Centre d'Information et de documentation du CRA Rhône-Alpes
CRA
Informations pratiques
-
Adresse
Centre d'information et de documentation
du CRA Rhône-Alpes
Centre Hospitalier le Vinatier
bât 211
95, Bd Pinel
69678 Bron CedexHoraires
Lundi au Vendredi
9h00-12h00 13h30-16h00Contact
Tél: +33(0)4 37 91 54 65
Mail
Fax: +33(0)4 37 91 54 37
-
Résultat de la recherche
6 recherche sur le mot-clé 'prenatal stress'
Affiner la recherche Générer le flux rss de la recherche
Partager le résultat de cette recherche Faire une suggestion
Maternal serotonin transporter genotype affects risk for ASD with exposure to prenatal stress / Patrick M. HECHT in Autism Research, 9-11 (November 2016)
[article]
Titre : Maternal serotonin transporter genotype affects risk for ASD with exposure to prenatal stress Type de document : Texte imprimé et/ou numérique Auteurs : Patrick M. HECHT, Auteur ; Melissa HUDSON, Auteur ; Susan L. CONNORS, Auteur ; Michael R. TILLEY, Auteur ; Xudong LIU, Auteur ; David Q. BEVERSDORF, Auteur Article en page(s) : p.1151-1160 Langues : Anglais (eng) Mots-clés : autism spectrum disorders serotonin stress prenatal stress development environmental influences Index. décimale : PER Périodiques Résumé : Stress exposure during gestation is implicated in several neuropsychiatric conditions, including autism spectrum disorder (ASD). Previous research showed that prenatal stress increases risk for ASD with peak exposure during the end of the second and the beginning of the third trimester. However, exposures to prenatal stress do not always result in ASD, suggesting that other factors may interact with environmental stressors to increase ASD risk. The present study examined a maternal genetic variation in the promoter region of the serotonin transporter gene (5-HTTLPR) affecting stress tolerance and its interaction with the effect of environmental stressors on risk for ASD. Two independent cohorts of mothers of ASD children recruited by the University of Missouri and Queen's University were surveyed regarding the prenatal environment and genotyping on 5-HTTLPR was performed to explore this relationship. In both samples, mothers of children with ASD carrying the stress susceptible short allele variant of 5-HTTLPR experienced a greater number of stressors and greater stress severity when compared to mothers carrying the long allele variant. The temporal peak of stressors during gestation in these mothers was consistent with previous findings. Additionally, increased exposure to prenatal stress was not reported in the pregnancies of typically developing siblings from the same mothers, regardless of maternal genotype, suggesting against the possibility that the short allele might increase the recall of stress during pregnancy. The present study provides further evidence of a specific maternal polymorphism that may affect the risk for ASD with exposure to prenatal stress. En ligne : http://dx.doi.org/10.1002/aur.1629 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=297
in Autism Research > 9-11 (November 2016) . - p.1151-1160[article] Maternal serotonin transporter genotype affects risk for ASD with exposure to prenatal stress [Texte imprimé et/ou numérique] / Patrick M. HECHT, Auteur ; Melissa HUDSON, Auteur ; Susan L. CONNORS, Auteur ; Michael R. TILLEY, Auteur ; Xudong LIU, Auteur ; David Q. BEVERSDORF, Auteur . - p.1151-1160.
Langues : Anglais (eng)
in Autism Research > 9-11 (November 2016) . - p.1151-1160
Mots-clés : autism spectrum disorders serotonin stress prenatal stress development environmental influences Index. décimale : PER Périodiques Résumé : Stress exposure during gestation is implicated in several neuropsychiatric conditions, including autism spectrum disorder (ASD). Previous research showed that prenatal stress increases risk for ASD with peak exposure during the end of the second and the beginning of the third trimester. However, exposures to prenatal stress do not always result in ASD, suggesting that other factors may interact with environmental stressors to increase ASD risk. The present study examined a maternal genetic variation in the promoter region of the serotonin transporter gene (5-HTTLPR) affecting stress tolerance and its interaction with the effect of environmental stressors on risk for ASD. Two independent cohorts of mothers of ASD children recruited by the University of Missouri and Queen's University were surveyed regarding the prenatal environment and genotyping on 5-HTTLPR was performed to explore this relationship. In both samples, mothers of children with ASD carrying the stress susceptible short allele variant of 5-HTTLPR experienced a greater number of stressors and greater stress severity when compared to mothers carrying the long allele variant. The temporal peak of stressors during gestation in these mothers was consistent with previous findings. Additionally, increased exposure to prenatal stress was not reported in the pregnancies of typically developing siblings from the same mothers, regardless of maternal genotype, suggesting against the possibility that the short allele might increase the recall of stress during pregnancy. The present study provides further evidence of a specific maternal polymorphism that may affect the risk for ASD with exposure to prenatal stress. En ligne : http://dx.doi.org/10.1002/aur.1629 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=297 Mothers' prenatal stress and their children's antisocial outcomes – a moderating role for the Dopamine D4 Receptor (DRD4) gene / Katrin ZOHSEL in Journal of Child Psychology and Psychiatry, 55-1 (January 2014)
[article]
Titre : Mothers' prenatal stress and their children's antisocial outcomes – a moderating role for the Dopamine D4 Receptor (DRD4) gene Type de document : Texte imprimé et/ou numérique Auteurs : Katrin ZOHSEL, Auteur ; Arlette F. BUCHMANN, Auteur ; Dorothea BLOMEYER, Auteur ; Erika HOHM, Auteur ; Martin H. SCHMIDT, Auteur ; Günter ESSER, Auteur ; Daniel BRANDEIS, Auteur ; Tobias BANASCHEWSKI, Auteur ; Manfred LAUCHT, Auteur Article en page(s) : p.69-76 Langues : Anglais (eng) Mots-clés : Prenatal stress antisocial conduct disorder DRD4 gene–environment interaction Index. décimale : PER Périodiques Résumé : Background Maternal distress during pregnancy has been linked to aggressive behavior in offspring. This effect has been interpreted in terms of ‘fetal programming’. The 7-repeat (7r) allele of a VNTR polymorphism in exon III of the human dopamine receptor D4 (DRD4) has consistently been associated with externalizing behavior problems, especially in the presence of adverse environmental factors. So far, it is not known whether the DRD4 genotype moderates the effect of prenatal maternal stress on the development of childhood antisocial behavior. Methods As part of an ongoing epidemiological cohort study, prenatal maternal stress was assessed using self-report 3 months following child birth. When children were 8, 11, and 15 years old, mothers rated their children's externalizing behavior, and diagnoses of conduct disorder and/or oppositional defiant disorder (CD/ODD) according to DSM-IV were obtained. In a sample of N = 308 participants, the effects of the DRD4 genotype, prenatal maternal stress, and the interaction thereof on antisocial outcome were tested. Results Under conditions of elevated prenatal maternal stress, children carrying one or two DRD4 7r alleles were at increased risk of a diagnosis of CD/ODD. Moreover, homozygous carriers of the DRD4 7r allele displayed more externalizing behavior following exposure to higher levels of prenatal maternal stress, while homozygous carriers of the DRD4 4r allele turned out to be insensitive to the effects of prenatal stress. Conclusions This study is the first to report a gene–environment interaction related to DRD4 and prenatal maternal stress using data from a prospective study, which extends earlier findings on the impact of prenatal maternal stress with respect to childhood antisocial behavior. En ligne : http://dx.doi.org/10.1111/jcpp.12138 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=220
in Journal of Child Psychology and Psychiatry > 55-1 (January 2014) . - p.69-76[article] Mothers' prenatal stress and their children's antisocial outcomes – a moderating role for the Dopamine D4 Receptor (DRD4) gene [Texte imprimé et/ou numérique] / Katrin ZOHSEL, Auteur ; Arlette F. BUCHMANN, Auteur ; Dorothea BLOMEYER, Auteur ; Erika HOHM, Auteur ; Martin H. SCHMIDT, Auteur ; Günter ESSER, Auteur ; Daniel BRANDEIS, Auteur ; Tobias BANASCHEWSKI, Auteur ; Manfred LAUCHT, Auteur . - p.69-76.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 55-1 (January 2014) . - p.69-76
Mots-clés : Prenatal stress antisocial conduct disorder DRD4 gene–environment interaction Index. décimale : PER Périodiques Résumé : Background Maternal distress during pregnancy has been linked to aggressive behavior in offspring. This effect has been interpreted in terms of ‘fetal programming’. The 7-repeat (7r) allele of a VNTR polymorphism in exon III of the human dopamine receptor D4 (DRD4) has consistently been associated with externalizing behavior problems, especially in the presence of adverse environmental factors. So far, it is not known whether the DRD4 genotype moderates the effect of prenatal maternal stress on the development of childhood antisocial behavior. Methods As part of an ongoing epidemiological cohort study, prenatal maternal stress was assessed using self-report 3 months following child birth. When children were 8, 11, and 15 years old, mothers rated their children's externalizing behavior, and diagnoses of conduct disorder and/or oppositional defiant disorder (CD/ODD) according to DSM-IV were obtained. In a sample of N = 308 participants, the effects of the DRD4 genotype, prenatal maternal stress, and the interaction thereof on antisocial outcome were tested. Results Under conditions of elevated prenatal maternal stress, children carrying one or two DRD4 7r alleles were at increased risk of a diagnosis of CD/ODD. Moreover, homozygous carriers of the DRD4 7r allele displayed more externalizing behavior following exposure to higher levels of prenatal maternal stress, while homozygous carriers of the DRD4 4r allele turned out to be insensitive to the effects of prenatal stress. Conclusions This study is the first to report a gene–environment interaction related to DRD4 and prenatal maternal stress using data from a prospective study, which extends earlier findings on the impact of prenatal maternal stress with respect to childhood antisocial behavior. En ligne : http://dx.doi.org/10.1111/jcpp.12138 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=220 Prenatal maternal stress during the COVID-19 pandemic and infant regulatory capacity at 3 months: A longitudinal study / Livio PROVENZI in Development and Psychopathology, 35-1 (February 2023)
[article]
Titre : Prenatal maternal stress during the COVID-19 pandemic and infant regulatory capacity at 3 months: A longitudinal study Type de document : Texte imprimé et/ou numérique Auteurs : Livio PROVENZI, Auteur ; Serena GRUMI, Auteur ; Lilia ALTIERI, Auteur ; Giulia BENSI, Auteur ; Emanuela BERTAZZOLI, Auteur ; Giacomo BIASUCCI, Auteur ; Anna CAVALLINI, Auteur ; Lidia DECEMBRINO, Auteur ; Rossana FALCONE, Auteur ; Anna FREDDI, Auteur ; Barbara GARDELLA, Auteur ; Roberta GIACCHERO, Auteur ; Roberto GIORDA, Auteur ; Elena GROSSI, Auteur ; Paola GUERINI, Auteur ; Maria Luisa MAGNANI, Auteur ; Paola MARTELLI, Auteur ; Mario MOTTA, Auteur ; Renata NACINOVICH, Auteur ; Dario PANTALEO, Auteur ; Camilla PISONI, Auteur ; Federico PREFUMO, Auteur ; Laura RIVA, Auteur ; Barbara SCELSA, Auteur ; Maria V. SPARTÀ, Auteur ; Arsenio SPINILLO, Auteur ; Patrizia VERGANI, Auteur ; Simona ORCESI, Auteur ; Renato BORGATTI, Auteur Article en page(s) : p.35-43 Langues : Anglais (eng) Mots-clés : anxiety COVID-19 epidemic maternal bonding prenatal stress regulatory capacity social support temperament Index. décimale : PER Périodiques Résumé : The COVID-19 pandemic is a global traumatic experience for citizens, especially during sensitive time windows of heightened plasticity such as pregnancy and neonatal life. Pandemic-related stress experienced by mothers during pregnancy may act as an early risk factor for infants' regulatory capacity development by altering maternal psychosocial well-being (e.g., increased anxiety, reduced social support) and caregiving environment (e.g., greater parenting stress, impaired mother-infant bonding). The aim of the present longitudinal study was to assess the consequences of pandemic-related prenatal stress on infants' regulatory capacity. A sample of 163 mother-infant dyads was enrolled at eight maternity units in northern Italy. They provided complete data about prenatal stress, perceived social support, postnatal anxiety symptoms, parenting stress, mother-infant bonding, and infants' regulatory capacity at 3 months of age. Women who experienced emotional stress and received partial social support during pregnancy reported higher anxious symptoms. Moreover, maternal postnatal anxiety was indirectly linked to the infants' regulatory capacity at 3 months, mediated by parenting stress and mother-infant bonding. Dedicated preventive interventions should be delivered to mothers and should be focused on protecting the mother-infant dyad from the detrimental effects of pandemic-related stress during the COVID-19 healthcare emergency. En ligne : https://doi.org/10.1017/S0954579421000766 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499
in Development and Psychopathology > 35-1 (February 2023) . - p.35-43[article] Prenatal maternal stress during the COVID-19 pandemic and infant regulatory capacity at 3 months: A longitudinal study [Texte imprimé et/ou numérique] / Livio PROVENZI, Auteur ; Serena GRUMI, Auteur ; Lilia ALTIERI, Auteur ; Giulia BENSI, Auteur ; Emanuela BERTAZZOLI, Auteur ; Giacomo BIASUCCI, Auteur ; Anna CAVALLINI, Auteur ; Lidia DECEMBRINO, Auteur ; Rossana FALCONE, Auteur ; Anna FREDDI, Auteur ; Barbara GARDELLA, Auteur ; Roberta GIACCHERO, Auteur ; Roberto GIORDA, Auteur ; Elena GROSSI, Auteur ; Paola GUERINI, Auteur ; Maria Luisa MAGNANI, Auteur ; Paola MARTELLI, Auteur ; Mario MOTTA, Auteur ; Renata NACINOVICH, Auteur ; Dario PANTALEO, Auteur ; Camilla PISONI, Auteur ; Federico PREFUMO, Auteur ; Laura RIVA, Auteur ; Barbara SCELSA, Auteur ; Maria V. SPARTÀ, Auteur ; Arsenio SPINILLO, Auteur ; Patrizia VERGANI, Auteur ; Simona ORCESI, Auteur ; Renato BORGATTI, Auteur . - p.35-43.
Langues : Anglais (eng)
in Development and Psychopathology > 35-1 (February 2023) . - p.35-43
Mots-clés : anxiety COVID-19 epidemic maternal bonding prenatal stress regulatory capacity social support temperament Index. décimale : PER Périodiques Résumé : The COVID-19 pandemic is a global traumatic experience for citizens, especially during sensitive time windows of heightened plasticity such as pregnancy and neonatal life. Pandemic-related stress experienced by mothers during pregnancy may act as an early risk factor for infants' regulatory capacity development by altering maternal psychosocial well-being (e.g., increased anxiety, reduced social support) and caregiving environment (e.g., greater parenting stress, impaired mother-infant bonding). The aim of the present longitudinal study was to assess the consequences of pandemic-related prenatal stress on infants' regulatory capacity. A sample of 163 mother-infant dyads was enrolled at eight maternity units in northern Italy. They provided complete data about prenatal stress, perceived social support, postnatal anxiety symptoms, parenting stress, mother-infant bonding, and infants' regulatory capacity at 3 months of age. Women who experienced emotional stress and received partial social support during pregnancy reported higher anxious symptoms. Moreover, maternal postnatal anxiety was indirectly linked to the infants' regulatory capacity at 3 months, mediated by parenting stress and mother-infant bonding. Dedicated preventive interventions should be delivered to mothers and should be focused on protecting the mother-infant dyad from the detrimental effects of pandemic-related stress during the COVID-19 healthcare emergency. En ligne : https://doi.org/10.1017/S0954579421000766 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499 Maternal caregiving ameliorates the consequences of prenatal maternal psychological distress on child development / Leah A. GRANDE in Development and Psychopathology, 34-4 (October 2022)
[article]
Titre : Maternal caregiving ameliorates the consequences of prenatal maternal psychological distress on child development Type de document : Texte imprimé et/ou numérique Auteurs : Leah A. GRANDE, Auteur ; Danielle A. SWALES, Auteur ; Curt A. SANDMAN, Auteur ; Laura M. GLYNN, Auteur ; Elysia Poggi DAVIS, Auteur Article en page(s) : p.1376-1385 Langues : Anglais (eng) Mots-clés : Child Development Child, Preschool Depression/psychology Female Humans Mothers/psychology Pregnancy Prenatal Exposure Delayed Effects/psychology Psychological Distress Stress, Psychological/psychology cognitive function depression fetal programming maternal care parenting prenatal stress resilience Index. décimale : PER Périodiques Résumé : Children exposed to prenatal maternal psychological distress are at elevated risk for a range of adverse outcomes; however, it remains poorly understood whether postnatal influences can ameliorate impairments related to prenatal distress. The current study evaluated if sensitivematernal care during the first postnatal year could mitigate child cognitive and emotional impairments associated with prenatal psychological distress. Prenatal maternal psychological distress was assessed via self-reports of anxiety, depression, and perceived stress for 136 mothers at five prenatal and four postpartum time points. Quality of maternal care (sensitivity to nondistress, positive regard, and intrusiveness reverse-scored) were assessed during a mother-child play interaction at 6 and 12 months. Child cognitive function and negative emotionality were assessed at 2 years, using The Bayley Scales and the Early Childhood Behavior Questionnaire. Elevated prenatal distress was associated with poorer child cognitive function and elevated negative emotionality. Children exposed to elevated prenatal maternal distress did not, however, display these outcomes if they received high-quality caregiving. Specifically, maternal care moderated the relation between prenatal psychological distress and child cognitive function and negative emotionality. This association remained after consideration of postnatal maternal psychological distress and relevant covariates. Sensitive maternal care was associated with altered offspring developmental trajectories, supporting child resilience following prenatal distress exposure. En ligne : http://dx.doi.org/10.1017/s0954579421000286 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488
in Development and Psychopathology > 34-4 (October 2022) . - p.1376-1385[article] Maternal caregiving ameliorates the consequences of prenatal maternal psychological distress on child development [Texte imprimé et/ou numérique] / Leah A. GRANDE, Auteur ; Danielle A. SWALES, Auteur ; Curt A. SANDMAN, Auteur ; Laura M. GLYNN, Auteur ; Elysia Poggi DAVIS, Auteur . - p.1376-1385.
Langues : Anglais (eng)
in Development and Psychopathology > 34-4 (October 2022) . - p.1376-1385
Mots-clés : Child Development Child, Preschool Depression/psychology Female Humans Mothers/psychology Pregnancy Prenatal Exposure Delayed Effects/psychology Psychological Distress Stress, Psychological/psychology cognitive function depression fetal programming maternal care parenting prenatal stress resilience Index. décimale : PER Périodiques Résumé : Children exposed to prenatal maternal psychological distress are at elevated risk for a range of adverse outcomes; however, it remains poorly understood whether postnatal influences can ameliorate impairments related to prenatal distress. The current study evaluated if sensitivematernal care during the first postnatal year could mitigate child cognitive and emotional impairments associated with prenatal psychological distress. Prenatal maternal psychological distress was assessed via self-reports of anxiety, depression, and perceived stress for 136 mothers at five prenatal and four postpartum time points. Quality of maternal care (sensitivity to nondistress, positive regard, and intrusiveness reverse-scored) were assessed during a mother-child play interaction at 6 and 12 months. Child cognitive function and negative emotionality were assessed at 2 years, using The Bayley Scales and the Early Childhood Behavior Questionnaire. Elevated prenatal distress was associated with poorer child cognitive function and elevated negative emotionality. Children exposed to elevated prenatal maternal distress did not, however, display these outcomes if they received high-quality caregiving. Specifically, maternal care moderated the relation between prenatal psychological distress and child cognitive function and negative emotionality. This association remained after consideration of postnatal maternal psychological distress and relevant covariates. Sensitive maternal care was associated with altered offspring developmental trajectories, supporting child resilience following prenatal distress exposure. En ligne : http://dx.doi.org/10.1017/s0954579421000286 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=488 The impacts of maternal childhood adversity, stress, and mental health on child development at 6 months in Taiwan: A follow-up study / Yi-Ting CHANG in Development and Psychopathology, 33-3 (August 2021)
[article]
Titre : The impacts of maternal childhood adversity, stress, and mental health on child development at 6 months in Taiwan: A follow-up study Type de document : Texte imprimé et/ou numérique Auteurs : Yi-Ting CHANG, Auteur ; Jui-Ying FENG, Auteur ; Hsin-Yi CHANG, Auteur ; Yu-Chun CHANG, Auteur ; Chia-Kuei LEE, Auteur Article en page(s) : p.970-979 Langues : Anglais (eng) Mots-clés : adverse childhood experiences intergenerational effect perinatal depression prenatal stress psychosocial risk Index. décimale : PER Périodiques Résumé : Exposure to adverse childhood experiences (ACEs) is not only associated with one's adverse health outcomes in adulthood but also increases the risk of child developmental problems in offspring. However, the mechanisms involved in the transmission of the effects of maternal ACEs to the offspring largely remain unexplored. This study sought to identify possible psychosocial pathways of intergenerational effects of maternal ACEs on child development at 6 months. Data from a longitudinal study on maternal childhood adversity and maternal psychosocial risk during pregnancy as well as maternal mental health problems and child development at 6 months postnatal were used. Structural equation modeling with bootstrapping was used to estimate the indirect effects of maternal ACEs on child development at 6 months. The model showed that maternal ACEs indirectly influenced offspring's development via maternal stressful events during pregnancy and pre- and postnatal mental health problems. This finding highlights the possible interventions at the prenatal and postnatal periods. Early identification of women who have ACEs or who are at psychosocial risk during pre- and postnatal periods is critical to provide interventions to buffer those negative effects on offspring's development. Future studies are needed to longitudinally assess the effects of maternal ACEs on child development over time. En ligne : http://dx.doi.org/10.1017/S0954579420000267 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457
in Development and Psychopathology > 33-3 (August 2021) . - p.970-979[article] The impacts of maternal childhood adversity, stress, and mental health on child development at 6 months in Taiwan: A follow-up study [Texte imprimé et/ou numérique] / Yi-Ting CHANG, Auteur ; Jui-Ying FENG, Auteur ; Hsin-Yi CHANG, Auteur ; Yu-Chun CHANG, Auteur ; Chia-Kuei LEE, Auteur . - p.970-979.
Langues : Anglais (eng)
in Development and Psychopathology > 33-3 (August 2021) . - p.970-979
Mots-clés : adverse childhood experiences intergenerational effect perinatal depression prenatal stress psychosocial risk Index. décimale : PER Périodiques Résumé : Exposure to adverse childhood experiences (ACEs) is not only associated with one's adverse health outcomes in adulthood but also increases the risk of child developmental problems in offspring. However, the mechanisms involved in the transmission of the effects of maternal ACEs to the offspring largely remain unexplored. This study sought to identify possible psychosocial pathways of intergenerational effects of maternal ACEs on child development at 6 months. Data from a longitudinal study on maternal childhood adversity and maternal psychosocial risk during pregnancy as well as maternal mental health problems and child development at 6 months postnatal were used. Structural equation modeling with bootstrapping was used to estimate the indirect effects of maternal ACEs on child development at 6 months. The model showed that maternal ACEs indirectly influenced offspring's development via maternal stressful events during pregnancy and pre- and postnatal mental health problems. This finding highlights the possible interventions at the prenatal and postnatal periods. Early identification of women who have ACEs or who are at psychosocial risk during pre- and postnatal periods is critical to provide interventions to buffer those negative effects on offspring's development. Future studies are needed to longitudinally assess the effects of maternal ACEs on child development over time. En ligne : http://dx.doi.org/10.1017/S0954579420000267 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 Breastfeeding continuation at 6 weeks postpartum remediates the negative effects of prenatal intimate partner violence on infant temperament / Laura MILLER-GRAFF in Development and Psychopathology, 32-2 (May 2020)
Permalink