The development of psychopathy / James R. BLAIR in Journal of Child Psychology and Psychiatry, 47-3/4 (March/April 2006)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 47-3/4 (March/April 2006) . - p.262–276 Titre : The development of psychopathy Type de document : Texte imprimé et/ou numérique Auteurs : James R. BLAIR, Auteur ; Karina S. PESCHARDT, Auteur ; Derek G.V. MITCHELL, Auteur ; Daniel Samuel PINE, Auteur Année de publication : 2006 Article en page(s) : p.262–276 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The current review focuses on the construct of psychopathy, conceptualized as a clinical entity that is fundamentally distinct from a heterogeneous collection of syndromes encompassed by the term 'conduct disorder'. We will provide an account of the development of psychopathy at multiple levels: ultimate causal (the genetic or social primary cause), molecular, neural, cognitive and behavioral. The following main claims will be made: (1) that there is a stronger genetic as opposed to social ultimate cause to this disorder. The types of social causes proposed (e.g., childhood sexual/physical abuse) should elevate emotional responsiveness, not lead to the specific form of reduced responsiveness seen in psychopathy; (2) The genetic influence leads to the emotional dysfunction that is the core of psychopathy; (3) The genetic influence at the molecular level remains unknown. However, it appears to impact the functional integrity of the amygdala and orbital/ventrolateral frontal cortex (and possibly additional systems); (4) Disruption within these two neural systems leads to impairment in the ability to form stimulus–reinforcement associations and to alter stimulus–response associations as a function of contingency change. These impairments disrupt the impact of standard socialization techniques and increase the risk for frustration-induced reactive aggression respectively. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01596.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=721 [article] The development of psychopathy [Texte imprimé et/ou numérique] / James R. BLAIR, Auteur ; Karina S. PESCHARDT, Auteur ; Derek G.V. MITCHELL, Auteur ; Daniel Samuel PINE, Auteur . - 2006 . - p.262–276. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 47-3/4 (March/April 2006) . - p.262–276 Index. décimale : PER Périodiques Résumé : The current review focuses on the construct of psychopathy, conceptualized as a clinical entity that is fundamentally distinct from a heterogeneous collection of syndromes encompassed by the term 'conduct disorder'. We will provide an account of the development of psychopathy at multiple levels: ultimate causal (the genetic or social primary cause), molecular, neural, cognitive and behavioral. The following main claims will be made: (1) that there is a stronger genetic as opposed to social ultimate cause to this disorder. The types of social causes proposed (e.g., childhood sexual/physical abuse) should elevate emotional responsiveness, not lead to the specific form of reduced responsiveness seen in psychopathy; (2) The genetic influence leads to the emotional dysfunction that is the core of psychopathy; (3) The genetic influence at the molecular level remains unknown. However, it appears to impact the functional integrity of the amygdala and orbital/ventrolateral frontal cortex (and possibly additional systems); (4) Disruption within these two neural systems leads to impairment in the ability to form stimulus–reinforcement associations and to alter stimulus–response associations as a function of contingency change. These impairments disrupt the impact of standard socialization techniques and increase the risk for frustration-induced reactive aggression respectively. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01596.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=721

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