False memory for trauma-related Deese–Roediger–McDermott lists in adolescents and adults with histories of child sexual abuse / Gail S. GOODMAN in Development and Psychopathology, 23-2 (May 2011)  

Public ISBD [article]  inDevelopment and Psychopathology > 23-2 (May 2011) . - p.423-438 Titre : False memory for trauma-related Deese–Roediger–McDermott lists in adolescents and adults with histories of child sexual abuse Type de document : Texte imprimé et/ou numérique Auteurs : Gail S. GOODMAN, Auteur ; Christin M. OGLE, Auteur ; Stephanie D. BLOCK, Auteur ; Latonya S. HARRIS, Auteur ; Rakel P. LARSON, Auteur ; Else-Marie AUGUSTI, Auteur ; Young Il CHO, Auteur ; Jonathan BEBER, Auteur ; Susan G. TIMMER, Auteur ; Anthony J. URQUIZA, Auteur Année de publication : 2011 Article en page(s) : p.423-438 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The purpose of the present research was to examine Deese–Roediger–McDermott false memory for trauma-related and nontrauma-related lists in adolescents and adults with and without documented histories of child sexual abuse (CSA). Individual differences in psychopathology and adult attachment were also explored. Participants were administered free recall and recognition tests after hearing CSA, negative, neutral, and positive Deese–Roediger–McDermott lists. In free recall, CSA and negative lists produced the most false memory. In sharp contrast, for recognition, CSA lists enjoyed the highest d′ scores. CSA-group adolescents who evinced greater posttraumatic stress disorder (PTSD) symptoms had higher rates of false memory compared to (a) non-CSA group adolescents with higher PTSD symptom scores (free recall), and (b) CSA-group adolescents with lower PTSD symptom scores (recognition). Regression analyses revealed that individuals with higher PTSD scores and greater fearful-avoidant attachment tendencies showed less proficient memory monitoring for CSA lists. Implications for trauma and memory development and for translational research are discussed. En ligne : http://dx.doi.org/10.1017/S0954579411000150 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=121 [article] False memory for trauma-related Deese–Roediger–McDermott lists in adolescents and adults with histories of child sexual abuse [Texte imprimé et/ou numérique] / Gail S. GOODMAN, Auteur ; Christin M. OGLE, Auteur ; Stephanie D. BLOCK, Auteur ; Latonya S. HARRIS, Auteur ; Rakel P. LARSON, Auteur ; Else-Marie AUGUSTI, Auteur ; Young Il CHO, Auteur ; Jonathan BEBER, Auteur ; Susan G. TIMMER, Auteur ; Anthony J. URQUIZA, Auteur . - 2011 . - p.423-438. Langues : Anglais (eng) in Development and Psychopathology > 23-2 (May 2011) . - p.423-438 Index. décimale : PER Périodiques Résumé : The purpose of the present research was to examine Deese–Roediger–McDermott false memory for trauma-related and nontrauma-related lists in adolescents and adults with and without documented histories of child sexual abuse (CSA). Individual differences in psychopathology and adult attachment were also explored. Participants were administered free recall and recognition tests after hearing CSA, negative, neutral, and positive Deese–Roediger–McDermott lists. In free recall, CSA and negative lists produced the most false memory. In sharp contrast, for recognition, CSA lists enjoyed the highest d′ scores. CSA-group adolescents who evinced greater posttraumatic stress disorder (PTSD) symptoms had higher rates of false memory compared to (a) non-CSA group adolescents with higher PTSD symptom scores (free recall), and (b) CSA-group adolescents with lower PTSD symptom scores (recognition). Regression analyses revealed that individuals with higher PTSD scores and greater fearful-avoidant attachment tendencies showed less proficient memory monitoring for CSA lists. Implications for trauma and memory development and for translational research are discussed. En ligne : http://dx.doi.org/10.1017/S0954579411000150 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=121

Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation / Laurie A. CHASSIN in Development and Psychopathology, 24-3 (August 2012)  

Public ISBD [article]  inDevelopment and Psychopathology > 24-3 (August 2012) . - p.953-67 Titre : Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation Type de document : Texte imprimé et/ou numérique Auteurs : Laurie A. CHASSIN, Auteur ; Matthew R. LEE, Auteur ; Young Il CHO, Auteur ; Frances L. WANG, Auteur ; Arpana AGRAWAL, Auteur ; Kenneth J. SHER, Auteur ; Michael T. LYNSKEY, Auteur Année de publication : 2012 Article en page(s) : p.953-67 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This study examined the interplay between the influence of peers who promote alcohol use and μ-opioid receptor M1 (OPRM1) genetic variation in the intergenerational transmission of alcohol use disorder (AUD) symptoms while separating the “traitlike” components of AUD symptoms from their age-specific manifestations at three ages from emerging adulthood (17–23 years) to adulthood (29–40 years). The results for males were consistent with genetically influenced peer selection mechanisms as mediators of parent alcoholism effects. Male children of alcoholics were less likely to be carriers of the G allele in single nucleotide polymorphism A118G (rs1799971), and those who were homozygous for the A allele were more likely to affiliate with alcohol use promoting peers who increased the risk for AUD symptoms at all ages. There was evidence for women of an interaction between OPRM1 variation and peer affiliations but only at the earliest age band. Peer influences had stronger effects among women who were G-carriers. These results illustrate the complex ways in which the interplay between influences at multiple levels of analysis can underlie the intergenerational transmission of alcohol disorders as well as the importance of considering age and gender differences in these pathways. En ligne : http://dx.doi.org/10.1017/S0954579412000478 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178 [article] Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation [Texte imprimé et/ou numérique] / Laurie A. CHASSIN, Auteur ; Matthew R. LEE, Auteur ; Young Il CHO, Auteur ; Frances L. WANG, Auteur ; Arpana AGRAWAL, Auteur ; Kenneth J. SHER, Auteur ; Michael T. LYNSKEY, Auteur . - 2012 . - p.953-67. Langues : Anglais (eng) in Development and Psychopathology > 24-3 (August 2012) . - p.953-67 Index. décimale : PER Périodiques Résumé : This study examined the interplay between the influence of peers who promote alcohol use and μ-opioid receptor M1 (OPRM1) genetic variation in the intergenerational transmission of alcohol use disorder (AUD) symptoms while separating the “traitlike” components of AUD symptoms from their age-specific manifestations at three ages from emerging adulthood (17–23 years) to adulthood (29–40 years). The results for males were consistent with genetically influenced peer selection mechanisms as mediators of parent alcoholism effects. Male children of alcoholics were less likely to be carriers of the G allele in single nucleotide polymorphism A118G (rs1799971), and those who were homozygous for the A allele were more likely to affiliate with alcohol use promoting peers who increased the risk for AUD symptoms at all ages. There was evidence for women of an interaction between OPRM1 variation and peer affiliations but only at the earliest age band. Peer influences had stronger effects among women who were G-carriers. These results illustrate the complex ways in which the interplay between influences at multiple levels of analysis can underlie the intergenerational transmission of alcohol disorders as well as the importance of considering age and gender differences in these pathways. En ligne : http://dx.doi.org/10.1017/S0954579412000478 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178

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