Altered neural response to rejection-related words in children exposed to maltreatment / Vanessa B. PUETZ in Journal of Child Psychology and Psychiatry, 57-10 (October 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-10 (October 2016) . - p.1165-1173 Titre : Altered neural response to rejection-related words in children exposed to maltreatment Type de document : Texte imprimé et/ou numérique Auteurs : Vanessa B. PUETZ, Auteur ; Essi VIDING, Auteur ; Amy L. PALMER, Auteur ; Philip A. KELLY, Auteur ; Rachael LICKLEY, Auteur ; Iakovina KOUTOUFA, Auteur ; Catherine L. SEBASTIAN, Auteur ; Eamon J. MCCRORY, Auteur Article en page(s) : p.1165-1173 Langues : Anglais (eng) Mots-clés : Child abuse  emotion regulation  fMRI  posttraumatic stress disorder  adolescence Index. décimale : PER Périodiques Résumé : Background Children exposed to maltreatment show neural sensitivity to facial cues signalling threat. However, little is known about how maltreatment influences the processing of social threat cues more broadly, and whether atypical processing of social threat cues relates to psychiatric risk. Methods Forty-one 10- to 14-year-old children underwent a social rejection-themed emotional Stroop task during functional magnetic resonance imaging: 21 children with a documented history of maltreatment (11 F) and 19 comparison children with no maltreatment history (11 F). Groups were matched on age, pubertal status, gender, IQ, socioeconomic status, ethnicity and reading ability. Classic colour Stroop stimuli were also administered in the same paradigm to investigate potential differences in general cognitive control. Results Compared with their peers, children who had experienced maltreatment showed reduced activation in the Rejection versus Neutral condition, across circuitry previously implicated in abuse-related posttraumatic stress disorder (PTSD), including the left anterior insula, extending into left ventrolateral prefrontal cortex/orbitofrontal cortex; left amygdala; left inferior parietal cortex (STS); and bilateral visual association cortex, encompassing the cuneus and lingual gyrus. No group differences in neural or behavioural responses were found for the classic colour Stroop conditions. Significant negative associations between activity in bilateral cuneus and STS during the rejection-themed Stroop and higher self-reported PTSD symptomatology, including dissociation, were observed in children exposed to maltreatment. Conclusion Our findings indicate a pattern of altered neural response to social rejection cues in maltreated children. Compared to their peers, these children displayed relative hypoactivation to rejection cues in regions previously associated with PTSD, potentially reflecting an avoidant coping response. It is suggested that such atypical processing of social threat may index latent vulnerability to future psychopathology in general and PTSD in particular. En ligne : http://dx.doi.org/10.1111/jcpp.12595 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=295 [article] Altered neural response to rejection-related words in children exposed to maltreatment [Texte imprimé et/ou numérique] / Vanessa B. PUETZ, Auteur ; Essi VIDING, Auteur ; Amy L. PALMER, Auteur ; Philip A. KELLY, Auteur ; Rachael LICKLEY, Auteur ; Iakovina KOUTOUFA, Auteur ; Catherine L. SEBASTIAN, Auteur ; Eamon J. MCCRORY, Auteur . - p.1165-1173. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-10 (October 2016) . - p.1165-1173 Mots-clés : Child abuse  emotion regulation  fMRI  posttraumatic stress disorder  adolescence Index. décimale : PER Périodiques Résumé : Background Children exposed to maltreatment show neural sensitivity to facial cues signalling threat. However, little is known about how maltreatment influences the processing of social threat cues more broadly, and whether atypical processing of social threat cues relates to psychiatric risk. Methods Forty-one 10- to 14-year-old children underwent a social rejection-themed emotional Stroop task during functional magnetic resonance imaging: 21 children with a documented history of maltreatment (11 F) and 19 comparison children with no maltreatment history (11 F). Groups were matched on age, pubertal status, gender, IQ, socioeconomic status, ethnicity and reading ability. Classic colour Stroop stimuli were also administered in the same paradigm to investigate potential differences in general cognitive control. Results Compared with their peers, children who had experienced maltreatment showed reduced activation in the Rejection versus Neutral condition, across circuitry previously implicated in abuse-related posttraumatic stress disorder (PTSD), including the left anterior insula, extending into left ventrolateral prefrontal cortex/orbitofrontal cortex; left amygdala; left inferior parietal cortex (STS); and bilateral visual association cortex, encompassing the cuneus and lingual gyrus. No group differences in neural or behavioural responses were found for the classic colour Stroop conditions. Significant negative associations between activity in bilateral cuneus and STS during the rejection-themed Stroop and higher self-reported PTSD symptomatology, including dissociation, were observed in children exposed to maltreatment. Conclusion Our findings indicate a pattern of altered neural response to social rejection cues in maltreated children. Compared to their peers, these children displayed relative hypoactivation to rejection cues in regions previously associated with PTSD, potentially reflecting an avoidant coping response. It is suggested that such atypical processing of social threat may index latent vulnerability to future psychopathology in general and PTSD in particular. En ligne : http://dx.doi.org/10.1111/jcpp.12595 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=295

Annual Research Review: Childhood maltreatment, latent vulnerability and the shift to preventative psychiatry – the contribution of functional brain imaging / Eamon J. MCCRORY in Journal of Child Psychology and Psychiatry, 58-4 (April 2017)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 58-4 (April 2017) . - p.338-357 Titre : Annual Research Review: Childhood maltreatment, latent vulnerability and the shift to preventative psychiatry – the contribution of functional brain imaging Type de document : Texte imprimé et/ou numérique Auteurs : Eamon J. MCCRORY, Auteur ; Mattia I. GERIN, Auteur ; Essi VIDING, Auteur Article en page(s) : p.338-357 Langues : Anglais (eng) Mots-clés : Child abuse  maltreatment  mental health  functional magnetic resonance imaging  resilience Index. décimale : PER Périodiques Résumé : Background Childhood maltreatment is a potent predictor of poor mental health across the life span. We argue that there is a need to improve the understanding of the mechanisms that confer psychiatric vulnerability following maltreatment, if we are to progress from simply treating those with a manifest disorder, to developing effective preventative approaches that can help offset the likelihood that such disorders will emerge in the first place. Methods We review extant functional neuroimaging studies of children and adolescents exposed to early neglect and/or maltreatment, including physical, sexual and emotional abuse across four neurocognitive domains: threat processing, reward processing, emotion regulation and executive control. Findings are discussed in the context of ‘latent vulnerability’, where alterations in neurocognitive function are considered to carry adaptive value in early adverse caregiving environments but confer long-term risk. Results Studies on threat processing indicate heightened as well as depressed neural responsiveness in maltreated samples, particularly in the amygdala, thought to reflect threat hypervigilance and avoidance respectively. Studies on reward processing generally report blunted neural response to anticipation and receipt of rewards, particularly in the striatum, patterns associated with depressive symptomatology. Studies on emotion regulation report increased activation of the anterior cingulate cortex (ACC) during active emotion regulation, possibly reflecting greater effortful processing. Finally, studies of executive control report increased dorsal ACC activity during error monitoring and inhibition. Conclusions An emerging body of work indicates that altered neurocognitive functioning following maltreatment: (a) is evident even in the absence of overt psychopathology; (b) is consistent with perturbations seen in individuals presenting with psychiatric disorder; (c) can predict future psychiatric symptomatology. These findings suggest that maltreatment leads to neurocognitive alterations that embed latent vulnerability to psychiatric disorder, establishing a compelling case for identifying those children at most risk and developing mechanistically informed models of preventative intervention. Such interventions should aim to offset the likelihood of any future psychiatric disorder. En ligne : http://dx.doi.org/10.1111/jcpp.12713 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=305 [article] Annual Research Review: Childhood maltreatment, latent vulnerability and the shift to preventative psychiatry – the contribution of functional brain imaging [Texte imprimé et/ou numérique] / Eamon J. MCCRORY, Auteur ; Mattia I. GERIN, Auteur ; Essi VIDING, Auteur . - p.338-357. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 58-4 (April 2017) . - p.338-357 Mots-clés : Child abuse  maltreatment  mental health  functional magnetic resonance imaging  resilience Index. décimale : PER Périodiques Résumé : Background Childhood maltreatment is a potent predictor of poor mental health across the life span. We argue that there is a need to improve the understanding of the mechanisms that confer psychiatric vulnerability following maltreatment, if we are to progress from simply treating those with a manifest disorder, to developing effective preventative approaches that can help offset the likelihood that such disorders will emerge in the first place. Methods We review extant functional neuroimaging studies of children and adolescents exposed to early neglect and/or maltreatment, including physical, sexual and emotional abuse across four neurocognitive domains: threat processing, reward processing, emotion regulation and executive control. Findings are discussed in the context of ‘latent vulnerability’, where alterations in neurocognitive function are considered to carry adaptive value in early adverse caregiving environments but confer long-term risk. Results Studies on threat processing indicate heightened as well as depressed neural responsiveness in maltreated samples, particularly in the amygdala, thought to reflect threat hypervigilance and avoidance respectively. Studies on reward processing generally report blunted neural response to anticipation and receipt of rewards, particularly in the striatum, patterns associated with depressive symptomatology. Studies on emotion regulation report increased activation of the anterior cingulate cortex (ACC) during active emotion regulation, possibly reflecting greater effortful processing. Finally, studies of executive control report increased dorsal ACC activity during error monitoring and inhibition. Conclusions An emerging body of work indicates that altered neurocognitive functioning following maltreatment: (a) is evident even in the absence of overt psychopathology; (b) is consistent with perturbations seen in individuals presenting with psychiatric disorder; (c) can predict future psychiatric symptomatology. These findings suggest that maltreatment leads to neurocognitive alterations that embed latent vulnerability to psychiatric disorder, establishing a compelling case for identifying those children at most risk and developing mechanistically informed models of preventative intervention. Such interventions should aim to offset the likelihood of any future psychiatric disorder. En ligne : http://dx.doi.org/10.1111/jcpp.12713 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=305

Antisocial behavior from a developmental psychopathology perspective / Paul J. FRICK in Development and Psychopathology, 21-4 (November 2009)  

Public ISBD [article]  inDevelopment and Psychopathology > 21-4 (November 2009) . - p.1111-1131 Titre : Antisocial behavior from a developmental psychopathology perspective Type de document : Texte imprimé et/ou numérique Auteurs : Paul J. FRICK, Auteur ; Essi VIDING, Auteur Année de publication : 2009 Article en page(s) : p.1111-1131 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : This paper reviews research on chronic patterns of antisocial behavior and places this research into a developmental psychopathology framework. Specifically, research suggests that there are at least three important pathways through which children and adolescents can develop severe antisocial behaviors. One group of youth shows antisocial behavior that begins in adolescence, and two groups show antisocial behavior that begins in childhood but differ on the presence or absence of callous–unemotional traits. In outlining these distinct pathways to antisocial behavior, we have tried to illustrate some key concepts from developmental psychopathology such as equifinality and multifinality, the importance of understanding the interface between normal and abnormal development, and the importance of using multiple levels of analyses to advance causal theories. Finally, we discuss how this development model can be used to enhance existing interventions for antisocial individuals. En ligne : http://dx.doi.org/10.1017/s0954579409990071 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=846 [article] Antisocial behavior from a developmental psychopathology perspective [Texte imprimé et/ou numérique] / Paul J. FRICK, Auteur ; Essi VIDING, Auteur . - 2009 . - p.1111-1131. Langues : Anglais (eng) in Development and Psychopathology > 21-4 (November 2009) . - p.1111-1131 Index. décimale : PER Périodiques Résumé : This paper reviews research on chronic patterns of antisocial behavior and places this research into a developmental psychopathology framework. Specifically, research suggests that there are at least three important pathways through which children and adolescents can develop severe antisocial behaviors. One group of youth shows antisocial behavior that begins in adolescence, and two groups show antisocial behavior that begins in childhood but differ on the presence or absence of callous–unemotional traits. In outlining these distinct pathways to antisocial behavior, we have tried to illustrate some key concepts from developmental psychopathology such as equifinality and multifinality, the importance of understanding the interface between normal and abnormal development, and the importance of using multiple levels of analyses to advance causal theories. Finally, we discuss how this development model can be used to enhance existing interventions for antisocial individuals. En ligne : http://dx.doi.org/10.1017/s0954579409990071 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=846

Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort / Christina O. CARLISI in Development and Psychopathology, 34-5 (December 2022)  

Public ISBD [article]  inDevelopment and Psychopathology > 34-5 (December 2022) . - p.2012-2022 Titre : Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort Type de document : Texte imprimé et/ou numérique Auteurs : Christina O. CARLISI, Auteur ; Terrie E. MOFFITT, Auteur ; Annchen R. KNODT, Auteur ; Honalee HARRINGTON, Auteur ; Stéphanie LANGEVIN, Auteur ; David IRELAND, Auteur ; Tracy R. MELZER, Auteur ; Richie POULTON, Auteur ; Sandhya RAMRAKHA, Auteur ; Avshalom CASPI, Auteur ; Ahmad R. HARIRI, Auteur ; Essi VIDING, Auteur Article en page(s) : p.2012-2022 Langues : Anglais (eng) Mots-clés : antisocial behavior  conduct disorder  development  longitudinal  structural MRI Index. décimale : PER Périodiques Résumé : Neuropsychological evidence supports the developmental taxonomy theory of antisocial behavior, suggesting that abnormal brain development distinguishes life-course-persistent from adolescence-limited antisocial behavior. Recent neuroimaging work confirmed that prospectively-measured life-course-persistent antisocial behavior is associated with differences in cortical brain structure. Whether this extends to subcortical brain structures remains uninvestigated. This study compared subcortical gray-matter volumes between 672 members of the Dunedin Study previously defined as exhibiting life-course-persistent, adolescence-limited or low-level antisocial behavior based on repeated assessments at ages 7 “26 years. Gray-matter volumes of 10 subcortical structures were compared across groups. The life-course-persistent group had lower volumes of amygdala, brain stem, cerebellum, hippocampus, pallidum, thalamus, and ventral diencephalon compared to the low-antisocial group. Differences between life-course-persistent and adolescence-limited individuals were comparable in effect size to differences between life-course-persistent and low-antisocial individuals, but were not statistically significant due to less statistical power. Gray-matter volumes in adolescence-limited individuals were near the norm in this population-representative cohort and similar to volumes in low-antisocial individuals. Although this study could not establish causal links between brain volume and antisocial behavior, it constitutes new biological evidence that all people with antisocial behavior are not the same, supporting a need for greater developmental and diagnostic precision in clinical, forensic, and policy-based interventions. En ligne : http://dx.doi.org/10.1017/S0954579421000377 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492 [article] Association of subcortical gray-matter volumes with life-course-persistent antisocial behavior in a population-representative longitudinal birth cohort [Texte imprimé et/ou numérique] / Christina O. CARLISI, Auteur ; Terrie E. MOFFITT, Auteur ; Annchen R. KNODT, Auteur ; Honalee HARRINGTON, Auteur ; Stéphanie LANGEVIN, Auteur ; David IRELAND, Auteur ; Tracy R. MELZER, Auteur ; Richie POULTON, Auteur ; Sandhya RAMRAKHA, Auteur ; Avshalom CASPI, Auteur ; Ahmad R. HARIRI, Auteur ; Essi VIDING, Auteur . - p.2012-2022. Langues : Anglais (eng) in Development and Psychopathology > 34-5 (December 2022) . - p.2012-2022 Mots-clés : antisocial behavior  conduct disorder  development  longitudinal  structural MRI Index. décimale : PER Périodiques Résumé : Neuropsychological evidence supports the developmental taxonomy theory of antisocial behavior, suggesting that abnormal brain development distinguishes life-course-persistent from adolescence-limited antisocial behavior. Recent neuroimaging work confirmed that prospectively-measured life-course-persistent antisocial behavior is associated with differences in cortical brain structure. Whether this extends to subcortical brain structures remains uninvestigated. This study compared subcortical gray-matter volumes between 672 members of the Dunedin Study previously defined as exhibiting life-course-persistent, adolescence-limited or low-level antisocial behavior based on repeated assessments at ages 7 “26 years. Gray-matter volumes of 10 subcortical structures were compared across groups. The life-course-persistent group had lower volumes of amygdala, brain stem, cerebellum, hippocampus, pallidum, thalamus, and ventral diencephalon compared to the low-antisocial group. Differences between life-course-persistent and adolescence-limited individuals were comparable in effect size to differences between life-course-persistent and low-antisocial individuals, but were not statistically significant due to less statistical power. Gray-matter volumes in adolescence-limited individuals were near the norm in this population-representative cohort and similar to volumes in low-antisocial individuals. Although this study could not establish causal links between brain volume and antisocial behavior, it constitutes new biological evidence that all people with antisocial behavior are not the same, supporting a need for greater developmental and diagnostic precision in clinical, forensic, and policy-based interventions. En ligne : http://dx.doi.org/10.1017/S0954579421000377 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=492

Autobiographical memory as a latent vulnerability mechanism following childhood maltreatment: Association with future depression symptoms and prosocial behavior / Vanessa B. PUETZ in Development and Psychopathology, 33-4 (October 2021)  

Public ISBD [article]  inDevelopment and Psychopathology > 33-4 (October 2021) . - p.1300-1307 Titre : Autobiographical memory as a latent vulnerability mechanism following childhood maltreatment: Association with future depression symptoms and prosocial behavior Type de document : Texte imprimé et/ou numérique Auteurs : Vanessa B. PUETZ, Auteur ; Essi VIDING, Auteur ; Ferdinand HOFFMANN, Auteur ; Mattia I. GERIN, Auteur ; Molly SHARP, Auteur ; Georgia RANKIN, Auteur ; Eleanor A. MAGUIRE, Auteur ; Andrea MECHELLI, Auteur ; Eamon J. MCCRORY, Auteur Article en page(s) : p.1300-1307 Langues : Anglais (eng) Mots-clés : autobiographical memory  conduct problems  depression  maltreatment  prosocial behavior Index. décimale : PER Périodiques Résumé : Objectives Childhood maltreatment is associated with altered neural reactivity during autobiographical memory (ABM) recall and a pattern of overgeneral memory (OGM). Altered ABM and OGM have been linked with psychopathology and poorer social functioning. The present study investigated the association between altered ABM and subsequent socio-emotional functioning (measured two years later) in a sample of adolescents with (N = 20; maltreatment group, MT) and without (N = 17; non-MT group) documented childhood maltreatment histories. Method At baseline, adolescents (aged 12.6 ± 1.45 years) were administered the Autobiographical Memory Test to measure OGM. Participants also recalled specific ABMs in response to emotionally valenced cue words during functional MRI. Adolescents in both groups underwent assessments measuring depressive symptoms and prosocial behavior at both timepoints. Regression analyses were carried out to predict outcome measures at follow-up controlling for baseline levels. Results In the MT group, greater OGM at baseline significantly predicted reduced prosocial behavior at follow-up and showed a trend level association with elevated depressive symptoms. Patterns of altered ABM-related brain activity did not significantly predict future psycho-social functioning. Conclusions The current findings highlight the potential value of OGM as a cognitive mechanism that could be targeted to reduce risk of depression in adolescents with prior histories of maltreatment. En ligne : http://dx.doi.org/10.1017/S0954579420000504 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 [article] Autobiographical memory as a latent vulnerability mechanism following childhood maltreatment: Association with future depression symptoms and prosocial behavior [Texte imprimé et/ou numérique] / Vanessa B. PUETZ, Auteur ; Essi VIDING, Auteur ; Ferdinand HOFFMANN, Auteur ; Mattia I. GERIN, Auteur ; Molly SHARP, Auteur ; Georgia RANKIN, Auteur ; Eleanor A. MAGUIRE, Auteur ; Andrea MECHELLI, Auteur ; Eamon J. MCCRORY, Auteur . - p.1300-1307. Langues : Anglais (eng) in Development and Psychopathology > 33-4 (October 2021) . - p.1300-1307 Mots-clés : autobiographical memory  conduct problems  depression  maltreatment  prosocial behavior Index. décimale : PER Périodiques Résumé : Objectives Childhood maltreatment is associated with altered neural reactivity during autobiographical memory (ABM) recall and a pattern of overgeneral memory (OGM). Altered ABM and OGM have been linked with psychopathology and poorer social functioning. The present study investigated the association between altered ABM and subsequent socio-emotional functioning (measured two years later) in a sample of adolescents with (N = 20; maltreatment group, MT) and without (N = 17; non-MT group) documented childhood maltreatment histories. Method At baseline, adolescents (aged 12.6 ± 1.45 years) were administered the Autobiographical Memory Test to measure OGM. Participants also recalled specific ABMs in response to emotionally valenced cue words during functional MRI. Adolescents in both groups underwent assessments measuring depressive symptoms and prosocial behavior at both timepoints. Regression analyses were carried out to predict outcome measures at follow-up controlling for baseline levels. Results In the MT group, greater OGM at baseline significantly predicted reduced prosocial behavior at follow-up and showed a trend level association with elevated depressive symptoms. Patterns of altered ABM-related brain activity did not significantly predict future psycho-social functioning. Conclusions The current findings highlight the potential value of OGM as a cognitive mechanism that could be targeted to reduce risk of depression in adolescents with prior histories of maltreatment. En ligne : http://dx.doi.org/10.1017/S0954579420000504 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457

Brief Report: No Increase in Criminal Convictions in Hans Asperger’s Original Cohort / Kathrin HIPPLER in Journal of Autism and Developmental Disorders, 40-6 (June 2010)  

Permalink

Callous-unemotional traits in children and mechanisms of impaired eye contact during expressions of love: a treatment target? / Mark R. DADDS in Journal of Child Psychology and Psychiatry, 55-7 (July 2014)  

Permalink

Commentary: Conduct disorder and perceiving harm to others – a reflection on Michalska et al. (2016) / Patricia L. LOCKWOOD in Journal of Child Psychology and Psychiatry, 57-4 (April 2016)  

Permalink

Commentary: Genetic influences on adolescent attachment security: an empirical reminder of biology and the complexities of development – a reply to Rutter (2014) / Richard M. PASCO FEARON in Journal of Child Psychology and Psychiatry, 55-9 (September 2014)  

Permalink

Commentary: Study of gene–environment interplay – a lesson in how to keep oneself busy for the foreseeable future / Essi VIDING in Journal of Child Psychology and Psychiatry, 54-10 (October 2013)  

Permalink

Commentary: Will genomics revolutionise research on gene-environment interplay? / Robert PLOMIN in Journal of Child Psychology and Psychiatry, 63-10 (October 2022)  

Permalink

Development of the ‘Extreme Demand Avoidance Questionnaire’ (EDA-Q): preliminary observations on a trait measure for Pathological Demand Avoidance / Elizabeth O'NIONS in Journal of Child Psychology and Psychiatry, 55-7 (July 2014)  

Permalink

Dimensions of Psychopathy and their Relationships to Cognitive Functioning in Children / Nathalie FONTAINE in Journal of Clinical Child & Adolescent Psychology, 37-3 (July-September 2008)  

Permalink

Double disadvantage: the influence of childhood maltreatment and community violence exposure on adolescent mental health / Charlotte A. M. CECIL in Journal of Child Psychology and Psychiatry, 55-7 (July 2014)  

Permalink

Effects of DTNBP1 genotype on brain development in children / Stefania TOGNIN in Journal of Child Psychology and Psychiatry, 52-12 (December 2011)  

Permalink

---

1 2 3 (1 - 15 / 36) Par page : 25 Tout afficher