A life span developmental investigation of marriage and problem-drinking reduction / Matthew R. LEE in Development and Psychopathology, 36-1 (February 2024)  

Public ISBD [article]  inDevelopment and Psychopathology > 36-1 (February 2024) . - p.1-11 Titre : A life span developmental investigation of marriage and problem-drinking reduction Type de document : Texte imprimé et/ou numérique Auteurs : Matthew R. LEE, Auteur ; Ellen W. YEUNG, Auteur ; Andrew K. LITTLEFIELD, Auteur ; Audrey STEPHENSON, Auteur ; Annabel KADY, Auteur ; Thomas KWAN, Auteur ; Laurie CHASSIN, Auteur ; Kenneth J. SHER, Auteur Article en page(s) : p.1-11 Langues : Anglais (eng) Mots-clés : alcohol use disorder  desistance  lifespan development  marriage  maturing out  natural recovery  problem drinking Index. décimale : PER Périodiques Résumé : While prior literature has largely focused on marriage effects during young adulthood, it is less clear whether these effects are as strong in middle adulthood. Thus, we investigated age differences in marriage effects on problem-drinking reduction. We employed parallel analyses with two independent samples (analytic-sample Ns of 577 and 441, respectively). Both are high-risk samples by design, with about 50% of participants having a parent with lifetime alcohol use disorder. Both samples have been assessed longitudinally from early young adulthood to the mid-to-late 30s. Separate parallel analyses with these two samples allowed evaluation of the reproducibility of results. Growth models of problem drinking tested marriage as a time-varying predictor and thereby assessed age differences in marriage effects. For both samples, results consistently showed marriage effects to be strongest in early young adulthood and to decrease somewhat monotonically thereafter with age, reaching very small (and nonsignificant) magnitudes by the 30s. Results may reflect that role transitions like marriage have more impact on problem drinking in earlier versus later adulthood, thereby highlighting the importance of life span developmental research for understanding problem-drinking desistance. Our findings can inform intervention strategies aimed at reducing problem drinking by jumpstarting or amplifying natural processes of adult role adaptation. En ligne : https://dx.doi.org/10.1017/S0954579422000827 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523 [article] A life span developmental investigation of marriage and problem-drinking reduction [Texte imprimé et/ou numérique] / Matthew R. LEE, Auteur ; Ellen W. YEUNG, Auteur ; Andrew K. LITTLEFIELD, Auteur ; Audrey STEPHENSON, Auteur ; Annabel KADY, Auteur ; Thomas KWAN, Auteur ; Laurie CHASSIN, Auteur ; Kenneth J. SHER, Auteur . - p.1-11. Langues : Anglais (eng) in Development and Psychopathology > 36-1 (February 2024) . - p.1-11 Mots-clés : alcohol use disorder  desistance  lifespan development  marriage  maturing out  natural recovery  problem drinking Index. décimale : PER Périodiques Résumé : While prior literature has largely focused on marriage effects during young adulthood, it is less clear whether these effects are as strong in middle adulthood. Thus, we investigated age differences in marriage effects on problem-drinking reduction. We employed parallel analyses with two independent samples (analytic-sample Ns of 577 and 441, respectively). Both are high-risk samples by design, with about 50% of participants having a parent with lifetime alcohol use disorder. Both samples have been assessed longitudinally from early young adulthood to the mid-to-late 30s. Separate parallel analyses with these two samples allowed evaluation of the reproducibility of results. Growth models of problem drinking tested marriage as a time-varying predictor and thereby assessed age differences in marriage effects. For both samples, results consistently showed marriage effects to be strongest in early young adulthood and to decrease somewhat monotonically thereafter with age, reaching very small (and nonsignificant) magnitudes by the 30s. Results may reflect that role transitions like marriage have more impact on problem drinking in earlier versus later adulthood, thereby highlighting the importance of life span developmental research for understanding problem-drinking desistance. Our findings can inform intervention strategies aimed at reducing problem drinking by jumpstarting or amplifying natural processes of adult role adaptation. En ligne : https://dx.doi.org/10.1017/S0954579422000827 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523

Maturing out of alcohol involvement: Transitions in latent drinking statuses from late adolescence to adulthood / Matthew R. LEE in Development and Psychopathology, 25-4 (November 2013)  

Public ISBD [article]  inDevelopment and Psychopathology > 25-4 (November 2013) . - p.1137-1153 Titre : Maturing out of alcohol involvement: Transitions in latent drinking statuses from late adolescence to adulthood Type de document : Texte imprimé et/ou numérique Auteurs : Matthew R. LEE, Auteur ; Laurie A. CHASSIN, Auteur ; Ian K. VILLALTA, Auteur Article en page(s) : p.1137-1153 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Research has shown a developmental process of “maturing out” of alcohol involvement beginning in young adulthood, but the precise nature of changes characterizing maturing out is unclear. We used latent transition analysis to investigate these changes in a high-risk sample from a longitudinal study of familial alcoholism (N = 844; 51% children of alcoholics; 53% male, 71% non-Hispanic Caucasian, 27% Hispanic). Analyses classified participants into latent drinking statuses during late adolescence (ages 17–22), young adulthood (ages 23–28), and adulthood (ages 29–40), and characterized transitions among these statuses over time. The resulting four statuses were abstainers, low-risk drinkers who typically drank less than weekly and rarely binged or showed drinking problems, moderate-risk drinkers who typically binged less than weekly and showed moderate risk for drinking problems, and high-risk drinkers who typically binged at least weekly and showed high risk for drinking problems. Maturing out between late adolescence and young adulthood was most common among initial high-risk drinkers, but they typically declined to moderate-risk drinking rather than to nonrisky drinking statuses. This suggests that the developmental phenomenon of maturing out pertains primarily to relatively high-risk initial drinkers and that many high-risk drinkers who mature out merely reduce rather than eliminate their risky drinking. En ligne : http://dx.doi.org/10.1017/S0954579413000424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=219 [article] Maturing out of alcohol involvement: Transitions in latent drinking statuses from late adolescence to adulthood [Texte imprimé et/ou numérique] / Matthew R. LEE, Auteur ; Laurie A. CHASSIN, Auteur ; Ian K. VILLALTA, Auteur . - p.1137-1153. Langues : Anglais (eng) in Development and Psychopathology > 25-4 (November 2013) . - p.1137-1153 Index. décimale : PER Périodiques Résumé : Research has shown a developmental process of “maturing out” of alcohol involvement beginning in young adulthood, but the precise nature of changes characterizing maturing out is unclear. We used latent transition analysis to investigate these changes in a high-risk sample from a longitudinal study of familial alcoholism (N = 844; 51% children of alcoholics; 53% male, 71% non-Hispanic Caucasian, 27% Hispanic). Analyses classified participants into latent drinking statuses during late adolescence (ages 17–22), young adulthood (ages 23–28), and adulthood (ages 29–40), and characterized transitions among these statuses over time. The resulting four statuses were abstainers, low-risk drinkers who typically drank less than weekly and rarely binged or showed drinking problems, moderate-risk drinkers who typically binged less than weekly and showed moderate risk for drinking problems, and high-risk drinkers who typically binged at least weekly and showed high risk for drinking problems. Maturing out between late adolescence and young adulthood was most common among initial high-risk drinkers, but they typically declined to moderate-risk drinking rather than to nonrisky drinking statuses. This suggests that the developmental phenomenon of maturing out pertains primarily to relatively high-risk initial drinkers and that many high-risk drinkers who mature out merely reduce rather than eliminate their risky drinking. En ligne : http://dx.doi.org/10.1017/S0954579413000424 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=219

Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation / Laurie A. CHASSIN in Development and Psychopathology, 24-3 (August 2012)  

Public ISBD [article]  inDevelopment and Psychopathology > 24-3 (August 2012) . - p.953-67 Titre : Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation Type de document : Texte imprimé et/ou numérique Auteurs : Laurie A. CHASSIN, Auteur ; Matthew R. LEE, Auteur ; Young Il CHO, Auteur ; Frances L. WANG, Auteur ; Arpana AGRAWAL, Auteur ; Kenneth J. SHER, Auteur ; Michael T. LYNSKEY, Auteur Année de publication : 2012 Article en page(s) : p.953-67 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This study examined the interplay between the influence of peers who promote alcohol use and μ-opioid receptor M1 (OPRM1) genetic variation in the intergenerational transmission of alcohol use disorder (AUD) symptoms while separating the “traitlike” components of AUD symptoms from their age-specific manifestations at three ages from emerging adulthood (17–23 years) to adulthood (29–40 years). The results for males were consistent with genetically influenced peer selection mechanisms as mediators of parent alcoholism effects. Male children of alcoholics were less likely to be carriers of the G allele in single nucleotide polymorphism A118G (rs1799971), and those who were homozygous for the A allele were more likely to affiliate with alcohol use promoting peers who increased the risk for AUD symptoms at all ages. There was evidence for women of an interaction between OPRM1 variation and peer affiliations but only at the earliest age band. Peer influences had stronger effects among women who were G-carriers. These results illustrate the complex ways in which the interplay between influences at multiple levels of analysis can underlie the intergenerational transmission of alcohol disorders as well as the importance of considering age and gender differences in these pathways. En ligne : http://dx.doi.org/10.1017/S0954579412000478 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178 [article] Testing multiple levels of influence in the intergenerational transmission of alcohol disorders from a developmental perspective: The example of alcohol use promoting peers and μ-opioid receptor M1 variation [Texte imprimé et/ou numérique] / Laurie A. CHASSIN, Auteur ; Matthew R. LEE, Auteur ; Young Il CHO, Auteur ; Frances L. WANG, Auteur ; Arpana AGRAWAL, Auteur ; Kenneth J. SHER, Auteur ; Michael T. LYNSKEY, Auteur . - 2012 . - p.953-67. Langues : Anglais (eng) in Development and Psychopathology > 24-3 (August 2012) . - p.953-67 Index. décimale : PER Périodiques Résumé : This study examined the interplay between the influence of peers who promote alcohol use and μ-opioid receptor M1 (OPRM1) genetic variation in the intergenerational transmission of alcohol use disorder (AUD) symptoms while separating the “traitlike” components of AUD symptoms from their age-specific manifestations at three ages from emerging adulthood (17–23 years) to adulthood (29–40 years). The results for males were consistent with genetically influenced peer selection mechanisms as mediators of parent alcoholism effects. Male children of alcoholics were less likely to be carriers of the G allele in single nucleotide polymorphism A118G (rs1799971), and those who were homozygous for the A allele were more likely to affiliate with alcohol use promoting peers who increased the risk for AUD symptoms at all ages. There was evidence for women of an interaction between OPRM1 variation and peer affiliations but only at the earliest age band. Peer influences had stronger effects among women who were G-carriers. These results illustrate the complex ways in which the interplay between influences at multiple levels of analysis can underlie the intergenerational transmission of alcohol disorders as well as the importance of considering age and gender differences in these pathways. En ligne : http://dx.doi.org/10.1017/S0954579412000478 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=178

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