Abnormalities in cerebellar subregions' volume and cerebellocerebral structural covariance in autism spectrum disorder / Yu WANG ; Aihua CAO ; Jing WANG ; He BAI ; Tianci LIU ; Chenxi SUN ; Zhuoran LI ; Yuchun TANG ; Feifei XU ; Shuwei LIU in Autism Research, 18-1 (January 2025)  

Public ISBD [article]  inAutism Research > 18-1 (January 2025) . - p.83-97 Titre : Abnormalities in cerebellar subregions' volume and cerebellocerebral structural covariance in autism spectrum disorder : Autism Research Type de document : texte imprimé Auteurs : Yu WANG, Auteur ; Aihua CAO, Auteur ; Jing WANG, Auteur ; He BAI, Auteur ; Tianci LIU, Auteur ; Chenxi SUN, Auteur ; Zhuoran LI, Auteur ; Yuchun TANG, Auteur ; Feifei XU, Auteur ; Shuwei LIU, Auteur Article en page(s) : p.83-97 Langues : Anglais (eng) Mots-clés : atypical patterns  autism  cerebellar subregions' volume  cerebellocerebral structural covariance  pars opercularis Index. décimale : PER Périodiques Résumé : Abstract The cerebellum plays a crucial role in functions, including sensory-motor coordination, cognition, and emotional processing. Compared to the neocortex, the human cerebellum exhibits a protracted developmental trajectory. This delayed developmental timeline may lead to increased sensitivity of the cerebellum to external influences, potentially extending the vulnerability period for neurological disorders. Abnormal cerebellar development in individuals with autism has been confirmed, and these atypical cerebellar changes may affect the development of the neocortex. However, due to the heterogeneity of autism spectrum disorder (ASD), the regional changes in the cerebellum and cerebellocerebral structural relationship remain unknown. To address these issues, we utilized imaging methods optimized for the cerebellum and cerebrum on 817 individuals aged 5 18 years in the ABIDE II dataset. After FDR correction, significant differences between groups were found in the right crus II/VIIB and vermis VI-VII. Structural covariance analysis revealed enhanced structural covariance in individuals with autism between the cerebellum and parahippocampal gyrus, pars opercularis, and transverse temporal gyrus in the right hemisphere after FDR correction. Furthermore, the structural covariance between the cerebellum and some regions of the cerebrum varied across sexes. A significant increase in structural covariance between the cerebellum and specific subcortical structures was also observed in individuals with ASD. Our study found atypical patterns in the structural covariance between the cerebellum and cerebrum in individuals with autism, which suggested that the underlying pathological processes of ASD might concurrently affect these brain regions. This study provided insight into the potential of cerebellocerebral pathways as therapeutic targets for ASD. En ligne : https://doi.org/10.1002/aur.3287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=546 [article] Abnormalities in cerebellar subregions' volume and cerebellocerebral structural covariance in autism spectrum disorder : Autism Research [texte imprimé] / Yu WANG, Auteur ; Aihua CAO, Auteur ; Jing WANG, Auteur ; He BAI, Auteur ; Tianci LIU, Auteur ; Chenxi SUN, Auteur ; Zhuoran LI, Auteur ; Yuchun TANG, Auteur ; Feifei XU, Auteur ; Shuwei LIU, Auteur . - p.83-97. Langues : Anglais (eng) in Autism Research > 18-1 (January 2025) . - p.83-97 Mots-clés : atypical patterns  autism  cerebellar subregions' volume  cerebellocerebral structural covariance  pars opercularis Index. décimale : PER Périodiques Résumé : Abstract The cerebellum plays a crucial role in functions, including sensory-motor coordination, cognition, and emotional processing. Compared to the neocortex, the human cerebellum exhibits a protracted developmental trajectory. This delayed developmental timeline may lead to increased sensitivity of the cerebellum to external influences, potentially extending the vulnerability period for neurological disorders. Abnormal cerebellar development in individuals with autism has been confirmed, and these atypical cerebellar changes may affect the development of the neocortex. However, due to the heterogeneity of autism spectrum disorder (ASD), the regional changes in the cerebellum and cerebellocerebral structural relationship remain unknown. To address these issues, we utilized imaging methods optimized for the cerebellum and cerebrum on 817 individuals aged 5 18 years in the ABIDE II dataset. After FDR correction, significant differences between groups were found in the right crus II/VIIB and vermis VI-VII. Structural covariance analysis revealed enhanced structural covariance in individuals with autism between the cerebellum and parahippocampal gyrus, pars opercularis, and transverse temporal gyrus in the right hemisphere after FDR correction. Furthermore, the structural covariance between the cerebellum and some regions of the cerebrum varied across sexes. A significant increase in structural covariance between the cerebellum and specific subcortical structures was also observed in individuals with ASD. Our study found atypical patterns in the structural covariance between the cerebellum and cerebrum in individuals with autism, which suggested that the underlying pathological processes of ASD might concurrently affect these brain regions. This study provided insight into the potential of cerebellocerebral pathways as therapeutic targets for ASD. En ligne : https://doi.org/10.1002/aur.3287 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=546

Attentional biases to faces with direct versus averted gaze in children without and with autism spectrum disorder: A dot-probe paradigm / Wei JING in Research in Autism Spectrum Disorders, 108 (October 2023)  

Public ISBD [article]  inResearch in Autism Spectrum Disorders > 108 (October 2023) . - p.102233 Titre : Attentional biases to faces with direct versus averted gaze in children without and with autism spectrum disorder: A dot-probe paradigm Type de document : texte imprimé Auteurs : Wei JING, Auteur ; Jing WANG, Auteur ; Jinxia FU, Auteur Article en page(s) : p.102233 Mots-clés : Children with ASD  Dot-probe paradigms  Direct gaze  Averted gaze  Facial attention Index. décimale : PER Périodiques Résumé : Previous research utilized a variety of paradigms to demonstrate attentional biases to faces with direct versus averted gaze in typical development (TD) and no such bias in autism spectrum disorder (ASD). However, little is known about whether the biases can be observed during automatic processing stages in TD and whether the lack of such bias in ASD is due to passive neglect or active avoidance of direct gaze. Therefore, we employed a dot-probe paradigm and manipulated stimulus onset asynchrony (SOA) to measure response times to probes replacing faces with direct gaze compared to faces with averted gaze in children without and with ASD. There was no evidence of attentional bias in either group when stimuli were presented during the automatic processing stage (200 ms SOA). However, during the controlled processing stage (1000 ms SOA), an attentional bias to faces with direct versus averted gaze was found in control children but not in those with ASD. The results indicate that the facilitation of direct gaze on facial attention occurs during controlled rather than automatic processing stages in TD individuals. In contrast, children with ASD respond indiscriminately to direct and averted gaze during both stages of cognitive processing, supporting the gaze indifference hypothesis. For TD children but not for children with ASD, direct gaze is an adaptively informative or socially salient signal. En ligne : https://doi.org/10.1016/j.rasd.2023.102233 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=514 [article] Attentional biases to faces with direct versus averted gaze in children without and with autism spectrum disorder: A dot-probe paradigm [texte imprimé] / Wei JING, Auteur ; Jing WANG, Auteur ; Jinxia FU, Auteur . - p.102233. in Research in Autism Spectrum Disorders > 108 (October 2023) . - p.102233 Mots-clés : Children with ASD  Dot-probe paradigms  Direct gaze  Averted gaze  Facial attention Index. décimale : PER Périodiques Résumé : Previous research utilized a variety of paradigms to demonstrate attentional biases to faces with direct versus averted gaze in typical development (TD) and no such bias in autism spectrum disorder (ASD). However, little is known about whether the biases can be observed during automatic processing stages in TD and whether the lack of such bias in ASD is due to passive neglect or active avoidance of direct gaze. Therefore, we employed a dot-probe paradigm and manipulated stimulus onset asynchrony (SOA) to measure response times to probes replacing faces with direct gaze compared to faces with averted gaze in children without and with ASD. There was no evidence of attentional bias in either group when stimuli were presented during the automatic processing stage (200 ms SOA). However, during the controlled processing stage (1000 ms SOA), an attentional bias to faces with direct versus averted gaze was found in control children but not in those with ASD. The results indicate that the facilitation of direct gaze on facial attention occurs during controlled rather than automatic processing stages in TD individuals. In contrast, children with ASD respond indiscriminately to direct and averted gaze during both stages of cognitive processing, supporting the gaze indifference hypothesis. For TD children but not for children with ASD, direct gaze is an adaptively informative or socially salient signal. En ligne : https://doi.org/10.1016/j.rasd.2023.102233 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=514

Changes in women's alcoholic, antisocial, and depressive symptomatology over 12 years: A multilevel network of individual, familial, and neighborhood influences / Anne BUU in Development and Psychopathology, 23-1 (January 2011)  

Public ISBD [article]  inDevelopment and Psychopathology > 23-1 (January 2011) . - p.325-337 Titre : Changes in women's alcoholic, antisocial, and depressive symptomatology over 12 years: A multilevel network of individual, familial, and neighborhood influences Type de document : texte imprimé Auteurs : Anne BUU, Auteur ; Wei WANG, Auteur ; Jing WANG, Auteur ; Leon I. PUTTLER, Auteur ; Hiram E. FITZGERALD, Auteur ; Robert A. ZUCKER, Auteur Année de publication : 2011 Article en page(s) : p.325-337 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : In a sample of 273 adult women and their families, we examined the effects of women's psychopathology history, their social support, their husbands' and children's symptomatology, family stress, and neighborhood environment on their alcohol problems, antisocial behavior, and depression over a 12-year period during their 30s and early 40s. Women's alcohol problems and antisocial behavior decreased but their depression symptoms increased over time. Women's disorder history and their partners' parallel symptomatology were associated with their symptoms. For women's antisocial behavior, their own history of alcoholism and their partners' alcohol problems were also significant risk factors. Higher levels of social support were associated with lower levels of depression in women. Children's externalizing behavior was positively correlated with their mothers' alcohol problems and antisocial behavior, whereas children's internalizing behavior was positively correlated with their mothers' depression. Neighborhood residential instability was associated with higher levels of alcoholic and depressive symptomatology in women. Intervention efforts might target women with young children by improving social support, educational or professional training opportunity, access to family counseling, and neighborhood environment. En ligne : http://dx.doi.org/10.1017/S0954579410000830 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=117 [article] Changes in women's alcoholic, antisocial, and depressive symptomatology over 12 years: A multilevel network of individual, familial, and neighborhood influences [texte imprimé] / Anne BUU, Auteur ; Wei WANG, Auteur ; Jing WANG, Auteur ; Leon I. PUTTLER, Auteur ; Hiram E. FITZGERALD, Auteur ; Robert A. ZUCKER, Auteur . - 2011 . - p.325-337. Langues : Anglais (eng) in Development and Psychopathology > 23-1 (January 2011) . - p.325-337 Index. décimale : PER Périodiques Résumé : In a sample of 273 adult women and their families, we examined the effects of women's psychopathology history, their social support, their husbands' and children's symptomatology, family stress, and neighborhood environment on their alcohol problems, antisocial behavior, and depression over a 12-year period during their 30s and early 40s. Women's alcohol problems and antisocial behavior decreased but their depression symptoms increased over time. Women's disorder history and their partners' parallel symptomatology were associated with their symptoms. For women's antisocial behavior, their own history of alcoholism and their partners' alcohol problems were also significant risk factors. Higher levels of social support were associated with lower levels of depression in women. Children's externalizing behavior was positively correlated with their mothers' alcohol problems and antisocial behavior, whereas children's internalizing behavior was positively correlated with their mothers' depression. Neighborhood residential instability was associated with higher levels of alcoholic and depressive symptomatology in women. Intervention efforts might target women with young children by improving social support, educational or professional training opportunity, access to family counseling, and neighborhood environment. En ligne : http://dx.doi.org/10.1017/S0954579410000830 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=117

Copy number variation in Han Chinese individuals with autism spectrum disorder / MatthewJ GAZZELLONE in Journal of Neurodevelopmental Disorders, 6-1 (December 2014)  

Public ISBD [article]  inJournal of Neurodevelopmental Disorders > 6-1 (December 2014) . - p.34 Titre : Copy number variation in Han Chinese individuals with autism spectrum disorder Type de document : texte imprimé Auteurs : MatthewJ GAZZELLONE, Auteur ; Xue ZHOU, Auteur ; Anath C. LIONEL, Auteur ; Mohammed UDDIN, Auteur ; Bhooma THIRUVAHINDRAPURAM, Auteur ; Shuang LIANG, Auteur ; Caihong SUN, Auteur ; Jing WANG, Auteur ; Mingyang ZOU, Auteur ; Kristiina TAMMIMIES, Auteur ; Susan WALKER, Auteur ; Thanuja SELVANAYAGAM, Auteur ; John WEI, Auteur ; Ziqi WANG, Auteur ; Lijie WU, Auteur ; Stephen SCHERER, Auteur Article en page(s) : p.34 Langues : Anglais (eng) Mots-clés : Autism spectrum disorder (ASD)  Copy number variations (CNVs)  Han Chinese  Microarray diagnostic testing Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorders (ASDs) are a group of neurodevelopmental conditions with a demonstrated genetic etiology. Rare (<1% frequency) copy number variations (CNVs) account for a proportion of the genetic events involved, but the contribution of these events in non-European ASD populations has not been well studied. Here, we report on rare CNVs detected in a cohort of individuals with ASD of Han Chinese background. METHODS: DNA samples were obtained from 104 ASD probands and their parents who were recruited from Harbin, China. Samples were genotyped on the Affymetrix CytoScan HD platform. Rare CNVs were identified by comparing data with 873 technology-matched controls from Ontario and 1,235 additional population controls of Han Chinese ethnicity. RESULTS: Of the probands, 8.6% had at least 1 de novo CNV (overlapping the GIGYF2, SPRY1, 16p13.3, 16p11.2, 17p13.3-17p13.2, DMD, and NAP1L6 genes/loci). Rare inherited CNVs affected other plausible neurodevelopmental candidate genes including GRID2, LINGO2, and SLC39A12. A 24-kb duplication was also identified at YWHAE, a gene previously implicated in ASD and other developmental disorders. This duplication is observed at a similar frequency in cases and in population controls and is likely a benign Asian-specific copy number polymorphism. CONCLUSIONS: Our findings help define genomic features relevant to ASD in the Han Chinese and emphasize the importance of using ancestry-matched controls in medical genetic interpretations. En ligne : http://dx.doi.org/10.1186/1866-1955-6-34 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=346 [article] Copy number variation in Han Chinese individuals with autism spectrum disorder [texte imprimé] / MatthewJ GAZZELLONE, Auteur ; Xue ZHOU, Auteur ; Anath C. LIONEL, Auteur ; Mohammed UDDIN, Auteur ; Bhooma THIRUVAHINDRAPURAM, Auteur ; Shuang LIANG, Auteur ; Caihong SUN, Auteur ; Jing WANG, Auteur ; Mingyang ZOU, Auteur ; Kristiina TAMMIMIES, Auteur ; Susan WALKER, Auteur ; Thanuja SELVANAYAGAM, Auteur ; John WEI, Auteur ; Ziqi WANG, Auteur ; Lijie WU, Auteur ; Stephen SCHERER, Auteur . - p.34. Langues : Anglais (eng) in Journal of Neurodevelopmental Disorders > 6-1 (December 2014) . - p.34 Mots-clés : Autism spectrum disorder (ASD)  Copy number variations (CNVs)  Han Chinese  Microarray diagnostic testing Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorders (ASDs) are a group of neurodevelopmental conditions with a demonstrated genetic etiology. Rare (<1% frequency) copy number variations (CNVs) account for a proportion of the genetic events involved, but the contribution of these events in non-European ASD populations has not been well studied. Here, we report on rare CNVs detected in a cohort of individuals with ASD of Han Chinese background. METHODS: DNA samples were obtained from 104 ASD probands and their parents who were recruited from Harbin, China. Samples were genotyped on the Affymetrix CytoScan HD platform. Rare CNVs were identified by comparing data with 873 technology-matched controls from Ontario and 1,235 additional population controls of Han Chinese ethnicity. RESULTS: Of the probands, 8.6% had at least 1 de novo CNV (overlapping the GIGYF2, SPRY1, 16p13.3, 16p11.2, 17p13.3-17p13.2, DMD, and NAP1L6 genes/loci). Rare inherited CNVs affected other plausible neurodevelopmental candidate genes including GRID2, LINGO2, and SLC39A12. A 24-kb duplication was also identified at YWHAE, a gene previously implicated in ASD and other developmental disorders. This duplication is observed at a similar frequency in cases and in population controls and is likely a benign Asian-specific copy number polymorphism. CONCLUSIONS: Our findings help define genomic features relevant to ASD in the Han Chinese and emphasize the importance of using ancestry-matched controls in medical genetic interpretations. En ligne : http://dx.doi.org/10.1186/1866-1955-6-34 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=346

Depicting the composition of gut microbiota in children with tic disorders: an exploratory study / Wenjie XI in Journal of Child Psychology and Psychiatry, 62-10 (October 2021)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 62-10 (October 2021) . - p.1246-1254 Titre : Depicting the composition of gut microbiota in children with tic disorders: an exploratory study Type de document : texte imprimé Auteurs : Wenjie XI, Auteur ; Xuefeng GAO, Auteur ; Huijun ZHAO, Auteur ; Xuerong LUO, Auteur ; Jun LI, Auteur ; Xinjie TAN, Auteur ; Lifang WANG, Auteur ; Jian-Bo ZHAO, Auteur ; Jing WANG, Auteur ; Guang YANG, Auteur ; Li-Ying LIU, Auteur ; Yang-Yang WANG, Auteur ; Lilhua PENG, Auteur ; Li-Ping ZOU, Auteur ; Yongjie YANG, Auteur Année de publication : 2021 Article en page(s) : p.1246-1254 Langues : Anglais (eng) Mots-clés : Bacteroides  Child  Gastrointestinal Microbiome  Humans  Prevotella  Ruminococcus  Streptococcus  Tic Disorders  dopamine receptor antagonists  gut microbiota  metabolic pathways  metagenomics Index. décimale : PER Périodiques Résumé : BACKGROUND: Symptom improvement in children with tic disorder (TD) following fecal microbiota transplantation led us to investigate the gut microbiota in TD. This exploratory study aims to depict the gut microbial profile in patients with TD and explore the impact of dopamine receptor antagonist (DRA) drugs on the composition and metabolic function of the gut microbiota. METHODS: The gut microbiota were profiled in fecal samples of 49 children with TD and 50 matched healthy controls (HC) using shotgun metagenomic sequencing. A random forest (RF) model was constructed using the gut bacterial species to distinguish TD from HC. Associations between clinical metadata and microbial abundance or function were analyzed using MaAsLin2 and Spearman correlation. RESULTS: The gut microbiota in children with TD was featured by higher abundances of Bacteroides plebeius and Ruminococcus lactaris (a potential pro-inflammatory taxon) and lower abundances of Prevotella stercorea and Streptococcus lutetiensis compared to HC. The constructed RF model accurately distinguished TD from HC based on the gut microbiota profile, resulting in an AUC of 0.884. Significant correlations were observed between tic symptom severity and the abundances of multiple bacterial species and gut microbiota metabolic functions. Multivariate analysis identified an upregulation of 4-aminobutanoate (GABA) degradation in the gut microbiota associated with TD status. The gut microbiota of DRA-treated TD children showed a distinct gut microbiota compared to the treatment-naïve group, represented by an increase in some potential enteric pathogens such as Escherichia coli, a decline in several species including Akkermansia muciniphila, and alterations in various metabolic functions. CONCLUSIONS: Bacterial species promoting inflammatory responses and those modulating neurotransmitters such as GABA may be involved in the pathogenesis of TD. The use of DRA drugs is likely to induce overgrowth of some enteric pathogens and alter the gut microbiota metabolism. En ligne : http://dx.doi.org/10.1111/jcpp.13409 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=456 [article] Depicting the composition of gut microbiota in children with tic disorders: an exploratory study [texte imprimé] / Wenjie XI, Auteur ; Xuefeng GAO, Auteur ; Huijun ZHAO, Auteur ; Xuerong LUO, Auteur ; Jun LI, Auteur ; Xinjie TAN, Auteur ; Lifang WANG, Auteur ; Jian-Bo ZHAO, Auteur ; Jing WANG, Auteur ; Guang YANG, Auteur ; Li-Ying LIU, Auteur ; Yang-Yang WANG, Auteur ; Lilhua PENG, Auteur ; Li-Ping ZOU, Auteur ; Yongjie YANG, Auteur . - 2021 . - p.1246-1254. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 62-10 (October 2021) . - p.1246-1254 Mots-clés : Bacteroides  Child  Gastrointestinal Microbiome  Humans  Prevotella  Ruminococcus  Streptococcus  Tic Disorders  dopamine receptor antagonists  gut microbiota  metabolic pathways  metagenomics Index. décimale : PER Périodiques Résumé : BACKGROUND: Symptom improvement in children with tic disorder (TD) following fecal microbiota transplantation led us to investigate the gut microbiota in TD. This exploratory study aims to depict the gut microbial profile in patients with TD and explore the impact of dopamine receptor antagonist (DRA) drugs on the composition and metabolic function of the gut microbiota. METHODS: The gut microbiota were profiled in fecal samples of 49 children with TD and 50 matched healthy controls (HC) using shotgun metagenomic sequencing. A random forest (RF) model was constructed using the gut bacterial species to distinguish TD from HC. Associations between clinical metadata and microbial abundance or function were analyzed using MaAsLin2 and Spearman correlation. RESULTS: The gut microbiota in children with TD was featured by higher abundances of Bacteroides plebeius and Ruminococcus lactaris (a potential pro-inflammatory taxon) and lower abundances of Prevotella stercorea and Streptococcus lutetiensis compared to HC. The constructed RF model accurately distinguished TD from HC based on the gut microbiota profile, resulting in an AUC of 0.884. Significant correlations were observed between tic symptom severity and the abundances of multiple bacterial species and gut microbiota metabolic functions. Multivariate analysis identified an upregulation of 4-aminobutanoate (GABA) degradation in the gut microbiota associated with TD status. The gut microbiota of DRA-treated TD children showed a distinct gut microbiota compared to the treatment-naïve group, represented by an increase in some potential enteric pathogens such as Escherichia coli, a decline in several species including Akkermansia muciniphila, and alterations in various metabolic functions. CONCLUSIONS: Bacterial species promoting inflammatory responses and those modulating neurotransmitters such as GABA may be involved in the pathogenesis of TD. The use of DRA drugs is likely to induce overgrowth of some enteric pathogens and alter the gut microbiota metabolism. En ligne : http://dx.doi.org/10.1111/jcpp.13409 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=456

Effects of a social stimulus on gene expression in a mouse model of fragile X syndrome / Tiffany D. ROGERS in Molecular Autism, 8 (2017)  

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Erratum to: Neural synchronization deficits linked to cortical hyper-excitability and auditory hypersensitivity in fragile X syndrome / Lauren E ETHRIDGE in Molecular Autism, 8 (2017)  

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Few individuals with Lennox-Gastaut syndrome have autism spectrum disorder: a comparison with Dravet syndrome / Na HE in Journal of Neurodevelopmental Disorders, 10-1 (December 2018)  

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Genomic sequencing of a dyslexia susceptibility haplotype encompassing ROBO1 / Satu MASSINEN in Journal of Neurodevelopmental Disorders, 8-1 (December 2016)  

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A granular perspective on inclusion: Objectively measured interactions of preschoolers with and without autism / Regina M. FASANO in Autism Research, 14-8 (August 2021)  

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Neural synchronization deficits linked to cortical hyper-excitability and auditory hypersensitivity in fragile X syndrome / Lauren E ETHRIDGE in Molecular Autism, 8 (2017)  

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Refractive Status and Amblyopia Risk Factors in Chinese Children with Autism Spectrum Disorder / Jing WANG in Journal of Autism and Developmental Disorders, 48-5 (May 2018)  

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A resting EEG study of neocortical hyperexcitability and altered functional connectivity in fragile X syndrome / Jing WANG in Journal of Neurodevelopmental Disorders, 9-1 (December 2017)  

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Resting state EEG abnormalities in autism spectrum disorders / Jing WANG in Journal of Neurodevelopmental Disorders, 5-1 (December 2013)  

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Shank3 deficiency elicits autistic-like behaviors by activating p38? in hypothalamic AgRP neurons / Shanshan WU in Molecular Autism, 15 (2024)  

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