Maternal perinatal depression and infant self-regulation: A meta-analytic review / Emily R. PADRUTT in Development and Psychopathology, 38-2 (May 2026)  

Public ISBD [article]  inDevelopment and Psychopathology > 38-2 (May 2026) . - p.925-944 Titre : Maternal perinatal depression and infant self-regulation: A meta-analytic review Type de document : texte imprimé Auteurs : Emily R. PADRUTT, Auteur ; Daniel BERRY, Auteur ; Ellie SCHWARTZMAN, Auteur ; Sylia WILSON, Auteur Article en page(s) : p.925-944 Langues : Anglais (eng) Mots-clés : Depression  infancy  meta-analysis  perinatal  self-regulation Index. décimale : PER Périodiques Résumé : Infant self-regulation is shaped by early physiological systems and caregiver-infant co-regulatory interactions. Maternal perinatal (pre- and/or postnatal) depression may affect these processes and infants’ development of this critical construct. However, literature addressing the association between maternal perinatal depression and infant self-regulation has been mixed. We conducted a pre-registered meta-analysis of the association between maternal perinatal depression and several self-regulation constructs (e.g., effortful control, executive function) measured during the first 2 years of life. We included 68 reports comprising 193 effect sizes and 16,722 mother-infant dyads. On average, studies included an equal number of male and female infants, and, for most (68%) studies, most participants were White. Average infant age ranged from 0 – 16 months. Three-level random effects meta-analytic models indicated a small, significant overall association, with higher levels of depression associated with lower self-regulation (r = −.10, 95% CI = −.14, −.06, p < .001). There was substantial heterogeneity in this pooled effect. Subsequent analyses indicated moderation by methodological and conceptual variables. Evidence that maternal perinatal depression is associated with lower infant self-regulation underscores the importance of supporting dyads experiencing perinatal depression. Clarifying this association highlights a critical next step of examining potential causal processes linking maternal and infant well-being. En ligne : https://dx.doi.org/10.1017/S0954579425100837 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=586 [article] Maternal perinatal depression and infant self-regulation: A meta-analytic review [texte imprimé] / Emily R. PADRUTT, Auteur ; Daniel BERRY, Auteur ; Ellie SCHWARTZMAN, Auteur ; Sylia WILSON, Auteur . - p.925-944. Langues : Anglais (eng) in Development and Psychopathology > 38-2 (May 2026) . - p.925-944 Mots-clés : Depression  infancy  meta-analysis  perinatal  self-regulation Index. décimale : PER Périodiques Résumé : Infant self-regulation is shaped by early physiological systems and caregiver-infant co-regulatory interactions. Maternal perinatal (pre- and/or postnatal) depression may affect these processes and infants’ development of this critical construct. However, literature addressing the association between maternal perinatal depression and infant self-regulation has been mixed. We conducted a pre-registered meta-analysis of the association between maternal perinatal depression and several self-regulation constructs (e.g., effortful control, executive function) measured during the first 2 years of life. We included 68 reports comprising 193 effect sizes and 16,722 mother-infant dyads. On average, studies included an equal number of male and female infants, and, for most (68%) studies, most participants were White. Average infant age ranged from 0 – 16 months. Three-level random effects meta-analytic models indicated a small, significant overall association, with higher levels of depression associated with lower self-regulation (r = −.10, 95% CI = −.14, −.06, p < .001). There was substantial heterogeneity in this pooled effect. Subsequent analyses indicated moderation by methodological and conceptual variables. Evidence that maternal perinatal depression is associated with lower infant self-regulation underscores the importance of supporting dyads experiencing perinatal depression. Clarifying this association highlights a critical next step of examining potential causal processes linking maternal and infant well-being. En ligne : https://dx.doi.org/10.1017/S0954579425100837 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=586

Pubertal timing and adolescent outcomes: investigating explanations for associations with a genetically informed design / Emily R. PADRUTT in Journal of Child Psychology and Psychiatry, 64-8 (August 2023)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 64-8 (August 2023) . - p.1232-1241 Titre : Pubertal timing and adolescent outcomes: investigating explanations for associations with a genetically informed design Type de document : texte imprimé Auteurs : Emily R. PADRUTT, Auteur ; Jeremy HARPER, Auteur ; Jonathan D. SCHAEFER, Auteur ; Kayla M. NELSON, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Sylia WILSON, Auteur Article en page(s) : p.1232-1241 Langues : Anglais (eng) Mots-clés : Pubertal timing  substance use  risky behavior  peer problems  psychopathology Index. décimale : PER Périodiques Résumé : Background Psychopathology and risky behaviors increase during adolescence, and understanding which adolescents are most at risk informs prevention and intervention efforts. Pubertal timing relative to same-sex, same-age peers is a known correlate of adolescent outcomes among both boys and girls. However, it remains unclear whether this relation is better explained by a plausible causal process or unobserved familial liability. Methods We extended previous research by examining associations between pubertal timing in early adolescence (age 14) and outcomes in later adolescence (age 17) in a community sample of 2,510 twins (49% boys, 51% girls). Results Earlier pubertal timing was associated with more substance use, risk behavior, internalizing and externalizing problems, and peer problems in later adolescence; these effects were small, consistent with previous literature. Follow-up co-twin control analyses indicated that within-twin-pair differences in pubertal timing were not associated with within-twin-pair differences in most adolescent outcomes after accounting for shared familial liability, suggesting that earlier pubertal timing and adolescent outcomes both reflect familial risk factors. Biometric models indicated that associations between earlier pubertal timing and negative adolescent outcomes were largely attributable to shared genetic liability. Conclusions Although earlier pubertal timing was associated with negative adolescent outcomes, our results suggests that these associations did not appear to be caused by earlier pubertal timing but were likely caused by shared genetic influences. En ligne : https://doi.org/10.1111/jcpp.13808 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=508 [article] Pubertal timing and adolescent outcomes: investigating explanations for associations with a genetically informed design [texte imprimé] / Emily R. PADRUTT, Auteur ; Jeremy HARPER, Auteur ; Jonathan D. SCHAEFER, Auteur ; Kayla M. NELSON, Auteur ; Matt MCGUE, Auteur ; William G. IACONO, Auteur ; Sylia WILSON, Auteur . - p.1232-1241. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 64-8 (August 2023) . - p.1232-1241 Mots-clés : Pubertal timing  substance use  risky behavior  peer problems  psychopathology Index. décimale : PER Périodiques Résumé : Background Psychopathology and risky behaviors increase during adolescence, and understanding which adolescents are most at risk informs prevention and intervention efforts. Pubertal timing relative to same-sex, same-age peers is a known correlate of adolescent outcomes among both boys and girls. However, it remains unclear whether this relation is better explained by a plausible causal process or unobserved familial liability. Methods We extended previous research by examining associations between pubertal timing in early adolescence (age 14) and outcomes in later adolescence (age 17) in a community sample of 2,510 twins (49% boys, 51% girls). Results Earlier pubertal timing was associated with more substance use, risk behavior, internalizing and externalizing problems, and peer problems in later adolescence; these effects were small, consistent with previous literature. Follow-up co-twin control analyses indicated that within-twin-pair differences in pubertal timing were not associated with within-twin-pair differences in most adolescent outcomes after accounting for shared familial liability, suggesting that earlier pubertal timing and adolescent outcomes both reflect familial risk factors. Biometric models indicated that associations between earlier pubertal timing and negative adolescent outcomes were largely attributable to shared genetic liability. Conclusions Although earlier pubertal timing was associated with negative adolescent outcomes, our results suggests that these associations did not appear to be caused by earlier pubertal timing but were likely caused by shared genetic influences. En ligne : https://doi.org/10.1111/jcpp.13808 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=508

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