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Auteur Dorothea L. FLORIS |
Documents disponibles écrits par cet auteur (6)
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Correction: Understanding the relationship between cerebellar structure and social abilities / Dorothea L. FLORIS ; Pierrick COUPÉ ; Edouard DUCHESNAY ; Angeline MIHAILOV ; Antoine GRIGIS ; Indrit BÈGUE ; Julie VICTOR ; Vincent FROUIN ; Marion LEBOYER ; Josselin HOUENOU ; Charles LAIDI in Molecular Autism, 14 (2023)
[article]
Titre : Correction: Understanding the relationship between cerebellar structure and social abilities Type de document : Texte imprimé et/ou numérique Auteurs : Dorothea L. FLORIS, Auteur ; Pierrick COUPÉ, Auteur ; Edouard DUCHESNAY, Auteur ; Angeline MIHAILOV, Auteur ; Antoine GRIGIS, Auteur ; Indrit BÈGUE, Auteur ; Julie VICTOR, Auteur ; Vincent FROUIN, Auteur ; Marion LEBOYER, Auteur ; Josselin HOUENOU, Auteur ; Charles LAIDI, Auteur Article en page(s) : 24 p. Langues : Anglais (eng) Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1186/s13229-023-00553-6 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513
in Molecular Autism > 14 (2023) . - 24 p.[article] Correction: Understanding the relationship between cerebellar structure and social abilities [Texte imprimé et/ou numérique] / Dorothea L. FLORIS, Auteur ; Pierrick COUPÉ, Auteur ; Edouard DUCHESNAY, Auteur ; Angeline MIHAILOV, Auteur ; Antoine GRIGIS, Auteur ; Indrit BÈGUE, Auteur ; Julie VICTOR, Auteur ; Vincent FROUIN, Auteur ; Marion LEBOYER, Auteur ; Josselin HOUENOU, Auteur ; Charles LAIDI, Auteur . - 24 p.
Langues : Anglais (eng)
in Molecular Autism > 14 (2023) . - 24 p.
Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1186/s13229-023-00553-6 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513 Linking functional and structural brain organisation with behaviour in autism: a multimodal EU-AIMS Longitudinal European Autism Project (LEAP) study / Alberto LLERA ; Ting MEI ; Koen HAAK ; Christina ISAKOGLOU ; Dorothea L. FLORIS ; Sarah DURSTON ; Carolin MOESSNANG ; Tobias BANASCHEWSKI ; Simon BARON-COHEN ; Eva LOTH ; Flavio DELL'ACQUA ; Tony CHARMAN ; Declan G. M. MURPHY ; Christine ECKER ; Jan K. BUITELAAR ; Christian F. BECKMANN in Molecular Autism, 14 (2023)
[article]
Titre : Linking functional and structural brain organisation with behaviour in autism: a multimodal EU-AIMS Longitudinal European Autism Project (LEAP) study Type de document : Texte imprimé et/ou numérique Auteurs : Alberto LLERA, Auteur ; Ting MEI, Auteur ; Koen HAAK, Auteur ; Christina ISAKOGLOU, Auteur ; Dorothea L. FLORIS, Auteur ; Sarah DURSTON, Auteur ; Carolin MOESSNANG, Auteur ; Tobias BANASCHEWSKI, Auteur ; Simon BARON-COHEN, Auteur ; Eva LOTH, Auteur ; Flavio DELL'ACQUA, Auteur ; Tony CHARMAN, Auteur ; Declan G. M. MURPHY, Auteur ; Christine ECKER, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur Article en page(s) : 32 p. Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Neuroimaging analyses of brain structure and function in autism have typically been conducted in isolation, missing the sensitivity gains of linking data across modalities. Here we focus on the integration of structural and functional organisational properties of brain regions. We aim to identify novel brain-organisation phenotypes of autism. We utilised multimodal MRI (T1-, diffusion-weighted and resting state functional), behavioural and clinical data from the EU AIMS Longitudinal European Autism Project (LEAP) from autistic (n=206) and non-autistic (n=196) participants. Of these, 97 had data from 2 timepoints resulting in a total scan number of 466. Grey matter density maps, probabilistic tractography connectivity matrices and connectopic maps were extracted from respective MRI modalities and were then integrated with Linked Independent Component Analysis. Linear mixed-effects models were used to evaluate the relationship between components and group while accounting for covariates and non-independence of participants with longitudinal data. Additional models were run to investigate associations with dimensional measures of behaviour. We identified one component that differed significantly between groups (coefficient=0.33, p(adj)=0.02). This was driven (99%) by variance of the right fusiform gyrus connectopic map 2. While there were multiple nominal (uncorrected p<0.05) associations with behavioural measures, none were significant following multiple comparison correction. Our analysis considered the relative contributions of both structural and functional brain phenotypes simultaneously, finding that functional phenotypes drive associations with autism. These findings expanded on previous unimodal studies by revealing the topographic organisation of functional connectivity patterns specific to autism and warrant further investigation. En ligne : http://dx.doi.org/10.1186/s13229-023-00564-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513
in Molecular Autism > 14 (2023) . - 32 p.[article] Linking functional and structural brain organisation with behaviour in autism: a multimodal EU-AIMS Longitudinal European Autism Project (LEAP) study [Texte imprimé et/ou numérique] / Alberto LLERA, Auteur ; Ting MEI, Auteur ; Koen HAAK, Auteur ; Christina ISAKOGLOU, Auteur ; Dorothea L. FLORIS, Auteur ; Sarah DURSTON, Auteur ; Carolin MOESSNANG, Auteur ; Tobias BANASCHEWSKI, Auteur ; Simon BARON-COHEN, Auteur ; Eva LOTH, Auteur ; Flavio DELL'ACQUA, Auteur ; Tony CHARMAN, Auteur ; Declan G. M. MURPHY, Auteur ; Christine ECKER, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur . - 32 p.
Langues : Anglais (eng)
in Molecular Autism > 14 (2023) . - 32 p.
Index. décimale : PER Périodiques Résumé : Neuroimaging analyses of brain structure and function in autism have typically been conducted in isolation, missing the sensitivity gains of linking data across modalities. Here we focus on the integration of structural and functional organisational properties of brain regions. We aim to identify novel brain-organisation phenotypes of autism. We utilised multimodal MRI (T1-, diffusion-weighted and resting state functional), behavioural and clinical data from the EU AIMS Longitudinal European Autism Project (LEAP) from autistic (n=206) and non-autistic (n=196) participants. Of these, 97 had data from 2 timepoints resulting in a total scan number of 466. Grey matter density maps, probabilistic tractography connectivity matrices and connectopic maps were extracted from respective MRI modalities and were then integrated with Linked Independent Component Analysis. Linear mixed-effects models were used to evaluate the relationship between components and group while accounting for covariates and non-independence of participants with longitudinal data. Additional models were run to investigate associations with dimensional measures of behaviour. We identified one component that differed significantly between groups (coefficient=0.33, p(adj)=0.02). This was driven (99%) by variance of the right fusiform gyrus connectopic map 2. While there were multiple nominal (uncorrected p<0.05) associations with behavioural measures, none were significant following multiple comparison correction. Our analysis considered the relative contributions of both structural and functional brain phenotypes simultaneously, finding that functional phenotypes drive associations with autism. These findings expanded on previous unimodal studies by revealing the topographic organisation of functional connectivity patterns specific to autism and warrant further investigation. En ligne : http://dx.doi.org/10.1186/s13229-023-00564-3 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513 Patterns of connectome variability in autism across five functional activation tasks: findings from the LEAP project / Tristan LOODEN in Molecular Autism, 13 (2022)
[article]
Titre : Patterns of connectome variability in autism across five functional activation tasks: findings from the LEAP project Type de document : Texte imprimé et/ou numérique Auteurs : Tristan LOODEN, Auteur ; Dorothea L. FLORIS, Auteur ; Alberto LLERA, Auteur ; Roselyne J. CHAUVIN, Auteur ; Tony CHARMAN, Auteur ; Tobias BANASCHEWSKI, Auteur ; Declan MURPHY, Auteur ; Andre F. MARQUAND, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur ; AIMS-2-TRIALS GROUP, Auteur Article en page(s) : 53 p. Langues : Anglais (eng) Mots-clés : Autism Canonical correlation analysis Functional connectivity Heterogeneity Normative modeling fMRI Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorder (autism) is a complex neurodevelopmental condition with pronounced behavioral, cognitive, and neural heterogeneities across individuals. Here, our goal was to characterize heterogeneity in autism by identifying patterns of neural diversity as reflected in BOLD fMRI in the way individuals with autism engage with a varied array of cognitive tasks. METHODS: All analyses were based on the EU-AIMS/AIMS-2-TRIALS multisite Longitudinal European Autism Project (LEAP) with participants with autism (n=282) and typically developing (TD) controls (n=221) between 6 and 30Â years of age. We employed a novel task potency approach which combines the unique aspects of both resting state fMRI and task-fMRI to quantify task-induced variations in the functional connectome. Normative modelling was used to map atypicality of features on an individual basis with respect to their distribution in neurotypical control participants. We applied robust out-of-sample canonical correlation analysis (CCA) to relate connectome data to behavioral data. RESULTS: Deviation from the normative ranges of global functional connectivity was greater for individuals with autism compared to TD in each fMRI task paradigm (all tasks p< 0.001). The similarity across individuals of the deviation pattern was significantly increased in autistic relative to TD individuals (p< 0.002). The CCA identified significant and robust brain-behavior covariation between functional connectivity atypicality and autism-related behavioral features. CONCLUSIONS: Individuals with autism engage with tasks in a globally atypical way, but the particular spatial pattern of this atypicality is nevertheless similar across tasks. Atypicalities in the tasks originate mostly from prefrontal cortex and default mode network regions, but also speech and auditory networks. We show how sophisticated modeling methods such as task potency and normative modeling can be used toward unravelling complex heterogeneous conditions like autism. En ligne : http://dx.doi.org/10.1186/s13229-022-00529-y Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=491
in Molecular Autism > 13 (2022) . - 53 p.[article] Patterns of connectome variability in autism across five functional activation tasks: findings from the LEAP project [Texte imprimé et/ou numérique] / Tristan LOODEN, Auteur ; Dorothea L. FLORIS, Auteur ; Alberto LLERA, Auteur ; Roselyne J. CHAUVIN, Auteur ; Tony CHARMAN, Auteur ; Tobias BANASCHEWSKI, Auteur ; Declan MURPHY, Auteur ; Andre F. MARQUAND, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur ; AIMS-2-TRIALS GROUP, Auteur . - 53 p.
Langues : Anglais (eng)
in Molecular Autism > 13 (2022) . - 53 p.
Mots-clés : Autism Canonical correlation analysis Functional connectivity Heterogeneity Normative modeling fMRI Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorder (autism) is a complex neurodevelopmental condition with pronounced behavioral, cognitive, and neural heterogeneities across individuals. Here, our goal was to characterize heterogeneity in autism by identifying patterns of neural diversity as reflected in BOLD fMRI in the way individuals with autism engage with a varied array of cognitive tasks. METHODS: All analyses were based on the EU-AIMS/AIMS-2-TRIALS multisite Longitudinal European Autism Project (LEAP) with participants with autism (n=282) and typically developing (TD) controls (n=221) between 6 and 30Â years of age. We employed a novel task potency approach which combines the unique aspects of both resting state fMRI and task-fMRI to quantify task-induced variations in the functional connectome. Normative modelling was used to map atypicality of features on an individual basis with respect to their distribution in neurotypical control participants. We applied robust out-of-sample canonical correlation analysis (CCA) to relate connectome data to behavioral data. RESULTS: Deviation from the normative ranges of global functional connectivity was greater for individuals with autism compared to TD in each fMRI task paradigm (all tasks p< 0.001). The similarity across individuals of the deviation pattern was significantly increased in autistic relative to TD individuals (p< 0.002). The CCA identified significant and robust brain-behavior covariation between functional connectivity atypicality and autism-related behavioral features. CONCLUSIONS: Individuals with autism engage with tasks in a globally atypical way, but the particular spatial pattern of this atypicality is nevertheless similar across tasks. Atypicalities in the tasks originate mostly from prefrontal cortex and default mode network regions, but also speech and auditory networks. We show how sophisticated modeling methods such as task potency and normative modeling can be used toward unravelling complex heterogeneous conditions like autism. En ligne : http://dx.doi.org/10.1186/s13229-022-00529-y Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=491 Psychological Correlates of Handedness and Corpus Callosum Asymmetry in Autism: The left Hemisphere Dysfunction Theory Revisited / Dorothea L. FLORIS in Journal of Autism and Developmental Disorders, 43-8 (August 2013)
[article]
Titre : Psychological Correlates of Handedness and Corpus Callosum Asymmetry in Autism: The left Hemisphere Dysfunction Theory Revisited Type de document : Texte imprimé et/ou numérique Auteurs : Dorothea L. FLORIS, Auteur ; Lindsay R. CHURA, Auteur ; Rosemary J. HOLT, Auteur ; John SUCKLING, Auteur ; Edward T. BULLMORE, Auteur ; Simon BARON-COHEN, Auteur ; Michael D. SPENCER, Auteur Article en page(s) : p.1758-1772 Langues : Anglais (eng) Mots-clés : Autism Corpus callosum Handedness Asymmetry Lateralization Broader autism phenotype Index. décimale : PER Périodiques Résumé : Rightward cerebral lateralization has been suggested to be involved in the neuropathology of autism spectrum conditions. We investigated functional and neuroanatomical asymmetry, in terms of handedness and corpus callosum measurements in male adolescents with autism, their unaffected siblings and controls, and their associations with executive dysfunction and symptom severity. Adolescents with autism did not differ from controls in functional asymmetry, but neuroanatomically showed the expected pattern of stronger rightward lateralization in the posterior and anterior midbody based on their hand-preference. Measures of symptom severity were related to rightward asymmetry in three subregions (splenium, posterior midbody and rostral body). We found the opposite pattern for the isthmus and rostrum with better cognitive and less severe clinical scores associated with rightward lateralization. En ligne : http://dx.doi.org/10.1007/s10803-012-1720-8 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=205
in Journal of Autism and Developmental Disorders > 43-8 (August 2013) . - p.1758-1772[article] Psychological Correlates of Handedness and Corpus Callosum Asymmetry in Autism: The left Hemisphere Dysfunction Theory Revisited [Texte imprimé et/ou numérique] / Dorothea L. FLORIS, Auteur ; Lindsay R. CHURA, Auteur ; Rosemary J. HOLT, Auteur ; John SUCKLING, Auteur ; Edward T. BULLMORE, Auteur ; Simon BARON-COHEN, Auteur ; Michael D. SPENCER, Auteur . - p.1758-1772.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 43-8 (August 2013) . - p.1758-1772
Mots-clés : Autism Corpus callosum Handedness Asymmetry Lateralization Broader autism phenotype Index. décimale : PER Périodiques Résumé : Rightward cerebral lateralization has been suggested to be involved in the neuropathology of autism spectrum conditions. We investigated functional and neuroanatomical asymmetry, in terms of handedness and corpus callosum measurements in male adolescents with autism, their unaffected siblings and controls, and their associations with executive dysfunction and symptom severity. Adolescents with autism did not differ from controls in functional asymmetry, but neuroanatomically showed the expected pattern of stronger rightward lateralization in the posterior and anterior midbody based on their hand-preference. Measures of symptom severity were related to rightward asymmetry in three subregions (splenium, posterior midbody and rostral body). We found the opposite pattern for the isthmus and rostrum with better cognitive and less severe clinical scores associated with rightward lateralization. En ligne : http://dx.doi.org/10.1007/s10803-012-1720-8 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=205 The neuroanatomical substrates of autism and ADHD and their link to putative genomic underpinnings / Caroline GURR ; Johanna LEYHAUSEN ; Hanna SEELEMEYER ; Anke BLETSCH ; Tim SCHAEFER ; Charlotte M. PRETZSCH ; Bethany OAKLEY ; Eva LOTH ; Dorothea L. FLORIS ; Jan K. BUITELAAR ; Christian F. BECKMANN ; Tobias BANASCHEWSKI ; Tony CHARMAN ; Emily J. H. JONES ; Julian TILLMANN ; Chris H CHATHAM ; Thomas BOURGERON ; EU-AIMS LEAP Group ; Declan G. M. MURPHY ; Christine ECKER in Molecular Autism, 14 (2023)
[article]
Titre : The neuroanatomical substrates of autism and ADHD and their link to putative genomic underpinnings Type de document : Texte imprimé et/ou numérique Auteurs : Caroline GURR, Auteur ; Johanna LEYHAUSEN, Auteur ; Hanna SEELEMEYER, Auteur ; Anke BLETSCH, Auteur ; Tim SCHAEFER, Auteur ; Charlotte M. PRETZSCH, Auteur ; Bethany OAKLEY, Auteur ; Eva LOTH, Auteur ; Dorothea L. FLORIS, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur ; Tobias BANASCHEWSKI, Auteur ; Tony CHARMAN, Auteur ; Emily J. H. JONES, Auteur ; Julian TILLMANN, Auteur ; Chris H CHATHAM, Auteur ; Thomas BOURGERON, Auteur ; EU-AIMS LEAP Group, Auteur ; Declan G. M. MURPHY, Auteur ; Christine ECKER, Auteur Article en page(s) : 36 p. Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorders (ASD) are neurodevelopmental conditions accompanied by differences in brain development. Neuroanatomical differences in autism are variable across individuals and likely underpin distinct clinical phenotypes. To parse heterogeneity, it is essential to establish how the neurobiology of ASD is modulated by differences associated with co-occurring conditions, such as attention-deficit/hyperactivity disorder (ADHD). This study aimed to (1) investigate between-group differences in autistic individuals with and without co-occurring ADHD, and to (2) link these variances to putative genomic underpinnings. METHODS: We examined differences in cortical thickness (CT) and surface area (SA) and their genomic associations in a sample of 533 individuals from the Longitudinal European Autism Project. Using a general linear model including main effects of autism and ADHD, and an ASD-by-ADHD interaction, we examined to which degree ADHD modulates the autism-related neuroanatomy. Further, leveraging the spatial gene expression data of the Allen Human Brain Atlas, we identified genes whose spatial expression patterns resemble our neuroimaging findings. RESULTS: In addition to significant main effects for ASD and ADHD in fronto-temporal, limbic, and occipital regions, we observed a significant ASD-by-ADHD interaction in the left precentral gyrus and the right frontal gyrus for measures of CT and SA, respectively. Moreover, individuals with ASD?+?ADHD differed in CT to those without. Both main effects and the interaction were enriched for ASD-but not for ADHD-related genes. LIMITATIONS: Although we employed a multicenter design to overcome single-site recruitment limitations, our sample size of N=25 individuals in the ADHD only group is relatively small compared to the other subgroups, which limits the generalizability of the results. Also, we assigned subjects into ADHD positive groupings according to the DSM-5 rating scale. While this is sufficient for obtaining a research diagnosis of ADHD, our approach did not take into account for how long the symptoms have been present, which is typically considered when assessing ADHD in the clinical setting. CONCLUSION: Thus, our findings suggest that the neuroanatomy of ASD is significantly modulated by ADHD, and that autistic individuals with co-occurring ADHD may have specific neuroanatomical underpinnings potentially mediated by atypical gene expression. En ligne : http://dx.doi.org/10.1186/s13229-023-00568-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513
in Molecular Autism > 14 (2023) . - 36 p.[article] The neuroanatomical substrates of autism and ADHD and their link to putative genomic underpinnings [Texte imprimé et/ou numérique] / Caroline GURR, Auteur ; Johanna LEYHAUSEN, Auteur ; Hanna SEELEMEYER, Auteur ; Anke BLETSCH, Auteur ; Tim SCHAEFER, Auteur ; Charlotte M. PRETZSCH, Auteur ; Bethany OAKLEY, Auteur ; Eva LOTH, Auteur ; Dorothea L. FLORIS, Auteur ; Jan K. BUITELAAR, Auteur ; Christian F. BECKMANN, Auteur ; Tobias BANASCHEWSKI, Auteur ; Tony CHARMAN, Auteur ; Emily J. H. JONES, Auteur ; Julian TILLMANN, Auteur ; Chris H CHATHAM, Auteur ; Thomas BOURGERON, Auteur ; EU-AIMS LEAP Group, Auteur ; Declan G. M. MURPHY, Auteur ; Christine ECKER, Auteur . - 36 p.
Langues : Anglais (eng)
in Molecular Autism > 14 (2023) . - 36 p.
Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorders (ASD) are neurodevelopmental conditions accompanied by differences in brain development. Neuroanatomical differences in autism are variable across individuals and likely underpin distinct clinical phenotypes. To parse heterogeneity, it is essential to establish how the neurobiology of ASD is modulated by differences associated with co-occurring conditions, such as attention-deficit/hyperactivity disorder (ADHD). This study aimed to (1) investigate between-group differences in autistic individuals with and without co-occurring ADHD, and to (2) link these variances to putative genomic underpinnings. METHODS: We examined differences in cortical thickness (CT) and surface area (SA) and their genomic associations in a sample of 533 individuals from the Longitudinal European Autism Project. Using a general linear model including main effects of autism and ADHD, and an ASD-by-ADHD interaction, we examined to which degree ADHD modulates the autism-related neuroanatomy. Further, leveraging the spatial gene expression data of the Allen Human Brain Atlas, we identified genes whose spatial expression patterns resemble our neuroimaging findings. RESULTS: In addition to significant main effects for ASD and ADHD in fronto-temporal, limbic, and occipital regions, we observed a significant ASD-by-ADHD interaction in the left precentral gyrus and the right frontal gyrus for measures of CT and SA, respectively. Moreover, individuals with ASD?+?ADHD differed in CT to those without. Both main effects and the interaction were enriched for ASD-but not for ADHD-related genes. LIMITATIONS: Although we employed a multicenter design to overcome single-site recruitment limitations, our sample size of N=25 individuals in the ADHD only group is relatively small compared to the other subgroups, which limits the generalizability of the results. Also, we assigned subjects into ADHD positive groupings according to the DSM-5 rating scale. While this is sufficient for obtaining a research diagnosis of ADHD, our approach did not take into account for how long the symptoms have been present, which is typically considered when assessing ADHD in the clinical setting. CONCLUSION: Thus, our findings suggest that the neuroanatomy of ASD is significantly modulated by ADHD, and that autistic individuals with co-occurring ADHD may have specific neuroanatomical underpinnings potentially mediated by atypical gene expression. En ligne : http://dx.doi.org/10.1186/s13229-023-00568-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=513 Understanding the relationship between cerebellar structure and social abilities / Dorothea L. FLORIS ; Pierrick COUPÉ ; Edouard DUCHESNAY ; Angeline MIHAILOV ; Antoine GRIGIS ; Indrit BÈGUE ; Julie VICTOR ; Vincent FROUIN ; Marion LEBOYER ; Josselin HOUENOU ; Charles LAIDI in Molecular Autism, 14 (2023)
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