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Exploring the possibility of parents’ broad internalizing phenotype acting through passive gene–environment correlations on daughters’ disordered eating / Shannon M. O’CONNOR in Development and Psychopathology, 34-5 (December 2022)
[article]
Titre : Exploring the possibility of parents’ broad internalizing phenotype acting through passive gene–environment correlations on daughters’ disordered eating Type de document : Texte imprimé et/ou numérique Auteurs : Shannon M. O’CONNOR, Auteur ; Megan MIKHAIL, Auteur ; Carolina ANAYA, Auteur ; Leora L. HALLER, Auteur ; S. Alexandra BURT, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Kelly L. KLUMP, Auteur Article en page(s) : p.1744-1755 Langues : Anglais (eng) Mots-clés : Disordered eating eating disorders family twin study internalizing Index. décimale : PER Périodiques Résumé : Twin studies demonstrate significant environmental influences and a lack of genetic effects on disordered eating before puberty in girls. However, genetic factors could act indirectly through passive gene “environment correlations (rGE; correlations between parents’ genes and an environment shaped by those genes) that inflate environmental (but not genetic) estimates. The only study to explore passive rGE did not find significant effects, but the full range of parental phenotypes (e.g., internalizing symptoms) that could impact daughters’ disordered eating was not examined. We addressed this gap by exploring whether parents’ internalizing symptoms (e.g., anxiety, depressive symptoms) contribute to daughters’ eating pathology through passive rGE. Participants were female twin pairs (aged 8 “14 years; M = 10.44) in pre-early puberty and their biological parents (n = 279 families) from the Michigan State University Twin Registry. Nuclear twin family models explored passive rGE for parents’ internalizing traits/symptoms and twins’ overall eating disorder symptoms. No evidence for passive rGE was found. Instead, environmental factors that create similarities between co-twins (but not with their parents) and unique environmental factors were important. In pre-early puberty, genetic factors do not influence daughters’ disordered eating, even indirectly through passive rGE. Future research should explore sibling-specific and unique environmental factors during this critical developmental period. En ligne : http://dx.doi.org/10.1017/S0954579422000608 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492
in Development and Psychopathology > 34-5 (December 2022) . - p.1744-1755[article] Exploring the possibility of parents’ broad internalizing phenotype acting through passive gene–environment correlations on daughters’ disordered eating [Texte imprimé et/ou numérique] / Shannon M. O’CONNOR, Auteur ; Megan MIKHAIL, Auteur ; Carolina ANAYA, Auteur ; Leora L. HALLER, Auteur ; S. Alexandra BURT, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Kelly L. KLUMP, Auteur . - p.1744-1755.
Langues : Anglais (eng)
in Development and Psychopathology > 34-5 (December 2022) . - p.1744-1755
Mots-clés : Disordered eating eating disorders family twin study internalizing Index. décimale : PER Périodiques Résumé : Twin studies demonstrate significant environmental influences and a lack of genetic effects on disordered eating before puberty in girls. However, genetic factors could act indirectly through passive gene “environment correlations (rGE; correlations between parents’ genes and an environment shaped by those genes) that inflate environmental (but not genetic) estimates. The only study to explore passive rGE did not find significant effects, but the full range of parental phenotypes (e.g., internalizing symptoms) that could impact daughters’ disordered eating was not examined. We addressed this gap by exploring whether parents’ internalizing symptoms (e.g., anxiety, depressive symptoms) contribute to daughters’ eating pathology through passive rGE. Participants were female twin pairs (aged 8 “14 years; M = 10.44) in pre-early puberty and their biological parents (n = 279 families) from the Michigan State University Twin Registry. Nuclear twin family models explored passive rGE for parents’ internalizing traits/symptoms and twins’ overall eating disorder symptoms. No evidence for passive rGE was found. Instead, environmental factors that create similarities between co-twins (but not with their parents) and unique environmental factors were important. In pre-early puberty, genetic factors do not influence daughters’ disordered eating, even indirectly through passive rGE. Future research should explore sibling-specific and unique environmental factors during this critical developmental period. En ligne : http://dx.doi.org/10.1017/S0954579422000608 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492 Mediating role of depressive symptoms linking insecure attachment and disordered eating in adolescents: A multiwave longitudinal study / L. CORTES-GARCIA in Development and Psychopathology, 34-1 (February 2022)
[article]
Titre : Mediating role of depressive symptoms linking insecure attachment and disordered eating in adolescents: A multiwave longitudinal study Type de document : Texte imprimé et/ou numérique Auteurs : L. CORTES-GARCIA, Auteur ; K. R. VIDDAL, Auteur ; L. WICHSTRØM, Auteur ; C. SENRA, Auteur Article en page(s) : p.115-127 Langues : Anglais (eng) Mots-clés : adolescents depressive symptoms disordered eating dynamic panel model insecure attachment Index. décimale : PER Périodiques Résumé : Research has supported a link between insecure attachment and disordered eating in adolescents; however, how this influence is exerted remains unclear. This study explored whether depressive symptoms constitute a pathway through which insecure attachment to parents predicts subsequent development of disordered eating in the transition from childhood to adolescence. The study also examines whether there are differential effects regarding the attachment figure, child's gender, or reciprocity between variables. A community-based sample of Spanish youth (n = 904; 49.4% girls) was followed biennially from age 10 to 16 years. Attachment, depressive symptoms, and disordered eating were measured using the Inventory of Parental and Peer Attachment, Children's Depression Inventory, and Children's Eating Attitudes Test, respectively. Prospective data were analyzed using a dynamic panel model, which accounts for unmeasured time-invariant factors. Whereas insecure attachment to the father did not predict later depression or disordered eating, higher insecure attachment to the mother at ages 10 and 12 years predicted more disordered eating at ages 14 and 16 years via increased depressive symptoms at ages 12 and 14 years. No child's gender-specific or reverse mediational effects were found. This study suggests that an increase in depressive symptoms might be one mechanism by which insecure attachment exerts its influence on the development of eating disorders symptomatology in adolescence. Intervention efforts aimed at strengthening particularly the mother?child attachment relationship may reduce the vulnerability to develop depressive symptoms and disordered eating. En ligne : http://dx.doi.org/10.1017/s0954579420001029 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474
in Development and Psychopathology > 34-1 (February 2022) . - p.115-127[article] Mediating role of depressive symptoms linking insecure attachment and disordered eating in adolescents: A multiwave longitudinal study [Texte imprimé et/ou numérique] / L. CORTES-GARCIA, Auteur ; K. R. VIDDAL, Auteur ; L. WICHSTRØM, Auteur ; C. SENRA, Auteur . - p.115-127.
Langues : Anglais (eng)
in Development and Psychopathology > 34-1 (February 2022) . - p.115-127
Mots-clés : adolescents depressive symptoms disordered eating dynamic panel model insecure attachment Index. décimale : PER Périodiques Résumé : Research has supported a link between insecure attachment and disordered eating in adolescents; however, how this influence is exerted remains unclear. This study explored whether depressive symptoms constitute a pathway through which insecure attachment to parents predicts subsequent development of disordered eating in the transition from childhood to adolescence. The study also examines whether there are differential effects regarding the attachment figure, child's gender, or reciprocity between variables. A community-based sample of Spanish youth (n = 904; 49.4% girls) was followed biennially from age 10 to 16 years. Attachment, depressive symptoms, and disordered eating were measured using the Inventory of Parental and Peer Attachment, Children's Depression Inventory, and Children's Eating Attitudes Test, respectively. Prospective data were analyzed using a dynamic panel model, which accounts for unmeasured time-invariant factors. Whereas insecure attachment to the father did not predict later depression or disordered eating, higher insecure attachment to the mother at ages 10 and 12 years predicted more disordered eating at ages 14 and 16 years via increased depressive symptoms at ages 12 and 14 years. No child's gender-specific or reverse mediational effects were found. This study suggests that an increase in depressive symptoms might be one mechanism by which insecure attachment exerts its influence on the development of eating disorders symptomatology in adolescence. Intervention efforts aimed at strengthening particularly the mother?child attachment relationship may reduce the vulnerability to develop depressive symptoms and disordered eating. En ligne : http://dx.doi.org/10.1017/s0954579420001029 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474 Risk factors and temporal patterns of disordered eating differ in adolescent boys and girls: Testing gender-specific appearance anxiety models / Melanie J. ZIMMER-GEMBECK in Development and Psychopathology, 33-3 (August 2021)
[article]
Titre : Risk factors and temporal patterns of disordered eating differ in adolescent boys and girls: Testing gender-specific appearance anxiety models Type de document : Texte imprimé et/ou numérique Auteurs : Melanie J. ZIMMER-GEMBECK, Auteur ; Haley J. WEBB, Auteur ; Jessica KERIN, Auteur ; Allison M. WATERS, Auteur ; Lara J. FARRELL, Auteur Article en page(s) : p.856-867 Langues : Anglais (eng) Mots-clés : appearance body dysmorphic symptoms body image disordered eating eating disorders peer victimization Index. décimale : PER Périodiques Résumé : Adolescent dieting and disordered eating (DE) are risks for clinical eating disorders. In this five-wave longitudinal study, we tested gender-specific models linking early risk factors to temporal patterns of DE, considering appearance anxiety as a mediator. Participants were 384 Australian students (age 10 to 13; 45% boys) who reported their purging and skipping meals, experience with appearance-related teasing, media pressure, and appearance anxiety. Parents reported pubertal maturation and height/weight was measured. Gender differences in temporal patterns of DE were found and predictive models were tested using latent-variable growth curve and path models. Boys’ DE was generally stable over time; girls showed stability in purging but an average increase in skipping meals. Peer teasing, media pressure, and pubertal maturation were associated with more elevated initial DE in girls, and pubertal maturation was associated with a steeper increase in DE. For boys, body mass index had a direct positive association with DE. Appearance anxiety was associated with more DE, but there was only one significant indirect effect via anxiety, which was for boys’ pubertal maturation. Findings support the dominant role of social interactions and messages, as well as pubertal maturation, for girls’ DE and the prominence of physical risk factors for explaining boys’ DE. En ligne : http://dx.doi.org/10.1017/S0954579420000188 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457
in Development and Psychopathology > 33-3 (August 2021) . - p.856-867[article] Risk factors and temporal patterns of disordered eating differ in adolescent boys and girls: Testing gender-specific appearance anxiety models [Texte imprimé et/ou numérique] / Melanie J. ZIMMER-GEMBECK, Auteur ; Haley J. WEBB, Auteur ; Jessica KERIN, Auteur ; Allison M. WATERS, Auteur ; Lara J. FARRELL, Auteur . - p.856-867.
Langues : Anglais (eng)
in Development and Psychopathology > 33-3 (August 2021) . - p.856-867
Mots-clés : appearance body dysmorphic symptoms body image disordered eating eating disorders peer victimization Index. décimale : PER Périodiques Résumé : Adolescent dieting and disordered eating (DE) are risks for clinical eating disorders. In this five-wave longitudinal study, we tested gender-specific models linking early risk factors to temporal patterns of DE, considering appearance anxiety as a mediator. Participants were 384 Australian students (age 10 to 13; 45% boys) who reported their purging and skipping meals, experience with appearance-related teasing, media pressure, and appearance anxiety. Parents reported pubertal maturation and height/weight was measured. Gender differences in temporal patterns of DE were found and predictive models were tested using latent-variable growth curve and path models. Boys’ DE was generally stable over time; girls showed stability in purging but an average increase in skipping meals. Peer teasing, media pressure, and pubertal maturation were associated with more elevated initial DE in girls, and pubertal maturation was associated with a steeper increase in DE. For boys, body mass index had a direct positive association with DE. Appearance anxiety was associated with more DE, but there was only one significant indirect effect via anxiety, which was for boys’ pubertal maturation. Findings support the dominant role of social interactions and messages, as well as pubertal maturation, for girls’ DE and the prominence of physical risk factors for explaining boys’ DE. En ligne : http://dx.doi.org/10.1017/S0954579420000188 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 The Association Between Autistic Traits and Disordered Eating is Moderated by Sex/Gender and Independent of Anxiety and Depression / A. BARNETT in Journal of Autism and Developmental Disorders, 51-6 (June 2021)
[article]
Titre : The Association Between Autistic Traits and Disordered Eating is Moderated by Sex/Gender and Independent of Anxiety and Depression Type de document : Texte imprimé et/ou numérique Auteurs : A. BARNETT, Auteur ; K. EDWARDS, Auteur ; R. HARPER, Auteur ; E. EVANS, Auteur ; D. ALEXANDER, Auteur ; M. CHOUDHARY, Auteur ; G. RICHARDS, Auteur Article en page(s) : p.1866-1879 Langues : Anglais (eng) Mots-clés : Adolescent Adult Aged Anxiety/psychology Autistic Disorder/psychology Cross-Sectional Studies Depression/psychology Feeding and Eating Disorders/psychology Female Gender Identity Humans Male Middle Aged Sex Factors Surveys and Questionnaires Young Adult Anorexia nervosa Anxiety Autism Autistic traits Bulimia nervosa Depression Disordered eating Eating disorders Gender differences Sex differences Index. décimale : PER Périodiques Résumé : Previous studies have reported positive correlations between autistic traits and disordered eating, though it is unclear whether the association is moderated by sex/gender or whether it is independent of anxiety or depression. We present the findings of an online survey of 691 participants who completed the Autism Spectrum Quotient (AQ), Hospital Anxiety and Depression Scale (HADS), and Eating Attitudes Test-26 (EAT-26). Following a pre-registered analysis plan, we observed positive correlations between AQ and EAT-26 in males and females, with the association being significantly stronger in females. AQ also remained a significant predictor of EAT-26 when anxiety and depression were controlled for statistically. These findings may be relevant when considering therapeutic interventions in disordered eating populations that exhibit autistic traits. En ligne : http://dx.doi.org/10.1007/s10803-020-04669-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=452
in Journal of Autism and Developmental Disorders > 51-6 (June 2021) . - p.1866-1879[article] The Association Between Autistic Traits and Disordered Eating is Moderated by Sex/Gender and Independent of Anxiety and Depression [Texte imprimé et/ou numérique] / A. BARNETT, Auteur ; K. EDWARDS, Auteur ; R. HARPER, Auteur ; E. EVANS, Auteur ; D. ALEXANDER, Auteur ; M. CHOUDHARY, Auteur ; G. RICHARDS, Auteur . - p.1866-1879.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 51-6 (June 2021) . - p.1866-1879
Mots-clés : Adolescent Adult Aged Anxiety/psychology Autistic Disorder/psychology Cross-Sectional Studies Depression/psychology Feeding and Eating Disorders/psychology Female Gender Identity Humans Male Middle Aged Sex Factors Surveys and Questionnaires Young Adult Anorexia nervosa Anxiety Autism Autistic traits Bulimia nervosa Depression Disordered eating Eating disorders Gender differences Sex differences Index. décimale : PER Périodiques Résumé : Previous studies have reported positive correlations between autistic traits and disordered eating, though it is unclear whether the association is moderated by sex/gender or whether it is independent of anxiety or depression. We present the findings of an online survey of 691 participants who completed the Autism Spectrum Quotient (AQ), Hospital Anxiety and Depression Scale (HADS), and Eating Attitudes Test-26 (EAT-26). Following a pre-registered analysis plan, we observed positive correlations between AQ and EAT-26 in males and females, with the association being significantly stronger in females. AQ also remained a significant predictor of EAT-26 when anxiety and depression were controlled for statistically. These findings may be relevant when considering therapeutic interventions in disordered eating populations that exhibit autistic traits. En ligne : http://dx.doi.org/10.1007/s10803-020-04669-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=452 Research Review: What we have learned about the causes of eating disorders – a synthesis of sociocultural, psychological, and biological research / Kristen M. CULBERT in Journal of Child Psychology and Psychiatry, 56-11 (November 2015)
[article]
Titre : Research Review: What we have learned about the causes of eating disorders – a synthesis of sociocultural, psychological, and biological research Type de document : Texte imprimé et/ou numérique Auteurs : Kristen M. CULBERT, Auteur ; Sarah E. RACINE, Auteur ; Kelly L. KLUMP, Auteur Article en page(s) : p.1141-1164 Langues : Anglais (eng) Mots-clés : Eating disorder disordered eating risk etiology biopsychosocial Index. décimale : PER Périodiques Résumé : Background Eating disorders are severe psychiatric disorders with a complex etiology involving transactions among sociocultural, psychological, and biological influences. Most research and reviews, however, focus on only one level of analysis. To address this gap, we provide a qualitative review and summary using an integrative biopsychosocial approach. Methods We selected variables for which there were available data using integrative methodologies (e.g., twin studies, gene-environment interactions) and/or data at the biological and behavioral level (e.g., neuroimaging). Factors that met these inclusion criteria were idealization of thinness, negative emotionality, perfectionism, negative urgency, inhibitory control, cognitive inflexibility, serotonin, dopamine, ovarian hormones. Literature searches were conducted using PubMed. Variables were classified as risk factors or correlates of eating disorder diagnoses and disordered eating symptoms using Kraemer et al.'s (1997) criteria. Findings Sociocultural idealization of thinness variables (media exposure, pressures for thinness, thin-ideal internalization, thinness expectancies) and personality traits (negative emotionality, perfectionism, negative urgency) attained ‘risk status’ for eating disorders and/or disordered eating symptoms. Other factors were identified as correlates of eating pathology or were not classified given limited data. Effect sizes for risk factors and correlates were generally small-to-moderate in magnitude. Conclusions Multiple biopsychosocial influences are implicated in eating disorders and/or disordered eating symptoms and several can now be considered established risk factors. Data suggest that psychological and environmental factors interact with and influence the expression of genetic risk to cause eating pathology. Additional studies that examine risk variables across multiple levels of analysis and that consider specific transactional processes amongst variables are needed to further elucidate the intersection of sociocultural, psychological, and biological influences on eating disorders. En ligne : http://dx.doi.org/10.1111/jcpp.12441 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=270
in Journal of Child Psychology and Psychiatry > 56-11 (November 2015) . - p.1141-1164[article] Research Review: What we have learned about the causes of eating disorders – a synthesis of sociocultural, psychological, and biological research [Texte imprimé et/ou numérique] / Kristen M. CULBERT, Auteur ; Sarah E. RACINE, Auteur ; Kelly L. KLUMP, Auteur . - p.1141-1164.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 56-11 (November 2015) . - p.1141-1164
Mots-clés : Eating disorder disordered eating risk etiology biopsychosocial Index. décimale : PER Périodiques Résumé : Background Eating disorders are severe psychiatric disorders with a complex etiology involving transactions among sociocultural, psychological, and biological influences. Most research and reviews, however, focus on only one level of analysis. To address this gap, we provide a qualitative review and summary using an integrative biopsychosocial approach. Methods We selected variables for which there were available data using integrative methodologies (e.g., twin studies, gene-environment interactions) and/or data at the biological and behavioral level (e.g., neuroimaging). Factors that met these inclusion criteria were idealization of thinness, negative emotionality, perfectionism, negative urgency, inhibitory control, cognitive inflexibility, serotonin, dopamine, ovarian hormones. Literature searches were conducted using PubMed. Variables were classified as risk factors or correlates of eating disorder diagnoses and disordered eating symptoms using Kraemer et al.'s (1997) criteria. Findings Sociocultural idealization of thinness variables (media exposure, pressures for thinness, thin-ideal internalization, thinness expectancies) and personality traits (negative emotionality, perfectionism, negative urgency) attained ‘risk status’ for eating disorders and/or disordered eating symptoms. Other factors were identified as correlates of eating pathology or were not classified given limited data. Effect sizes for risk factors and correlates were generally small-to-moderate in magnitude. Conclusions Multiple biopsychosocial influences are implicated in eating disorders and/or disordered eating symptoms and several can now be considered established risk factors. Data suggest that psychological and environmental factors interact with and influence the expression of genetic risk to cause eating pathology. Additional studies that examine risk variables across multiple levels of analysis and that consider specific transactional processes amongst variables are needed to further elucidate the intersection of sociocultural, psychological, and biological influences on eating disorders. En ligne : http://dx.doi.org/10.1111/jcpp.12441 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=270 One size does not fit all: Trajectories of body image development and their predictors in early adolescence / Emilie LACROIX in Development and Psychopathology, 34-1 (February 2022)
PermalinkReexamining the restraint pathway as a conditional process among adolescent girls: When does dieting link body dissatisfaction to bulimia? / Rebecca A. MORRISSEY in Development and Psychopathology, 32-3 (August 2020)
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