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Détail de l'auteur
Auteur Jonathan L. WILLIAMS |
Documents disponibles écrits par cet auteur (2)
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Neurosonology of the infant and newborn / Jonathan L. WILLIAMS in Developmental Medicine & Child Neurology, 24-3 (June 1982)
[article]
Titre : Neurosonology of the infant and newborn Type de document : Texte imprimé et/ou numérique Auteurs : Jonathan L. WILLIAMS, Auteur Année de publication : 1982 Article en page(s) : p.390-393 Langues : Anglais (eng) Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=530
in Developmental Medicine & Child Neurology > 24-3 (June 1982) . - p.390-393[article] Neurosonology of the infant and newborn [Texte imprimé et/ou numérique] / Jonathan L. WILLIAMS, Auteur . - 1982 . - p.390-393.
Langues : Anglais (eng)
in Developmental Medicine & Child Neurology > 24-3 (June 1982) . - p.390-393
Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=530 Working toward a neurobiological account of ADHD: Commentary on Gail Tripp and Jeff Wickens, Dopamine transfer deficit / Jonathan L. WILLIAMS in Journal of Child Psychology and Psychiatry, 49-7 (July 2008)
[article]
Titre : Working toward a neurobiological account of ADHD: Commentary on Gail Tripp and Jeff Wickens, Dopamine transfer deficit Type de document : Texte imprimé et/ou numérique Auteurs : Jonathan L. WILLIAMS, Auteur Année de publication : 2008 Article en page(s) : p.705 - 711 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The dopamine transfer deficit model of attention deficit hyperactivity disorder (ADHD) is compared and contrasted with the existing dynamic developmental theory and the extended temporal difference (TD) model. The first two both identify learning deficits as a key problem in ADHD, but this mechanism would seem at least as likely to cause other neurodevelopmental disorders. Learning deficits also do not provide a natural account of ADHD performance worsening in longer trials, nor of stimulant action, nor of the high rate of test–retest inconsistency. The extended TD model is based on the quite different premise that there are very many underlying causes for the disorder, and computational simulation of it has supported the existence of two subtypes that involve no dopamine transfer deficit, as well as several subtypes that do. The three models have markedly different ways of accounting for key aspects of ADHD, including intra-individual variability, stimulant effects, and extinction deficits. The most important shortcoming of all the models is their limited treatment of state-regulation processes, which are very important in ADHD; this is partially addressed by the extended TD model's proposed dopamine appetite. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01921.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=540
in Journal of Child Psychology and Psychiatry > 49-7 (July 2008) . - p.705 - 711[article] Working toward a neurobiological account of ADHD: Commentary on Gail Tripp and Jeff Wickens, Dopamine transfer deficit [Texte imprimé et/ou numérique] / Jonathan L. WILLIAMS, Auteur . - 2008 . - p.705 - 711.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 49-7 (July 2008) . - p.705 - 711
Index. décimale : PER Périodiques Résumé : The dopamine transfer deficit model of attention deficit hyperactivity disorder (ADHD) is compared and contrasted with the existing dynamic developmental theory and the extended temporal difference (TD) model. The first two both identify learning deficits as a key problem in ADHD, but this mechanism would seem at least as likely to cause other neurodevelopmental disorders. Learning deficits also do not provide a natural account of ADHD performance worsening in longer trials, nor of stimulant action, nor of the high rate of test–retest inconsistency. The extended TD model is based on the quite different premise that there are very many underlying causes for the disorder, and computational simulation of it has supported the existence of two subtypes that involve no dopamine transfer deficit, as well as several subtypes that do. The three models have markedly different ways of accounting for key aspects of ADHD, including intra-individual variability, stimulant effects, and extinction deficits. The most important shortcoming of all the models is their limited treatment of state-regulation processes, which are very important in ADHD; this is partially addressed by the extended TD model's proposed dopamine appetite. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01921.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=540