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Interactive effects of early and recent exposure to stressful contexts on cortisol reactivity in middle childhood / Sara R. JAFFEE in Journal of Child Psychology and Psychiatry, 56-2 (February 2015)
[article]
Titre : Interactive effects of early and recent exposure to stressful contexts on cortisol reactivity in middle childhood Type de document : Texte imprimé et/ou numérique Auteurs : Sara R. JAFFEE, Auteur ; Tara MCFARQUHAR, Auteur ; Suzanne E. STEVENS, Auteur ; Isabelle OUELLET-MORIN, Auteur ; Edward MELHUISH, Auteur ; Jay BELSKY, Auteur Article en page(s) : p.138-146 Langues : Anglais (eng) Mots-clés : Cortisol reactivity stress parenting internalizing externalizing Index. décimale : PER Périodiques Résumé : Background Given mixed findings as to whether stressful experiences and relationships are associated with increases or decreases in children's cortisol reactivity, we tested whether a child's developmental history of risk exposure explained variation in cortisol reactivity to an experimentally induced task. We also tested whether the relationship between cortisol reactivity and children's internalizing and externalizing problems varied as a function of their developmental history of stressful experiences and relationships. Method Participants included 400 children (M = 9.99 years, SD = 0.74 years) from the Children's Experiences and Development Study. Early risk exposure was measured by children's experiences of harsh, nonresponsive parenting at 3 years. Recent risk exposure was measured by children's exposure to traumatic events in the past year. Children's cortisol reactivity was measured in response to a social provocation task and parents and teachers described children's internalizing and externalizing problems. Results The effect of recent exposure to traumatic events was partially dependent upon a child's early experiences of harsh, nonresponsive parenting: the more traumatic events children had recently experienced, the greater their cortisol reactivity if they had experienced lower (but not higher) levels of harsh, nonresponsive parenting at age 3. The lowest levels of cortisol reactivity were observed among children who had experienced the most traumatic events in the past year and higher (vs. lower) levels of harsh, nonresponsive parenting in early childhood. Among youth who experienced harsh, nonresponsive parent–child relationships in early childhood and later traumatic events, lower levels of cortisol reactivity were associated with higher levels of internalizing and externalizing problems. Conclusions Hypothalamic–pituitary–adrenal (HPA) axis reactivity to psychological stressors and the relationship between HPA axis reactivity and children's internalizing and externalizing problems vary as a function of a child's developmental history of exposure to stressful relationships and experiences. En ligne : http://dx.doi.org/10.1111/jcpp.12287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.138-146[article] Interactive effects of early and recent exposure to stressful contexts on cortisol reactivity in middle childhood [Texte imprimé et/ou numérique] / Sara R. JAFFEE, Auteur ; Tara MCFARQUHAR, Auteur ; Suzanne E. STEVENS, Auteur ; Isabelle OUELLET-MORIN, Auteur ; Edward MELHUISH, Auteur ; Jay BELSKY, Auteur . - p.138-146.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.138-146
Mots-clés : Cortisol reactivity stress parenting internalizing externalizing Index. décimale : PER Périodiques Résumé : Background Given mixed findings as to whether stressful experiences and relationships are associated with increases or decreases in children's cortisol reactivity, we tested whether a child's developmental history of risk exposure explained variation in cortisol reactivity to an experimentally induced task. We also tested whether the relationship between cortisol reactivity and children's internalizing and externalizing problems varied as a function of their developmental history of stressful experiences and relationships. Method Participants included 400 children (M = 9.99 years, SD = 0.74 years) from the Children's Experiences and Development Study. Early risk exposure was measured by children's experiences of harsh, nonresponsive parenting at 3 years. Recent risk exposure was measured by children's exposure to traumatic events in the past year. Children's cortisol reactivity was measured in response to a social provocation task and parents and teachers described children's internalizing and externalizing problems. Results The effect of recent exposure to traumatic events was partially dependent upon a child's early experiences of harsh, nonresponsive parenting: the more traumatic events children had recently experienced, the greater their cortisol reactivity if they had experienced lower (but not higher) levels of harsh, nonresponsive parenting at age 3. The lowest levels of cortisol reactivity were observed among children who had experienced the most traumatic events in the past year and higher (vs. lower) levels of harsh, nonresponsive parenting in early childhood. Among youth who experienced harsh, nonresponsive parent–child relationships in early childhood and later traumatic events, lower levels of cortisol reactivity were associated with higher levels of internalizing and externalizing problems. Conclusions Hypothalamic–pituitary–adrenal (HPA) axis reactivity to psychological stressors and the relationship between HPA axis reactivity and children's internalizing and externalizing problems vary as a function of a child's developmental history of exposure to stressful relationships and experiences. En ligne : http://dx.doi.org/10.1111/jcpp.12287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259 Life stress and cortisol reactivity: An exploratory analysis of the effects of stress exposure across life on HPA-axis functioning / Ethan S. YOUNG in Development and Psychopathology, 33-1 (February 2021)
[article]
Titre : Life stress and cortisol reactivity: An exploratory analysis of the effects of stress exposure across life on HPA-axis functioning Type de document : Texte imprimé et/ou numérique Auteurs : Ethan S. YOUNG, Auteur ; Jenalee R. DOOM, Auteur ; Allison K. FARRELL, Auteur ; Elizabeth A. CARLSON, Auteur ; Michelle M. ENGLUND, Auteur ; Gregory E. MILLER, Auteur ; Megan R. GUNNAR, Auteur ; Glenn I. ROISMAN, Auteur ; Jeffry A. SIMPSON, Auteur Article en page(s) : p.301-312 Langues : Anglais (eng) Mots-clés : Trier Social Stress Test cortisol reactivity cumulative stress development life stress Index. décimale : PER Périodiques Résumé : Stressful experiences affect biological stress systems, such as the hypothalamic-pituitary-adrenal (HPA) axis. Life stress can potentially alter regulation of the HPA axis and has been associated with poorer physical and mental health. Little, however, is known about the relative influence of stressors that are encountered at different developmental periods on acute stress reactions in adulthood. In this study, we explored three models of the influence of stress exposure on cortisol reactivity to a modified version of the Trier Social Stress Test (TSST) by leveraging 37 years of longitudinal data in a high-risk birth cohort (N = 112). The cumulative stress model suggests that accumulated stress across the lifespan leads to dysregulated reactivity, whereas the biological embedding model implicates early childhood as a critical period. The sensitization model assumes that dysregulation should only occur when stress is high in both early childhood and concurrently. All of the models predicted altered reactivity, but do not anticipate its exact form. We found support for both cumulative and biological embedding effects. However, when pitted against each other, early life stress predicted more blunted cortisol responses at age 37 over and above cumulative life stress. Additional analyses revealed that stress exposure in middle childhood also predicted more blunted cortisol reactivity. En ligne : http://dx.doi.org/10.1017/s0954579419001779 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=442
in Development and Psychopathology > 33-1 (February 2021) . - p.301-312[article] Life stress and cortisol reactivity: An exploratory analysis of the effects of stress exposure across life on HPA-axis functioning [Texte imprimé et/ou numérique] / Ethan S. YOUNG, Auteur ; Jenalee R. DOOM, Auteur ; Allison K. FARRELL, Auteur ; Elizabeth A. CARLSON, Auteur ; Michelle M. ENGLUND, Auteur ; Gregory E. MILLER, Auteur ; Megan R. GUNNAR, Auteur ; Glenn I. ROISMAN, Auteur ; Jeffry A. SIMPSON, Auteur . - p.301-312.
Langues : Anglais (eng)
in Development and Psychopathology > 33-1 (February 2021) . - p.301-312
Mots-clés : Trier Social Stress Test cortisol reactivity cumulative stress development life stress Index. décimale : PER Périodiques Résumé : Stressful experiences affect biological stress systems, such as the hypothalamic-pituitary-adrenal (HPA) axis. Life stress can potentially alter regulation of the HPA axis and has been associated with poorer physical and mental health. Little, however, is known about the relative influence of stressors that are encountered at different developmental periods on acute stress reactions in adulthood. In this study, we explored three models of the influence of stress exposure on cortisol reactivity to a modified version of the Trier Social Stress Test (TSST) by leveraging 37 years of longitudinal data in a high-risk birth cohort (N = 112). The cumulative stress model suggests that accumulated stress across the lifespan leads to dysregulated reactivity, whereas the biological embedding model implicates early childhood as a critical period. The sensitization model assumes that dysregulation should only occur when stress is high in both early childhood and concurrently. All of the models predicted altered reactivity, but do not anticipate its exact form. We found support for both cumulative and biological embedding effects. However, when pitted against each other, early life stress predicted more blunted cortisol responses at age 37 over and above cumulative life stress. Additional analyses revealed that stress exposure in middle childhood also predicted more blunted cortisol reactivity. En ligne : http://dx.doi.org/10.1017/s0954579419001779 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=442