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Détail de l'auteur
Auteur Rishi PURI |
Documents disponibles écrits par cet auteur (1)
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Ultrastructural analyses in the hippocampus CA1 field in Shank3-deficient mice / Neha UPPAL in Molecular Autism, (June 2015)
[article]
Titre : Ultrastructural analyses in the hippocampus CA1 field in Shank3-deficient mice Type de document : Texte imprimé et/ou numérique Auteurs : Neha UPPAL, Auteur ; Rishi PURI, Auteur ; Frank YUK, Auteur ; William G. M. JANSSEN, Auteur ; Ozlem BOZDAGI-GUNAL, Auteur ; Hala HARONY-NICOLAS, Auteur ; Dara L. DICKSTEIN, Auteur ; Joseph D. BUXBAUM, Auteur ; Patrick R. HOF, Auteur Article en page(s) : p.1-10 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The genetics of autism spectrum disorder (hereafter referred to as “autism”) are rapidly unfolding, with a significant increase in the identification of genes implicated in the disorder. Many of these genes are part of a complex landscape of genetic variants that are thought to act together to cause the behavioral phenotype associated with autism. One of the few single-locus causes of autism involves a mutation in the SH3 and multiple ankyrin repeat domains 3 (SHANK3) gene. Previous electrophysiological studies in mice with Shank3 mutations demonstrated impairment in synaptic long-term potentiation, suggesting a potential disruption at the synapse. En ligne : http://dx.doi.org/10.1186/s13229-015-0036-x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=277
in Molecular Autism > (June 2015) . - p.1-10[article] Ultrastructural analyses in the hippocampus CA1 field in Shank3-deficient mice [Texte imprimé et/ou numérique] / Neha UPPAL, Auteur ; Rishi PURI, Auteur ; Frank YUK, Auteur ; William G. M. JANSSEN, Auteur ; Ozlem BOZDAGI-GUNAL, Auteur ; Hala HARONY-NICOLAS, Auteur ; Dara L. DICKSTEIN, Auteur ; Joseph D. BUXBAUM, Auteur ; Patrick R. HOF, Auteur . - p.1-10.
Langues : Anglais (eng)
in Molecular Autism > (June 2015) . - p.1-10
Index. décimale : PER Périodiques Résumé : The genetics of autism spectrum disorder (hereafter referred to as “autism”) are rapidly unfolding, with a significant increase in the identification of genes implicated in the disorder. Many of these genes are part of a complex landscape of genetic variants that are thought to act together to cause the behavioral phenotype associated with autism. One of the few single-locus causes of autism involves a mutation in the SH3 and multiple ankyrin repeat domains 3 (SHANK3) gene. Previous electrophysiological studies in mice with Shank3 mutations demonstrated impairment in synaptic long-term potentiation, suggesting a potential disruption at the synapse. En ligne : http://dx.doi.org/10.1186/s13229-015-0036-x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=277