Attention difficulties and mood-related ruminative response style in adolescents with unipolar depression / Paul O. WILKINSON in Journal of Child Psychology and Psychiatry, 47-12 (December 2006)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 47-12 (December 2006) . - p.1284–1291 Titre : Attention difficulties and mood-related ruminative response style in adolescents with unipolar depression Type de document : Texte imprimé et/ou numérique Auteurs : Paul O. WILKINSON, Auteur ; Ian M. GOODYER, Auteur Année de publication : 2007 Article en page(s) : p.1284–1291 Langues : Anglais (eng) Mots-clés : Adolescence depression executive-function neuropsychology rumination Index. décimale : PER Périodiques Résumé : Background: Depressed adults may show impairment in switching attention from one task to another. Rumination on negative thoughts is associated with the onset and persistence of depressive episodes. It is unclear if such mood-related ruminations are specifically associated with slowed ability in switching attention from one task to another. Method: Adolescents with current unipolar depression (n = 40) were compared with age and sex-matched controls (n = 38) on tests of attention and general cognitive abilities and completed the mood-related ruminative response style questionnaire. Results: Compared to controls, depressed participants were significantly slower at switching attention. There was no association between attentional switching and mood-related ruminations and both processes contributed to the likelihood of being depressed. Findings were not accounted for by slowness in reading and speaking, the effects of antidepressants nor being more careful on tasks. Conclusions: Depressed adolescents demonstrate dual deficits in mood-related ruminative thinking and attention. Further studies are needed to investigate whether higher ruminations are associated with attention impairments in emotionally-valent attentional switching tasks, and whether both processes precede the emergence of clinical disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01660.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=818 [article] Attention difficulties and mood-related ruminative response style in adolescents with unipolar depression [Texte imprimé et/ou numérique] / Paul O. WILKINSON, Auteur ; Ian M. GOODYER, Auteur . - 2007 . - p.1284–1291. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 47-12 (December 2006) . - p.1284–1291 Mots-clés : Adolescence depression executive-function neuropsychology rumination Index. décimale : PER Périodiques Résumé : Background: Depressed adults may show impairment in switching attention from one task to another. Rumination on negative thoughts is associated with the onset and persistence of depressive episodes. It is unclear if such mood-related ruminations are specifically associated with slowed ability in switching attention from one task to another. Method: Adolescents with current unipolar depression (n = 40) were compared with age and sex-matched controls (n = 38) on tests of attention and general cognitive abilities and completed the mood-related ruminative response style questionnaire. Results: Compared to controls, depressed participants were significantly slower at switching attention. There was no association between attentional switching and mood-related ruminations and both processes contributed to the likelihood of being depressed. Findings were not accounted for by slowness in reading and speaking, the effects of antidepressants nor being more careful on tasks. Conclusions: Depressed adolescents demonstrate dual deficits in mood-related ruminative thinking and attention. Further studies are needed to investigate whether higher ruminations are associated with attention impairments in emotionally-valent attentional switching tasks, and whether both processes precede the emergence of clinical disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01660.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=818

Childhood adversity and allostatic overload of the hypothalamic–pituitary–adrenal axis: A vulnerability model for depressive disorders / Paul O. WILKINSON in Development and Psychopathology, 23-4 (November 2011)  

Public ISBD [article]  inDevelopment and Psychopathology > 23-4 (November 2011) . - p.1017-1037 Titre : Childhood adversity and allostatic overload of the hypothalamic–pituitary–adrenal axis: A vulnerability model for depressive disorders Type de document : Texte imprimé et/ou numérique Auteurs : Paul O. WILKINSON, Auteur ; Ian M. GOODYER, Auteur Année de publication : 2011 Article en page(s) : p.1017-1037 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Childhood adversity is associated with increased risk for onset of depressive episodes. This review will present evidence that allostatic overload of the hypothalamic–pituitary–adrenal axis (HPAA) partially mediates this association. The HPAA is the physiological system that regulates levels of the stress hormone cortisol. First, data from animals and humans has shown that early environmental adversity is associated with long-term dysregulation of the HPAA. This may occur due to permanent epigenetic modification of the glucocorticoid receptor. Second, data from humans has demonstrated that HPAA dysregulation is associated with increased risk of future depression onset in healthy individuals, and pharmacological correction of HPAA dysregulation reduces depressive symptoms. HPAA dysregulation may result in corticoid-mediated abnormalities in neurogenesis in early life and/or neurotoxicity on neural systems that subserve emotion and cognition. En ligne : http://dx.doi.org/10.1017/S0954579411000472 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=146 [article] Childhood adversity and allostatic overload of the hypothalamic–pituitary–adrenal axis: A vulnerability model for depressive disorders [Texte imprimé et/ou numérique] / Paul O. WILKINSON, Auteur ; Ian M. GOODYER, Auteur . - 2011 . - p.1017-1037. Langues : Anglais (eng) in Development and Psychopathology > 23-4 (November 2011) . - p.1017-1037 Index. décimale : PER Périodiques Résumé : Childhood adversity is associated with increased risk for onset of depressive episodes. This review will present evidence that allostatic overload of the hypothalamic–pituitary–adrenal axis (HPAA) partially mediates this association. The HPAA is the physiological system that regulates levels of the stress hormone cortisol. First, data from animals and humans has shown that early environmental adversity is associated with long-term dysregulation of the HPAA. This may occur due to permanent epigenetic modification of the glucocorticoid receptor. Second, data from humans has demonstrated that HPAA dysregulation is associated with increased risk of future depression onset in healthy individuals, and pharmacological correction of HPAA dysregulation reduces depressive symptoms. HPAA dysregulation may result in corticoid-mediated abnormalities in neurogenesis in early life and/or neurotoxicity on neural systems that subserve emotion and cognition. En ligne : http://dx.doi.org/10.1017/S0954579411000472 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=146

The role of gene–environment correlations and interactions in middle childhood depressive symptoms / Paul O. WILKINSON in Development and Psychopathology, 25-1 (February 2013)  

Public ISBD [article]  inDevelopment and Psychopathology > 25-1 (February 2013) . - p.93-104 Titre : The role of gene–environment correlations and interactions in middle childhood depressive symptoms Type de document : Texte imprimé et/ou numérique Auteurs : Paul O. WILKINSON, Auteur ; Maciej TRZASKOWSKI, Auteur ; Claire Margaret Alison HAWORTH, Auteur ; Thalia C. ELEY, Auteur Article en page(s) : p.93-104 Index. décimale : PER Périodiques Résumé : Depression is known to be associated with a wide array of environmental factors. Such associations are due at least in part to genetic influences on both. This issue has been little explored with preadolescent children. Measures of family chaos and parenting style at age 9 and child depressive symptoms at age 12 were completed by 3,258 twin pairs from the Twins Early Development Study and their parents. Quantitative genetic modeling was used to explore common and unique genetic and environmental influences on both family environment and later depressive symptoms. Depressive symptoms at age 12 were significantly heritable. Moderate genetic effects influenced parenting style and family chaos at the age of 9, indicating gene–environment correlation. There were significant genetic correlations between family environment and depressive symptoms. There was some evidence of a Gene × Environment interaction, with stronger genetic effects on depressive symptoms for children with more suboptimal family environment. There was an Environment × Environment interaction, with effects of nonshared environment on depressive symptoms stronger for twins with more adverse parenting experiences. There is some evidence for gene–environment correlation between aspects of family environment in middle childhood and subsequent depressive symptoms. This suggests that one of the mechanisms by which genes lead to depressive symptoms may be by themselves influencing depressogenic environments. En ligne : http://dx.doi.org/10.1017/S0954579412000922 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=190 [article] The role of gene–environment correlations and interactions in middle childhood depressive symptoms [Texte imprimé et/ou numérique] / Paul O. WILKINSON, Auteur ; Maciej TRZASKOWSKI, Auteur ; Claire Margaret Alison HAWORTH, Auteur ; Thalia C. ELEY, Auteur . - p.93-104. in Development and Psychopathology > 25-1 (February 2013) . - p.93-104 Index. décimale : PER Périodiques Résumé : Depression is known to be associated with a wide array of environmental factors. Such associations are due at least in part to genetic influences on both. This issue has been little explored with preadolescent children. Measures of family chaos and parenting style at age 9 and child depressive symptoms at age 12 were completed by 3,258 twin pairs from the Twins Early Development Study and their parents. Quantitative genetic modeling was used to explore common and unique genetic and environmental influences on both family environment and later depressive symptoms. Depressive symptoms at age 12 were significantly heritable. Moderate genetic effects influenced parenting style and family chaos at the age of 9, indicating gene–environment correlation. There were significant genetic correlations between family environment and depressive symptoms. There was some evidence of a Gene × Environment interaction, with stronger genetic effects on depressive symptoms for children with more suboptimal family environment. There was an Environment × Environment interaction, with effects of nonshared environment on depressive symptoms stronger for twins with more adverse parenting experiences. There is some evidence for gene–environment correlation between aspects of family environment in middle childhood and subsequent depressive symptoms. This suggests that one of the mechanisms by which genes lead to depressive symptoms may be by themselves influencing depressogenic environments. En ligne : http://dx.doi.org/10.1017/S0954579412000922 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=190

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