Continuities and discontinuities in psychopathology between childhood and adult life / Michael RUTTER in Journal of Child Psychology and Psychiatry, 47-3/4 (March/April 2006)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 47-3/4 (March/April 2006) . - p.276–295 Titre : Continuities and discontinuities in psychopathology between childhood and adult life Type de document : Texte imprimé et/ou numérique Auteurs : Michael RUTTER, Auteur ; Julia KIM-COHEN, Auteur ; Barbara MAUGHAN, Auteur Année de publication : 2006 Article en page(s) : p.276–295 Langues : Anglais (eng) Mots-clés : Heterotypic-continuity precursors/prodromata age-at-onset genetics adolescence Index. décimale : PER Périodiques Résumé : The possible mechanisms involved in continuities and discontinuities in psychopathology between childhood and adult life are considered in relation to the findings from systematic, prospective, long-term longitudinal studies. Findings on schizophrenia, neurodevelopmental disorders, emotional disturbances, antisocial behaviour and substance abuse are used as conditions illustrating the key issues. The overarching themes are then discussed in relation to heterotypic continuity and psychopathologic progression, early age at onset and a range of possible mediating mechanisms – including genetic mediation, 'kindling' effects, environmental influences, coping mechanisms and cognitive processing of experiences. Some of the key research challenges that remain concern the testing of competing hypotheses on mediating processes, the changes involved in adolescence, the transition from prodromal phase to overt schizophrenia and the emergence of adolescent-limited antisocial behaviour. Greater use needs to be made of genetic research strategies and of the testing of possible cognitive processing mediation effects. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01614.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=722 [article] Continuities and discontinuities in psychopathology between childhood and adult life [Texte imprimé et/ou numérique] / Michael RUTTER, Auteur ; Julia KIM-COHEN, Auteur ; Barbara MAUGHAN, Auteur . - 2006 . - p.276–295. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 47-3/4 (March/April 2006) . - p.276–295 Mots-clés : Heterotypic-continuity precursors/prodromata age-at-onset genetics adolescence Index. décimale : PER Périodiques Résumé : The possible mechanisms involved in continuities and discontinuities in psychopathology between childhood and adult life are considered in relation to the findings from systematic, prospective, long-term longitudinal studies. Findings on schizophrenia, neurodevelopmental disorders, emotional disturbances, antisocial behaviour and substance abuse are used as conditions illustrating the key issues. The overarching themes are then discussed in relation to heterotypic continuity and psychopathologic progression, early age at onset and a range of possible mediating mechanisms – including genetic mediation, 'kindling' effects, environmental influences, coping mechanisms and cognitive processing of experiences. Some of the key research challenges that remain concern the testing of competing hypotheses on mediating processes, the changes involved in adolescence, the transition from prodromal phase to overt schizophrenia and the emergence of adolescent-limited antisocial behaviour. Greater use needs to be made of genetic research strategies and of the testing of possible cognitive processing mediation effects. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2006.01614.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=722

Meta-analysis of gene–environment interactions in developmental psychopathology / Alan TAYLOR in Development and Psychopathology, 19-4 (Fall 2007)  

Public ISBD [article]  inDevelopment and Psychopathology > 19-4 (Fall 2007) . - p.1029-1037 Titre : Meta-analysis of gene–environment interactions in developmental psychopathology Type de document : Texte imprimé et/ou numérique Auteurs : Alan TAYLOR, Auteur ; Julia KIM-COHEN, Auteur Année de publication : 2007 Article en page(s) : p.1029-1037 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : As studies of measured gene–environment interactions (G × E) in developmental psychopathology gain momentum, methods for systematically and quantitatively summarizing effects across multiple studies are urgently needed. Meta-analyses of G × E findings are critical for evaluating the overall statistical and theoretical significance of any given G × E based on cumulative and systematically combined knowledge. Although meta-analytic methods for the combination of study findings based on single effect measures such as odds ratios and mean differences are well established, equivalent methods for the meta-analysis of studies investigating interactions are not well developed. This article describes one simple approach to the meta-analysis of G × E effects using, as a contemporaneous example, the interaction of the monoamine oxidase A (MAOA) gene and the impact of childhood maltreatment on risk for developing antisocial behavior. En ligne : http://dx.doi.org/10.1017/s095457940700051x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=181 [article] Meta-analysis of gene–environment interactions in developmental psychopathology [Texte imprimé et/ou numérique] / Alan TAYLOR, Auteur ; Julia KIM-COHEN, Auteur . - 2007 . - p.1029-1037. Langues : Anglais (eng) in Development and Psychopathology > 19-4 (Fall 2007) . - p.1029-1037 Index. décimale : PER Périodiques Résumé : As studies of measured gene–environment interactions (G × E) in developmental psychopathology gain momentum, methods for systematically and quantitatively summarizing effects across multiple studies are urgently needed. Meta-analyses of G × E findings are critical for evaluating the overall statistical and theoretical significance of any given G × E based on cumulative and systematically combined knowledge. Although meta-analytic methods for the combination of study findings based on single effect measures such as odds ratios and mean differences are well established, equivalent methods for the meta-analysis of studies investigating interactions are not well developed. This article describes one simple approach to the meta-analysis of G × E effects using, as a contemporaneous example, the interaction of the monoamine oxidase A (MAOA) gene and the impact of childhood maltreatment on risk for developing antisocial behavior. En ligne : http://dx.doi.org/10.1017/s095457940700051x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=181

Research Review: DSM-V conduct disorder: research needs for an evidence base / Terrie E. MOFFITT in Journal of Child Psychology and Psychiatry, 49-1 (January 2008)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 49-1 (January 2008) . - p.3–33 Titre : Research Review: DSM-V conduct disorder: research needs for an evidence base Type de document : Texte imprimé et/ou numérique Auteurs : Terrie E. MOFFITT, Auteur ; Julia KIM-COHEN, Auteur ; Essi VIDING, Auteur ; Sara R. JAFFEE, Auteur ; Louise ARSENEAULT, Auteur ; Karestan C. KOENEN, Auteur ; Candice L. ODGERS, Auteur ; Wendy S. SLUTSKE, Auteur Année de publication : 2007 Article en page(s) : p.3–33 Langues : Anglais (eng) Mots-clés : Conduct-disorder DSM-V Index. décimale : PER Périodiques Résumé : This article charts a strategic research course toward an empirical foundation for the diagnosis of conduct disorder in the forthcoming DSM-V. Since the DSM-IV appeared in 1994, an impressive amount of new information about conduct disorder has emerged. As a result of this new knowledge, reasonable rationales have been put forward for adding to the conduct disorder diagnostic protocol: a childhood-limited subtype, family psychiatric history, callous-unemotional traits, female-specific criteria, preschool-specific criteria, early substance use, and biomarkers from genetics, neuroimaging, and physiology research. This article reviews the evidence for these and other potential changes to the conduct disorder diagnosis. We report that although there is a great deal of exciting research into each of the topics, very little of it provides the precise sort of evidence base required to justify any alteration to the DSM-V. We outline specific research questions and study designs needed to build the lacking evidence base for or against proposed changes to DSM-V conduct disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2007.01823.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=310 [article] Research Review: DSM-V conduct disorder: research needs for an evidence base [Texte imprimé et/ou numérique] / Terrie E. MOFFITT, Auteur ; Julia KIM-COHEN, Auteur ; Essi VIDING, Auteur ; Sara R. JAFFEE, Auteur ; Louise ARSENEAULT, Auteur ; Karestan C. KOENEN, Auteur ; Candice L. ODGERS, Auteur ; Wendy S. SLUTSKE, Auteur . - 2007 . - p.3–33. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 49-1 (January 2008) . - p.3–33 Mots-clés : Conduct-disorder DSM-V Index. décimale : PER Périodiques Résumé : This article charts a strategic research course toward an empirical foundation for the diagnosis of conduct disorder in the forthcoming DSM-V. Since the DSM-IV appeared in 1994, an impressive amount of new information about conduct disorder has emerged. As a result of this new knowledge, reasonable rationales have been put forward for adding to the conduct disorder diagnostic protocol: a childhood-limited subtype, family psychiatric history, callous-unemotional traits, female-specific criteria, preschool-specific criteria, early substance use, and biomarkers from genetics, neuroimaging, and physiology research. This article reviews the evidence for these and other potential changes to the conduct disorder diagnosis. We report that although there is a great deal of exciting research into each of the topics, very little of it provides the precise sort of evidence base required to justify any alteration to the DSM-V. We outline specific research questions and study designs needed to build the lacking evidence base for or against proposed changes to DSM-V conduct disorder. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2007.01823.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=310

Resilience and measured gene–environment interactions / Julia KIM-COHEN in Development and Psychopathology, 24-4 (November 2012)  

Public ISBD [article]  inDevelopment and Psychopathology > 24-4 (November 2012) . - p.1297-1306 Titre : Resilience and measured gene–environment interactions Type de document : Texte imprimé et/ou numérique Auteurs : Julia KIM-COHEN, Auteur ; Rebecca TURKEWITZ, Auteur Année de publication : 2012 Article en page(s) : p.1297-1306 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The past decade has witnessed an exponential growth in studies that have attempted to identify the genetic polymporphisms that moderate the influence of environmental risks on mental disorders. What tends to be neglected in these Gene × Environment (G × E) interaction studies has been a focus on resilience, which refers to a dynamic pattern of positive adaptation despite the experience of a significant trauma or adversity. In this article, we argue that one step toward advancing the field of developmental psychopathology would be for G × E research to consider resilience instead of focusing almost exclusively on mental disorders. After providing an up-to-date summary on the expanding definitions and models of resilience, and the available evidence regarding measured G × E studies of childhood maltreatment, we discuss why resilience would be a worthwhile phenotype for studies of measured G × E. First, although G × E hypotheses require that there be an environmental risk (e-risk) involved in a causal process that leads to psychopathology, e-risks are typically not included in the diagnostic criteria for most psychiatric disorders. In contrast, resilience by definition includes an e-risk. Second, G × E hypotheses require that there is evidence of variability in response to an environmental stressor, and resilience often represents the positive end on this continuum of adaptation. Third, both resilience and G × E are best understood from a developmental perspective. Fourth, although resilient outcomes are not public health concerns, the types of adversities (e.g., childhood maltreatment, poverty, or exposure to natural disasters) that are often investigated in studies of resilience certainly are. Understanding how some individuals, perhaps because of their genetic makeup, are able to withstand such adversities can inform prevention and intervention efforts to improve mental health. En ligne : http://dx.doi.org/10.1017/S0954579412000715 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182 [article] Resilience and measured gene–environment interactions [Texte imprimé et/ou numérique] / Julia KIM-COHEN, Auteur ; Rebecca TURKEWITZ, Auteur . - 2012 . - p.1297-1306. Langues : Anglais (eng) in Development and Psychopathology > 24-4 (November 2012) . - p.1297-1306 Index. décimale : PER Périodiques Résumé : The past decade has witnessed an exponential growth in studies that have attempted to identify the genetic polymporphisms that moderate the influence of environmental risks on mental disorders. What tends to be neglected in these Gene × Environment (G × E) interaction studies has been a focus on resilience, which refers to a dynamic pattern of positive adaptation despite the experience of a significant trauma or adversity. In this article, we argue that one step toward advancing the field of developmental psychopathology would be for G × E research to consider resilience instead of focusing almost exclusively on mental disorders. After providing an up-to-date summary on the expanding definitions and models of resilience, and the available evidence regarding measured G × E studies of childhood maltreatment, we discuss why resilience would be a worthwhile phenotype for studies of measured G × E. First, although G × E hypotheses require that there be an environmental risk (e-risk) involved in a causal process that leads to psychopathology, e-risks are typically not included in the diagnostic criteria for most psychiatric disorders. In contrast, resilience by definition includes an e-risk. Second, G × E hypotheses require that there is evidence of variability in response to an environmental stressor, and resilience often represents the positive end on this continuum of adaptation. Third, both resilience and G × E are best understood from a developmental perspective. Fourth, although resilient outcomes are not public health concerns, the types of adversities (e.g., childhood maltreatment, poverty, or exposure to natural disasters) that are often investigated in studies of resilience certainly are. Understanding how some individuals, perhaps because of their genetic makeup, are able to withstand such adversities can inform prevention and intervention efforts to improve mental health. En ligne : http://dx.doi.org/10.1017/S0954579412000715 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=182

The development of the person: The Minnesota Study of Risk and Adaptation from Birth to Adulthood / Julia KIM-COHEN in Journal of Child Psychology and Psychiatry, 49-6 (June 2008)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 49-6 (June 2008) . - p.686 Titre : The development of the person: The Minnesota Study of Risk and Adaptation from Birth to Adulthood Type de document : Texte imprimé et/ou numérique Auteurs : Julia KIM-COHEN, Auteur Année de publication : 2008 Article en page(s) : p.686 Langues : Anglais (eng) Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/j.1469-7610.2007.01794.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 [article] The development of the person: The Minnesota Study of Risk and Adaptation from Birth to Adulthood [Texte imprimé et/ou numérique] / Julia KIM-COHEN, Auteur . - 2008 . - p.686. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 49-6 (June 2008) . - p.686 Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/j.1469-7610.2007.01794.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457

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