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Life stress, the dopamine receptor gene, and emerging adult drug use trajectories: A longitudinal, multilevel, mediated moderation analysis / Gene H. BRODY in Development and Psychopathology, 24-3 (August 2012)
[article]
Titre : Life stress, the dopamine receptor gene, and emerging adult drug use trajectories: A longitudinal, multilevel, mediated moderation analysis Type de document : Texte imprimé et/ou numérique Auteurs : Gene H. BRODY, Auteur ; Yi-Fu CHEN, Auteur ; Tianyi YU, Auteur ; Steven R. H. BEACH, Auteur ; Steven M. KOGAN, Auteur ; Ronald L. SIMONS, Auteur ; Michael WINDLE, Auteur ; Robert A. PHILIBERT, Auteur Année de publication : 2012 Article en page(s) : p.941-51 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This study was designed to examine the prospective relations of life stress and genetic status with increases in drug use. African Americans (N = 399) in rural Georgia (Wave 1 mean age = 17 years) provided three waves of data across 27.5 months and a saliva sample from which the dopamine receptor D4 (DRD4) gene was genotyped. Multilevel growth curve modeling analysis indicated that emerging adults manifested the highest escalations in drug use when they reported high life stress and carried an allele of DRD4 with 7 or more repeats (7 + R allele). In addition, emerging adults who reported high life stress and carried the 7 + R allele evinced the largest increases in two proximal risk factors for drug use: affiliations with drug-using companions and drug use vulnerability cognitions. Furthermore, when the Gene × Environment interaction effects on the increases in affiliations with drug-using companions and vulnerability cognitions were entered into the model forecasting drug use, the Life Stress × DRD4 Status interaction on drug use became nonsignificant in the presence of the risk mechanisms. This finding provides an example of “second generation” Gene × Environment interaction research in which the interaction's effects on proximal risk mechanisms account for its effects on outcomes. En ligne : http://dx.doi.org/10.1017/S0954579412000466 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178
in Development and Psychopathology > 24-3 (August 2012) . - p.941-51[article] Life stress, the dopamine receptor gene, and emerging adult drug use trajectories: A longitudinal, multilevel, mediated moderation analysis [Texte imprimé et/ou numérique] / Gene H. BRODY, Auteur ; Yi-Fu CHEN, Auteur ; Tianyi YU, Auteur ; Steven R. H. BEACH, Auteur ; Steven M. KOGAN, Auteur ; Ronald L. SIMONS, Auteur ; Michael WINDLE, Auteur ; Robert A. PHILIBERT, Auteur . - 2012 . - p.941-51.
Langues : Anglais (eng)
in Development and Psychopathology > 24-3 (August 2012) . - p.941-51
Index. décimale : PER Périodiques Résumé : This study was designed to examine the prospective relations of life stress and genetic status with increases in drug use. African Americans (N = 399) in rural Georgia (Wave 1 mean age = 17 years) provided three waves of data across 27.5 months and a saliva sample from which the dopamine receptor D4 (DRD4) gene was genotyped. Multilevel growth curve modeling analysis indicated that emerging adults manifested the highest escalations in drug use when they reported high life stress and carried an allele of DRD4 with 7 or more repeats (7 + R allele). In addition, emerging adults who reported high life stress and carried the 7 + R allele evinced the largest increases in two proximal risk factors for drug use: affiliations with drug-using companions and drug use vulnerability cognitions. Furthermore, when the Gene × Environment interaction effects on the increases in affiliations with drug-using companions and vulnerability cognitions were entered into the model forecasting drug use, the Life Stress × DRD4 Status interaction on drug use became nonsignificant in the presence of the risk mechanisms. This finding provides an example of “second generation” Gene × Environment interaction research in which the interaction's effects on proximal risk mechanisms account for its effects on outcomes. En ligne : http://dx.doi.org/10.1017/S0954579412000466 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178 Linking prenatal maternal adversity to developmental outcomes in infants: The role of epigenetic pathways / Catherine MONK in Development and Psychopathology, 24-4 (November 2012)
[article]
Titre : Linking prenatal maternal adversity to developmental outcomes in infants: The role of epigenetic pathways Type de document : Texte imprimé et/ou numérique Auteurs : Catherine MONK, Auteur ; Julie A. SPICER, Auteur ; Frances A. CHAMPAGNE, Auteur Année de publication : 2012 Article en page(s) : p.1361-1376 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Prenatal exposure to maternal stress, anxiety, and depression can have lasting effects on infant development with risk of psychopathology. Although the impact of prenatal maternal distress has been well documented, the potential mechanisms through which maternal psychosocial variables shape development have yet to be fully elucidated. Advances in molecular biology have highlighted the role of epigenetic mechanisms in regulating gene activity, neurobiology, and behavior and the potential role of environmentally induced epigenetic variation in linking early life exposures to long-term biobehavioral outcomes. In this article, we discuss evidence illustrating the association between maternal prenatal distress and both fetal and infant developmental trajectories and the potential role of epigenetic mechanisms in mediating these effects. Postnatal experiences may have a critical moderating influence on prenatal effects, and we review findings illustrating prenatal–postnatal interplay and the developmental and epigenetic consequences of postnatal mother–infant interactions. The in utero environment is regulated by placental function and there is emerging evidence that the placenta is highly susceptible to maternal distress and a target of epigenetic dysregulation. Integrating studies of prenatal exposures, placental function, and postnatal maternal care with the exploration of epigenetic mechanisms may provide novel insights into the pathophysiology induced by maternal distress. En ligne : http://dx.doi.org/10.1017/S0954579412000764 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182
in Development and Psychopathology > 24-4 (November 2012) . - p.1361-1376[article] Linking prenatal maternal adversity to developmental outcomes in infants: The role of epigenetic pathways [Texte imprimé et/ou numérique] / Catherine MONK, Auteur ; Julie A. SPICER, Auteur ; Frances A. CHAMPAGNE, Auteur . - 2012 . - p.1361-1376.
Langues : Anglais (eng)
in Development and Psychopathology > 24-4 (November 2012) . - p.1361-1376
Index. décimale : PER Périodiques Résumé : Prenatal exposure to maternal stress, anxiety, and depression can have lasting effects on infant development with risk of psychopathology. Although the impact of prenatal maternal distress has been well documented, the potential mechanisms through which maternal psychosocial variables shape development have yet to be fully elucidated. Advances in molecular biology have highlighted the role of epigenetic mechanisms in regulating gene activity, neurobiology, and behavior and the potential role of environmentally induced epigenetic variation in linking early life exposures to long-term biobehavioral outcomes. In this article, we discuss evidence illustrating the association between maternal prenatal distress and both fetal and infant developmental trajectories and the potential role of epigenetic mechanisms in mediating these effects. Postnatal experiences may have a critical moderating influence on prenatal effects, and we review findings illustrating prenatal–postnatal interplay and the developmental and epigenetic consequences of postnatal mother–infant interactions. The in utero environment is regulated by placental function and there is emerging evidence that the placenta is highly susceptible to maternal distress and a target of epigenetic dysregulation. Integrating studies of prenatal exposures, placental function, and postnatal maternal care with the exploration of epigenetic mechanisms may provide novel insights into the pathophysiology induced by maternal distress. En ligne : http://dx.doi.org/10.1017/S0954579412000764 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182 LPHN3 and attention-deficit/hyperactivity disorder: interaction with maternal stress during pregnancy / Zia CHOUDHRY in Journal of Child Psychology and Psychiatry, 53-8 (August 2012)
[article]
Titre : LPHN3 and attention-deficit/hyperactivity disorder: interaction with maternal stress during pregnancy Type de document : Texte imprimé et/ou numérique Auteurs : Zia CHOUDHRY, Auteur ; Sarojini M. SENGUPTA, Auteur ; Natalie GRIZENKO, Auteur ; Marie-Eve FORTIER, Auteur ; Geeta A. THAKUR, Auteur ; Johanne BELLINGHAM, Auteur ; Ridha JOOBER, Auteur Année de publication : 2012 Article en page(s) : p.892-902 Langues : Anglais (eng) Mots-clés : ADHD LPHN3 maternal stress environmental factors GXE genetic association Index. décimale : PER Périodiques Résumé : Background: Attention-deficit/hyperactivity disorder (ADHD) is a heterogeneous behavioral disorder, complex both in etiology and clinical expression. Both genetic and environmental factors have been implicated, and it has been suggested that gene-environment interactions may play a pivotal role in the disorder. Recently, a significant association was reported between ADHD and LPHN3 (which codes for latrophilin 3), and replicated in independent samples. Methods: We have examined the association between tag single nucleotide polymorphisms (SNPs) in LPHN3 within the region previously implicated in ADHD. Family based association tests (FBAT) were conducted (n = 380 families) with the categorical diagnosis of ADHD, behavioral and cognitive phenotypes related to ADHD, and response to treatment (given a fixed dose of methylphenidate, 0.5 mg/day). Stratified FBAT analyses, based on maternal smoking and stress during pregnancy, was conducted. Results: Whereas limited association was observed in the total sample, highly significant interaction between four LPHN3 tag SNPs (rs6551665, rs1947274, rs6858066, rs2345039) and maternal stress during pregnancy was noted. Analysis conducted in the sub-group of mothers exposed to minimal stress during pregnancy showed significant associations with ADHD, behavioral and cognitive dimensions related to ADHD, as well as treatment response. Although extensive association was observed with the candidate SNPs, the findings are partially inconsistent with previously published results with the opposite alleles over-transmitted in these studies. Conclusions: These results provide evidence for the interaction between a genetic and environmental factor independently shown to be associated with ADHD. If confirmed in independent large studies, they may present a step forward in unraveling the complex etiology of ADHD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2012.02551.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=177
in Journal of Child Psychology and Psychiatry > 53-8 (August 2012) . - p.892-902[article] LPHN3 and attention-deficit/hyperactivity disorder: interaction with maternal stress during pregnancy [Texte imprimé et/ou numérique] / Zia CHOUDHRY, Auteur ; Sarojini M. SENGUPTA, Auteur ; Natalie GRIZENKO, Auteur ; Marie-Eve FORTIER, Auteur ; Geeta A. THAKUR, Auteur ; Johanne BELLINGHAM, Auteur ; Ridha JOOBER, Auteur . - 2012 . - p.892-902.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 53-8 (August 2012) . - p.892-902
Mots-clés : ADHD LPHN3 maternal stress environmental factors GXE genetic association Index. décimale : PER Périodiques Résumé : Background: Attention-deficit/hyperactivity disorder (ADHD) is a heterogeneous behavioral disorder, complex both in etiology and clinical expression. Both genetic and environmental factors have been implicated, and it has been suggested that gene-environment interactions may play a pivotal role in the disorder. Recently, a significant association was reported between ADHD and LPHN3 (which codes for latrophilin 3), and replicated in independent samples. Methods: We have examined the association between tag single nucleotide polymorphisms (SNPs) in LPHN3 within the region previously implicated in ADHD. Family based association tests (FBAT) were conducted (n = 380 families) with the categorical diagnosis of ADHD, behavioral and cognitive phenotypes related to ADHD, and response to treatment (given a fixed dose of methylphenidate, 0.5 mg/day). Stratified FBAT analyses, based on maternal smoking and stress during pregnancy, was conducted. Results: Whereas limited association was observed in the total sample, highly significant interaction between four LPHN3 tag SNPs (rs6551665, rs1947274, rs6858066, rs2345039) and maternal stress during pregnancy was noted. Analysis conducted in the sub-group of mothers exposed to minimal stress during pregnancy showed significant associations with ADHD, behavioral and cognitive dimensions related to ADHD, as well as treatment response. Although extensive association was observed with the candidate SNPs, the findings are partially inconsistent with previously published results with the opposite alleles over-transmitted in these studies. Conclusions: These results provide evidence for the interaction between a genetic and environmental factor independently shown to be associated with ADHD. If confirmed in independent large studies, they may present a step forward in unraveling the complex etiology of ADHD. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2012.02551.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=177 Mal élevé / Suzanne B. ROBERT-OUVRAY
Titre : Mal élevé : Le drame de l'enfant sans limites Type de document : Texte imprimé et/ou numérique Auteurs : Suzanne B. ROBERT-OUVRAY, Auteur Editeur : Paris [France] : Desclée de Brouwer Année de publication : 2003 Collection : Psychologie, ISSN 1620-0977 Importance : 237 p. Format : 13,0cm x 20,5cm x 1,6cm ISBN/ISSN/EAN : 978-2-220-05255-7 Langues : Français (fre) Index. décimale : TRO-B TRO-B - Troubles du Comportement Résumé : Beaucoup d'enfants se conduisent aujourd'hui comme des " tyrans ".
Capricieux, violents, en un mot : mal élevés. Pourtant, leurs parents leur ont donné de l'amour, ont voulu satisfaire leurs désirs, et même devancer leurs attentes. Peine perdue. Déconcertés, ne sachant plus comment asseoir leur autorité, ils répondent aux réactions de l'enfant par des agressions verbales ou physiques. Pourtant, ces manifestations sont bien souvent le signe d'angoisses refoulées et de frustration des besoins fondamentaux.
Ce livre de Suzanne Robert Ouvray éclaire les adultes sur ces besoins. Il pointe en particulier celui des limites émotionnelles qui pose le plus de problèmes car il réveille chez les parents des souffrances précoces oubliées et met en jeu des croyances éducatives désuètes. S'appuyant sur de nombreux exemples, l'ouvrage aidera les parents à mieux comprendre l'organisation tonique et émotionnelle de leur bébé, à acquérir des connaissances simples sur son développement et à assimiler le rôle prépondérant des limites dans la construction personnelle de l'enfant.Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=106 Mal élevé : Le drame de l'enfant sans limites [Texte imprimé et/ou numérique] / Suzanne B. ROBERT-OUVRAY, Auteur . - Paris [France] : Desclée de Brouwer, 2003 . - 237 p. ; 13,0cm x 20,5cm x 1,6cm. - (Psychologie, ISSN 1620-0977) .
ISBN : 978-2-220-05255-7
Langues : Français (fre)
Index. décimale : TRO-B TRO-B - Troubles du Comportement Résumé : Beaucoup d'enfants se conduisent aujourd'hui comme des " tyrans ".
Capricieux, violents, en un mot : mal élevés. Pourtant, leurs parents leur ont donné de l'amour, ont voulu satisfaire leurs désirs, et même devancer leurs attentes. Peine perdue. Déconcertés, ne sachant plus comment asseoir leur autorité, ils répondent aux réactions de l'enfant par des agressions verbales ou physiques. Pourtant, ces manifestations sont bien souvent le signe d'angoisses refoulées et de frustration des besoins fondamentaux.
Ce livre de Suzanne Robert Ouvray éclaire les adultes sur ces besoins. Il pointe en particulier celui des limites émotionnelles qui pose le plus de problèmes car il réveille chez les parents des souffrances précoces oubliées et met en jeu des croyances éducatives désuètes. S'appuyant sur de nombreux exemples, l'ouvrage aidera les parents à mieux comprendre l'organisation tonique et émotionnelle de leur bébé, à acquérir des connaissances simples sur son développement et à assimiler le rôle prépondérant des limites dans la construction personnelle de l'enfant.Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=106 Exemplaires (1)
Code-barres Cote Support Localisation Section Disponibilité DOC0000304 TRO-B ROB Livre Centre d'Information et de Documentation du CRA Rhône-Alpes TRO - Autres Troubles Disponible Managing autism spectrum disorders in current education provision / Rita JORDAN
Titre : Managing autism spectrum disorders in current education provision Type de document : Texte imprimé et/ou numérique Auteurs : Rita JORDAN, Auteur Année de publication : 2011 Importance : p.364-392 Langues : Anglais (eng) Index. décimale : AUT-B AUT-B - L'Autisme - Ouvrages généraux et scientifiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=141 Managing autism spectrum disorders in current education provision [Texte imprimé et/ou numérique] / Rita JORDAN, Auteur . - 2011 . - p.364-392.
Langues : Anglais (eng)
Index. décimale : AUT-B AUT-B - L'Autisme - Ouvrages généraux et scientifiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=141 Exemplaires
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