Familial and temperamental predictors of resilience in children at risk for conduct disorder and depression / Katherine E. SHANNON in Development and Psychopathology, 19-3 (Summer 2007)  

Public ISBD [article]  inDevelopment and Psychopathology > 19-3 (Summer 2007) . - p.701-727 Titre : Familial and temperamental predictors of resilience in children at risk for conduct disorder and depression Type de document : Texte imprimé et/ou numérique Auteurs : Katherine E. SHANNON, Auteur ; Theodore P. BEAUCHAINE, Auteur ; Sharon L. BRENNER, Auteur ; Emily NEUHAUS, Auteur ; Lisa GATZKE-KOPP, Auteur Année de publication : 2007 Article en page(s) : p.701-727 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : In this study, we evaluated predictors of resilience among 8- to 12-year-old children recruited from primarily low socioeconomic status neighborhoods, 117 of whom suffered from clinical levels of conduct problems and/or depression, and 63 of whom suffered from no significant symptoms. Tests of interactions were conducted between (a) paternal antisocial behavior and maternal depression and (b) several physiological indices of child temperament and emotionality in predicting (c) children's conduct problems and depression. Both internalizing and externalizing outcomes among children were associated specifically with maternal melancholic depression, and not with nonmelancholic depression. In addition, low levels of respiratory sinus arrhythmia (RSA) among children conferred significant risk for depression, regardless of maternal melancholia, whereas high RSA offered partial protection. Furthermore, high levels of maternal melancholia conferred significant risk for child depression, regardless of paternal antisocial behavior, whereas low levels of maternal melancholia offered partial protection. Finally, low levels of electrodermal responding (EDR) conferred significant risk for conduct problems, regardless of paternal antisocial behavior, whereas high EDR offered partial protection. None of the identified protective factors offered complete immunity from psychopathology. These findings underscore the complexity of resilience and resilience-related processes, and suggest several potential avenues for future longitudinal research. En ligne : http://dx.doi.org/10.1017/s0954579407000351 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=166 [article] Familial and temperamental predictors of resilience in children at risk for conduct disorder and depression [Texte imprimé et/ou numérique] / Katherine E. SHANNON, Auteur ; Theodore P. BEAUCHAINE, Auteur ; Sharon L. BRENNER, Auteur ; Emily NEUHAUS, Auteur ; Lisa GATZKE-KOPP, Auteur . - 2007 . - p.701-727. Langues : Anglais (eng) in Development and Psychopathology > 19-3 (Summer 2007) . - p.701-727 Index. décimale : PER Périodiques Résumé : In this study, we evaluated predictors of resilience among 8- to 12-year-old children recruited from primarily low socioeconomic status neighborhoods, 117 of whom suffered from clinical levels of conduct problems and/or depression, and 63 of whom suffered from no significant symptoms. Tests of interactions were conducted between (a) paternal antisocial behavior and maternal depression and (b) several physiological indices of child temperament and emotionality in predicting (c) children's conduct problems and depression. Both internalizing and externalizing outcomes among children were associated specifically with maternal melancholic depression, and not with nonmelancholic depression. In addition, low levels of respiratory sinus arrhythmia (RSA) among children conferred significant risk for depression, regardless of maternal melancholia, whereas high RSA offered partial protection. Furthermore, high levels of maternal melancholia conferred significant risk for child depression, regardless of paternal antisocial behavior, whereas low levels of maternal melancholia offered partial protection. Finally, low levels of electrodermal responding (EDR) conferred significant risk for conduct problems, regardless of paternal antisocial behavior, whereas high EDR offered partial protection. None of the identified protective factors offered complete immunity from psychopathology. These findings underscore the complexity of resilience and resilience-related processes, and suggest several potential avenues for future longitudinal research. En ligne : http://dx.doi.org/10.1017/s0954579407000351 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=166

Ten good reasons to consider biological processes in prevention and intervention research / Theodore P. BEAUCHAINE in Development and Psychopathology, 20-3 (Summer 2008)  

Public ISBD [article]  inDevelopment and Psychopathology > 20-3 (Summer 2008) . - p.745-774 Titre : Ten good reasons to consider biological processes in prevention and intervention research Type de document : Texte imprimé et/ou numérique Auteurs : Theodore P. BEAUCHAINE, Auteur ; Sharon L. BRENNER, Auteur ; Emily NEUHAUS, Auteur ; Lisa GATZKE-KOPP, Auteur Année de publication : 2008 Article en page(s) : p.745-774 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Most contemporary accounts of psychopathology acknowledge the importance of both biological and environmental influences on behavior. In developmental psychopathology, multiple etiological mechanisms for psychiatric disturbance are well recognized, including those operating at genetic, neurobiological, and environmental levels of analysis. However, neuroscientific principles are rarely considered in current approaches to prevention or intervention. In this article, we explain why a deeper understanding of the genetic and neural substrates of behavior is essential for the next generation of preventive interventions, and we outline 10 specific reasons why considering biological processes can improve treatment efficacy. Among these, we discuss (a) the role of biomarkers and endophenotypes in identifying those most in need of prevention; (b) implications for treatment of genetic and neural mechanisms of homotypic comorbidity, heterotypic comorbidity, and heterotypic continuity; (c) ways in which biological vulnerabilities moderate the effects of environmental experience; (d) situations in which Biology × Environment interactions account for more variance in key outcomes than main effects; and (e) sensitivity of neural systems, via epigenesis, programming, and neural plasticity, to environmental moderation across the life span. For each of the 10 reasons outlined we present an example from current literature and discuss critical implications for prevention. En ligne : http://dx.doi.org/10.1017/s0954579408000369 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=543 [article] Ten good reasons to consider biological processes in prevention and intervention research [Texte imprimé et/ou numérique] / Theodore P. BEAUCHAINE, Auteur ; Sharon L. BRENNER, Auteur ; Emily NEUHAUS, Auteur ; Lisa GATZKE-KOPP, Auteur . - 2008 . - p.745-774. Langues : Anglais (eng) in Development and Psychopathology > 20-3 (Summer 2008) . - p.745-774 Index. décimale : PER Périodiques Résumé : Most contemporary accounts of psychopathology acknowledge the importance of both biological and environmental influences on behavior. In developmental psychopathology, multiple etiological mechanisms for psychiatric disturbance are well recognized, including those operating at genetic, neurobiological, and environmental levels of analysis. However, neuroscientific principles are rarely considered in current approaches to prevention or intervention. In this article, we explain why a deeper understanding of the genetic and neural substrates of behavior is essential for the next generation of preventive interventions, and we outline 10 specific reasons why considering biological processes can improve treatment efficacy. Among these, we discuss (a) the role of biomarkers and endophenotypes in identifying those most in need of prevention; (b) implications for treatment of genetic and neural mechanisms of homotypic comorbidity, heterotypic comorbidity, and heterotypic continuity; (c) ways in which biological vulnerabilities moderate the effects of environmental experience; (d) situations in which Biology × Environment interactions account for more variance in key outcomes than main effects; and (e) sensitivity of neural systems, via epigenesis, programming, and neural plasticity, to environmental moderation across the life span. For each of the 10 reasons outlined we present an example from current literature and discuss critical implications for prevention. En ligne : http://dx.doi.org/10.1017/s0954579408000369 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=543

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