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SHANK3 haploinsufficiency: a common but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders / Catalina BETANCUR in Molecular Autism, (June 2013)
[article]
Titre : SHANK3 haploinsufficiency: a common but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders Type de document : Texte imprimé et/ou numérique Auteurs : Catalina BETANCUR, Auteur ; Joseph D. BUXBAUM, Auteur Année de publication : 2013 Article en page(s) : 8 p. Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Autism spectrum disorders (ASD) are etiologically heterogeneous, with hundreds of rare, highly penetrant mutations and genomic imbalances involved, each contributing to a very small fraction of cases. As reviewed here, Phelan-McDermid syndrome, caused by deletion of 22q13.33 or SHANK3 mutations, is one of the more prevalent monogenic causes of ASD, explaining at least 0.5% of cases. In this issue of Molecular Autism, Soorya and colleagues evaluated 32 patients with SHANK3 haploinsufficiency using gold-standard diagnostic assessments and showed that 84% met criteria for ASD, including 75% meeting criteria for autism. This study and prior studies demonstrate that this syndrome is also one of the more penetrant causes of ASD. We note that SHANK3 haploinsufficiency remains underdiagnosed, although with the increasingly widespread use of chromosomal microarray analysis and targeted sequencing of SHANK3, the number of cases is bound to rise. En ligne : http://dx.doi.org/10.1186/2040-2392-4-17 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=202
in Molecular Autism > (June 2013) . - 8 p.[article] SHANK3 haploinsufficiency: a common but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders [Texte imprimé et/ou numérique] / Catalina BETANCUR, Auteur ; Joseph D. BUXBAUM, Auteur . - 2013 . - 8 p.
Langues : Anglais (eng)
in Molecular Autism > (June 2013) . - 8 p.
Index. décimale : PER Périodiques Résumé : Autism spectrum disorders (ASD) are etiologically heterogeneous, with hundreds of rare, highly penetrant mutations and genomic imbalances involved, each contributing to a very small fraction of cases. As reviewed here, Phelan-McDermid syndrome, caused by deletion of 22q13.33 or SHANK3 mutations, is one of the more prevalent monogenic causes of ASD, explaining at least 0.5% of cases. In this issue of Molecular Autism, Soorya and colleagues evaluated 32 patients with SHANK3 haploinsufficiency using gold-standard diagnostic assessments and showed that 84% met criteria for ASD, including 75% meeting criteria for autism. This study and prior studies demonstrate that this syndrome is also one of the more penetrant causes of ASD. We note that SHANK3 haploinsufficiency remains underdiagnosed, although with the increasingly widespread use of chromosomal microarray analysis and targeted sequencing of SHANK3, the number of cases is bound to rise. En ligne : http://dx.doi.org/10.1186/2040-2392-4-17 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=202 Shaping long-term primate development: Telomere length trajectory as an indicator of early maternal maltreatment and predictor of future physiologic regulation / Stacy S. DRURY in Development and Psychopathology, 29-5 (December 2017)
[article]
Titre : Shaping long-term primate development: Telomere length trajectory as an indicator of early maternal maltreatment and predictor of future physiologic regulation Type de document : Texte imprimé et/ou numérique Auteurs : Stacy S. DRURY, Auteur ; Brittany R. HOWELL, Auteur ; Christopher JONES, Auteur ; Kyle ESTEVES, Auteur ; Elyse MORIN, Auteur ; Reid SCHLESINGER, Auteur ; Jerrold S. MEYER, Auteur ; Kate BAKER, Auteur ; Mar M. SANCHEZ, Auteur Article en page(s) : p.1539-1551 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The molecular, neurobiological, and physical health impacts of child maltreatment are well established, yet mechanistic pathways remain inadequately defined. Telomere length (TL) decline is an emerging molecular indicator of stress exposure with definitive links to negative health outcomes in maltreated individuals. The multiple confounders endemic to human maltreatment research impede the identification of causal pathways. This study leverages a unique randomized, cross-foster, study design in a naturalistic translational nonhuman primate model of infant maltreatment. At birth, newborn macaques were randomly assigned to either a maltreating or a competent control mother, balancing for sex, biological mother parenting history, and social rank. Offspring TL was measured longitudinally across the first 6 months of life (infancy) from peripheral blood. Hair cortisol accumulation was also determined at 6, 12, and 18 months of age. TL decline was greater in animals randomized to maltreatment, but also interacted with biological mother group. Shorter TL at 6 months was associated with higher mean cortisol levels through 18 months (juvenile period) when controlling for relevant covariates. These results suggest that even under the equivalent social, nutritional, and environmental conditions feasible in naturalistic translational nonhuman primate models, early adverse caregiving results in lasting molecular scars that foreshadow elevated health risk and physiologic dysregulation. En ligne : http://dx.doi.org/10.1017/S0954579417001225 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=323
in Development and Psychopathology > 29-5 (December 2017) . - p.1539-1551[article] Shaping long-term primate development: Telomere length trajectory as an indicator of early maternal maltreatment and predictor of future physiologic regulation [Texte imprimé et/ou numérique] / Stacy S. DRURY, Auteur ; Brittany R. HOWELL, Auteur ; Christopher JONES, Auteur ; Kyle ESTEVES, Auteur ; Elyse MORIN, Auteur ; Reid SCHLESINGER, Auteur ; Jerrold S. MEYER, Auteur ; Kate BAKER, Auteur ; Mar M. SANCHEZ, Auteur . - p.1539-1551.
Langues : Anglais (eng)
in Development and Psychopathology > 29-5 (December 2017) . - p.1539-1551
Index. décimale : PER Périodiques Résumé : The molecular, neurobiological, and physical health impacts of child maltreatment are well established, yet mechanistic pathways remain inadequately defined. Telomere length (TL) decline is an emerging molecular indicator of stress exposure with definitive links to negative health outcomes in maltreated individuals. The multiple confounders endemic to human maltreatment research impede the identification of causal pathways. This study leverages a unique randomized, cross-foster, study design in a naturalistic translational nonhuman primate model of infant maltreatment. At birth, newborn macaques were randomly assigned to either a maltreating or a competent control mother, balancing for sex, biological mother parenting history, and social rank. Offspring TL was measured longitudinally across the first 6 months of life (infancy) from peripheral blood. Hair cortisol accumulation was also determined at 6, 12, and 18 months of age. TL decline was greater in animals randomized to maltreatment, but also interacted with biological mother group. Shorter TL at 6 months was associated with higher mean cortisol levels through 18 months (juvenile period) when controlling for relevant covariates. These results suggest that even under the equivalent social, nutritional, and environmental conditions feasible in naturalistic translational nonhuman primate models, early adverse caregiving results in lasting molecular scars that foreshadow elevated health risk and physiologic dysregulation. En ligne : http://dx.doi.org/10.1017/S0954579417001225 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=323 Shared and disorder-specific prefrontal abnormalities in boys with pure attention-deficit/hyperactivity disorder compared to boys with pure CD during interference inhibition and attention allocation / Katya RUBIA in Journal of Child Psychology and Psychiatry, 50-6 (June 2009)
[article]
Titre : Shared and disorder-specific prefrontal abnormalities in boys with pure attention-deficit/hyperactivity disorder compared to boys with pure CD during interference inhibition and attention allocation Type de document : Texte imprimé et/ou numérique Auteurs : Katya RUBIA, Auteur ; Michael BRAMMER, Auteur ; Stephen SCOTT, Auteur ; Anna B. SMITH, Auteur ; Rozmin HALARI, Auteur ; Majeed MOHAMMAD, Auteur Année de publication : 2009 Article en page(s) : p.669-678 Langues : Anglais (eng) Mots-clés : ADHD Conduct-disorder-(CD) fMRI interference-inhibition Simon-task oddball-task attention-allocation frontal-lobes Index. décimale : PER Périodiques Résumé : Background: Inhibitory and attention deficits have been suggested to be shared problems of disruptive behaviour disorders. Patients with attention deficit hyperactivity disorder (ADHD) and patients with conduct disorder (CD) show deficits in tasks of attention allocation and interference inhibition. However, functional magnetic resonance imaging (fMRI) of inhibitory and attention control has only been investigated in patients with ADHD, including comorbidity with CD, finding fronto-striatal and temporo-parietal dysfunction. This study investigates differences and commonalities in functional neural networks mediating interference inhibition and attention allocation between medication-naïve children and adolescents with pure CD and those with pure ADHD.
Methods: Event-related fMRI was used to compare brain activation of 13 boys with non-comorbid CD, 20 boys with non-comorbid ADHD and 20 healthy comparison boys during a Simon task that measures interference inhibition and controls for attention allocation, thus tapping into interference inhibition and selective attention networks.
Results: During interference inhibition, both patient groups shared reduced activation compared to controls in right superior temporal lobe and in predominantly right precuneus. During the oddball condition, both patient groups showed reduced activation compared to healthy control children in right medial prefrontal lobe. However, only ADHD patients showed a disorder-specific under-activation compared to the other two groups in an extensive activation cluster in left inferior prefrontal cortex.
Conclusions: This study shows shared dysfunction in both patients groups in right hemispheric temporal and parietal brain regions during interference inhibition and in right dorsolateral prefrontal cortex during attention allocation. Ventrolateral prefrontal dysfunction, however, was specific to ADHD and not observed in patients with CD in the context of attention allocation. The findings suggest that the typically reduced functional activation in patients with ADHD in ventrolateral prefrontal cortex may be specific to the disorder, at least when compared to patients with CD.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.02022.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=755
in Journal of Child Psychology and Psychiatry > 50-6 (June 2009) . - p.669-678[article] Shared and disorder-specific prefrontal abnormalities in boys with pure attention-deficit/hyperactivity disorder compared to boys with pure CD during interference inhibition and attention allocation [Texte imprimé et/ou numérique] / Katya RUBIA, Auteur ; Michael BRAMMER, Auteur ; Stephen SCOTT, Auteur ; Anna B. SMITH, Auteur ; Rozmin HALARI, Auteur ; Majeed MOHAMMAD, Auteur . - 2009 . - p.669-678.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 50-6 (June 2009) . - p.669-678
Mots-clés : ADHD Conduct-disorder-(CD) fMRI interference-inhibition Simon-task oddball-task attention-allocation frontal-lobes Index. décimale : PER Périodiques Résumé : Background: Inhibitory and attention deficits have been suggested to be shared problems of disruptive behaviour disorders. Patients with attention deficit hyperactivity disorder (ADHD) and patients with conduct disorder (CD) show deficits in tasks of attention allocation and interference inhibition. However, functional magnetic resonance imaging (fMRI) of inhibitory and attention control has only been investigated in patients with ADHD, including comorbidity with CD, finding fronto-striatal and temporo-parietal dysfunction. This study investigates differences and commonalities in functional neural networks mediating interference inhibition and attention allocation between medication-naïve children and adolescents with pure CD and those with pure ADHD.
Methods: Event-related fMRI was used to compare brain activation of 13 boys with non-comorbid CD, 20 boys with non-comorbid ADHD and 20 healthy comparison boys during a Simon task that measures interference inhibition and controls for attention allocation, thus tapping into interference inhibition and selective attention networks.
Results: During interference inhibition, both patient groups shared reduced activation compared to controls in right superior temporal lobe and in predominantly right precuneus. During the oddball condition, both patient groups showed reduced activation compared to healthy control children in right medial prefrontal lobe. However, only ADHD patients showed a disorder-specific under-activation compared to the other two groups in an extensive activation cluster in left inferior prefrontal cortex.
Conclusions: This study shows shared dysfunction in both patients groups in right hemispheric temporal and parietal brain regions during interference inhibition and in right dorsolateral prefrontal cortex during attention allocation. Ventrolateral prefrontal dysfunction, however, was specific to ADHD and not observed in patients with CD in the context of attention allocation. The findings suggest that the typically reduced functional activation in patients with ADHD in ventrolateral prefrontal cortex may be specific to the disorder, at least when compared to patients with CD.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.02022.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=755 Shared and distinct developmental pathways to ASD and ADHD phenotypes among infants at familial risk / Meghan MILLER in Development and Psychopathology, 32-4 (October 2020)
[article]
Titre : Shared and distinct developmental pathways to ASD and ADHD phenotypes among infants at familial risk Type de document : Texte imprimé et/ou numérique Auteurs : Meghan MILLER, Auteur ; Shane AUSTIN, Auteur ; Ana-Maria IOSIF, Auteur ; Leiana DE LA PAZ, Auteur ; Annie CHUANG, Auteur ; Burt HATCH, Auteur ; Sally OZONOFF, Auteur Article en page(s) : p.1323-1334 Langues : Anglais (eng) Mots-clés : Adhd autism early childhood infancy latent profile analysis Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) are believed to share partially overlapping causal mechanisms suggesting that early risk markers may also overlap. Using latent profile analysis (LPA) in a sample of infants enriched for ASD and ADHD, we first examined the number of distinct groups of 3-year-old children, based on ADHD and ASD symptomatology. To investigate early predictors of ASD and ADHD symptom profiles, we next examined differences in trajectories of infant behaviors among the LPA classes spanning general development, negative affect, attention, activity level, impulsivity, and social behavior. Participants included 166 infants at familial risk for ASD (n = 89), ADHD (n = 38), or low-risk for both (n = 39) evaluated at 12, 18, 24, and 36 months of age. A three-class solution was selected reflecting a Typically Developing (TD) class (low symptoms; n = 108), an ADHD class (high ADHD/low ASD symptoms; n = 39), and an ASD class (high ASD/ADHD symptoms; n = 19). Trajectories of infant behaviors were generally suggestive of a gradient pattern of differences, with the greatest impairment within the ASD class followed by the ADHD class. These findings indicate a mixture of overlapping and distinct early markers of preschool ASD- and ADHD-like profiles that can be difficult to disentangle early in life. En ligne : http://dx.doi.org/10.1017/s0954579420000735 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=433
in Development and Psychopathology > 32-4 (October 2020) . - p.1323-1334[article] Shared and distinct developmental pathways to ASD and ADHD phenotypes among infants at familial risk [Texte imprimé et/ou numérique] / Meghan MILLER, Auteur ; Shane AUSTIN, Auteur ; Ana-Maria IOSIF, Auteur ; Leiana DE LA PAZ, Auteur ; Annie CHUANG, Auteur ; Burt HATCH, Auteur ; Sally OZONOFF, Auteur . - p.1323-1334.
Langues : Anglais (eng)
in Development and Psychopathology > 32-4 (October 2020) . - p.1323-1334
Mots-clés : Adhd autism early childhood infancy latent profile analysis Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) are believed to share partially overlapping causal mechanisms suggesting that early risk markers may also overlap. Using latent profile analysis (LPA) in a sample of infants enriched for ASD and ADHD, we first examined the number of distinct groups of 3-year-old children, based on ADHD and ASD symptomatology. To investigate early predictors of ASD and ADHD symptom profiles, we next examined differences in trajectories of infant behaviors among the LPA classes spanning general development, negative affect, attention, activity level, impulsivity, and social behavior. Participants included 166 infants at familial risk for ASD (n = 89), ADHD (n = 38), or low-risk for both (n = 39) evaluated at 12, 18, 24, and 36 months of age. A three-class solution was selected reflecting a Typically Developing (TD) class (low symptoms; n = 108), an ADHD class (high ADHD/low ASD symptoms; n = 39), and an ASD class (high ASD/ADHD symptoms; n = 19). Trajectories of infant behaviors were generally suggestive of a gradient pattern of differences, with the greatest impairment within the ASD class followed by the ADHD class. These findings indicate a mixture of overlapping and distinct early markers of preschool ASD- and ADHD-like profiles that can be difficult to disentangle early in life. En ligne : http://dx.doi.org/10.1017/s0954579420000735 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=433 Shared and idiosyncratic cortical activation patterns in autism revealed under continuous real-life viewing conditions / Uri HASSON in Autism Research, 2-4 (August 2009)
[article]
Titre : Shared and idiosyncratic cortical activation patterns in autism revealed under continuous real-life viewing conditions Type de document : Texte imprimé et/ou numérique Auteurs : Uri HASSON, Auteur ; Nancy J. MINSHEW, Auteur ; Marlene BEHRMANN, Auteur ; Galia AVIDAN, Auteur ; Hagar GELBARD, Auteur ; Ignacio VALLINES, Auteur ; Michal HAREL, Auteur Année de publication : 2009 Article en page(s) : p.220-231 Langues : Anglais (eng) Mots-clés : autism inter-subject-correlation functional-magnetic-resonance-imaging-(fMRI) hyperconnectivity Index. décimale : PER Périodiques Résumé : Although widespread alterations in cortical structure have been documented in individuals with autism, the functional implications of these alterations remain to be determined. Here, we adopted a novel inter-subject correlation (inter-SC) and intra-subject correlation (intra-SC) technique to quantify the reliability of the spatio-temporal responses of functional MR activity in adults with autism during free-viewing of a popular audio-visual movie. Whereas these complex stimuli evoke highly reliable shared response time courses in typical individuals, cortical activity was more variable across individuals with autism (low inter-SC). Interestingly, when we measured the responses within an autistic individual across repeated presentations of the movie, we observed a unique, idiosyncratic response time course that was reliably replicated within each individual (high intra-SC). Encouragingly, after filtering out the idiosyncratic responses from each individual time course, we were able to uncover a more typical response profile, which resembles the shared responses seen in the typical subjects. These findings indicate that, under conditions approximating real-life situations, the neural activity of individuals with autism is characterized by individualistic responses that, although reliable within an autistic individual, are both highly variable across autistic individuals and different from the responses observed within the typical subjects. These idiosyncratic responses may underlie the atypical behaviors observed in autism. At the same time, we are encouraged by the presence of the more typical activation pattern lurking beneath these idiosyncratic fluctuations. Taken together, these findings may pave the way to future research aimed at characterizing the idiosyncratic response profiles, which, in turn, might contribute to a better understanding of the heterogeneity of the autism spectrum and its diagnosis. En ligne : http://dx.doi.org/10.1002/aur.89 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=937
in Autism Research > 2-4 (August 2009) . - p.220-231[article] Shared and idiosyncratic cortical activation patterns in autism revealed under continuous real-life viewing conditions [Texte imprimé et/ou numérique] / Uri HASSON, Auteur ; Nancy J. MINSHEW, Auteur ; Marlene BEHRMANN, Auteur ; Galia AVIDAN, Auteur ; Hagar GELBARD, Auteur ; Ignacio VALLINES, Auteur ; Michal HAREL, Auteur . - 2009 . - p.220-231.
Langues : Anglais (eng)
in Autism Research > 2-4 (August 2009) . - p.220-231
Mots-clés : autism inter-subject-correlation functional-magnetic-resonance-imaging-(fMRI) hyperconnectivity Index. décimale : PER Périodiques Résumé : Although widespread alterations in cortical structure have been documented in individuals with autism, the functional implications of these alterations remain to be determined. Here, we adopted a novel inter-subject correlation (inter-SC) and intra-subject correlation (intra-SC) technique to quantify the reliability of the spatio-temporal responses of functional MR activity in adults with autism during free-viewing of a popular audio-visual movie. Whereas these complex stimuli evoke highly reliable shared response time courses in typical individuals, cortical activity was more variable across individuals with autism (low inter-SC). Interestingly, when we measured the responses within an autistic individual across repeated presentations of the movie, we observed a unique, idiosyncratic response time course that was reliably replicated within each individual (high intra-SC). Encouragingly, after filtering out the idiosyncratic responses from each individual time course, we were able to uncover a more typical response profile, which resembles the shared responses seen in the typical subjects. These findings indicate that, under conditions approximating real-life situations, the neural activity of individuals with autism is characterized by individualistic responses that, although reliable within an autistic individual, are both highly variable across autistic individuals and different from the responses observed within the typical subjects. These idiosyncratic responses may underlie the atypical behaviors observed in autism. At the same time, we are encouraged by the presence of the more typical activation pattern lurking beneath these idiosyncratic fluctuations. Taken together, these findings may pave the way to future research aimed at characterizing the idiosyncratic response profiles, which, in turn, might contribute to a better understanding of the heterogeneity of the autism spectrum and its diagnosis. En ligne : http://dx.doi.org/10.1002/aur.89 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=937 Shared atypical default mode and salience network functional connectivity between autism and schizophrenia / Heng CHEN in Autism Research, 10-11 (November 2017)
PermalinkShared Book Reading Behaviors of Parents and Their Verbal Preschoolers on the Autism Spectrum / Marleen F. WESTERVELD in Journal of Autism and Developmental Disorders, 50-8 (August 2020)
PermalinkShared familial risk factors between attention-deficit/hyperactivity disorder and overweight/obesity – a population-based familial coaggregation study in Sweden / Qi CHEN in Journal of Child Psychology and Psychiatry, 58-6 (June 2017)
PermalinkShared familial risk factors between autism spectrum disorder and obesity - a register-based familial coaggregation cohort study / Richard AHLBERG in Journal of Child Psychology and Psychiatry, 63-8 (August 2022)
PermalinkShared familial transmission of autism spectrum and attention-deficit/hyperactivity disorders / Erica D. MUSSER in Journal of Child Psychology and Psychiatry, 55-7 (July 2014)
PermalinkShared Features or Co-occurrence? Evaluating Symptoms of Developmental Coordination Disorder in Children and Adolescents with Autism Spectrum Disorder / H. L. MILLER in Journal of Autism and Developmental Disorders, 51-10 (October 2021)
PermalinkShared genetic influences between dimensional ASD and ADHD symptoms during child and adolescent development / E. STERGIAKOULI in Molecular Autism, 8 (2017)
PermalinkShared genetic influences on ADHD symptoms and very low-frequency EEG activity: a twin study / Charlotte TYE in Journal of Child Psychology and Psychiatry, 53-6 (June 2012)
PermalinkShedding Light on a Pervasive Problem: A Review of Research on Bullying Experiences Among Children with Autism Spectrum Disorders / Jessica H. SCHROEDER in Journal of Autism and Developmental Disorders, 44-7 (July 2014)
PermalinkShift-&-Persist and discrimination predicting depression across the life course: An accelerated longitudinal design using MIDUSI-III / N. Keita CHRISTOPHE in Development and Psychopathology, 34-4 (October 2022)
PermalinkShifted phase of EEG cross-frequency coupling in individuals with Phelan-McDermid syndrome / M. G. MARISCAL in Molecular Autism, 12 (2021)
PermalinkShifting age of child eating disorder hospitalizations during the Covid-19 pandemic / Nathalie AUGER in Journal of Child Psychology and Psychiatry, 64-8 (August 2023)
PermalinkShifting Diagnostic Systems for Defining Intellectual Disability in Death Penalty Cases: Hall vs. Florida / Mina MUKHERJEE in Journal of Autism and Developmental Disorders, 45-7 (July 2015)
PermalinkShifting Preferences for Primate Faces in Neurotypical Infants and Infants Later Diagnosed With ASD / A. YAMASHIRO in Autism Research, 12-2 (February 2019)
PermalinkShifting the centre of gravity: Towards a truly global representation in autism research / Chung-Hsin CHIANG ; Michele VILLALOBOS ; Muideen BAKARE ; Rosa A. HOEKSTRA in Autism, 28-1 (January 2024)
PermalinkShifting visual attention to social and non-social stimuli in Autism Spectrum Disorders / Shuliang MO in Research in Autism Spectrum Disorders, 65 (September 2019)
PermalinkShifts in Behavioral Synchrony in Response to an Interaction Partner's Distress in Adolescents With and Without ASD / Ester ZADOK in Journal of Autism and Developmental Disorders, 52-10 (October 2022)
Permalink‘Shine bright like a diamond!’: is research on high?functioning ADHD at last entering the mainstream? / Klaus?Peter LESCH in Journal of Child Psychology and Psychiatry, 59-3 (March 2018)
PermalinkShining a Light on a Hidden Population: Social Functioning and Mental Health in Women Reporting Autistic Traits But Lacking Diagnosis / Hannah L. BELCHER in Journal of Autism and Developmental Disorders, 53-8 (August 2023)
PermalinkShort- and longer-term impacts of Child Friendly Space Interventions in Rwamwanja Refugee Settlement, Uganda / J. METZLER in Journal of Child Psychology and Psychiatry, 60-11 (November 2019)
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