Blunted feedback processing during risky decision making in adolescents with a parental history of substance use disorders / Anja S. EUSER in Development and Psychopathology, 25-4 (November 2013)  

Public ISBD [article]  inDevelopment and Psychopathology > 25-4 (November 2013) . - p.1119-1136 Titre : Blunted feedback processing during risky decision making in adolescents with a parental history of substance use disorders Type de document : texte imprimé Auteurs : Anja S. EUSER, Auteur ; Kirstin GREAVES-LORD, Auteur ; Michael J. CROWLEY, Auteur ; Brittany E. EVANS, Auteur ; Anja C. HUIZINK, Auteur ; Ingmar H.A. FRANKEN, Auteur Article en page(s) : p.1119-1136 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Risky decision making, a hallmark phenotype of substance use disorders (SUD), is thought to be associated with deficient feedback processing. Whether these aberrations are present prior to SUD onset or reflect merely a consequence of chronic substance use on the brain remains unclear. The present study investigated whether blunted feedback processing during risky decision making reflects a biological predisposition to SUD. We assessed event-related potentials elicited by positive and negative feedback during performance of a modified version of the Balloon Analogue Risk Task (BART) among high-risk adolescents with a parental history of SUD (HR; n = 61) and normal-risk controls (NR; n = 91). HR males made significantly more risky and faster decisions during the BART than did NR controls. Moreover, HR adolescents showed significantly reduced P300 amplitudes in response to both positive and negative feedback as compared to NR controls. These differences were not secondary to prolonged substance use exposure. Results are discussed in terms of feedback-specific processes. Reduced P300 amplitudes in the BART may reflect poor processing of feedback at the level of overall salience, which may keep people from effectively predicting the probability of future gains and losses. Though conclusions are tentative, blunted feedback processing during risky decision making may represent a promising endophenotypic vulnerability marker for SUD. En ligne : http://dx.doi.org/10.1017/S0954579413000412 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=219 [article] Blunted feedback processing during risky decision making in adolescents with a parental history of substance use disorders [texte imprimé] / Anja S. EUSER, Auteur ; Kirstin GREAVES-LORD, Auteur ; Michael J. CROWLEY, Auteur ; Brittany E. EVANS, Auteur ; Anja C. HUIZINK, Auteur ; Ingmar H.A. FRANKEN, Auteur . - p.1119-1136. Langues : Anglais (eng) in Development and Psychopathology > 25-4 (November 2013) . - p.1119-1136 Index. décimale : PER Périodiques Résumé : Risky decision making, a hallmark phenotype of substance use disorders (SUD), is thought to be associated with deficient feedback processing. Whether these aberrations are present prior to SUD onset or reflect merely a consequence of chronic substance use on the brain remains unclear. The present study investigated whether blunted feedback processing during risky decision making reflects a biological predisposition to SUD. We assessed event-related potentials elicited by positive and negative feedback during performance of a modified version of the Balloon Analogue Risk Task (BART) among high-risk adolescents with a parental history of SUD (HR; n = 61) and normal-risk controls (NR; n = 91). HR males made significantly more risky and faster decisions during the BART than did NR controls. Moreover, HR adolescents showed significantly reduced P300 amplitudes in response to both positive and negative feedback as compared to NR controls. These differences were not secondary to prolonged substance use exposure. Results are discussed in terms of feedback-specific processes. Reduced P300 amplitudes in the BART may reflect poor processing of feedback at the level of overall salience, which may keep people from effectively predicting the probability of future gains and losses. Though conclusions are tentative, blunted feedback processing during risky decision making may represent a promising endophenotypic vulnerability marker for SUD. En ligne : http://dx.doi.org/10.1017/S0954579413000412 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=219

Gene × Environment contributions to autonomic stress reactivity in youth / Andrea G. ALLEGRINI in Development and Psychopathology, 31-1 (February 2019)  

Public ISBD [article]  inDevelopment and Psychopathology > 31-1 (February 2019) . - p.293-307 Titre : Gene × Environment contributions to autonomic stress reactivity in youth Type de document : texte imprimé Auteurs : Andrea G. ALLEGRINI, Auteur ; Brittany E. EVANS, Auteur ; Susanne DE ROOIJ, Auteur ; Kirstin GREAVES-LORD, Auteur ; Anja C. HUIZINK, Auteur Article en page(s) : p.293-307 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Dysregulated physiological stress reactivity has been suggested to impact the development of children and adolescents with important health consequences throughout the life span. Both environmental adversity and genetic predispositions can lead to physiological imbalances in stress systems, which in turn lead to developmental differences. We investigated genetic and environmental contributions to autonomic nervous system reactivity to a psychosocial stressor. Furthermore, we tested whether these effects were consistent with the differential susceptibility framework. Composite measures of adverse life events combined with socioeconomic status were constructed. Effects of these adversity scores in interaction with a polygenic score summarizing six genetic variants, which were hypothesized to work as susceptibility factors, were tested on autonomic nervous system measures as indexed by heart rate and heart rate variability. Results showed that carriers of more genetic variants and exposed to high adversity manifested enhanced heart rate variability reactivity to a psychosocial stressor compared to carriers of fewer genetic variants. Conversely, the stress procedure elicited a more moderate response in these individuals compared to carriers of fewer variants when adversity was low. En ligne : http://dx.doi.org/10.1017/S095457941700181X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=383 [article] Gene × Environment contributions to autonomic stress reactivity in youth [texte imprimé] / Andrea G. ALLEGRINI, Auteur ; Brittany E. EVANS, Auteur ; Susanne DE ROOIJ, Auteur ; Kirstin GREAVES-LORD, Auteur ; Anja C. HUIZINK, Auteur . - p.293-307. Langues : Anglais (eng) in Development and Psychopathology > 31-1 (February 2019) . - p.293-307 Index. décimale : PER Périodiques Résumé : Dysregulated physiological stress reactivity has been suggested to impact the development of children and adolescents with important health consequences throughout the life span. Both environmental adversity and genetic predispositions can lead to physiological imbalances in stress systems, which in turn lead to developmental differences. We investigated genetic and environmental contributions to autonomic nervous system reactivity to a psychosocial stressor. Furthermore, we tested whether these effects were consistent with the differential susceptibility framework. Composite measures of adverse life events combined with socioeconomic status were constructed. Effects of these adversity scores in interaction with a polygenic score summarizing six genetic variants, which were hypothesized to work as susceptibility factors, were tested on autonomic nervous system measures as indexed by heart rate and heart rate variability. Results showed that carriers of more genetic variants and exposed to high adversity manifested enhanced heart rate variability reactivity to a psychosocial stressor compared to carriers of fewer genetic variants. Conversely, the stress procedure elicited a more moderate response in these individuals compared to carriers of fewer variants when adversity was low. En ligne : http://dx.doi.org/10.1017/S095457941700181X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=383

Heart rate (variability) and the association between relational peer victimization and internalizing symptoms in elementary school children / Pia BEHNSEN in Development and Psychopathology, 32-2 (May 2020)  

Public ISBD [article]  inDevelopment and Psychopathology > 32-2 (May 2020) . - p.521-529 Titre : Heart rate (variability) and the association between relational peer victimization and internalizing symptoms in elementary school children Type de document : texte imprimé Auteurs : Pia BEHNSEN, Auteur ; Joanne Marieke BUIL, Auteur ; Susanne KOOT, Auteur ; Anja C. HUIZINK, Auteur ; Pol VAN LIER, Auteur Article en page(s) : p.521-529 Langues : Anglais (eng) Mots-clés : autonomic nervous system  children  internalizing symptoms  relational victimization Index. décimale : PER Périodiques Résumé : Relational victimization typically emerges first during the elementary school period, and has been associated with increased levels of internalizing symptoms in children. Individual differences in autonomic nervous system functioning have been suggested as a potential factor linking social stressors and internalizing symptoms. The aim of this study was therefore to examine whether heart rate and heart rate variability mediated the association between relational victimization and internalizing symptoms in 373 mainstream elementary school children. Children were assessed in 2015 (T0; Grades 3-5, M age = 9.78 years, 51% boys) and reassessed in 2016 (T1). Heart rate and heart rate variability were assessed during a regular school day at T1. A multi-informant (teacher and peer report) cross-time measure of relational victimization, and a multi-informant (self- and teacher report) measure of internalizing problems at T1 was used. Results showed that heart rate variability, but not heart rate, mediated the association between relational victimization and internalizing symptoms. This study provides tentative support that in children from a general population sample, a psychobiological factor may mediate the association of relational victimization with internalizing symptoms. En ligne : http://dx.doi.org/10.1017/s0954579419000269 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=426 [article] Heart rate (variability) and the association between relational peer victimization and internalizing symptoms in elementary school children [texte imprimé] / Pia BEHNSEN, Auteur ; Joanne Marieke BUIL, Auteur ; Susanne KOOT, Auteur ; Anja C. HUIZINK, Auteur ; Pol VAN LIER, Auteur . - p.521-529. Langues : Anglais (eng) in Development and Psychopathology > 32-2 (May 2020) . - p.521-529 Mots-clés : autonomic nervous system  children  internalizing symptoms  relational victimization Index. décimale : PER Périodiques Résumé : Relational victimization typically emerges first during the elementary school period, and has been associated with increased levels of internalizing symptoms in children. Individual differences in autonomic nervous system functioning have been suggested as a potential factor linking social stressors and internalizing symptoms. The aim of this study was therefore to examine whether heart rate and heart rate variability mediated the association between relational victimization and internalizing symptoms in 373 mainstream elementary school children. Children were assessed in 2015 (T0; Grades 3-5, M age = 9.78 years, 51% boys) and reassessed in 2016 (T1). Heart rate and heart rate variability were assessed during a regular school day at T1. A multi-informant (teacher and peer report) cross-time measure of relational victimization, and a multi-informant (self- and teacher report) measure of internalizing problems at T1 was used. Results showed that heart rate variability, but not heart rate, mediated the association between relational victimization and internalizing symptoms. This study provides tentative support that in children from a general population sample, a psychobiological factor may mediate the association of relational victimization with internalizing symptoms. En ligne : http://dx.doi.org/10.1017/s0954579419000269 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=426

Mild dermatoglyphic deviations in adolescents with autism spectrum disorders and average intellectual abilities as compared to typically developing boys / Esther I. DE BRUIN in Autism Research and Treatment, 2014 (2014)  

Public ISBD [article]  inAutism Research and Treatment > 2014 (2014) Titre : Mild dermatoglyphic deviations in adolescents with autism spectrum disorders and average intellectual abilities as compared to typically developing boys Type de document : texte imprimé Auteurs : Esther I. DE BRUIN, Auteur ; James H. GRAHAM, Auteur ; Anneke LOUWERSE, Auteur ; Anja C. HUIZINK, Auteur Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Dermatoglyphics, ridge constellations on the hands and feet, are permanently formed by the second trimester of pregnancy. Consequently, they are considered "fossilized" evidence of a specific prenatal period. A high frequency of dermatoglyphic anomalies, or a high rate of dermatoglyphic asymmetry (discordance), is an indication of developmental instability (prenatal disturbances) prior to 24-week gestation. Most dermatoglyphic studies in psychiatry focus on adult schizophrenia. Studies on dermatoglyphic deviances and autism are sparse, include severely disturbed and intellectually retarded patients with autism, and are carried out mainly in non-Western European populations. In this study, finger print patterns, atd-angles, and palmar flexion crease patterns (PFCs) are compared between Western European adolescent teenage males, of average intellect, with Autism Spectrum Disorders (ASD; n = 46) and typically developing adolescent teenage males (TD; n = 49). Boys with ASD had a higher rate of discordance in their finger print patterns than TD boys. Thus, the hypothesized prenatal disturbances that play a role in the etiology of schizophrenia and severe autism might not be specific to these severe psychiatric disorders but might also be involved in the etiology of varying degrees of ASD. En ligne : http://dx.doi.org/10.1155/2014/968134 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=331 [article] Mild dermatoglyphic deviations in adolescents with autism spectrum disorders and average intellectual abilities as compared to typically developing boys [texte imprimé] / Esther I. DE BRUIN, Auteur ; James H. GRAHAM, Auteur ; Anneke LOUWERSE, Auteur ; Anja C. HUIZINK, Auteur. Langues : Anglais (eng) in Autism Research and Treatment > 2014 (2014) Index. décimale : PER Périodiques Résumé : Dermatoglyphics, ridge constellations on the hands and feet, are permanently formed by the second trimester of pregnancy. Consequently, they are considered "fossilized" evidence of a specific prenatal period. A high frequency of dermatoglyphic anomalies, or a high rate of dermatoglyphic asymmetry (discordance), is an indication of developmental instability (prenatal disturbances) prior to 24-week gestation. Most dermatoglyphic studies in psychiatry focus on adult schizophrenia. Studies on dermatoglyphic deviances and autism are sparse, include severely disturbed and intellectually retarded patients with autism, and are carried out mainly in non-Western European populations. In this study, finger print patterns, atd-angles, and palmar flexion crease patterns (PFCs) are compared between Western European adolescent teenage males, of average intellect, with Autism Spectrum Disorders (ASD; n = 46) and typically developing adolescent teenage males (TD; n = 49). Boys with ASD had a higher rate of discordance in their finger print patterns than TD boys. Thus, the hypothesized prenatal disturbances that play a role in the etiology of schizophrenia and severe autism might not be specific to these severe psychiatric disorders but might also be involved in the etiology of varying degrees of ASD. En ligne : http://dx.doi.org/10.1155/2014/968134 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=331

Prenatal smoking predicts non-responsiveness to an intervention targeting attention-deficit/hyperactivity symptoms in elementary schoolchildren / Patricia VUIJK in Journal of Child Psychology and Psychiatry, 47-9 (September 2006)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 47-9 (September 2006) . - p.891–901 Titre : Prenatal smoking predicts non-responsiveness to an intervention targeting attention-deficit/hyperactivity symptoms in elementary schoolchildren Type de document : texte imprimé Auteurs : Patricia VUIJK, Auteur ; Pol A.C. VAN LIER, Auteur ; Frank C. VERHULST, Auteur ; Anja C. HUIZINK, Auteur ; Alfons A.M. CRIJNEN, Auteur Année de publication : 2006 Article en page(s) : p.891–901 Langues : Anglais (eng) Mots-clés : Prenatal-smoking fetal-programming preventive-interventions ADHD-symptoms early-onset-experimentation-with-smoking Index. décimale : PER Périodiques Résumé : Background: Some evidence suggests that prenatal exposure to maternal smoking contributes to the etiology of Attention-Deficit/Hyperactivity Disorder (ADHD). The present study tested an intervention targeting disruptive behavior to establish whether exposure to maternal smoking during pregnancy predicts intervention failure in elementary schoolchildren. Methods: Five hundred and eight elementary schoolchildren were followed from ages 7 to 11. At ages 8 and 9, they were randomly assigned to a control condition, or to a two-year universal classroom-based intervention targeting disruptive behavior. Measures included maternal reports of smoking during pregnancy, teacher-rated symptoms of ADHD from ages 7 to 9 years, and children's self-report of experimentation with smoking at ages 10 and 11 years. Results: At age 7, prenatally exposed children had higher ADHD symptom scores. The intervention did not affect the course of their ADHD symptoms and the probability of early-onset experimentation with smoking. Among non-exposed children, the intervention positively affected the course of ADHD symptoms, and reduced the probability of early-onset experimentation with smoking. Conclusions: Prenatally exposed children are most prone to follow a path of high levels of ADHD symptoms and associated elevated risk for early-onset experimentation with smoking, which is unreceptive to a universal preventive intervention. In these children, the developmental course of ADHD symptoms seems to have been influenced by their prenatal exposure to maternal smoking. Future research should further explore whether prenatal smoking is a proxy measure that indexes another risk factor, or a causal factor for adverse developmental outcomes. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01647.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=775 [article] Prenatal smoking predicts non-responsiveness to an intervention targeting attention-deficit/hyperactivity symptoms in elementary schoolchildren [texte imprimé] / Patricia VUIJK, Auteur ; Pol A.C. VAN LIER, Auteur ; Frank C. VERHULST, Auteur ; Anja C. HUIZINK, Auteur ; Alfons A.M. CRIJNEN, Auteur . - 2006 . - p.891–901. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 47-9 (September 2006) . - p.891–901 Mots-clés : Prenatal-smoking fetal-programming preventive-interventions ADHD-symptoms early-onset-experimentation-with-smoking Index. décimale : PER Périodiques Résumé : Background: Some evidence suggests that prenatal exposure to maternal smoking contributes to the etiology of Attention-Deficit/Hyperactivity Disorder (ADHD). The present study tested an intervention targeting disruptive behavior to establish whether exposure to maternal smoking during pregnancy predicts intervention failure in elementary schoolchildren. Methods: Five hundred and eight elementary schoolchildren were followed from ages 7 to 11. At ages 8 and 9, they were randomly assigned to a control condition, or to a two-year universal classroom-based intervention targeting disruptive behavior. Measures included maternal reports of smoking during pregnancy, teacher-rated symptoms of ADHD from ages 7 to 9 years, and children's self-report of experimentation with smoking at ages 10 and 11 years. Results: At age 7, prenatally exposed children had higher ADHD symptom scores. The intervention did not affect the course of their ADHD symptoms and the probability of early-onset experimentation with smoking. Among non-exposed children, the intervention positively affected the course of ADHD symptoms, and reduced the probability of early-onset experimentation with smoking. Conclusions: Prenatally exposed children are most prone to follow a path of high levels of ADHD symptoms and associated elevated risk for early-onset experimentation with smoking, which is unreceptive to a universal preventive intervention. In these children, the developmental course of ADHD symptoms seems to have been influenced by their prenatal exposure to maternal smoking. Future research should further explore whether prenatal smoking is a proxy measure that indexes another risk factor, or a causal factor for adverse developmental outcomes. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01647.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=775

Prenatal stress and models explaining risk for psychopathology revisited: Generic vulnerability and divergent pathways / Anja C. HUIZINK in Development and Psychopathology, 30-3 (August 2018)  

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Social Skills as Precursors of Cannabis Use in Young Adolescents: A Trails Study / Merel F.H. GRIFFITH-LENDERING in Journal of Clinical Child & Adolescent Psychology, 40-5 (September-October 2011)  

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The developmental course of anxiety symptoms during adolescence: the TRAILS study / Floor V.A. VAN OORT in Journal of Child Psychology and Psychiatry, 50-10 (October 2009)  

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