Centre d'Information et de documentation du CRA Rhône-Alpes
CRA
Informations pratiques
-
Adresse
Centre d'information et de documentation
du CRA Rhône-Alpes
Centre Hospitalier le Vinatier
bât 211
95, Bd Pinel
69678 Bron CedexHoraires
Lundi au Vendredi
9h00-12h00 13h30-16h00Contact
Tél: +33(0)4 37 91 54 65
Mail
Fax: +33(0)4 37 91 54 37
-
Résultat de la recherche
1 recherche sur le mot-clé 'Autistic-disorder Cerebrum Corpus-callosum Dyslexia Gyral-window'
Affiner la recherche Générer le flux rss de la recherche
Partager le résultat de cette recherche Faire une suggestion
Increased White Matter Gyral Depth in Dyslexia: Implications for Corticocortical Connectivity / Manuel F. CASANOVA in Journal of Autism and Developmental Disorders, 40-1 (January 2010)
[article]
Titre : Increased White Matter Gyral Depth in Dyslexia: Implications for Corticocortical Connectivity Type de document : Texte imprimé et/ou numérique Auteurs : Manuel F. CASANOVA, Auteur ; Jay N. GIEDD, Auteur ; Andrew E. SWITALA, Auteur ; Ayman S. EL-BAZ, Auteur ; Judith M. RUMSEY, Auteur Année de publication : 2010 Article en page(s) : p.21-29 Langues : Anglais (eng) Mots-clés : Autistic-disorder Cerebrum Corpus-callosum Dyslexia Gyral-window Index. décimale : PER Périodiques Résumé : Recent studies provide credence to the minicolumnar origin of several developmental conditions, including dyslexia. Characteristics of minicolumnopathies include abnormalities in how the cortex expands and folds. This study examines the depth of the gyral white matter measured in an MRI series of 15 dyslexic adult men and eleven age-matched comparison subjects. Measurements were based upon the 3D Euclidean distance map inside the segmented cerebral white matter surface. Mean gyral white matter depth was 3.05 mm (SD ± 0.30 mm) in dyslexic subjects and 1.63 mm (SD ± 0.15 mm) in the controls. The results add credence to the growing literature suggesting that the attained reading circuit in dyslexia is abnormal because it is inefficient. Otherwise the anatomical substratum (i.e., corticocortical connectivity) underlying this inefficient circuit is normal. A deficit in very short-range connectivity (e.g., angular gyrus, striate cortex), consistent with results of a larger gyral window, could help explain reading difficulties in patients with dyslexia. The structural findings hereby reported are diametrically opposed to those reported for autism. En ligne : http://dx.doi.org/10.1007/s10803-009-0817-1 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=963
in Journal of Autism and Developmental Disorders > 40-1 (January 2010) . - p.21-29[article] Increased White Matter Gyral Depth in Dyslexia: Implications for Corticocortical Connectivity [Texte imprimé et/ou numérique] / Manuel F. CASANOVA, Auteur ; Jay N. GIEDD, Auteur ; Andrew E. SWITALA, Auteur ; Ayman S. EL-BAZ, Auteur ; Judith M. RUMSEY, Auteur . - 2010 . - p.21-29.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 40-1 (January 2010) . - p.21-29
Mots-clés : Autistic-disorder Cerebrum Corpus-callosum Dyslexia Gyral-window Index. décimale : PER Périodiques Résumé : Recent studies provide credence to the minicolumnar origin of several developmental conditions, including dyslexia. Characteristics of minicolumnopathies include abnormalities in how the cortex expands and folds. This study examines the depth of the gyral white matter measured in an MRI series of 15 dyslexic adult men and eleven age-matched comparison subjects. Measurements were based upon the 3D Euclidean distance map inside the segmented cerebral white matter surface. Mean gyral white matter depth was 3.05 mm (SD ± 0.30 mm) in dyslexic subjects and 1.63 mm (SD ± 0.15 mm) in the controls. The results add credence to the growing literature suggesting that the attained reading circuit in dyslexia is abnormal because it is inefficient. Otherwise the anatomical substratum (i.e., corticocortical connectivity) underlying this inefficient circuit is normal. A deficit in very short-range connectivity (e.g., angular gyrus, striate cortex), consistent with results of a larger gyral window, could help explain reading difficulties in patients with dyslexia. The structural findings hereby reported are diametrically opposed to those reported for autism. En ligne : http://dx.doi.org/10.1007/s10803-009-0817-1 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=963