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Childhood psychopathology in children of women with eating disorders: understanding risk mechanisms / Nadia MICALI in Journal of Child Psychology and Psychiatry, 55-2 (February 2014)
[article]
Titre : Childhood psychopathology in children of women with eating disorders: understanding risk mechanisms Type de document : Texte imprimé et/ou numérique Auteurs : Nadia MICALI, Auteur ; Daniel STAHL, Auteur ; Janet TREASURE, Auteur ; Emily SIMONOFF, Auteur Article en page(s) : p.124-134 Langues : Anglais (eng) Mots-clés : ALSPAC child psychopathology eating disorders parental mental health risk mechanisms Index. décimale : PER Périodiques Résumé : Background Very few studies have investigated psychopathology in children of mothers with eating disorders (ED). We aimed to determine the effect of maternal ED on childhood psychopathology in a large population-based cohort and investigate relevant risk pathways using structural equation modeling (SEM). Methods Data on emotional and behavioral problems at 3½ years were obtained prospectively on 8,622 children from the Avon Longitudinal Study of Parents and Children (ALSPAC). Children of exposed women who self-reported lifetime anorexia nervosa (AN, N = 193) or bulimia nervosa (BN, N = 158) in pregnancy were compared with children of unexposed women (N = 8,271) using linear and logistic regression models. SEM was used to determine best-fitting risk models by child gender. Results There was evidence that girls of AN women were more likely to have emotional, conduct, and hyperactivity disorders [Odds Ratio (OR): 1.7 (95% Confidence Intervals 1.0–3.0); OR: 2.2 (1.2–4.0); OR: 1.8 (1.1–3.1), respectively] and boys of AN women to have emotional disorders compared with unexposed [OR: 2.0(1.2–3.4)]. Girls of women with BN were more likely to show hyperactivity [OR: 1.7 (1.0–3.1)]; and boys to show emotional and conduct disorders compared with unexposed [OR: 2.2 (1.2–3.9); OR: 2.4 (1.4–4.2), respectively]. SEM models showed that pregnancy anxiety and depression mediated the effect of maternal ED on child psychopathology. Conclusions Maternal ED are associated with different childhood psychopathology outcomes in boys and girls. Pregnancy anxiety and depression and active ED symptoms are important mediators of risk and are preventable; the direct effect of maternal lifetime ED was small. En ligne : http://dx.doi.org/10.1111/jcpp.12112 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=221
in Journal of Child Psychology and Psychiatry > 55-2 (February 2014) . - p.124-134[article] Childhood psychopathology in children of women with eating disorders: understanding risk mechanisms [Texte imprimé et/ou numérique] / Nadia MICALI, Auteur ; Daniel STAHL, Auteur ; Janet TREASURE, Auteur ; Emily SIMONOFF, Auteur . - p.124-134.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 55-2 (February 2014) . - p.124-134
Mots-clés : ALSPAC child psychopathology eating disorders parental mental health risk mechanisms Index. décimale : PER Périodiques Résumé : Background Very few studies have investigated psychopathology in children of mothers with eating disorders (ED). We aimed to determine the effect of maternal ED on childhood psychopathology in a large population-based cohort and investigate relevant risk pathways using structural equation modeling (SEM). Methods Data on emotional and behavioral problems at 3½ years were obtained prospectively on 8,622 children from the Avon Longitudinal Study of Parents and Children (ALSPAC). Children of exposed women who self-reported lifetime anorexia nervosa (AN, N = 193) or bulimia nervosa (BN, N = 158) in pregnancy were compared with children of unexposed women (N = 8,271) using linear and logistic regression models. SEM was used to determine best-fitting risk models by child gender. Results There was evidence that girls of AN women were more likely to have emotional, conduct, and hyperactivity disorders [Odds Ratio (OR): 1.7 (95% Confidence Intervals 1.0–3.0); OR: 2.2 (1.2–4.0); OR: 1.8 (1.1–3.1), respectively] and boys of AN women to have emotional disorders compared with unexposed [OR: 2.0(1.2–3.4)]. Girls of women with BN were more likely to show hyperactivity [OR: 1.7 (1.0–3.1)]; and boys to show emotional and conduct disorders compared with unexposed [OR: 2.2 (1.2–3.9); OR: 2.4 (1.4–4.2), respectively]. SEM models showed that pregnancy anxiety and depression mediated the effect of maternal ED on child psychopathology. Conclusions Maternal ED are associated with different childhood psychopathology outcomes in boys and girls. Pregnancy anxiety and depression and active ED symptoms are important mediators of risk and are preventable; the direct effect of maternal lifetime ED was small. En ligne : http://dx.doi.org/10.1111/jcpp.12112 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=221 "For Me, the Anorexia is Just a Symptom, and the Cause is the Autism": Investigating Restrictive Eating Disorders in Autistic Women / Janina BREDE in Journal of Autism and Developmental Disorders, 50-12 (December 2020)
[article]
Titre : "For Me, the Anorexia is Just a Symptom, and the Cause is the Autism": Investigating Restrictive Eating Disorders in Autistic Women Type de document : Texte imprimé et/ou numérique Auteurs : Janina BREDE, Auteur ; Charli BABB, Auteur ; Catherine R. G. JONES, Auteur ; Mair ELLIOTT, Auteur ; Cathy ZANKER, Auteur ; Kate TCHANTURIA, Auteur ; Lucy SERPELL, Auteur ; John FOX, Auteur ; Will MANDY, Auteur Article en page(s) : p.4280-4296 Langues : Anglais (eng) Mots-clés : Anorexia nervosa Autism Co-occurrence Eating disorders Females Qualitative research Index. décimale : PER Périodiques Résumé : Autistic women are overrepresented among people in treatment for Anorexia Nervosa (AN). The current study aimed to: (1) better understand how AN develops and persists in autistic individuals from the perspective of autistic women, parents and healthcare professionals; (2) derive a theoretical model of restrictive eating difficulties in autism. We conducted 44 semi-structured interviews and used Thematic Analysis to identify patterns of meaning across the data. Themes related to sensory sensitivities, social interaction and relationships, sense of self and identity, difficulties with emotions, thinking styles, and a need for control and predictability. We developed a model of potential autism-specific mechanisms underlying restrictive eating difficulties. This study generated novel insights, which have the potential to inform treatment adaptations following empirical testing. En ligne : http://dx.doi.org/10.1007/s10803-020-04479-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434
in Journal of Autism and Developmental Disorders > 50-12 (December 2020) . - p.4280-4296[article] "For Me, the Anorexia is Just a Symptom, and the Cause is the Autism": Investigating Restrictive Eating Disorders in Autistic Women [Texte imprimé et/ou numérique] / Janina BREDE, Auteur ; Charli BABB, Auteur ; Catherine R. G. JONES, Auteur ; Mair ELLIOTT, Auteur ; Cathy ZANKER, Auteur ; Kate TCHANTURIA, Auteur ; Lucy SERPELL, Auteur ; John FOX, Auteur ; Will MANDY, Auteur . - p.4280-4296.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 50-12 (December 2020) . - p.4280-4296
Mots-clés : Anorexia nervosa Autism Co-occurrence Eating disorders Females Qualitative research Index. décimale : PER Périodiques Résumé : Autistic women are overrepresented among people in treatment for Anorexia Nervosa (AN). The current study aimed to: (1) better understand how AN develops and persists in autistic individuals from the perspective of autistic women, parents and healthcare professionals; (2) derive a theoretical model of restrictive eating difficulties in autism. We conducted 44 semi-structured interviews and used Thematic Analysis to identify patterns of meaning across the data. Themes related to sensory sensitivities, social interaction and relationships, sense of self and identity, difficulties with emotions, thinking styles, and a need for control and predictability. We developed a model of potential autism-specific mechanisms underlying restrictive eating difficulties. This study generated novel insights, which have the potential to inform treatment adaptations following empirical testing. En ligne : http://dx.doi.org/10.1007/s10803-020-04479-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=434 Maternal eating disorders and infant feeding difficulties: maternal and child mediators in a longitudinal general population study / Nadia MICALI in Journal of Child Psychology and Psychiatry, 52-7 (July 2011)
[article]
Titre : Maternal eating disorders and infant feeding difficulties: maternal and child mediators in a longitudinal general population study Type de document : Texte imprimé et/ou numérique Auteurs : Nadia MICALI, Auteur ; Emily SIMONOFF, Auteur ; Daniel STAHL, Auteur ; Janet TREASURE, Auteur Année de publication : 2011 Article en page(s) : p.800-807 Langues : Anglais (eng) Mots-clés : ALSPAC eating disorders feeding anxiety depression;infants Index. décimale : PER Périodiques Résumé : Background: Maternal eating disorders (ED) have been shown to increase the risk of feeding difficulties in the offspring. Very few studies, however, have investigated whether the effect of a maternal ED on childhood feeding is a direct effect or whether it can be ascribed to other child or maternal factors. We aimed to determine the role of maternal anxiety and depression in mediating the risk for feeding difficulties in infants of women with ED.
Methods: A prospective study comparing women with lifetime ED (441) and without any lifetime psychiatric disorder (10,461) and their infants from the Avon Longitudinal Study of Parents and Children (ALSPAC). We investigated the effect of: maternal anxiety and depression in late pregnancy (32 weeks) and the post-partum (8 weeks), child temperament and developmental status on infant feeding difficulties at 1 and 6 months. We also investigated the effect of active pregnancy ED symptoms. We tested 3 models and their fit to the data using structured equation modelling: a direct effect model, a fully mediational model and an integrated (partial meditational) model.
Results: The integrated model including a direct effect of maternal lifetime ED on infant feeding and a mediational path via maternal distress (a latent variable combining anxiety and depression) fitted the data best. This also applied to maternal pregnancy ED symptoms. Feeding difficulties in turn increased maternal distress over time.
Conclusions: Lifetime ED and active pregnancy ED increase the risk for infant feeding difficulties and do so via maternal distress (i.e., depression and anxiety). This has important implications for prevention and early intervention in relation to infant feeding difficulties, as well as for future research in the field.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2010.02341.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=126
in Journal of Child Psychology and Psychiatry > 52-7 (July 2011) . - p.800-807[article] Maternal eating disorders and infant feeding difficulties: maternal and child mediators in a longitudinal general population study [Texte imprimé et/ou numérique] / Nadia MICALI, Auteur ; Emily SIMONOFF, Auteur ; Daniel STAHL, Auteur ; Janet TREASURE, Auteur . - 2011 . - p.800-807.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 52-7 (July 2011) . - p.800-807
Mots-clés : ALSPAC eating disorders feeding anxiety depression;infants Index. décimale : PER Périodiques Résumé : Background: Maternal eating disorders (ED) have been shown to increase the risk of feeding difficulties in the offspring. Very few studies, however, have investigated whether the effect of a maternal ED on childhood feeding is a direct effect or whether it can be ascribed to other child or maternal factors. We aimed to determine the role of maternal anxiety and depression in mediating the risk for feeding difficulties in infants of women with ED.
Methods: A prospective study comparing women with lifetime ED (441) and without any lifetime psychiatric disorder (10,461) and their infants from the Avon Longitudinal Study of Parents and Children (ALSPAC). We investigated the effect of: maternal anxiety and depression in late pregnancy (32 weeks) and the post-partum (8 weeks), child temperament and developmental status on infant feeding difficulties at 1 and 6 months. We also investigated the effect of active pregnancy ED symptoms. We tested 3 models and their fit to the data using structured equation modelling: a direct effect model, a fully mediational model and an integrated (partial meditational) model.
Results: The integrated model including a direct effect of maternal lifetime ED on infant feeding and a mediational path via maternal distress (a latent variable combining anxiety and depression) fitted the data best. This also applied to maternal pregnancy ED symptoms. Feeding difficulties in turn increased maternal distress over time.
Conclusions: Lifetime ED and active pregnancy ED increase the risk for infant feeding difficulties and do so via maternal distress (i.e., depression and anxiety). This has important implications for prevention and early intervention in relation to infant feeding difficulties, as well as for future research in the field.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2010.02341.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=126 Neurobehavioural and cognitive development in infants born to mothers with eating disorders / Manuela BARONA in Journal of Child Psychology and Psychiatry, 58-8 (August 2017)
[article]
Titre : Neurobehavioural and cognitive development in infants born to mothers with eating disorders Type de document : Texte imprimé et/ou numérique Auteurs : Manuela BARONA, Auteur ; Emma TABORELLI, Auteur ; Freya CORFIELD, Auteur ; Susan PAWLBY, Auteur ; Abigail EASTER, Auteur ; Ulrike SCHMIDT, Auteur ; Janet TREASURE, Auteur ; Nadia MICALI, Auteur Article en page(s) : p.931-938 Langues : Anglais (eng) Mots-clés : Brazelton Neonatal Behavioural Assessment Scale Bayley Scales of Infant and Toddler Development eating disorders child development Index. décimale : PER Périodiques Résumé : Introduction Although recent research has focused on the effects of maternal eating disorders (EDs) on children, little is known about the effect of maternal EDs on neurobiological outcomes in newborns and infants. This study is the first to investigate neurobehavioural regulation and cognitive development in newborns and infants of mothers with EDs. Methods Women with an active and past ED and healthy controls were recruited to a prospective longitudinal study during their first trimester or second trimester of pregnancy. Newborns and infants of mothers with ED were compared with newborns and infants of healthy controls on (a) neurobehavioural dysregulation using the Brazelton Neonatal Behavioural Assessment Scale at 8 days postpartum (active ED, n = 15; past ED, n = 20; healthy controls, n = 28); and (b) cognitive development using the Bayley Scales of Infant and Toddler Development at 1-year postpartum (active ED, n = 18; past ED, n = 19; healthy controls, n = 28). In order to maintain the largest possible sample at each time point, sample size varied across time points. Results Newborns of mothers with an active ED had worse autonomic stability when compared with newborns of healthy controls [B = ?0.34 (?1.81, ?0.26)]. Infants of mothers with a past ED had poorer language [B = ?0.33 (?13.6, ?1.9)] and motor development [B = ?0.32 (?18.4, ?1.3)] compared with healthy controls. Conclusions Children of mothers with ED display neurobehavioural dysregulation early after birth and poorer language and motor development at 1 year. These characteristics suggest evidence of early neurobiological markers in children at risk. Differential outcomes in children of women with active versus past ED suggest that active symptomatology during pregnancy might have an effect on physiological reactivity while cognitive characteristics might be more stable markers of risk for ED. En ligne : http://dx.doi.org/10.1111/jcpp.12736 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=316
in Journal of Child Psychology and Psychiatry > 58-8 (August 2017) . - p.931-938[article] Neurobehavioural and cognitive development in infants born to mothers with eating disorders [Texte imprimé et/ou numérique] / Manuela BARONA, Auteur ; Emma TABORELLI, Auteur ; Freya CORFIELD, Auteur ; Susan PAWLBY, Auteur ; Abigail EASTER, Auteur ; Ulrike SCHMIDT, Auteur ; Janet TREASURE, Auteur ; Nadia MICALI, Auteur . - p.931-938.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 58-8 (August 2017) . - p.931-938
Mots-clés : Brazelton Neonatal Behavioural Assessment Scale Bayley Scales of Infant and Toddler Development eating disorders child development Index. décimale : PER Périodiques Résumé : Introduction Although recent research has focused on the effects of maternal eating disorders (EDs) on children, little is known about the effect of maternal EDs on neurobiological outcomes in newborns and infants. This study is the first to investigate neurobehavioural regulation and cognitive development in newborns and infants of mothers with EDs. Methods Women with an active and past ED and healthy controls were recruited to a prospective longitudinal study during their first trimester or second trimester of pregnancy. Newborns and infants of mothers with ED were compared with newborns and infants of healthy controls on (a) neurobehavioural dysregulation using the Brazelton Neonatal Behavioural Assessment Scale at 8 days postpartum (active ED, n = 15; past ED, n = 20; healthy controls, n = 28); and (b) cognitive development using the Bayley Scales of Infant and Toddler Development at 1-year postpartum (active ED, n = 18; past ED, n = 19; healthy controls, n = 28). In order to maintain the largest possible sample at each time point, sample size varied across time points. Results Newborns of mothers with an active ED had worse autonomic stability when compared with newborns of healthy controls [B = ?0.34 (?1.81, ?0.26)]. Infants of mothers with a past ED had poorer language [B = ?0.33 (?13.6, ?1.9)] and motor development [B = ?0.32 (?18.4, ?1.3)] compared with healthy controls. Conclusions Children of mothers with ED display neurobehavioural dysregulation early after birth and poorer language and motor development at 1 year. These characteristics suggest evidence of early neurobiological markers in children at risk. Differential outcomes in children of women with active versus past ED suggest that active symptomatology during pregnancy might have an effect on physiological reactivity while cognitive characteristics might be more stable markers of risk for ED. En ligne : http://dx.doi.org/10.1111/jcpp.12736 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=316 Alexithymia may explain the relationship between autistic traits and eating disorder psychopathology / L. VUILLIER in Molecular Autism, 11 (2020)
[article]
Titre : Alexithymia may explain the relationship between autistic traits and eating disorder psychopathology Type de document : Texte imprimé et/ou numérique Auteurs : L. VUILLIER, Auteur ; Z. CARTER, Auteur ; A. R. TEIXEIRA, Auteur ; R. L. MOSELEY, Auteur Article en page(s) : 63 p. Langues : Anglais (eng) Mots-clés : Alexithymia Anorexia nervosa Autism Autistic traits Eating disorders Index. décimale : PER Périodiques Résumé : BACKGROUND: Autistic people are disproportionately vulnerable to anorexia nervosa and other eating disorders (ED), and within the general population, autistic traits correlate with ED psychopathology. A putative mechanism which may underpin this heightened risk is alexithymia, a difficulty identifying and describing emotional states which is observed in both autism and ED. In two experiments with independent non-clinical samples, we explored whether alexithymia might mediate the heightened risk of eating psychopathology in individuals high in autistic traits. METHODS: Our first experiment used the PROCESS macro for SPSS to examine relationships between alexithymia (measured by the Toronto Alexithymia Scale (TAS-20)), autistic traits (autism quotient (AQ)), and eating psychopathology (Eating Attitudes Test (EAT-26)) in 121 participants. Our second experiment (n = 300) replicated and furthered this analysis by examining moderating effects of sex and controlling for anxiety and depression as covariates. We also included an additional performance-based measure of alexithymia, the Levels of Emotional Awareness Scale (LEAS). RESULTS: Study 1 suggested that TAS-20 scores mediated the relationship between heightened autistic traits and eating psychopathology. Replication and further scrutiny of this finding, in study 2, revealed that this mediation effect was partial and specific to the female participants in this sample. The mediation effect appeared to be carried by the difficulty identifying feelings subscale of the TAS-20, even when depression and anxiety were controlled for. LEAS scores, however, were not significantly related to autistic traits or eating psychopathology. LIMITATIONS: Cross-sectional data prevents any conclusions around the direction and causality of relationships between alexithymia, autistic traits, and eating psychopathology (alongside depression and anxiety), necessitating longitudinal research. Our non-clinical sample was predominantly Caucasian undergraduate students, so it remains to be seen if these results would extrapolate to clinical and/or autistic samples. Divergence between the TAS-20 and LEAS raises crucial questions regarding the construct validity of these measures. CONCLUSIONS: Our findings with respect to autistic traits suggest that alexithymia could partially explain the prevalence of ED in autistic people and may as such be an important consideration in the pathogenesis and treatment of ED in autistic and non-autistic people alike. Further research with clinical samples is critical to explore these ideas. Differences between men and women, furthermore, emphasize the importance of looking for sex-specific as well as generic risk factors in autistic and non-autistic men and women. En ligne : http://dx.doi.org/10.1186/s13229-020-00364-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=433
in Molecular Autism > 11 (2020) . - 63 p.[article] Alexithymia may explain the relationship between autistic traits and eating disorder psychopathology [Texte imprimé et/ou numérique] / L. VUILLIER, Auteur ; Z. CARTER, Auteur ; A. R. TEIXEIRA, Auteur ; R. L. MOSELEY, Auteur . - 63 p.
Langues : Anglais (eng)
in Molecular Autism > 11 (2020) . - 63 p.
Mots-clés : Alexithymia Anorexia nervosa Autism Autistic traits Eating disorders Index. décimale : PER Périodiques Résumé : BACKGROUND: Autistic people are disproportionately vulnerable to anorexia nervosa and other eating disorders (ED), and within the general population, autistic traits correlate with ED psychopathology. A putative mechanism which may underpin this heightened risk is alexithymia, a difficulty identifying and describing emotional states which is observed in both autism and ED. In two experiments with independent non-clinical samples, we explored whether alexithymia might mediate the heightened risk of eating psychopathology in individuals high in autistic traits. METHODS: Our first experiment used the PROCESS macro for SPSS to examine relationships between alexithymia (measured by the Toronto Alexithymia Scale (TAS-20)), autistic traits (autism quotient (AQ)), and eating psychopathology (Eating Attitudes Test (EAT-26)) in 121 participants. Our second experiment (n = 300) replicated and furthered this analysis by examining moderating effects of sex and controlling for anxiety and depression as covariates. We also included an additional performance-based measure of alexithymia, the Levels of Emotional Awareness Scale (LEAS). RESULTS: Study 1 suggested that TAS-20 scores mediated the relationship between heightened autistic traits and eating psychopathology. Replication and further scrutiny of this finding, in study 2, revealed that this mediation effect was partial and specific to the female participants in this sample. The mediation effect appeared to be carried by the difficulty identifying feelings subscale of the TAS-20, even when depression and anxiety were controlled for. LEAS scores, however, were not significantly related to autistic traits or eating psychopathology. LIMITATIONS: Cross-sectional data prevents any conclusions around the direction and causality of relationships between alexithymia, autistic traits, and eating psychopathology (alongside depression and anxiety), necessitating longitudinal research. Our non-clinical sample was predominantly Caucasian undergraduate students, so it remains to be seen if these results would extrapolate to clinical and/or autistic samples. Divergence between the TAS-20 and LEAS raises crucial questions regarding the construct validity of these measures. CONCLUSIONS: Our findings with respect to autistic traits suggest that alexithymia could partially explain the prevalence of ED in autistic people and may as such be an important consideration in the pathogenesis and treatment of ED in autistic and non-autistic people alike. Further research with clinical samples is critical to explore these ideas. Differences between men and women, furthermore, emphasize the importance of looking for sex-specific as well as generic risk factors in autistic and non-autistic men and women. En ligne : http://dx.doi.org/10.1186/s13229-020-00364-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=433 Eating disturbances in adults with autism spectrum disorder without intellectual disabilities / B. DEMARTINI in Autism Research, 14-7 (July 2021)
PermalinkRisk factors and temporal patterns of disordered eating differ in adolescent boys and girls: Testing gender-specific appearance anxiety models / Melanie J. ZIMMER-GEMBECK in Development and Psychopathology, 33-3 (August 2021)
PermalinkTrajectories of autistic social traits in childhood and adolescence and disordered eating behaviours at age 14 years: A UK general population cohort study / Francesca SOLMI in Journal of Child Psychology and Psychiatry, 62-1 (January 2021)
PermalinkUnravelling risks for child psychopathologies – general lessons from eating disorder research / Barbara MAUGHAN in Journal of Child Psychology and Psychiatry, 56-11 (November 2015)
PermalinkUsing the Ritvo Autism Asperger Diagnostic Scale-Revised (RAADS-R) disentangle the heterogeneity of autistic traits in an Italian eating disorder population / David VAGNI in Research in Autism Spectrum Disorders, 32 (December 2016)
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