Maternal age at childbirth and offspring disruptive behaviors: testing the causal hypothesis / Brian M. D'ONOFRIO in Journal of Child Psychology and Psychiatry, 50-8 (August 2009)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 50-8 (August 2009) . - p.1018-1028 Titre : Maternal age at childbirth and offspring disruptive behaviors: testing the causal hypothesis Type de document : Texte imprimé et/ou numérique Auteurs : Brian M. D'ONOFRIO, Auteur ; Benjamin B. LAHEY, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur ; Paul J. RATHOUZ, Auteur ; Jackson A. GOODNIGHT, Auteur Année de publication : 2009 Article en page(s) : p.1018-1028 Langues : Anglais (eng) Mots-clés : Disruptive-behaviors conduct-problems maternal-age-at-childbearing sibling-comparisons quasi-experiments Index. décimale : PER Périodiques Résumé : Background: Recent studies suggest that the association between maternal age at childbearing (MAC) and children's disruptive behaviors is the result of family factors that are confounded with both variables, rather than a casual effect of environmental factors specifically related to MAC. These studies, however, relied on restricted samples and did not use the strongest approach to test causal influences. Methods: Using data on 9,171 4–9-year-old and 6,592 10–13-year-old offspring of women from a nationally representative sample of US households, we conducted sibling-comparison analyses. The analyses ruled out all genetic factors that could confound the association, as well as all environmental confounds that differ between unrelated nuclear families, providing a strong test of the causal hypothesis that the environments of children born at different maternal ages influence mother- and self-reported disruptive behaviors. Results: When these genetic and environmental confounds were ruled out as alternative explanations, the relation between environments within nuclear families specifically associated with MAC and disruptive behaviors was robust, with the association being stronger for second- and third-born children. Conclusions: Environmental factors specifically associated with early MAC within nuclear families account for increased risk of offspring disruptive behaviors, especially in later-born children. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02068.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=788 [article] Maternal age at childbirth and offspring disruptive behaviors: testing the causal hypothesis [Texte imprimé et/ou numérique] / Brian M. D'ONOFRIO, Auteur ; Benjamin B. LAHEY, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur ; Paul J. RATHOUZ, Auteur ; Jackson A. GOODNIGHT, Auteur . - 2009 . - p.1018-1028. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 50-8 (August 2009) . - p.1018-1028 Mots-clés : Disruptive-behaviors conduct-problems maternal-age-at-childbearing sibling-comparisons quasi-experiments Index. décimale : PER Périodiques Résumé : Background: Recent studies suggest that the association between maternal age at childbearing (MAC) and children's disruptive behaviors is the result of family factors that are confounded with both variables, rather than a casual effect of environmental factors specifically related to MAC. These studies, however, relied on restricted samples and did not use the strongest approach to test causal influences. Methods: Using data on 9,171 4–9-year-old and 6,592 10–13-year-old offspring of women from a nationally representative sample of US households, we conducted sibling-comparison analyses. The analyses ruled out all genetic factors that could confound the association, as well as all environmental confounds that differ between unrelated nuclear families, providing a strong test of the causal hypothesis that the environments of children born at different maternal ages influence mother- and self-reported disruptive behaviors. Results: When these genetic and environmental confounds were ruled out as alternative explanations, the relation between environments within nuclear families specifically associated with MAC and disruptive behaviors was robust, with the association being stronger for second- and third-born children. Conclusions: Environmental factors specifically associated with early MAC within nuclear families account for increased risk of offspring disruptive behaviors, especially in later-born children. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02068.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=788

Population density and youth antisocial behavior / K. Paige HARDEN in Journal of Child Psychology and Psychiatry, 50-8 (August 2009)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 50-8 (August 2009) . - p.999-1008 Titre : Population density and youth antisocial behavior Type de document : Texte imprimé et/ou numérique Auteurs : K. Paige HARDEN, Auteur ; Benjamin B. LAHEY, Auteur ; Brian M. D'ONOFRIO, Auteur ; Eric TURKHEIMER, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur Année de publication : 2009 Article en page(s) : p.999-1008 Langues : Anglais (eng) Mots-clés : Behavior-problems delinquency demography environmental-influences epidemiology Index. décimale : PER Périodiques Résumé : Theoretical models concerning how neighborhood contexts adversely influence juvenile antisocial behavior frequently focus on urban neighborhoods; however, previous studies comparing urban and rural areas on the prevalence of youth antisocial behavior have yielded mixed results. The current study uses longitudinal data on the offspring of a nationally representative sample of mothers (N = 4,886) in the US. There was no relation between density and mother-reported child conduct problems across ages 4–13 years, but youth living in areas of greater population density exhibited more youth self-reported delinquency across 10–17 years. Families often moved to counties with greater or lesser population density, but longitudinal analyses treating population density as a time-varying covariate did not support the hypothesis that living in densely populated counties influenced youth delinquency. Rather, the association between population density and delinquency appears to be due to unmeasured selection variables that differ between families who live in more or less densely populated counties. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02044.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=788 [article] Population density and youth antisocial behavior [Texte imprimé et/ou numérique] / K. Paige HARDEN, Auteur ; Benjamin B. LAHEY, Auteur ; Brian M. D'ONOFRIO, Auteur ; Eric TURKHEIMER, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur . - 2009 . - p.999-1008. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 50-8 (August 2009) . - p.999-1008 Mots-clés : Behavior-problems delinquency demography environmental-influences epidemiology Index. décimale : PER Périodiques Résumé : Theoretical models concerning how neighborhood contexts adversely influence juvenile antisocial behavior frequently focus on urban neighborhoods; however, previous studies comparing urban and rural areas on the prevalence of youth antisocial behavior have yielded mixed results. The current study uses longitudinal data on the offspring of a nationally representative sample of mothers (N = 4,886) in the US. There was no relation between density and mother-reported child conduct problems across ages 4–13 years, but youth living in areas of greater population density exhibited more youth self-reported delinquency across 10–17 years. Families often moved to counties with greater or lesser population density, but longitudinal analyses treating population density as a time-varying covariate did not support the hypothesis that living in densely populated counties influenced youth delinquency. Rather, the association between population density and delinquency appears to be due to unmeasured selection variables that differ between families who live in more or less densely populated counties. En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02044.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=788

Smoking during pregnancy and offspring externalizing problems: An exploration of genetic and environmental confounds / Brian M. D'ONOFRIO in Development and Psychopathology, 20-1 (Winter 2008)  

Public ISBD [article]  inDevelopment and Psychopathology > 20-1 (Winter 2008) . - p.139-164 Titre : Smoking during pregnancy and offspring externalizing problems: An exploration of genetic and environmental confounds Type de document : Texte imprimé et/ou numérique Auteurs : Brian M. D'ONOFRIO, Auteur ; Benjamin B. LAHEY, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur ; K. Paige HARDEN, Auteur ; Paul J. RATHOUZ, Auteur Année de publication : 2008 Article en page(s) : p.139-164 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Previous studies have documented that smoking during pregnancy (SDP) is associated with offspring externalizing problems, even when measured covariates were used to control for possible confounds. However, the association may be because of nonmeasured environmental and genetic factors that increase risk for offspring externalizing problems. The current project used the National Longitudinal Survey of Youth and their children, ages 4–10 years, to explore the relations between SDP and offspring conduct problems (CPs), oppositional defiant problems (ODPs), and attention-deficit/hyperactivity problems (ADHPs) using methodological and statistical controls for confounds. When offspring were compared to their own siblings who differed in their exposure to prenatal nicotine, there was no effect of SDP on offspring CP and ODP. This suggests that SDP does not have a causal effect on offspring CP and ODP. There was a small association between SDP and ADHP, consistent with a causal effect of SDP, but the magnitude of the association was greatly reduced by methodological and statistical controls. Genetically informed analyses suggest that unmeasured environmental variables influencing both SDP and offspring externalizing behaviors account for the previously observed associations. That is, the current analyses imply that important unidentified environmental factors account for the association between SDP and offspring externalizing problems, not teratogenic effects of SDP. En ligne : http://dx.doi.org/10.1017/s0954579408000072 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=332 [article] Smoking during pregnancy and offspring externalizing problems: An exploration of genetic and environmental confounds [Texte imprimé et/ou numérique] / Brian M. D'ONOFRIO, Auteur ; Benjamin B. LAHEY, Auteur ; Carol A. VAN HULLE, Auteur ; Irwin D. WALDMAN, Auteur ; Joseph Lee RODGERS, Auteur ; K. Paige HARDEN, Auteur ; Paul J. RATHOUZ, Auteur . - 2008 . - p.139-164. Langues : Anglais (eng) in Development and Psychopathology > 20-1 (Winter 2008) . - p.139-164 Index. décimale : PER Périodiques Résumé : Previous studies have documented that smoking during pregnancy (SDP) is associated with offspring externalizing problems, even when measured covariates were used to control for possible confounds. However, the association may be because of nonmeasured environmental and genetic factors that increase risk for offspring externalizing problems. The current project used the National Longitudinal Survey of Youth and their children, ages 4–10 years, to explore the relations between SDP and offspring conduct problems (CPs), oppositional defiant problems (ODPs), and attention-deficit/hyperactivity problems (ADHPs) using methodological and statistical controls for confounds. When offspring were compared to their own siblings who differed in their exposure to prenatal nicotine, there was no effect of SDP on offspring CP and ODP. This suggests that SDP does not have a causal effect on offspring CP and ODP. There was a small association between SDP and ADHP, consistent with a causal effect of SDP, but the magnitude of the association was greatly reduced by methodological and statistical controls. Genetically informed analyses suggest that unmeasured environmental variables influencing both SDP and offspring externalizing behaviors account for the previously observed associations. That is, the current analyses imply that important unidentified environmental factors account for the association between SDP and offspring externalizing problems, not teratogenic effects of SDP. En ligne : http://dx.doi.org/10.1017/s0954579408000072 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=332

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