum Disorders: A Meta-Analysis Type de document : Texte imprimé et/ou numérique Auteurs : Danhua PU, Auteur ; Yiping SHEN, Auteur ; Jie WU, Auteur Article en page(s) : p.384-392 Langues : Anglais (eng) Mots-clés : methylenetetrahydrofolate reductase polymorphism autism spectrum disorders folic acidl meta-analysis Index. décimale : PER Périodiques Résumé : Methylenetetrahydrofolate reductase (MTHFR) is essential for DNA biosynthesis and the epigenetic process of DNA methylation, and its gene polymorphisms have been implicated as risk factors for birth defects, neurological disorders, and cancers. However, reports on the association of MTHFR polymorphisms with autism spectrum disorders (ASD) are inconclusive. Therefore, we investigated the relationship of the MTHFR polymorphisms (C677T and A1298C) and the risk of ASD by meta-analysis. Up to December 2012, eight case-control studies involving 1672 patients with ASD and 6760 controls were included for meta-analysis. The results showed that the C677T polymorphism was associated with significantly increased ASD risk in all the comparison models [T vs. C allele (frequency of allele): odds ratio (OR)?=?1.42, 95% confidence interval (CI): 1.09–1.85; CT vs. CC (heterozygote): OR?=?1.48, 95% CI: 1.09–2.00; TT vs. CC (homozygote): OR?=?1.86, 95% CI: 1.08–3.20; CT+TT vs. CC (dominant model): OR?=?1.56, 95% CI: 1.12–2.18; and TT vs. CC+CT (recessive model): OR?=?1.51, 95% CI: 1.02–2.22], whereas the A1298C polymorphism was found to be significantly associated with reduced ASD risk but only in a recessive model (CC vs. AA+AC: OR?=?0.73, 95% CI: 0.56–0.97). In addition, we stratified the patient population based on whether they were from a country with food fortification of folic acid or not. The meta-analysis showed that the C677T polymorphism was found to be associated with ASD only in children from countries without food fortification. Our study indicated that the MTHFR C677T polymorphism contributes to increased ASD risk, and periconceptional folic acid may reduce ASD risk in those with MTHFR 677CT polymorphism. En ligne : http://dx.doi.org/10.1002/aur.1300 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=218
in Autism Research > 6-5 (October 2013) . - p.384-392[article] Association between MTHFR Gene Polymorphisms and the Risk of Autism Spectrum Disorders: A Meta-Analysis [Texte imprimé et/ou numérique] / Danhua PU, Auteur ; Yiping SHEN, Auteur ; Jie WU, Auteur . - p.384-392.
Langues : Anglais (eng)
in Autism Research > 6-5 (October 2013) . - p.384-392
Mots-clés : methylenetetrahydrofolate reductase polymorphism autism spectrum disorders folic acidl meta-analysis Index. décimale : PER Périodiques Résumé : Methylenetetrahydrofolate reductase (MTHFR) is essential for DNA biosynthesis and the epigenetic process of DNA methylation, and its gene polymorphisms have been implicated as risk factors for birth defects, neurological disorders, and cancers. However, reports on the association of MTHFR polymorphisms with autism spectrum disorders (ASD) are inconclusive. Therefore, we investigated the relationship of the MTHFR polymorphisms (C677T and A1298C) and the risk of ASD by meta-analysis. Up to December 2012, eight case-control studies involving 1672 patients with ASD and 6760 controls were included for meta-analysis. The results showed that the C677T polymorphism was associated with significantly increased ASD risk in all the comparison models [T vs. C allele (frequency of allele): odds ratio (OR)?=?1.42, 95% confidence interval (CI): 1.09–1.85; CT vs. CC (heterozygote): OR?=?1.48, 95% CI: 1.09–2.00; TT vs. CC (homozygote): OR?=?1.86, 95% CI: 1.08–3.20; CT+TT vs. CC (dominant model): OR?=?1.56, 95% CI: 1.12–2.18; and TT vs. CC+CT (recessive model): OR?=?1.51, 95% CI: 1.02–2.22], whereas the A1298C polymorphism was found to be significantly associated with reduced ASD risk but only in a recessive model (CC vs. AA+AC: OR?=?0.73, 95% CI: 0.56–0.97). In addition, we stratified the patient population based on whether they were from a country with food fortification of folic acid or not. The meta-analysis showed that the C677T polymorphism was found to be associated with ASD only in children from countries without food fortification. Our study indicated that the MTHFR C677T polymorphism contributes to increased ASD risk, and periconceptional folic acid may reduce ASD risk in those with MTHFR 677CT polymorphism. En ligne : http://dx.doi.org/10.1002/aur.1300 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=218 Glutamate receptor, metabotropic 7 (GRM7) gene variations and susceptibility to autism: A case–control study / Rezvan NOROOZI in Autism Research, 9-11 (November 2016)
[article]
Titre : Glutamate receptor, metabotropic 7 (GRM7) gene variations and susceptibility to autism: A case–control study Type de document : Texte imprimé et/ou numérique Auteurs : Rezvan NOROOZI, Auteur ; Mohammad TAHERI, Auteur ; Abolfazl MOVAFAGH, Auteur ; Reza MIRFAKHRAIE, Auteur ; Ghasem SOLGI, Auteur ; Arezou SAYAD, Auteur ; Mehrdokht MAZDEH, Auteur ; Hossein DARVISH, Auteur Article en page(s) : p.1161-1168 Langues : Anglais (eng) Mots-clés : autism spectrum disorders metabotropic glutamate receptor glutamatergic polymorphism GRM7 Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) as a synaptopathy is revealed to be pertained to aberrant glutamatergic neurotransmission. Glutamate receptor, metabotropic 7 (GRM7), a receptor coding gene of this pathway, is a new candidate gene for autism. The aim of this study was to examine if there is a relationship between genetic variants rs779867 and rs6782011 of GRM7 with ASD. The present research was designed as a population-based, case–control study including 518 ASD patients versus 472 control individuals. The results showed that the frequency of rs779867 G/G genotype was significantly higher in ASD patients compared to healthy controls (P?=?0.0001). Also, the G allele of this SNP was found to be significantly more frequent in the patients than control group (P?=?0.0001). Haplotype analysis exhibited significant association of two estimated block of rs6782011/rs779867 in ASD patients versus control group. We found higher significant frequency of GT haplotype and lower frequencies of AT and AC haplotypes in the patients group compared to healthy controls (P?=?0.001, P?=?0.006, and P?=?0.05, respectively). Our study indicated that the rs779867 polymorphism is associated with ASD; thus, results of this study provide supportive evidence of association of the GRM7 gene with ASD. En ligne : http://dx.doi.org/10.1002/aur.1640 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=297
in Autism Research > 9-11 (November 2016) . - p.1161-1168[article] Glutamate receptor, metabotropic 7 (GRM7) gene variations and susceptibility to autism: A case–control study [Texte imprimé et/ou numérique] / Rezvan NOROOZI, Auteur ; Mohammad TAHERI, Auteur ; Abolfazl MOVAFAGH, Auteur ; Reza MIRFAKHRAIE, Auteur ; Ghasem SOLGI, Auteur ; Arezou SAYAD, Auteur ; Mehrdokht MAZDEH, Auteur ; Hossein DARVISH, Auteur . - p.1161-1168.
Langues : Anglais (eng)
in Autism Research > 9-11 (November 2016) . - p.1161-1168
Mots-clés : autism spectrum disorders metabotropic glutamate receptor glutamatergic polymorphism GRM7 Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) as a synaptopathy is revealed to be pertained to aberrant glutamatergic neurotransmission. Glutamate receptor, metabotropic 7 (GRM7), a receptor coding gene of this pathway, is a new candidate gene for autism. The aim of this study was to examine if there is a relationship between genetic variants rs779867 and rs6782011 of GRM7 with ASD. The present research was designed as a population-based, case–control study including 518 ASD patients versus 472 control individuals. The results showed that the frequency of rs779867 G/G genotype was significantly higher in ASD patients compared to healthy controls (P?=?0.0001). Also, the G allele of this SNP was found to be significantly more frequent in the patients than control group (P?=?0.0001). Haplotype analysis exhibited significant association of two estimated block of rs6782011/rs779867 in ASD patients versus control group. We found higher significant frequency of GT haplotype and lower frequencies of AT and AC haplotypes in the patients group compared to healthy controls (P?=?0.001, P?=?0.006, and P?=?0.05, respectively). Our study indicated that the rs779867 polymorphism is associated with ASD; thus, results of this study provide supportive evidence of association of the GRM7 gene with ASD. En ligne : http://dx.doi.org/10.1002/aur.1640 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=297 Genetic Analysis of UGT1A1 Polymorphisms Using Preserved Dried Umbilical Cord for Assessing the Potential of Neonatal Jaundice as a Risk Factor for Autism Spectrum Disorder in Children / T. HORINOUCHI in Journal of Autism and Developmental Disorders, 52-2 (February 2022)
[article]
Titre : Genetic Analysis of UGT1A1 Polymorphisms Using Preserved Dried Umbilical Cord for Assessing the Potential of Neonatal Jaundice as a Risk Factor for Autism Spectrum Disorder in Children Type de document : Texte imprimé et/ou numérique Auteurs : T. HORINOUCHI, Auteur ; K. MAEYAMA, Auteur ; M. NAGAI, Auteur ; M. MIZOBUCHI, Auteur ; Y. TAKAGI, Auteur ; Y. OKADA, Auteur ; T. KATO, Auteur ; M. NISHIMURA, Auteur ; Y. KAWASAKI, Auteur ; M. YOSHIOKA, Auteur ; S. TAKADA, Auteur ; H. MATSUMOTO, Auteur ; Y. NAKAMACHI, Auteur ; J. SAEGUSA, Auteur ; S. FUKUSHIMA, Auteur ; K. FUJIOKA, Auteur ; K. TOMIOKA, Auteur ; H. NAGASE, Auteur ; K. NOZU, Auteur ; K. IIJIMA, Auteur ; N. NISHIMURA, Auteur Article en page(s) : p.483-489 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/diagnosis/genetics Child Female Glucuronosyltransferase/genetics Humans Infant, Newborn Jaundice, Neonatal/complications Polymorphism, Genetic Pregnancy Risk Factors Umbilical Cord Autism spectrum disorder Dried umbilical cord Neonatal jaundice Polymorphism Ugt1a1 Index. décimale : PER Périodiques Résumé : Neonatal jaundice has been suggested as a perinatal risk factor for autism spectrum disorder (ASD). We examined UGT1A1 polymorphisms to assess the potential of neonatal jaundice as a risk factor for ASD in children by using DNA extracted from preserved umbilical cord. In total, 79 children with ASD were genotyped for UGT1A1*28 (c.-41-40dup), UGT1A1*6 (c.211 G?>?A), and UGT1A1*27 (c.686 C?>?A). The allele frequency of UGT1A1*6 (OR?=?1.34, p?=?0.26) and UGT1A1*28 (OR?=?0.80, p?=?0.54) and the prevalence of UGT1A1*28/*6 diplotypes did not differ significantly from those in the control population. No UGT1A1*27 allele was detected in the subjects. ASD symptom assessment scores were not associated with UGT1A1*28/*6/*27 genotypes or UGT1A1*28/*6 diplotypes. These results suggest that neonatal jaundice is not significantly associated with ASD. En ligne : http://dx.doi.org/10.1007/s10803-021-04941-w Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455
in Journal of Autism and Developmental Disorders > 52-2 (February 2022) . - p.483-489[article] Genetic Analysis of UGT1A1 Polymorphisms Using Preserved Dried Umbilical Cord for Assessing the Potential of Neonatal Jaundice as a Risk Factor for Autism Spectrum Disorder in Children [Texte imprimé et/ou numérique] / T. HORINOUCHI, Auteur ; K. MAEYAMA, Auteur ; M. NAGAI, Auteur ; M. MIZOBUCHI, Auteur ; Y. TAKAGI, Auteur ; Y. OKADA, Auteur ; T. KATO, Auteur ; M. NISHIMURA, Auteur ; Y. KAWASAKI, Auteur ; M. YOSHIOKA, Auteur ; S. TAKADA, Auteur ; H. MATSUMOTO, Auteur ; Y. NAKAMACHI, Auteur ; J. SAEGUSA, Auteur ; S. FUKUSHIMA, Auteur ; K. FUJIOKA, Auteur ; K. TOMIOKA, Auteur ; H. NAGASE, Auteur ; K. NOZU, Auteur ; K. IIJIMA, Auteur ; N. NISHIMURA, Auteur . - p.483-489.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 52-2 (February 2022) . - p.483-489
Mots-clés : Autism Spectrum Disorder/diagnosis/genetics Child Female Glucuronosyltransferase/genetics Humans Infant, Newborn Jaundice, Neonatal/complications Polymorphism, Genetic Pregnancy Risk Factors Umbilical Cord Autism spectrum disorder Dried umbilical cord Neonatal jaundice Polymorphism Ugt1a1 Index. décimale : PER Périodiques Résumé : Neonatal jaundice has been suggested as a perinatal risk factor for autism spectrum disorder (ASD). We examined UGT1A1 polymorphisms to assess the potential of neonatal jaundice as a risk factor for ASD in children by using DNA extracted from preserved umbilical cord. In total, 79 children with ASD were genotyped for UGT1A1*28 (c.-41-40dup), UGT1A1*6 (c.211 G?>?A), and UGT1A1*27 (c.686 C?>?A). The allele frequency of UGT1A1*6 (OR?=?1.34, p?=?0.26) and UGT1A1*28 (OR?=?0.80, p?=?0.54) and the prevalence of UGT1A1*28/*6 diplotypes did not differ significantly from those in the control population. No UGT1A1*27 allele was detected in the subjects. ASD symptom assessment scores were not associated with UGT1A1*28/*6/*27 genotypes or UGT1A1*28/*6 diplotypes. These results suggest that neonatal jaundice is not significantly associated with ASD. En ligne : http://dx.doi.org/10.1007/s10803-021-04941-w Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=455 Association analysis of two synapse-related gene mutations with autism spectrum disorder in a Chinese population / Fang HOU in Research in Autism Spectrum Disorders, 53 (September 2018)
[article]
Titre : Association analysis of two synapse-related gene mutations with autism spectrum disorder in a Chinese population Type de document : Texte imprimé et/ou numérique Auteurs : Fang HOU, Auteur ; Li LI, Auteur ; Jianhua GONG, Auteur ; Yanlin CHEN, Auteur ; Jia WANG, Auteur ; Lingfei LIU, Auteur ; Xiu LUO, Auteur ; HuaiTing GU, Auteur ; Jiajia ZHANG, Auteur ; Ranran SONG, Auteur Article en page(s) : p.67-72 Langues : Anglais (eng) Mots-clés : Polymorphism Autism spectrum disorder Index. décimale : PER Périodiques Résumé : Background Autism spectrum disorder (ASD) is a complex neurodevelopmental disorder with a strong genetic basis. Recently, synaptic abnormality has been proved to have a strong association with the etiology of ASD. PSD95 and DLGAP2 are the members of postsynaptic scaffolding proteins that play crucial roles in synaptic plasticity and function. This study evaluated the association of the genetic variants in PSD95 and DLGAP2 with ASD. Methods We performed a case-control study in a Chinese population with samples of 529 cases and 1923 controls. We extracted genomic DNA from oral swabs and determined the SNP genotypes by using a PCR-RFLP assay. Results We sequenced five SNPs (rs7005715, rs2301963 and rs2906569 in DLGAP2; rs2521985 and rs2017365 in PSD95). Genetic analysis suggested the GA genotype and GG genotype of rs7005715 were significantly associated with increased risk of ASD (respectively: OR?=?1.357, 95%CI?=?1.103–1.669, P?=?0.016; OR?=?1.860, 95%CI?=?1.359–2.551, P?0.001). The dominant model (OR?=?1.444, 95%CI?=?1.186–1.758, P?0.001) and recessive model (OR?=?1.597, 95%CI?=?1.187–2.149, P?=?0.011) also showed the same trend. We did not detect any significant association between other SNPs and ASD. Conclusions The genetic variant of rs7005715 in DLGAP2 increased susceptibility to the risk of ASD in a Chinese Han population. En ligne : https://doi.org/10.1016/j.rasd.2018.06.005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368
in Research in Autism Spectrum Disorders > 53 (September 2018) . - p.67-72[article] Association analysis of two synapse-related gene mutations with autism spectrum disorder in a Chinese population [Texte imprimé et/ou numérique] / Fang HOU, Auteur ; Li LI, Auteur ; Jianhua GONG, Auteur ; Yanlin CHEN, Auteur ; Jia WANG, Auteur ; Lingfei LIU, Auteur ; Xiu LUO, Auteur ; HuaiTing GU, Auteur ; Jiajia ZHANG, Auteur ; Ranran SONG, Auteur . - p.67-72.
Langues : Anglais (eng)
in Research in Autism Spectrum Disorders > 53 (September 2018) . - p.67-72
Mots-clés : Polymorphism Autism spectrum disorder Index. décimale : PER Périodiques Résumé : Background Autism spectrum disorder (ASD) is a complex neurodevelopmental disorder with a strong genetic basis. Recently, synaptic abnormality has been proved to have a strong association with the etiology of ASD. PSD95 and DLGAP2 are the members of postsynaptic scaffolding proteins that play crucial roles in synaptic plasticity and function. This study evaluated the association of the genetic variants in PSD95 and DLGAP2 with ASD. Methods We performed a case-control study in a Chinese population with samples of 529 cases and 1923 controls. We extracted genomic DNA from oral swabs and determined the SNP genotypes by using a PCR-RFLP assay. Results We sequenced five SNPs (rs7005715, rs2301963 and rs2906569 in DLGAP2; rs2521985 and rs2017365 in PSD95). Genetic analysis suggested the GA genotype and GG genotype of rs7005715 were significantly associated with increased risk of ASD (respectively: OR?=?1.357, 95%CI?=?1.103–1.669, P?=?0.016; OR?=?1.860, 95%CI?=?1.359–2.551, P?0.001). The dominant model (OR?=?1.444, 95%CI?=?1.186–1.758, P?0.001) and recessive model (OR?=?1.597, 95%CI?=?1.187–2.149, P?=?0.011) also showed the same trend. We did not detect any significant association between other SNPs and ASD. Conclusions The genetic variant of rs7005715 in DLGAP2 increased susceptibility to the risk of ASD in a Chinese Han population. En ligne : https://doi.org/10.1016/j.rasd.2018.06.005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368 Association and gene–gene interactions study of reelin signaling pathway related genes with autism in the Han Chinese population / Yidong SHEN in Autism Research, 9-4 (April 2016)
PermalinkAssociation between CNTNAP2 polymorphisms and autism: A family-based study in the chinese han population and a meta-analysis combined with GWAS data of psychiatric genomics consortium / T. ZHANG in Autism Research, 12-4 (April 2019)
PermalinkComprehensive Integrative Analyses Identify TIGD5 rs75547282 as a Risk Variant for Autism Spectrum Disorder / Xinyan XIE in Autism Research, 14-4 (April 2021)
PermalinkModeling rare gene variation to gain insight into the oldest biomarker in autism: construction of the serotonin transporter Gly56Ala knock-in mouse / J. VEENSTRA-VANDERWEELE in Journal of Neurodevelopmental Disorders, 1-2 (June 2009)
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