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Détail de l'auteur
Auteur Kate R. KUHLMAN |
Documents disponibles écrits par cet auteur (2)
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Facial emotion expression recognition by children at familial risk for depression: high-risk boys are oversensitive to sadness / Nestor L. LOPEZ-DURAN in Journal of Child Psychology and Psychiatry, 54-5 (May 2013)
[article]
Titre : Facial emotion expression recognition by children at familial risk for depression: high-risk boys are oversensitive to sadness Type de document : Texte imprimé et/ou numérique Auteurs : Nestor L. LOPEZ-DURAN, Auteur ; Kate R. KUHLMAN, Auteur ; Charles J. GEORGE, Auteur ; Maria KOVACS, Auteur Article en page(s) : p.565-574 Langues : Anglais (eng) Mots-clés : Facial expressions children depression information processing children of depressed parents Index. décimale : PER Périodiques Résumé : Background: Offspring of depressed parents are at greatly increased risk for mood disorders. Among potential mechanisms of risk, recent studies have focused on information processing anomalies, such as attention and memory biases, in the offspring of depressed parents. In this study we examined another information processing domain, perceptual sensitivity to emotion cues in facial expressions, as a potential mechanism of risk that characterizes the offspring of depressed parents. Methods: The study included 64 children at familial-risk for depression and 40 low-risk peers between the ages 7 and 13(Mage = 9.51; SD = 2.27). Participants were presented with pictures of facial expressions that varied in emotional intensity from neutral to full-intensity sadness or anger (i.e., emotion recognition), or pictures of faces morphing from anger to sadness (emotion discrimination). After each picture was presented, children indicated whether the face showed a specific emotion (i.e., sadness, anger) or no emotion at all (neutral) using a forced choice paradigm. We examined group differences in the intensity of emotion that suggested greater sensitivity to specific emotions. Results: In the emotion recognition task, boys (but not girls) at familial-risk for depression identified sadness at significantly lower levels of emotional intensity than did their low-risk peers. The high and low-risk groups did not differ with regard to identification of anger. In the emotion discrimination task, both groups displayed over-identification of sadness in ambiguous mixed faces but high-risk youth were less likely to show this labeling bias than their peers. Conclusion: Our findings are consistent with the hypothesis that enhanced perceptual sensitivity to subtle traces of sadness in facial expressions may be a potential mechanism of risk among boys at familial-risk for depression. This enhanced perceptual sensitivity does not appear to be due to biases in the labeling of ambiguous faces. En ligne : http://dx.doi.org/10.1111/jcpp.12005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=196
in Journal of Child Psychology and Psychiatry > 54-5 (May 2013) . - p.565-574[article] Facial emotion expression recognition by children at familial risk for depression: high-risk boys are oversensitive to sadness [Texte imprimé et/ou numérique] / Nestor L. LOPEZ-DURAN, Auteur ; Kate R. KUHLMAN, Auteur ; Charles J. GEORGE, Auteur ; Maria KOVACS, Auteur . - p.565-574.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 54-5 (May 2013) . - p.565-574
Mots-clés : Facial expressions children depression information processing children of depressed parents Index. décimale : PER Périodiques Résumé : Background: Offspring of depressed parents are at greatly increased risk for mood disorders. Among potential mechanisms of risk, recent studies have focused on information processing anomalies, such as attention and memory biases, in the offspring of depressed parents. In this study we examined another information processing domain, perceptual sensitivity to emotion cues in facial expressions, as a potential mechanism of risk that characterizes the offspring of depressed parents. Methods: The study included 64 children at familial-risk for depression and 40 low-risk peers between the ages 7 and 13(Mage = 9.51; SD = 2.27). Participants were presented with pictures of facial expressions that varied in emotional intensity from neutral to full-intensity sadness or anger (i.e., emotion recognition), or pictures of faces morphing from anger to sadness (emotion discrimination). After each picture was presented, children indicated whether the face showed a specific emotion (i.e., sadness, anger) or no emotion at all (neutral) using a forced choice paradigm. We examined group differences in the intensity of emotion that suggested greater sensitivity to specific emotions. Results: In the emotion recognition task, boys (but not girls) at familial-risk for depression identified sadness at significantly lower levels of emotional intensity than did their low-risk peers. The high and low-risk groups did not differ with regard to identification of anger. In the emotion discrimination task, both groups displayed over-identification of sadness in ambiguous mixed faces but high-risk youth were less likely to show this labeling bias than their peers. Conclusion: Our findings are consistent with the hypothesis that enhanced perceptual sensitivity to subtle traces of sadness in facial expressions may be a potential mechanism of risk among boys at familial-risk for depression. This enhanced perceptual sensitivity does not appear to be due to biases in the labeling of ambiguous faces. En ligne : http://dx.doi.org/10.1111/jcpp.12005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=196 Sleep problems in adolescence are prospectively linked to later depressive symptoms via the cortisol awakening response / Kate R. KUHLMAN in Development and Psychopathology, 32-3 (August 2020)
[article]
Titre : Sleep problems in adolescence are prospectively linked to later depressive symptoms via the cortisol awakening response Type de document : Texte imprimé et/ou numérique Auteurs : Kate R. KUHLMAN, Auteur ; Jessica J. CHIANG, Auteur ; Julienne E. BOWER, Auteur ; Michael R. IRWIN, Auteur ; Teresa E. SEEMAN, Auteur ; Heather E. MCCREATH, Auteur ; David M. ALMEIDA, Auteur ; Ronald E. DAHL, Auteur ; Andrew J. FULIGNI, Auteur Article en page(s) : p.997-1006 Langues : Anglais (eng) Mots-clés : HPA axis adolescence cortisol awakening response depression sleep Index. décimale : PER Périodiques Résumé : Sleep disturbance is a symptom of and a well-known risk factor for depression. Further, atypical functioning of the HPA axis has been linked to the pathogenesis of depression. The purpose of this study was to examine the role of adolescent HPA axis functioning in the link between adolescent sleep problems and later depressive symptoms. Methods: A sample of 157 17-18 year old adolescents (61.8% female) completed the Pittsburgh Sleep Quality Inventory (PSQI) and provided salivary cortisol samples throughout the day for three consecutive days. Two years later, adolescents reported their depressive symptoms via the Center for Epidemiological Studies Depression Scale (CES-D). Results: Individuals (age 17-18) with greater sleep disturbance reported greater depressive symptoms two years later (age 19-20). This association occurred through the indirect effect of sleep disturbance on the cortisol awakening response (CAR) (indirect effect = 0.14, 95%CI [.02 -.39]). Conclusions: One pathway through which sleep problems may lead to depressive symptoms is by up-regulating components of the body's physiological stress response system that can be measured through the cortisol awakening response. Behavioral interventions that target sleep disturbance in adolescents may mitigate this neurobiological pathway to depression during this high-risk developmental phase. En ligne : http://dx.doi.org/10.1017/s0954579419000762 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=429
in Development and Psychopathology > 32-3 (August 2020) . - p.997-1006[article] Sleep problems in adolescence are prospectively linked to later depressive symptoms via the cortisol awakening response [Texte imprimé et/ou numérique] / Kate R. KUHLMAN, Auteur ; Jessica J. CHIANG, Auteur ; Julienne E. BOWER, Auteur ; Michael R. IRWIN, Auteur ; Teresa E. SEEMAN, Auteur ; Heather E. MCCREATH, Auteur ; David M. ALMEIDA, Auteur ; Ronald E. DAHL, Auteur ; Andrew J. FULIGNI, Auteur . - p.997-1006.
Langues : Anglais (eng)
in Development and Psychopathology > 32-3 (August 2020) . - p.997-1006
Mots-clés : HPA axis adolescence cortisol awakening response depression sleep Index. décimale : PER Périodiques Résumé : Sleep disturbance is a symptom of and a well-known risk factor for depression. Further, atypical functioning of the HPA axis has been linked to the pathogenesis of depression. The purpose of this study was to examine the role of adolescent HPA axis functioning in the link between adolescent sleep problems and later depressive symptoms. Methods: A sample of 157 17-18 year old adolescents (61.8% female) completed the Pittsburgh Sleep Quality Inventory (PSQI) and provided salivary cortisol samples throughout the day for three consecutive days. Two years later, adolescents reported their depressive symptoms via the Center for Epidemiological Studies Depression Scale (CES-D). Results: Individuals (age 17-18) with greater sleep disturbance reported greater depressive symptoms two years later (age 19-20). This association occurred through the indirect effect of sleep disturbance on the cortisol awakening response (CAR) (indirect effect = 0.14, 95%CI [.02 -.39]). Conclusions: One pathway through which sleep problems may lead to depressive symptoms is by up-regulating components of the body's physiological stress response system that can be measured through the cortisol awakening response. Behavioral interventions that target sleep disturbance in adolescents may mitigate this neurobiological pathway to depression during this high-risk developmental phase. En ligne : http://dx.doi.org/10.1017/s0954579419000762 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=429