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Auteur Jean Baptiste PINGAULT |
Documents disponibles écrits par cet auteur (1)
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Editorial: Does the polygenic revolution herald a watershed in the study of GE interplay in developmental psychopathology? Some considerations for the Special Issue reader / Edward D. BARKER in Journal of Child Psychology and Psychiatry, 63-10 (October 2022)
[article]
Titre : Editorial: Does the polygenic revolution herald a watershed in the study of GE interplay in developmental psychopathology? Some considerations for the Special Issue reader Type de document : Texte imprimé et/ou numérique Auteurs : Edward D. BARKER, Auteur ; Barbara MAUGHAN, Auteur ; Andrea G. ALLEGRINI, Auteur ; Jean Baptiste PINGAULT, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur Année de publication : 2022 Article en page(s) : p.1107-1110 Langues : Anglais (eng) Mots-clés : Child Humans Mental Disorders/genetics/psychology Psychopathology Receptor for Advanced Glycation End Products Risk Factors Index. décimale : PER Périodiques Résumé : The primary goal motivating the scientific field of Developmental Psychopathology is to discover why some individuals develop mental health and neuro-developmental difficulties while others do not. This is not simply a 'blue skies' preoccupation: the underlying hope, of course, is to translate such discoveries to the benefit of individuals, families and communities, reducing poor outcomes for those at risk and - in the best case scenario - ensuring that they thrive. A core tenet of the bio-psycho-social framework within which this field of enquiry operates is that children's difficulties are determined by the interplay of predisposing genetic risk and resilience factors and the environments and experiences to which individuals are exposed. From this perspective, understanding gene-environment (GE) interplay is a necessary condition for explaining and, as importantly predicting, why one individual is at risk while another is not. If we believe this, then the risk calculators designed to show who will and will not get a particular disorder - all the rage at the moment - are doomed to fail until they can go beyond modelling the main effects of genes and environments, and reliably estimate GE processes too. Despite significant progress, we remain a considerable way off cracking this problem. En ligne : http://dx.doi.org/10.1111/jcpp.13692 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=486
in Journal of Child Psychology and Psychiatry > 63-10 (October 2022) . - p.1107-1110[article] Editorial: Does the polygenic revolution herald a watershed in the study of GE interplay in developmental psychopathology? Some considerations for the Special Issue reader [Texte imprimé et/ou numérique] / Edward D. BARKER, Auteur ; Barbara MAUGHAN, Auteur ; Andrea G. ALLEGRINI, Auteur ; Jean Baptiste PINGAULT, Auteur ; Edmund J. S. SONUGA-BARKE, Auteur . - 2022 . - p.1107-1110.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 63-10 (October 2022) . - p.1107-1110
Mots-clés : Child Humans Mental Disorders/genetics/psychology Psychopathology Receptor for Advanced Glycation End Products Risk Factors Index. décimale : PER Périodiques Résumé : The primary goal motivating the scientific field of Developmental Psychopathology is to discover why some individuals develop mental health and neuro-developmental difficulties while others do not. This is not simply a 'blue skies' preoccupation: the underlying hope, of course, is to translate such discoveries to the benefit of individuals, families and communities, reducing poor outcomes for those at risk and - in the best case scenario - ensuring that they thrive. A core tenet of the bio-psycho-social framework within which this field of enquiry operates is that children's difficulties are determined by the interplay of predisposing genetic risk and resilience factors and the environments and experiences to which individuals are exposed. From this perspective, understanding gene-environment (GE) interplay is a necessary condition for explaining and, as importantly predicting, why one individual is at risk while another is not. If we believe this, then the risk calculators designed to show who will and will not get a particular disorder - all the rage at the moment - are doomed to fail until they can go beyond modelling the main effects of genes and environments, and reliably estimate GE processes too. Despite significant progress, we remain a considerable way off cracking this problem. En ligne : http://dx.doi.org/10.1111/jcpp.13692 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=486