Dépouillements

Berard Auditory Integration Training: Behavior Changes Related to Sensory Modulation / Sally S. BROCKETT in Autism Insights, (February 2014)  

Public ISBD [article]  inAutism Insights > (February 2014) . - 10 p. Titre : Berard Auditory Integration Training: Behavior Changes Related to Sensory Modulation Type de document : Texte imprimé et/ou numérique Auteurs : Sally S. BROCKETT, Auteur ; Nancy K. LAWTON-SHIRLEY, Auteur ; Judith GIENCKE KIMBALL, Auteur Année de publication : 2014 Article en page(s) : 10 p. Langues : Anglais (eng) Mots-clés : autism  auditory processing  behavior  sensory integration  sensory modulation Index. décimale : PER Périodiques Résumé : Objective: The purpose of this study was to determine if behaviors specifically related to sensory modulation showed positive changes following 10 days of Berard auditory integration training (AIT). Method: Cases of 54 children with disabilities (34 with autism), ages 3–10 years, who received Berard AIT, were reviewed. Children received 30 minutes of training twice a day, separated by a minimum of three hours, for 10 consecutive days. Data were collected within one week before intervention and at one, three, and six months post-intervention. Results: Analysis of variance (ANOVA) indicated that Short Sensory Profile (SSP) total test scores and individual factor sections improved from pre-test to post-test (P , 0.01). Behavioral problems reduced on all five factors of the Aberrant Behavior Checklist (ABC) (P , 0.01). Most changes occurred within one month of intervention and maintained at three and six months. Correlations among the ABC and SSP factors indicate that sensory modulation as measured by the SSP is a significant contributor to four of the behavioral factors measured by the ABC. Conclusions: Although causality cannot be determined using this study design, scores on the SSP and ABC improved in a group of children who received Berard AIT. En ligne : http://dx.doi.org/10.4137/AUI.S13574 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=228 [article] Berard Auditory Integration Training: Behavior Changes Related to Sensory Modulation [Texte imprimé et/ou numérique] / Sally S. BROCKETT, Auteur ; Nancy K. LAWTON-SHIRLEY, Auteur ; Judith GIENCKE KIMBALL, Auteur . - 2014 . - 10 p. Langues : Anglais (eng) in Autism Insights > (February 2014) . - 10 p. Mots-clés : autism  auditory processing  behavior  sensory integration  sensory modulation Index. décimale : PER Périodiques Résumé : Objective: The purpose of this study was to determine if behaviors specifically related to sensory modulation showed positive changes following 10 days of Berard auditory integration training (AIT). Method: Cases of 54 children with disabilities (34 with autism), ages 3–10 years, who received Berard AIT, were reviewed. Children received 30 minutes of training twice a day, separated by a minimum of three hours, for 10 consecutive days. Data were collected within one week before intervention and at one, three, and six months post-intervention. Results: Analysis of variance (ANOVA) indicated that Short Sensory Profile (SSP) total test scores and individual factor sections improved from pre-test to post-test (P , 0.01). Behavioral problems reduced on all five factors of the Aberrant Behavior Checklist (ABC) (P , 0.01). Most changes occurred within one month of intervention and maintained at three and six months. Correlations among the ABC and SSP factors indicate that sensory modulation as measured by the SSP is a significant contributor to four of the behavioral factors measured by the ABC. Conclusions: Although causality cannot be determined using this study design, scores on the SSP and ABC improved in a group of children who received Berard AIT. En ligne : http://dx.doi.org/10.4137/AUI.S13574 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=228

Oncogenes Involved in the Etiology of Autism / A.J. RUSSO in Autism Insights, (February 2014)  

Public ISBD [article]  inAutism Insights > (February 2014) . - p.11-13 Titre : Oncogenes Involved in the Etiology of Autism Type de document : Texte imprimé et/ou numérique Auteurs : A.J. RUSSO, Auteur Article en page(s) : p.11-13 Langues : Anglais (eng) Mots-clés : epidermal growth factor receptor (EGFR)  epidermal growth factor (EGF)  hepatocyte growth factor receptor (HGFR)  cMet  hepatocyte growth factor (HGF)  autism Index. décimale : PER Périodiques Résumé : Epidermal growth factor receptor (EGFR) gene and Hepatocyte Growth Factor Receptor gene, c-Met, are proto-oncogenes which code for cell-surface tyrosine kinase receptors, EGFR and hepatocyte growth factor (HGF), respectively. These membrane receptors are stimulated by ligands, epidermal growth factor (EGF) and hepatocyte growth factor (HGF), and then, through a cascade of reactions, regulate several important cell events such as growth, motility, and morphogenesis. This editorial presents a cursory look at the evidence that these proto-oncogenes, and their products, are involved in the etiology of neurobehavioral disorders, such as autism. En ligne : https://dx.doi.org/10.4137/AUI.S14280 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=409 [article] Oncogenes Involved in the Etiology of Autism [Texte imprimé et/ou numérique] / A.J. RUSSO, Auteur . - p.11-13. Langues : Anglais (eng) in Autism Insights > (February 2014) . - p.11-13 Mots-clés : epidermal growth factor receptor (EGFR)  epidermal growth factor (EGF)  hepatocyte growth factor receptor (HGFR)  cMet  hepatocyte growth factor (HGF)  autism Index. décimale : PER Périodiques Résumé : Epidermal growth factor receptor (EGFR) gene and Hepatocyte Growth Factor Receptor gene, c-Met, are proto-oncogenes which code for cell-surface tyrosine kinase receptors, EGFR and hepatocyte growth factor (HGF), respectively. These membrane receptors are stimulated by ligands, epidermal growth factor (EGF) and hepatocyte growth factor (HGF), and then, through a cascade of reactions, regulate several important cell events such as growth, motility, and morphogenesis. This editorial presents a cursory look at the evidence that these proto-oncogenes, and their products, are involved in the etiology of neurobehavioral disorders, such as autism. En ligne : https://dx.doi.org/10.4137/AUI.S14280 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=409

Role of Environmental Exposure to Toxins and Microbial Infections in Autism / Rajendram V. RAJNARAYANAN in Autism Insights, (February 2014)  

Public ISBD [article]  inAutism Insights > (February 2014) . - p.15-25 Titre : Role of Environmental Exposure to Toxins and Microbial Infections in Autism Type de document : Texte imprimé et/ou numérique Auteurs : Rajendram V. RAJNARAYANAN, Auteur ; Nataliya KOSTYUK, Auteur ; Hari H.P. COHLY, Auteur Article en page(s) : p.15-25 Langues : Anglais (eng) Mots-clés : environmental chemicals  toxins  microbial infections  autism Index. décimale : PER Périodiques Résumé : Autism is a complex developmental neurological disorder causing impaired function and structure of brain development. According to a recent report from the Centers for Disease Control and Prevention (CDC), autism is estimated to affect 1 in 88 children in USA.1 In spite of several reports linking prenatal exposure to environmental toxins and to microbial agents via infections to a spectrum of autism and autism-like disorders, to date, neither the associated risk factor nor the pathophysiological mechanisms have been established unequivocally. The impact of these environmental agents is believed to be similar to that of other neuropsychiatric disorders. Earlier, we have reported the impact and immunological implications of mercury and viral infections in autism. In this review, we highlight the current incidence of autism, discuss brain development in autism, present the prominent features of neuroanatomy in autism, describe neurodegenerative findings in autistic individuals, summarize the hypotheses to explain autism, and provide a perspective of the molecular events in autism and autism spectrum disorders (ASD). The early events that trigger this complex cluster of neurological disorders may involve the breach of cellular interface, which leads to the influx of water which in turn damages the developing neurons during the early stages of brain development. Alternatively, neurodegenerative disorders can be caused by the interaction of environmental agents like heavy metals with transport proteins like aquaporins and gap junction protein complexes embedded in the neuronal network during synaptogenesis. En ligne : https://dx.doi.org/10.4137/AUI.S13839 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=409 [article] Role of Environmental Exposure to Toxins and Microbial Infections in Autism [Texte imprimé et/ou numérique] / Rajendram V. RAJNARAYANAN, Auteur ; Nataliya KOSTYUK, Auteur ; Hari H.P. COHLY, Auteur . - p.15-25. Langues : Anglais (eng) in Autism Insights > (February 2014) . - p.15-25 Mots-clés : environmental chemicals  toxins  microbial infections  autism Index. décimale : PER Périodiques Résumé : Autism is a complex developmental neurological disorder causing impaired function and structure of brain development. According to a recent report from the Centers for Disease Control and Prevention (CDC), autism is estimated to affect 1 in 88 children in USA.1 In spite of several reports linking prenatal exposure to environmental toxins and to microbial agents via infections to a spectrum of autism and autism-like disorders, to date, neither the associated risk factor nor the pathophysiological mechanisms have been established unequivocally. The impact of these environmental agents is believed to be similar to that of other neuropsychiatric disorders. Earlier, we have reported the impact and immunological implications of mercury and viral infections in autism. In this review, we highlight the current incidence of autism, discuss brain development in autism, present the prominent features of neuroanatomy in autism, describe neurodegenerative findings in autistic individuals, summarize the hypotheses to explain autism, and provide a perspective of the molecular events in autism and autism spectrum disorders (ASD). The early events that trigger this complex cluster of neurological disorders may involve the breach of cellular interface, which leads to the influx of water which in turn damages the developing neurons during the early stages of brain development. Alternatively, neurodegenerative disorders can be caused by the interaction of environmental agents like heavy metals with transport proteins like aquaporins and gap junction protein complexes embedded in the neuronal network during synaptogenesis. En ligne : https://dx.doi.org/10.4137/AUI.S13839 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=409