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Auteur Lisa A. CROEN |
Documents disponibles écrits par cet auteur (68)
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Increased mid-gestational IFN-gamma, IL-4, and IL-5 in women giving birth to a child with autism: a case-control study / Paula GOINES in Molecular Autism, (August 2011)
[article]
Titre : Increased mid-gestational IFN-gamma, IL-4, and IL-5 in women giving birth to a child with autism: a case-control study Type de document : Texte imprimé et/ou numérique Auteurs : Paula GOINES, Auteur ; Lisa A. CROEN, Auteur ; Daniel BRAUNSCHWEIG, Auteur ; Cathleen K. YOSHIDA, Auteur ; Judith K. GRETHER, Auteur ; David J. HANSEN, Auteur ; Martin KHARRAZI, Auteur ; Paul ASHWOOD, Auteur ; Judy VAN DE WATER, Auteur Année de publication : 2011 Article en page(s) : 41 p. Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Background
Immune anomalies have been documented in individuals with autism spectrum disorders (ASDs) and their family members. It is unknown whether the maternal immune profile during pregnancy is associated with the risk of bearing a child with ASD or other neurodevelopmental disorders.
Methods
Using Luminex technology, levels of 17 cytokines and chemokines were measured in banked serum collected from women at 15 to 19 weeks of gestation who gave birth to a child ultimately diagnosed with (1) ASD (n = 84), (2) a developmental delay (DD) but not autism (n = 49) or (3) no known developmental disability (general population (GP); n = 159). ASD and DD risk associated with maternal cytokine and chemokine levels was estimated by using multivariable logistic regression analysis.
Results
Elevated concentrations of IFN-gamma, IL-4 and IL-5 in midgestation maternal serum were significantly associated with a 50% increased risk of ASD, regardless of ASD onset type and the presence of intellectual disability. By contrast, elevated concentrations of IL-2, IL-4 and IL-6 were significantly associated with an increased risk of DD without autism.
Conclusion
The profile of elevated serum IFN-gamma, IL-4 and IL-5 was more common in women who gave birth to a child subsequently diagnosed with ASD. An alternative profile of increased IL-2, IL-4 and IL-6 was more common for women who gave birth to a child subsequently diagnosed with DD without autism. Further investigation is needed to characterize the relationship between these divergent maternal immunological phenotypes and to evaluate their effect on neurodevelopment.En ligne : http://dx.doi.org/10.1186/2040-2392-2-13 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=141
in Molecular Autism > (August 2011) . - 41 p.[article] Increased mid-gestational IFN-gamma, IL-4, and IL-5 in women giving birth to a child with autism: a case-control study [Texte imprimé et/ou numérique] / Paula GOINES, Auteur ; Lisa A. CROEN, Auteur ; Daniel BRAUNSCHWEIG, Auteur ; Cathleen K. YOSHIDA, Auteur ; Judith K. GRETHER, Auteur ; David J. HANSEN, Auteur ; Martin KHARRAZI, Auteur ; Paul ASHWOOD, Auteur ; Judy VAN DE WATER, Auteur . - 2011 . - 41 p.
Langues : Anglais (eng)
in Molecular Autism > (August 2011) . - 41 p.
Index. décimale : PER Périodiques Résumé : Background
Immune anomalies have been documented in individuals with autism spectrum disorders (ASDs) and their family members. It is unknown whether the maternal immune profile during pregnancy is associated with the risk of bearing a child with ASD or other neurodevelopmental disorders.
Methods
Using Luminex technology, levels of 17 cytokines and chemokines were measured in banked serum collected from women at 15 to 19 weeks of gestation who gave birth to a child ultimately diagnosed with (1) ASD (n = 84), (2) a developmental delay (DD) but not autism (n = 49) or (3) no known developmental disability (general population (GP); n = 159). ASD and DD risk associated with maternal cytokine and chemokine levels was estimated by using multivariable logistic regression analysis.
Results
Elevated concentrations of IFN-gamma, IL-4 and IL-5 in midgestation maternal serum were significantly associated with a 50% increased risk of ASD, regardless of ASD onset type and the presence of intellectual disability. By contrast, elevated concentrations of IL-2, IL-4 and IL-6 were significantly associated with an increased risk of DD without autism.
Conclusion
The profile of elevated serum IFN-gamma, IL-4 and IL-5 was more common in women who gave birth to a child subsequently diagnosed with ASD. An alternative profile of increased IL-2, IL-4 and IL-6 was more common for women who gave birth to a child subsequently diagnosed with DD without autism. Further investigation is needed to characterize the relationship between these divergent maternal immunological phenotypes and to evaluate their effect on neurodevelopment.En ligne : http://dx.doi.org/10.1186/2040-2392-2-13 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=141 Infant siblings and the investigation of autism risk factors / C. J. NEWSCHAFFER in Journal of Neurodevelopmental Disorders, 4-1 (December 2012)
[article]
Titre : Infant siblings and the investigation of autism risk factors Type de document : Texte imprimé et/ou numérique Auteurs : C. J. NEWSCHAFFER, Auteur ; Lisa A. CROEN, Auteur ; M. D. FALLIN, Auteur ; I. HERTZ-PICCIOTTO, Auteur ; D. V. NGUYEN, Auteur ; N. L. LEE, Auteur ; C. A. BERRY, Auteur ; H. FARZADEGAN, Auteur ; H. N. HESS, Auteur ; R. J. LANDA, Auteur ; S. E. LEVY, Auteur ; M. L. MASSOLO, Auteur ; S. C. MEYERER, Auteur ; S. M. MOHAMMED, Auteur ; M. C. OLIVER, Auteur ; S. OZONOFF, Auteur ; J. PANDEY, Auteur ; A. SCHROEDER, Auteur ; K. M. SHEDD-WISE, Auteur Article en page(s) : p.7 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Infant sibling studies have been at the vanguard of autism spectrum disorders (ASD) research over the past decade, providing important new knowledge about the earliest emerging signs of ASD and expanding our understanding of the developmental course of this complex disorder. Studies focused on siblings of children with ASD also have unrealized potential for contributing to ASD etiologic research. Moving targeted time of enrollment back from infancy toward conception creates tremendous opportunities for optimally studying risk factors and risk biomarkers during the pre-, peri- and neonatal periods. By doing so, a traditional sibling study, which already incorporates close developmental follow-up of at-risk infants through the third year of life, is essentially reconfigured as an enriched-risk pregnancy cohort study. This review considers the enriched-risk pregnancy cohort approach of studying infant siblings in the context of current thinking on ASD etiologic mechanisms. It then discusses the key features of this approach and provides a description of the design and implementation strategy of one major ASD enriched-risk pregnancy cohort study: the Early Autism Risk Longitudinal Investigation (EARLI). En ligne : http://dx.doi.org/10.1186/1866-1955-4-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=344
in Journal of Neurodevelopmental Disorders > 4-1 (December 2012) . - p.7[article] Infant siblings and the investigation of autism risk factors [Texte imprimé et/ou numérique] / C. J. NEWSCHAFFER, Auteur ; Lisa A. CROEN, Auteur ; M. D. FALLIN, Auteur ; I. HERTZ-PICCIOTTO, Auteur ; D. V. NGUYEN, Auteur ; N. L. LEE, Auteur ; C. A. BERRY, Auteur ; H. FARZADEGAN, Auteur ; H. N. HESS, Auteur ; R. J. LANDA, Auteur ; S. E. LEVY, Auteur ; M. L. MASSOLO, Auteur ; S. C. MEYERER, Auteur ; S. M. MOHAMMED, Auteur ; M. C. OLIVER, Auteur ; S. OZONOFF, Auteur ; J. PANDEY, Auteur ; A. SCHROEDER, Auteur ; K. M. SHEDD-WISE, Auteur . - p.7.
Langues : Anglais (eng)
in Journal of Neurodevelopmental Disorders > 4-1 (December 2012) . - p.7
Index. décimale : PER Périodiques Résumé : Infant sibling studies have been at the vanguard of autism spectrum disorders (ASD) research over the past decade, providing important new knowledge about the earliest emerging signs of ASD and expanding our understanding of the developmental course of this complex disorder. Studies focused on siblings of children with ASD also have unrealized potential for contributing to ASD etiologic research. Moving targeted time of enrollment back from infancy toward conception creates tremendous opportunities for optimally studying risk factors and risk biomarkers during the pre-, peri- and neonatal periods. By doing so, a traditional sibling study, which already incorporates close developmental follow-up of at-risk infants through the third year of life, is essentially reconfigured as an enriched-risk pregnancy cohort study. This review considers the enriched-risk pregnancy cohort approach of studying infant siblings in the context of current thinking on ASD etiologic mechanisms. It then discusses the key features of this approach and provides a description of the design and implementation strategy of one major ASD enriched-risk pregnancy cohort study: the Early Autism Risk Longitudinal Investigation (EARLI). En ligne : http://dx.doi.org/10.1186/1866-1955-4-7 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=344 Infection and Fever in Pregnancy and Autism Spectrum Disorders: Findings from the Study to Explore Early Development / Lisa A. CROEN in Autism Research, 12-10 (October 2019)
[article]
Titre : Infection and Fever in Pregnancy and Autism Spectrum Disorders: Findings from the Study to Explore Early Development Type de document : Texte imprimé et/ou numérique Auteurs : Lisa A. CROEN, Auteur ; Y. QIAN, Auteur ; Paul ASHWOOD, Auteur ; O. ZERBO, Auteur ; Diana SCHENDEL, Auteur ; J. PINTO-MARTIN, Auteur ; M. DANIELE FALLIN, Auteur ; S. LEVY, Auteur ; Laura A. SCHIEVE, Auteur ; M. YEARGIN-ALLSOPP, Auteur ; Katherine R. SABOURIN, Auteur ; Jennifer L. AMES, Auteur Article en page(s) : p.1551-1561 Langues : Anglais (eng) Mots-clés : autism developmental disorder immune function infection neurodevelopment prenatal Index. décimale : PER Périodiques Résumé : Maternal infection and fever during pregnancy have been implicated in the etiology of autism spectrum disorder (ASD); however, studies have not been able to separate the effects of fever itself from the impact of a specific infectious organism on the developing brain. We utilized data from the Study to Explore Early Development (SEED), a case-control study among 2- to 5-year-old children born between 2003 and 2006 in the United States, to explore a possible association between maternal infection and fever during pregnancy and risk of ASD and other developmental disorders (DDs). Three groups of children were included: children with ASD (N = 606) and children with DDs (N = 856), ascertained from clinical and educational sources, and children from the general population (N = 796), randomly sampled from state birth records. Information about infection and fever during pregnancy was obtained from a telephone interview with the mother shortly after study enrollment and maternal prenatal and labor/delivery medical records. ASD and DD status was determined by an in-person standardized developmental assessment of the child at 3-5 years of age. After adjustment for covariates, maternal infection anytime during pregnancy was not associated with ASD or DDs. However, second trimester infection accompanied by fever elevated risk for ASD approximately twofold (aOR = 2.19, 95% confidence interval 1.14-4.23). These findings of an association between maternal infection with fever in the second trimester and increased risk of ASD in the offspring suggest that the inflammatory response to the infectious agent may be etiologically relevant. Autism Res 2019, 12: 1551-1561. (c) 2019 International Society for Autism Research, Wiley Periodicals, Inc. LAY SUMMARY: Using data from a large multisite study in the United States-the Study to Explore Early Development-we found that women who had an infection during the second trimester of pregnancy accompanied by a fever are more likely to have children with ASD. These findings suggest the possibility that only more severe infections accompanied by a robust inflammatory response increase the risk of ASD. En ligne : http://dx.doi.org/10.1002/aur.2175 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=408
in Autism Research > 12-10 (October 2019) . - p.1551-1561[article] Infection and Fever in Pregnancy and Autism Spectrum Disorders: Findings from the Study to Explore Early Development [Texte imprimé et/ou numérique] / Lisa A. CROEN, Auteur ; Y. QIAN, Auteur ; Paul ASHWOOD, Auteur ; O. ZERBO, Auteur ; Diana SCHENDEL, Auteur ; J. PINTO-MARTIN, Auteur ; M. DANIELE FALLIN, Auteur ; S. LEVY, Auteur ; Laura A. SCHIEVE, Auteur ; M. YEARGIN-ALLSOPP, Auteur ; Katherine R. SABOURIN, Auteur ; Jennifer L. AMES, Auteur . - p.1551-1561.
Langues : Anglais (eng)
in Autism Research > 12-10 (October 2019) . - p.1551-1561
Mots-clés : autism developmental disorder immune function infection neurodevelopment prenatal Index. décimale : PER Périodiques Résumé : Maternal infection and fever during pregnancy have been implicated in the etiology of autism spectrum disorder (ASD); however, studies have not been able to separate the effects of fever itself from the impact of a specific infectious organism on the developing brain. We utilized data from the Study to Explore Early Development (SEED), a case-control study among 2- to 5-year-old children born between 2003 and 2006 in the United States, to explore a possible association between maternal infection and fever during pregnancy and risk of ASD and other developmental disorders (DDs). Three groups of children were included: children with ASD (N = 606) and children with DDs (N = 856), ascertained from clinical and educational sources, and children from the general population (N = 796), randomly sampled from state birth records. Information about infection and fever during pregnancy was obtained from a telephone interview with the mother shortly after study enrollment and maternal prenatal and labor/delivery medical records. ASD and DD status was determined by an in-person standardized developmental assessment of the child at 3-5 years of age. After adjustment for covariates, maternal infection anytime during pregnancy was not associated with ASD or DDs. However, second trimester infection accompanied by fever elevated risk for ASD approximately twofold (aOR = 2.19, 95% confidence interval 1.14-4.23). These findings of an association between maternal infection with fever in the second trimester and increased risk of ASD in the offspring suggest that the inflammatory response to the infectious agent may be etiologically relevant. Autism Res 2019, 12: 1551-1561. (c) 2019 International Society for Autism Research, Wiley Periodicals, Inc. LAY SUMMARY: Using data from a large multisite study in the United States-the Study to Explore Early Development-we found that women who had an infection during the second trimester of pregnancy accompanied by a fever are more likely to have children with ASD. These findings suggest the possibility that only more severe infections accompanied by a robust inflammatory response increase the risk of ASD. En ligne : http://dx.doi.org/10.1002/aur.2175 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=408 Infections in children with autism spectrum disorder: Study to Explore Early Development (SEED) / Katherine R. SABOURIN in Autism Research, 12-1 (January 2019)
[article]
Titre : Infections in children with autism spectrum disorder: Study to Explore Early Development (SEED) Type de document : Texte imprimé et/ou numérique Auteurs : Katherine R. SABOURIN, Auteur ; A. REYNOLDS, Auteur ; Diana SCHENDEL, Auteur ; S. ROSENBERG, Auteur ; Lisa A. CROEN, Auteur ; J. A. PINTO-MARTIN, Auteur ; Laura A. SCHIEVE, Auteur ; C. NEWSCHAFFER, Auteur ; L. C. LEE, Auteur ; Carolyn G. DIGUISEPPI, Auteur Article en page(s) : p.136-146 Langues : Anglais (eng) Mots-clés : autism regression autism spectrum disorder childhood infection developmental disabilities temperature dysregulation Index. décimale : PER Périodiques Résumé : Immune system abnormalities have been widely reported among children with autism spectrum disorder (ASD), which may increase the risk of childhood infections. The Study to Explore Early Development (SEED) is a multisite case-control study of children aged 30-69 months, born in 2003-2006. Cases are children previously diagnosed and newly identified with ASD enrolled from education and clinical settings. Children with a previously diagnosed non-ASD developmental condition were included in the developmental delay/disorder (DD) control group. The population (POP) control group included children randomly sampled from birth certificates. Clinical illness from infection during the first 28 days ("neonatal," from medical records) and first three years of life (caregiver report) in cases was compared to DD and POP controls; and between cases with and without regression. Children with ASD had greater odds of neonatal (OR = 1.8; 95%CI: 1.1, 2.9) and early childhood infection (OR = 1.7; 95%CI: 1.5, 1.9) compared to POP children, and greater odds of neonatal infection (OR = 1.5; 95%CI: 1.1, 2.0) compared to DD children. Cases with regression had 1.6 times the odds (95%CI: 1.1, 2.3) of caregiver-reported infection during the first year of life compared to cases without regression, but neonatal infection risk and overall early childhood infection risk did not differ. Our results support the hypothesis that children with ASD are more likely to have infection early in life compared to the general population and to children with other developmental conditions. Future studies should examine the contributions of different causes, timing, frequency, and severity of infection to ASD risk. Autism Research 2019, 12: 136-146. (c) 2018 International Society for Autism Research, Wiley Periodicals, Inc. LAY SUMMARY: We looked at infections during early childhood in relation to autism spectrum disorder (ASD). We found that children with ASD were more likely to have an infection in the first 28 days of life and before age three compared to children with typical development. Children with ASD were also more likely than children with other developmental delays or disorders to have an infection in the first 28 days of life. En ligne : http://dx.doi.org/10.1002/aur.2012 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=376
in Autism Research > 12-1 (January 2019) . - p.136-146[article] Infections in children with autism spectrum disorder: Study to Explore Early Development (SEED) [Texte imprimé et/ou numérique] / Katherine R. SABOURIN, Auteur ; A. REYNOLDS, Auteur ; Diana SCHENDEL, Auteur ; S. ROSENBERG, Auteur ; Lisa A. CROEN, Auteur ; J. A. PINTO-MARTIN, Auteur ; Laura A. SCHIEVE, Auteur ; C. NEWSCHAFFER, Auteur ; L. C. LEE, Auteur ; Carolyn G. DIGUISEPPI, Auteur . - p.136-146.
Langues : Anglais (eng)
in Autism Research > 12-1 (January 2019) . - p.136-146
Mots-clés : autism regression autism spectrum disorder childhood infection developmental disabilities temperature dysregulation Index. décimale : PER Périodiques Résumé : Immune system abnormalities have been widely reported among children with autism spectrum disorder (ASD), which may increase the risk of childhood infections. The Study to Explore Early Development (SEED) is a multisite case-control study of children aged 30-69 months, born in 2003-2006. Cases are children previously diagnosed and newly identified with ASD enrolled from education and clinical settings. Children with a previously diagnosed non-ASD developmental condition were included in the developmental delay/disorder (DD) control group. The population (POP) control group included children randomly sampled from birth certificates. Clinical illness from infection during the first 28 days ("neonatal," from medical records) and first three years of life (caregiver report) in cases was compared to DD and POP controls; and between cases with and without regression. Children with ASD had greater odds of neonatal (OR = 1.8; 95%CI: 1.1, 2.9) and early childhood infection (OR = 1.7; 95%CI: 1.5, 1.9) compared to POP children, and greater odds of neonatal infection (OR = 1.5; 95%CI: 1.1, 2.0) compared to DD children. Cases with regression had 1.6 times the odds (95%CI: 1.1, 2.3) of caregiver-reported infection during the first year of life compared to cases without regression, but neonatal infection risk and overall early childhood infection risk did not differ. Our results support the hypothesis that children with ASD are more likely to have infection early in life compared to the general population and to children with other developmental conditions. Future studies should examine the contributions of different causes, timing, frequency, and severity of infection to ASD risk. Autism Research 2019, 12: 136-146. (c) 2018 International Society for Autism Research, Wiley Periodicals, Inc. LAY SUMMARY: We looked at infections during early childhood in relation to autism spectrum disorder (ASD). We found that children with ASD were more likely to have an infection in the first 28 days of life and before age three compared to children with typical development. Children with ASD were also more likely than children with other developmental delays or disorders to have an infection in the first 28 days of life. En ligne : http://dx.doi.org/10.1002/aur.2012 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=376 Investigation of Shifts in Autism Reporting in the California Department of Developmental Services / Judith K. GRETHER in Journal of Autism and Developmental Disorders, 39-10 (October 2009)
[article]
Titre : Investigation of Shifts in Autism Reporting in the California Department of Developmental Services Type de document : Texte imprimé et/ou numérique Auteurs : Judith K. GRETHER, Auteur ; Nila J. ROSEN, Auteur ; Karen S. SMITH, Auteur ; Lisa A. CROEN, Auteur Année de publication : 2009 Article en page(s) : p.1412-1419 Langues : Anglais (eng) Mots-clés : Autism Autism-reporting California-DDS-autism Index. décimale : PER Périodiques Résumé : We investigated if shifts in the coding of qualifying conditions in the California Department of Developmental Services (DDS) have contributed to the increase in California children with autism observed in recent years. Qualifying condition codes for mental retardation (MR) and autism in DDS electronic files were compared to hard-copy records for samples of children born 1987, 1990, 1994, and 1997. Contrary to expectations, we did not find evidence of a coding shift from “MR only” to “both MR and autism” or an increase in the proportion of children with coded autism who lacked supportive diagnostic documentation in records (possible “misclassifications”). These results indicate that changes in DDS coding practices are unlikely to explain the increase in DDS clients with autism. En ligne : http://dx.doi.org/10.1007/s10803-009-0754-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=839
in Journal of Autism and Developmental Disorders > 39-10 (October 2009) . - p.1412-1419[article] Investigation of Shifts in Autism Reporting in the California Department of Developmental Services [Texte imprimé et/ou numérique] / Judith K. GRETHER, Auteur ; Nila J. ROSEN, Auteur ; Karen S. SMITH, Auteur ; Lisa A. CROEN, Auteur . - 2009 . - p.1412-1419.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 39-10 (October 2009) . - p.1412-1419
Mots-clés : Autism Autism-reporting California-DDS-autism Index. décimale : PER Périodiques Résumé : We investigated if shifts in the coding of qualifying conditions in the California Department of Developmental Services (DDS) have contributed to the increase in California children with autism observed in recent years. Qualifying condition codes for mental retardation (MR) and autism in DDS electronic files were compared to hard-copy records for samples of children born 1987, 1990, 1994, and 1997. Contrary to expectations, we did not find evidence of a coding shift from “MR only” to “both MR and autism” or an increase in the proportion of children with coded autism who lacked supportive diagnostic documentation in records (possible “misclassifications”). These results indicate that changes in DDS coding practices are unlikely to explain the increase in DDS clients with autism. En ligne : http://dx.doi.org/10.1007/s10803-009-0754-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=839 Is Infertility Associated with Childhood Autism? / Judith K. GRETHER in Journal of Autism and Developmental Disorders, 43-3 (March 2013)
PermalinkMaternal and Paternal Infertility Disorders and Treatments and Autism Spectrum Disorder: Findings from the Study to Explore Early Development / Laura A. SCHIEVE in Journal of Autism and Developmental Disorders, 47-12 (December 2017)
PermalinkMaternal diabetes and hypertensive disorders in association with autism spectrum disorder / C. CORDERO in Autism Research, 12-6 (June 2019)
PermalinkMaternal Exposure to Occupational Asthmagens During Pregnancy and Autism Spectrum Disorder in the Study to Explore Early Development / Alison B. SINGER in Journal of Autism and Developmental Disorders, 46-11 (November 2016)
PermalinkMaternal Infection During Pregnancy and Autism Spectrum Disorders / Ousseny ZERBO in Journal of Autism and Developmental Disorders, 45-12 (December 2015)
PermalinkMaternal Pre-pregnancy Body Mass Index and Gestational Weight Gain in Relation to Autism Spectrum Disorder and other Developmental Disorders in Offspring / G. C. WINDHAM in Autism Research, 12-2 (February 2019)
PermalinkMaternal tobacco smoking and offspring autism spectrum disorder or traits in ECHO cohorts / Irva HERTZ-PICCIOTTO in Autism Research, 15-3 (March 2022)
PermalinkMaternal Vitamin D Levels During Pregnancy in Association With Autism Spectrum Disorders (ASD) or Intellectual Disability (ID) in Offspring; Exploring Non-linear Patterns and Demographic Sub-groups / Gayle C. WINDHAM in Autism Research, 13-12 (December 2020)
PermalinkMeconium androgens are correlated with ASD-related phenotypic traits in early childhood in a familial enriched risk cohort / Dina TERLOYEVA in Molecular Autism, 11 (2020)
PermalinkMedical Conditions in the First Years of Life Associated with Future Diagnosis of ASD in Children / Stacey E. ALEXEEFF in Journal of Autism and Developmental Disorders, 47-7 (July 2017)
PermalinkA meta-analysis of two high-risk prospective cohort studies reveals autism-specific transcriptional changes to chromatin, autoimmune, and environmental response genes in umbilical cord blood / C. E. MORDAUNT in Molecular Autism, 10 (2019)
PermalinkNeonatal Thyroid Stimulating Hormone and Subsequent Diagnosis of Autism Spectrum Disorders and Intellectual Disability / Jennifer L. AMES in Autism Research, 13-3 (March 2020)
PermalinkNeonatally measured immunoglobulins and risk of autism / Judith K. GRETHER in Autism Research, 3-6 (December 2010)
PermalinkNewborn vitamin D levels in relation to autism spectrum disorders and intellectual disability: A case-control study in california / G. C. WINDHAM in Autism Research, 12-6 (June 2019)
PermalinkOpportunities for Inclusion and Engagement in the Transition of Autistic Youth from Pediatric to Adult Healthcare: A Qualitative Study / Jennifer L. AMES in Journal of Autism and Developmental Disorders, 53-5 (May 2023)
PermalinkParental First Concerns and Timing of Autism Spectrum Disorder Diagnosis / Tracy A. BECERRA-CULQUI in Journal of Autism and Developmental Disorders, 48-10 (October 2018)
PermalinkPeri-Pregnancy Cannabis Use and Autism Spectrum Disorder in the Offspring: Findings from the Study to Explore Early Development / Carolyn G. DIGUISEPPI in Journal of Autism and Developmental Disorders, 52-11 (November 2022)
PermalinkPrenatal and Neonatal Thyroid Stimulating Hormone Levels and Autism Spectrum Disorders / Vincent M. YAU in Journal of Autism and Developmental Disorders, 45-3 (March 2015)
PermalinkPrenatal depression and risk of child autism-related traits among participants in the Environmental influences on Child Health Outcomes program / Lyndsay A. AVALOS in Autism Research, 16-9 (September 2023)
PermalinkPrenatal exposure to beta2-adrenergic receptor agonists and risk of autism spectrum disorders / Lisa A. CROEN in Journal of Neurodevelopmental Disorders, 3-4 (December 2011)
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