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Détail de l'auteur
Auteur Russell G. PORT |
Documents disponibles écrits par cet auteur (2)
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Auditory encoding abnormalities in children with autism spectrum disorder suggest delayed development of auditory cortex / J. Christopher EDGAR in Molecular Autism, (December 2015)
[article]
Titre : Auditory encoding abnormalities in children with autism spectrum disorder suggest delayed development of auditory cortex Type de document : Texte imprimé et/ou numérique Auteurs : J. Christopher EDGAR, Auteur ; Charles L. FISK IV, Auteur ; Jeffrey I. BERMAN, Auteur ; Darina CHUDNOVSKAYA, Auteur ; Song LIU, Auteur ; Juhi PANDEY, Auteur ; John D. HERRINGTON, Auteur ; Russell G. PORT, Auteur ; Robert T. SCHULTZ, Auteur ; Timothy P. L. ROBERTS, Auteur Article en page(s) : p.1-14 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Findings of auditory abnormalities in children with autism spectrum disorder (ASD) include delayed superior temporal gyrus auditory responses, pre- and post-stimulus superior temporal gyrus (STG) auditory oscillatory abnormalities, and atypical hemispheric lateralization. These abnormalities are likely associated with abnormal brain maturation. To better understand changes in brain activity as a function of age, the present study investigated associations between age and STG auditory time-domain and time-frequency neural activity. En ligne : http://dx.doi.org/10.1186/s13229-015-0065-5 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=277
in Molecular Autism > (December 2015) . - p.1-14[article] Auditory encoding abnormalities in children with autism spectrum disorder suggest delayed development of auditory cortex [Texte imprimé et/ou numérique] / J. Christopher EDGAR, Auteur ; Charles L. FISK IV, Auteur ; Jeffrey I. BERMAN, Auteur ; Darina CHUDNOVSKAYA, Auteur ; Song LIU, Auteur ; Juhi PANDEY, Auteur ; John D. HERRINGTON, Auteur ; Russell G. PORT, Auteur ; Robert T. SCHULTZ, Auteur ; Timothy P. L. ROBERTS, Auteur . - p.1-14.
Langues : Anglais (eng)
in Molecular Autism > (December 2015) . - p.1-14
Index. décimale : PER Périodiques Résumé : Findings of auditory abnormalities in children with autism spectrum disorder (ASD) include delayed superior temporal gyrus auditory responses, pre- and post-stimulus superior temporal gyrus (STG) auditory oscillatory abnormalities, and atypical hemispheric lateralization. These abnormalities are likely associated with abnormal brain maturation. To better understand changes in brain activity as a function of age, the present study investigated associations between age and STG auditory time-domain and time-frequency neural activity. En ligne : http://dx.doi.org/10.1186/s13229-015-0065-5 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=277 Exploring the relationship between cortical GABA concentrations, auditory gamma-band responses and development in ASD: Evidence for an altered maturational trajectory in ASD / Russell G. PORT in Autism Research, 10-4 (April 2017)
[article]
Titre : Exploring the relationship between cortical GABA concentrations, auditory gamma-band responses and development in ASD: Evidence for an altered maturational trajectory in ASD Type de document : Texte imprimé et/ou numérique Auteurs : Russell G. PORT, Auteur ; William GAETZ, Auteur ; Luke BLOY, Auteur ; Dah-Jyuu WANG, Auteur ; Lisa BLASKEY, Auteur ; Emily S. KUSCHNER, Auteur ; Susan E. LEVY, Auteur ; Edward S. BRODKIN, Auteur ; Timothy P. L. ROBERTS, Auteur Article en page(s) : p.593-607 Langues : Anglais (eng) Mots-clés : magnetoencephalography gamma-band GABA MEGA-PRESS magnetic resonance spectroscopy auditory autism spectrum disorder excitatory/inhibitory Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) is hypothesized to arise from imbalances between excitatory and inhibitory neurotransmission (E/I imbalance). Studies have demonstrated E/I imbalance in individuals with ASD and also corresponding rodent models. One neural process thought to be reliant on E/I balance is gamma-band activity (Gamma), with support arising from observed correlations between motor, as well as visual, Gamma and underlying GABA concentrations in healthy adults. Additionally, decreased Gamma has been observed in ASD individuals and relevant animal models, though the direct relationship between Gamma and GABA concentrations in ASD remains unexplored. This study combined magnetoencephalography (MEG) and edited magnetic resonance spectroscopy (MRS) in 27 typically developing individuals (TD) and 30 individuals with ASD. Auditory cortex localized phase-locked Gamma was compared to resting Superior Temporal Gyrus relative cortical GABA concentrations for both children/adolescents and adults. Children/adolescents with ASD exhibited significantly decreased GABA+/Creatine (Cr) levels, though typical Gamma. Additionally, these children/adolescents lacked the typical maturation of GABA+/Cr concentrations and gamma-band coherence. Furthermore, children/adolescents with ASD additionally failed to exhibit the typical GABA+/Cr to gamma-band coherence association. This altered coupling during childhood/adolescence may result in Gamma decreases observed in the adults with ASD. Therefore, individuals with ASD exhibit improper local neuronal circuitry maturation during a childhood/adolescence critical period, when GABA is involved in configuring of such circuit functioning. Provocatively a novel line of treatment is suggested (with a critical time window); by increasing neural GABA levels in children/adolescents with ASD, proper local circuitry maturation may be restored resulting in typical Gamma in adulthood. En ligne : http://dx.doi.org/10.1002/aur.1686 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=307
in Autism Research > 10-4 (April 2017) . - p.593-607[article] Exploring the relationship between cortical GABA concentrations, auditory gamma-band responses and development in ASD: Evidence for an altered maturational trajectory in ASD [Texte imprimé et/ou numérique] / Russell G. PORT, Auteur ; William GAETZ, Auteur ; Luke BLOY, Auteur ; Dah-Jyuu WANG, Auteur ; Lisa BLASKEY, Auteur ; Emily S. KUSCHNER, Auteur ; Susan E. LEVY, Auteur ; Edward S. BRODKIN, Auteur ; Timothy P. L. ROBERTS, Auteur . - p.593-607.
Langues : Anglais (eng)
in Autism Research > 10-4 (April 2017) . - p.593-607
Mots-clés : magnetoencephalography gamma-band GABA MEGA-PRESS magnetic resonance spectroscopy auditory autism spectrum disorder excitatory/inhibitory Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) is hypothesized to arise from imbalances between excitatory and inhibitory neurotransmission (E/I imbalance). Studies have demonstrated E/I imbalance in individuals with ASD and also corresponding rodent models. One neural process thought to be reliant on E/I balance is gamma-band activity (Gamma), with support arising from observed correlations between motor, as well as visual, Gamma and underlying GABA concentrations in healthy adults. Additionally, decreased Gamma has been observed in ASD individuals and relevant animal models, though the direct relationship between Gamma and GABA concentrations in ASD remains unexplored. This study combined magnetoencephalography (MEG) and edited magnetic resonance spectroscopy (MRS) in 27 typically developing individuals (TD) and 30 individuals with ASD. Auditory cortex localized phase-locked Gamma was compared to resting Superior Temporal Gyrus relative cortical GABA concentrations for both children/adolescents and adults. Children/adolescents with ASD exhibited significantly decreased GABA+/Creatine (Cr) levels, though typical Gamma. Additionally, these children/adolescents lacked the typical maturation of GABA+/Cr concentrations and gamma-band coherence. Furthermore, children/adolescents with ASD additionally failed to exhibit the typical GABA+/Cr to gamma-band coherence association. This altered coupling during childhood/adolescence may result in Gamma decreases observed in the adults with ASD. Therefore, individuals with ASD exhibit improper local neuronal circuitry maturation during a childhood/adolescence critical period, when GABA is involved in configuring of such circuit functioning. Provocatively a novel line of treatment is suggested (with a critical time window); by increasing neural GABA levels in children/adolescents with ASD, proper local circuitry maturation may be restored resulting in typical Gamma in adulthood. En ligne : http://dx.doi.org/10.1002/aur.1686 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=307