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Détail de l'auteur
Auteur Keith SCHOFIELD |
Documents disponibles écrits par cet auteur (1)
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Autism, Chemicals, Probable Cause and Mitigation: A New Examination / Keith SCHOFIELD in Autism - Open Access, 6-4 ([01/07/2016])
[article]
Titre : Autism, Chemicals, Probable Cause and Mitigation: A New Examination Type de document : Texte imprimé et/ou numérique Auteurs : Keith SCHOFIELD, Auteur Article en page(s) : 27 p. Langues : Anglais (eng) Mots-clés : Autism Triggered growth Neurotoxins Body-burden Blood testing Selenium importance Multisource roles Index. décimale : PER Périodiques Résumé : This extensive overview has addressed in a scientific manner the probable cause of autism. Although seemingly an impossible task, an important clue has always been autism’s triggered growth from the late 90s. This triggering eliminated genetics as a major cause and redirected it to changes in the anthropogenic environment. Global availability for ingestion by women of child-bearing years of all neurotoxic chemicals has been reviewed and potential changes examined. Surprisingly, these simple criteria eliminate almost everything except the neurotoxic metal elements. These are all present in recent blood analyses of pregnant women and also in the fetal cord with some at risk level concentrations. However, the two predominant neurotoxic elements that clearly stand out are Al and Hg. Furthermore, isotopically labeled experiments on animals coupled to fetal autopsies indicate that these in the forms of Al3+, HgCH3+ and HgC2H5+ can penetrate the usually tight blood/brain barrier and enter the brain with now known half-lives. Normally, these elements are controlled and neutralized by brain seleno-cysteine proteins. However, an overloading burden has become plausible in current times and even a temporary failure leaves the fetal brain vulnerable. For the first time, the burden is seen to arise not from a single source, but from the combination of medicine and diet. If these neurological illnesses are to be controlled, the body-burden for these and all the other toxins that the body has to constantly manage has to be reduced. To begin with, this will have to involve modifying currently high inoculation rates and procedures particularly in the US, and minimizing risks from consuming fish especially in a Japanese sushi life-style. This is not only of importance for women of child-bearing ages but for the whole population considering the consequences of such body burdens throughout life. En ligne : https://dx.doi.org/10.4172/2165-7890.1000184 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=410
in Autism - Open Access > 6-4 [01/07/2016] . - 27 p.[article] Autism, Chemicals, Probable Cause and Mitigation: A New Examination [Texte imprimé et/ou numérique] / Keith SCHOFIELD, Auteur . - 27 p.
Langues : Anglais (eng)
in Autism - Open Access > 6-4 [01/07/2016] . - 27 p.
Mots-clés : Autism Triggered growth Neurotoxins Body-burden Blood testing Selenium importance Multisource roles Index. décimale : PER Périodiques Résumé : This extensive overview has addressed in a scientific manner the probable cause of autism. Although seemingly an impossible task, an important clue has always been autism’s triggered growth from the late 90s. This triggering eliminated genetics as a major cause and redirected it to changes in the anthropogenic environment. Global availability for ingestion by women of child-bearing years of all neurotoxic chemicals has been reviewed and potential changes examined. Surprisingly, these simple criteria eliminate almost everything except the neurotoxic metal elements. These are all present in recent blood analyses of pregnant women and also in the fetal cord with some at risk level concentrations. However, the two predominant neurotoxic elements that clearly stand out are Al and Hg. Furthermore, isotopically labeled experiments on animals coupled to fetal autopsies indicate that these in the forms of Al3+, HgCH3+ and HgC2H5+ can penetrate the usually tight blood/brain barrier and enter the brain with now known half-lives. Normally, these elements are controlled and neutralized by brain seleno-cysteine proteins. However, an overloading burden has become plausible in current times and even a temporary failure leaves the fetal brain vulnerable. For the first time, the burden is seen to arise not from a single source, but from the combination of medicine and diet. If these neurological illnesses are to be controlled, the body-burden for these and all the other toxins that the body has to constantly manage has to be reduced. To begin with, this will have to involve modifying currently high inoculation rates and procedures particularly in the US, and minimizing risks from consuming fish especially in a Japanese sushi life-style. This is not only of importance for women of child-bearing ages but for the whole population considering the consequences of such body burdens throughout life. En ligne : https://dx.doi.org/10.4172/2165-7890.1000184 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=410