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Book Reviews in Journal of Child Psychology and Psychiatry, 40-6 (September 1999)
[article]
Titre : Book Reviews Type de document : Texte imprimé et/ou numérique Année de publication : 1999 Article en page(s) : p.981-983 Langues : Anglais (eng) Mots-clés : Behaviour problems distress eating behaviour endocrinology growth retardation Index. décimale : PER Périodiques Résumé : Books Reviewed:
Philip Graham., Cognitive-Behaviour Therapy for Children and Families
D. Aldridge. Jessica Kingsley, Suicide: The Tragedy of Hopelessness.
D. Cicchetti, & S. L. Toth, Developmental Perspectives on Trauma: Theory, Research, and Intervention.
M. Lindblad-Goldberg, M. M. Dore & L. Stern. W. W. Norton, Creating Competence from Chaos:A Comprehensive Guide to Home-based Services.
O. Stevenson., Neglected Children: Issues and Dilemmas.
C. Garland. Duckworth, Understanding Trauma: A Psychoanalytical Approach.
M. Rutter, H. Giller, & A. Hagell, Antisocial Behaviour by Young People.Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=124
in Journal of Child Psychology and Psychiatry > 40-6 (September 1999) . - p.981-983[article] Book Reviews [Texte imprimé et/ou numérique] . - 1999 . - p.981-983.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 40-6 (September 1999) . - p.981-983
Mots-clés : Behaviour problems distress eating behaviour endocrinology growth retardation Index. décimale : PER Périodiques Résumé : Books Reviewed:
Philip Graham., Cognitive-Behaviour Therapy for Children and Families
D. Aldridge. Jessica Kingsley, Suicide: The Tragedy of Hopelessness.
D. Cicchetti, & S. L. Toth, Developmental Perspectives on Trauma: Theory, Research, and Intervention.
M. Lindblad-Goldberg, M. M. Dore & L. Stern. W. W. Norton, Creating Competence from Chaos:A Comprehensive Guide to Home-based Services.
O. Stevenson., Neglected Children: Issues and Dilemmas.
C. Garland. Duckworth, Understanding Trauma: A Psychoanalytical Approach.
M. Rutter, H. Giller, & A. Hagell, Antisocial Behaviour by Young People.Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=124 A Case-comparison Study of the Characteristics of Children with a Short Stature Syndrome Induced by Stress (Hyperphagic Short Stature) and a Consecutive Series of Unaffected “Stressed” Children / Jane GILMOUR in Journal of Child Psychology and Psychiatry, 40-6 (September 1999)
[article]
Titre : A Case-comparison Study of the Characteristics of Children with a Short Stature Syndrome Induced by Stress (Hyperphagic Short Stature) and a Consecutive Series of Unaffected “Stressed” Children Type de document : Texte imprimé et/ou numérique Auteurs : Jane GILMOUR, Auteur ; David H. SKUSE, Auteur Année de publication : 1999 Article en page(s) : p.969-978 Langues : Anglais (eng) Mots-clés : Behaviour problems distress eating behaviour endocrinology growth retardation Index. décimale : PER Périodiques Résumé : Recently a type of growth failure (Hyperphagic Short Stature) has been described, in which there is potentially reversible severe impairment of growth hormone secretion, in association with excessively high levels of psychosocial stress. This condition is a variant of the disorder formerly known as Psychosocial Dwarfism. In the present study we compared children with Hyperphagic Short Stature (N= 25, aged 9.04 years±3.78, 72% male) and a closely matched sample with normal height, drawn from comparably stressful family circumstances (N= 25, aged 10.61±3.04, 60% male). Measures of the psychosocial environment, anthropometry, and developmental history from infancy were obtained. Many symptoms thought previously to be characteristics of psychosocial dwarfism were found to be nonspecific stress responses. Hypotonia (p < .05), enuresis/encopresis (p < .01), and sleep cycle disruption (p < .05) did differentiate the groups. Growth, appetite, and sleep are all influenced by hypothalamic nuclei, suggesting hypothalamic pathology could account for most of the clinical features of Hyperphagic Short Stature. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=124
in Journal of Child Psychology and Psychiatry > 40-6 (September 1999) . - p.969-978[article] A Case-comparison Study of the Characteristics of Children with a Short Stature Syndrome Induced by Stress (Hyperphagic Short Stature) and a Consecutive Series of Unaffected “Stressed” Children [Texte imprimé et/ou numérique] / Jane GILMOUR, Auteur ; David H. SKUSE, Auteur . - 1999 . - p.969-978.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 40-6 (September 1999) . - p.969-978
Mots-clés : Behaviour problems distress eating behaviour endocrinology growth retardation Index. décimale : PER Périodiques Résumé : Recently a type of growth failure (Hyperphagic Short Stature) has been described, in which there is potentially reversible severe impairment of growth hormone secretion, in association with excessively high levels of psychosocial stress. This condition is a variant of the disorder formerly known as Psychosocial Dwarfism. In the present study we compared children with Hyperphagic Short Stature (N= 25, aged 9.04 years±3.78, 72% male) and a closely matched sample with normal height, drawn from comparably stressful family circumstances (N= 25, aged 10.61±3.04, 60% male). Measures of the psychosocial environment, anthropometry, and developmental history from infancy were obtained. Many symptoms thought previously to be characteristics of psychosocial dwarfism were found to be nonspecific stress responses. Hypotonia (p < .05), enuresis/encopresis (p < .01), and sleep cycle disruption (p < .05) did differentiate the groups. Growth, appetite, and sleep are all influenced by hypothalamic nuclei, suggesting hypothalamic pathology could account for most of the clinical features of Hyperphagic Short Stature. Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=124 Pubertal recalibration of cortisol reactivity following early life stress: a cross-sectional analysis / Carrie E. DEPASQUALE in Journal of Child Psychology and Psychiatry, 60-5 (May 2019)
[article]
Titre : Pubertal recalibration of cortisol reactivity following early life stress: a cross-sectional analysis Type de document : Texte imprimé et/ou numérique Auteurs : Carrie E. DEPASQUALE, Auteur ; B. DONZELLA, Auteur ; M. R. GUNNAR, Auteur Article en page(s) : p.566-575 Langues : Anglais (eng) Mots-clés : Stress endocrinology institutions puberty Index. décimale : PER Périodiques Résumé : BACKGROUND: Children adopted from orphanages or other such institutions tend to display blunted reactivity to stressors - even years after arriving in their generally supportive and highly resourced postadoption homes. Puberty, a proposed sensitive period for environmental influences on stress-mediating systems, may provide an opportunity for postinstitutionalized children to recalibrate stress response systems in accordance with their now more supportive living situations. METHODS: This cross-sectional study examined the hypothalamic-pituitary-adrenocortical (HPA)-axis reactivity of 280 children ages 7 through 14 years; 122 children were adopted from institutions in 14 countries between the ages of 6 months and 5 years, after spending an average of 95% of their lives in institutional care, and 158 children of similarly high socioeconomic status in their biological families served as the nonadopted comparison group. All of the children were assessed by nurses for Tanner stage and, on a different day, completed the Trier Social Stress Test for Children. RESULTS: Using a linear mixed-effects model and seven measures of salivary cortisol, results indicated that early-pubertal postinstitutionalized children showed blunted HPA axis reactivity compared to nonadopted children, but mid/late-pubertal postinstitutionalized children displayed higher reactivity similar to the nonadopted comparison children. CONCLUSIONS: This is the first evidence of possible pubertal recalibration of HPA axis reactivity to a psychosocial stressor in postinstitutionalized children, which provides a promising avenue for future research regarding the protective factors of the postadoption environment and subsequent physiological, behavioral, and psychopathological outcomes. En ligne : http://dx.doi.org/10.1111/jcpp.12992 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=392
in Journal of Child Psychology and Psychiatry > 60-5 (May 2019) . - p.566-575[article] Pubertal recalibration of cortisol reactivity following early life stress: a cross-sectional analysis [Texte imprimé et/ou numérique] / Carrie E. DEPASQUALE, Auteur ; B. DONZELLA, Auteur ; M. R. GUNNAR, Auteur . - p.566-575.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 60-5 (May 2019) . - p.566-575
Mots-clés : Stress endocrinology institutions puberty Index. décimale : PER Périodiques Résumé : BACKGROUND: Children adopted from orphanages or other such institutions tend to display blunted reactivity to stressors - even years after arriving in their generally supportive and highly resourced postadoption homes. Puberty, a proposed sensitive period for environmental influences on stress-mediating systems, may provide an opportunity for postinstitutionalized children to recalibrate stress response systems in accordance with their now more supportive living situations. METHODS: This cross-sectional study examined the hypothalamic-pituitary-adrenocortical (HPA)-axis reactivity of 280 children ages 7 through 14 years; 122 children were adopted from institutions in 14 countries between the ages of 6 months and 5 years, after spending an average of 95% of their lives in institutional care, and 158 children of similarly high socioeconomic status in their biological families served as the nonadopted comparison group. All of the children were assessed by nurses for Tanner stage and, on a different day, completed the Trier Social Stress Test for Children. RESULTS: Using a linear mixed-effects model and seven measures of salivary cortisol, results indicated that early-pubertal postinstitutionalized children showed blunted HPA axis reactivity compared to nonadopted children, but mid/late-pubertal postinstitutionalized children displayed higher reactivity similar to the nonadopted comparison children. CONCLUSIONS: This is the first evidence of possible pubertal recalibration of HPA axis reactivity to a psychosocial stressor in postinstitutionalized children, which provides a promising avenue for future research regarding the protective factors of the postadoption environment and subsequent physiological, behavioral, and psychopathological outcomes. En ligne : http://dx.doi.org/10.1111/jcpp.12992 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=392 Research Review: Intergenerational transmission of disadvantage: epigenetics and parents' childhoods as the first exposure / P. SCORZA in Journal of Child Psychology and Psychiatry, 60-2 (February 2019)
[article]
Titre : Research Review: Intergenerational transmission of disadvantage: epigenetics and parents' childhoods as the first exposure Type de document : Texte imprimé et/ou numérique Auteurs : P. SCORZA, Auteur ; C. S. DUARTE, Auteur ; A. E. HIPWELL, Auteur ; J. POSNER, Auteur ; A. ORTIN, Auteur ; Glorisa CANINO, Auteur ; C. MONK, Auteur Article en page(s) : p.119-132 Langues : Anglais (eng) Mots-clés : Development adversity early life experience endocrinology epigenetics gene-environment interaction stress Index. décimale : PER Périodiques Résumé : BACKGROUND: For decades, economists and sociologists have documented intergenerational transmission of socioeconomic disadvantage, demonstrating that economic, political, and social factors contribute to 'inherited hardship'. Drawing on biological factors, the developmental origins of adult health and disease model posits that fetal exposure to maternal prenatal distress associated with socioeconomic disadvantage compromises offspring's neurodevelopment, affecting short- and long-term physical and mental health, and thereby psychosocial standing and resources. Increasing evidence suggests that mother-to-child influence occurs prenatally, in part via maternal and offspring atypical HPA axis regulation, with negative effects on the maturation of prefrontal and subcortical neural circuits in the offspring. However, even this in utero timeframe may be insufficient to understand biological aspects of the transmission of factors contributing to disadvantage across generations. METHODS: We review animal studies and emerging human research indicating that parents' childhood experiences may transfer epigenetic marks that could impact the development of their offspring independently of and in interaction with their offspring's perinatal and early childhood direct exposures to stress stemming from socioeconomic disadvantage and adversity. RESULTS: Animal models point to epigenetic mechanisms by which traits that could contribute to disadvantage may be transmitted across generations. However, epigenetic pathways of parental childhood experiences influencing child outcomes in the next generation are only beginning to be studied in humans. With a focus on translational research, we point to design features and methodological considerations for human cohort studies to be able to test the intergenerational transmission hypothesis, and we illustrate this with existing longitudinal studies. CONCLUSIONS: Epigenetic intergenerational transmission, if at play in human populations, could have policy implications in terms of reducing the continuation of disadvantage across generations. Further research is needed to address this gap in the understanding of the perpetuation of compromised lives across generations. En ligne : http://dx.doi.org/10.1111/jcpp.12877 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=381
in Journal of Child Psychology and Psychiatry > 60-2 (February 2019) . - p.119-132[article] Research Review: Intergenerational transmission of disadvantage: epigenetics and parents' childhoods as the first exposure [Texte imprimé et/ou numérique] / P. SCORZA, Auteur ; C. S. DUARTE, Auteur ; A. E. HIPWELL, Auteur ; J. POSNER, Auteur ; A. ORTIN, Auteur ; Glorisa CANINO, Auteur ; C. MONK, Auteur . - p.119-132.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 60-2 (February 2019) . - p.119-132
Mots-clés : Development adversity early life experience endocrinology epigenetics gene-environment interaction stress Index. décimale : PER Périodiques Résumé : BACKGROUND: For decades, economists and sociologists have documented intergenerational transmission of socioeconomic disadvantage, demonstrating that economic, political, and social factors contribute to 'inherited hardship'. Drawing on biological factors, the developmental origins of adult health and disease model posits that fetal exposure to maternal prenatal distress associated with socioeconomic disadvantage compromises offspring's neurodevelopment, affecting short- and long-term physical and mental health, and thereby psychosocial standing and resources. Increasing evidence suggests that mother-to-child influence occurs prenatally, in part via maternal and offspring atypical HPA axis regulation, with negative effects on the maturation of prefrontal and subcortical neural circuits in the offspring. However, even this in utero timeframe may be insufficient to understand biological aspects of the transmission of factors contributing to disadvantage across generations. METHODS: We review animal studies and emerging human research indicating that parents' childhood experiences may transfer epigenetic marks that could impact the development of their offspring independently of and in interaction with their offspring's perinatal and early childhood direct exposures to stress stemming from socioeconomic disadvantage and adversity. RESULTS: Animal models point to epigenetic mechanisms by which traits that could contribute to disadvantage may be transmitted across generations. However, epigenetic pathways of parental childhood experiences influencing child outcomes in the next generation are only beginning to be studied in humans. With a focus on translational research, we point to design features and methodological considerations for human cohort studies to be able to test the intergenerational transmission hypothesis, and we illustrate this with existing longitudinal studies. CONCLUSIONS: Epigenetic intergenerational transmission, if at play in human populations, could have policy implications in terms of reducing the continuation of disadvantage across generations. Further research is needed to address this gap in the understanding of the perpetuation of compromised lives across generations. En ligne : http://dx.doi.org/10.1111/jcpp.12877 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=381