Childhood adversity moderates the influence of proximal episodic stress on the cortisol awakening response and depressive symptoms in adolescents / Lisa R. STARR in Development and Psychopathology, 29-5 (December 2017)  

Public ISBD [article]  inDevelopment and Psychopathology > 29-5 (December 2017) . - p.1877-1893 Titre : Childhood adversity moderates the influence of proximal episodic stress on the cortisol awakening response and depressive symptoms in adolescents Type de document : Texte imprimé et/ou numérique Auteurs : Lisa R. STARR, Auteur ; Kimberly DIENES, Auteur ; Catherine B. STROUD, Auteur ; Zoey A. SHAW, Auteur ; Y. Irina LI, Auteur ; Fanny MLAWER, Auteur ; Meghan HUANG, Auteur Article en page(s) : p.1877-1893 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Childhood adversity (CA) is known to predict sensitization to proximal stressors. Researchers have suggested that disruptions in hypothalamus–pituitary–adrenal axis functioning may be a biological mechanism. If so, CA may predict altered associations between proximal life stress and markers of cortisol secretion. We examined whether CA moderates associations between recent episodic stress and (a) the cortisol awakening response (CAR), and (b) depressive symptoms, in 241 adolescents aged 14–17 years (cortisol n = 196). Salivary cortisol was sampled at 0, 30, and 60 min postawakening for 2 days. The CAR was calculated as the area under the curve with respect to increase and waking cortisol. CA and episodic stress were assessed using contextual-threat-method-coded objective interviews. CA significantly interacted with episodic stress to predict both the CAR and depression. Among those with low CA, episodic stress predicted increased CAR but did not predict depression. For adolescents with high CA, episodic stress predicted lower CAR and higher depression. These interactions were found only for independent (uncontrollable, fateful) events, and not for dependent (self-generated) stress. Increased allostatic load resulting from CA exposure may interfere with adolescents' ability to optimally regulate their CAR in relation to recent stress, contributing to increased depression risk. En ligne : http://dx.doi.org/10.1017/S0954579417001468 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=324 [article] Childhood adversity moderates the influence of proximal episodic stress on the cortisol awakening response and depressive symptoms in adolescents [Texte imprimé et/ou numérique] / Lisa R. STARR, Auteur ; Kimberly DIENES, Auteur ; Catherine B. STROUD, Auteur ; Zoey A. SHAW, Auteur ; Y. Irina LI, Auteur ; Fanny MLAWER, Auteur ; Meghan HUANG, Auteur . - p.1877-1893. Langues : Anglais (eng) in Development and Psychopathology > 29-5 (December 2017) . - p.1877-1893 Index. décimale : PER Périodiques Résumé : Childhood adversity (CA) is known to predict sensitization to proximal stressors. Researchers have suggested that disruptions in hypothalamus–pituitary–adrenal axis functioning may be a biological mechanism. If so, CA may predict altered associations between proximal life stress and markers of cortisol secretion. We examined whether CA moderates associations between recent episodic stress and (a) the cortisol awakening response (CAR), and (b) depressive symptoms, in 241 adolescents aged 14–17 years (cortisol n = 196). Salivary cortisol was sampled at 0, 30, and 60 min postawakening for 2 days. The CAR was calculated as the area under the curve with respect to increase and waking cortisol. CA and episodic stress were assessed using contextual-threat-method-coded objective interviews. CA significantly interacted with episodic stress to predict both the CAR and depression. Among those with low CA, episodic stress predicted increased CAR but did not predict depression. For adolescents with high CA, episodic stress predicted lower CAR and higher depression. These interactions were found only for independent (uncontrollable, fateful) events, and not for dependent (self-generated) stress. Increased allostatic load resulting from CA exposure may interfere with adolescents' ability to optimally regulate their CAR in relation to recent stress, contributing to increased depression risk. En ligne : http://dx.doi.org/10.1017/S0954579417001468 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=324

Commentary: Defining and Understanding Adolescent Romantic Competence: Progress, Challenges, and Implications / Joanne DAVILA in Journal of Clinical Child & Adolescent Psychology, 36-4 (October-December 2007)  

Public ISBD [article]  inJournal of Clinical Child & Adolescent Psychology > 36-4 (October-December 2007) . - p.534-540 Titre : Commentary: Defining and Understanding Adolescent Romantic Competence: Progress, Challenges, and Implications Type de document : Texte imprimé et/ou numérique Auteurs : Joanne DAVILA, Auteur ; Catherine B. STROUD, Auteur ; Melissa RAMSAY MILLER, Auteur ; Sara J. STEINBERG, Auteur Année de publication : 2007 Article en page(s) : p.534-540 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Commentary on the articles in this special section is provided. Following a brief review of the articles in this section, discussion focuses on the conceptualization of adolescent romantic competence, the importance of studying competence, and the implications of research in this area for knowing how to help young people have more successful romantic relationships. En ligne : http://dx.doi.org/10.1080/15374410701662147 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=312 [article] Commentary: Defining and Understanding Adolescent Romantic Competence: Progress, Challenges, and Implications [Texte imprimé et/ou numérique] / Joanne DAVILA, Auteur ; Catherine B. STROUD, Auteur ; Melissa RAMSAY MILLER, Auteur ; Sara J. STEINBERG, Auteur . - 2007 . - p.534-540. Langues : Anglais (eng) in Journal of Clinical Child & Adolescent Psychology > 36-4 (October-December 2007) . - p.534-540 Index. décimale : PER Périodiques Résumé : Commentary on the articles in this special section is provided. Following a brief review of the articles in this section, discussion focuses on the conceptualization of adolescent romantic competence, the importance of studying competence, and the implications of research in this area for knowing how to help young people have more successful romantic relationships. En ligne : http://dx.doi.org/10.1080/15374410701662147 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=312

Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress / Catherine B. STROUD in Development and Psychopathology, 35-3 (August 2023)  

Public ISBD [article]  inDevelopment and Psychopathology > 35-3 (August 2023) . - p.1272-1287 Titre : Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress Type de document : Texte imprimé et/ou numérique Auteurs : Catherine B. STROUD, Auteur ; Frances R. CHEN, Auteur ; Erin E. DUNNING, Auteur ; Julia CHENG, Auteur ; Carey MARR, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur Article en page(s) : p.1272-1287 Langues : Anglais (eng) Mots-clés : adolescents  depression  early adversity  interpersonal stress  stressful life events Index. décimale : PER Périodiques Résumé : Early adversity confers risk for depression in part through its association with recent (i.e., proximal) acute stress. However, it remains unresolved whether: a) early adversity predicts increases in recent acute stress over time; b) all - or only certain types - of recent events mediate the relationship between early adversity and depression; and c) early adversity places individuals at greater risk for depression via greater exposure to independent (i.e., fateful) interpersonal events or via greater generation of dependent (i.e., partially self-initiated) interpersonal events (i.e., stress generation) or both. These questions were examined in a 3-wave longitudinal study of early adolescent girls (N = 125; M = 12.35 years [SD = .77]) with no history of diagnosable depression using contextual life stress and diagnostic interviews. Path analyses indicated that increases in past-year acute interpersonal, but not non-interpersonal, stress mediated the link between early adversity and depressive symptoms. The mediating role of interpersonal events was limited to independent ones, suggesting increases in interpersonal event exposure, not interpersonal stress generation, acted as a mediator. Finally, findings support prior evidence that early adversity may not directly predict future depressive symptoms. Implications for understanding the role of recent stress in the association between early adversity and adolescent depression are discussed. En ligne : http://dx.doi.org/10.1017/S0954579421001176 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=511 [article] Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress [Texte imprimé et/ou numérique] / Catherine B. STROUD, Auteur ; Frances R. CHEN, Auteur ; Erin E. DUNNING, Auteur ; Julia CHENG, Auteur ; Carey MARR, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur . - p.1272-1287. Langues : Anglais (eng) in Development and Psychopathology > 35-3 (August 2023) . - p.1272-1287 Mots-clés : adolescents  depression  early adversity  interpersonal stress  stressful life events Index. décimale : PER Périodiques Résumé : Early adversity confers risk for depression in part through its association with recent (i.e., proximal) acute stress. However, it remains unresolved whether: a) early adversity predicts increases in recent acute stress over time; b) all - or only certain types - of recent events mediate the relationship between early adversity and depression; and c) early adversity places individuals at greater risk for depression via greater exposure to independent (i.e., fateful) interpersonal events or via greater generation of dependent (i.e., partially self-initiated) interpersonal events (i.e., stress generation) or both. These questions were examined in a 3-wave longitudinal study of early adolescent girls (N = 125; M = 12.35 years [SD = .77]) with no history of diagnosable depression using contextual life stress and diagnostic interviews. Path analyses indicated that increases in past-year acute interpersonal, but not non-interpersonal, stress mediated the link between early adversity and depressive symptoms. The mediating role of interpersonal events was limited to independent ones, suggesting increases in interpersonal event exposure, not interpersonal stress generation, acted as a mediator. Finally, findings support prior evidence that early adversity may not directly predict future depressive symptoms. Implications for understanding the role of recent stress in the association between early adversity and adolescent depression are discussed. En ligne : http://dx.doi.org/10.1017/S0954579421001176 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=511

Early adversity and internalizing symptoms in adolescence: Mediation by individual differences in latent trait cortisol / Catherine B. STROUD in Development and Psychopathology, 31-2 (May 2019)  

Public ISBD [article]  inDevelopment and Psychopathology > 31-2 (May 2019) . - p.509-524 Titre : Early adversity and internalizing symptoms in adolescence: Mediation by individual differences in latent trait cortisol Type de document : Texte imprimé et/ou numérique Auteurs : Catherine B. STROUD, Auteur ; Frances S. CHEN, Auteur ; Leah D. DOANE, Auteur ; Douglas A. GRANGER, Auteur Article en page(s) : p.509-524 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Research suggests that early adversity places individuals at risk for psychopathology across the life span. Guided by concepts of allostasis and allostatic load, the present study examined whether early adversity contributes to the development of subsequent internalizing symptoms through its association with traitlike individual differences in hypothalamic–pituitary–adrenal axis regulation. Early adolescent girls (n = 113; M age = 12.30 years) provided saliva samples at waking, 30 min postwaking, and bedtime over 3 days (later assayed for cortisol). Objective contextual stress interviews with adolescents and their mothers were used to assess the accumulation of nine types of early adversity within the family environment. Greater early adversity predicted subsequent increases in internalizing symptoms through lower levels of latent trait cortisol. Traitlike individual differences in hypothalamic–pituitary–adrenal axis activity may be among the mechanisms through which early adversity confers risk for the development of psychopathology. En ligne : http://dx.doi.org/10.1017/S0954579418000044 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=393 [article] Early adversity and internalizing symptoms in adolescence: Mediation by individual differences in latent trait cortisol [Texte imprimé et/ou numérique] / Catherine B. STROUD, Auteur ; Frances S. CHEN, Auteur ; Leah D. DOANE, Auteur ; Douglas A. GRANGER, Auteur . - p.509-524. Langues : Anglais (eng) in Development and Psychopathology > 31-2 (May 2019) . - p.509-524 Index. décimale : PER Périodiques Résumé : Research suggests that early adversity places individuals at risk for psychopathology across the life span. Guided by concepts of allostasis and allostatic load, the present study examined whether early adversity contributes to the development of subsequent internalizing symptoms through its association with traitlike individual differences in hypothalamic–pituitary–adrenal axis regulation. Early adolescent girls (n = 113; M age = 12.30 years) provided saliva samples at waking, 30 min postwaking, and bedtime over 3 days (later assayed for cortisol). Objective contextual stress interviews with adolescents and their mothers were used to assess the accumulation of nine types of early adversity within the family environment. Greater early adversity predicted subsequent increases in internalizing symptoms through lower levels of latent trait cortisol. Traitlike individual differences in hypothalamic–pituitary–adrenal axis activity may be among the mechanisms through which early adversity confers risk for the development of psychopathology. En ligne : http://dx.doi.org/10.1017/S0954579418000044 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=393

Stress sensitization to depression following childhood adversity: Moderation by HPA axis and serotonergic multilocus profile scores / Lisa R. STARR in Development and Psychopathology, 33-4 (October 2021)  

Public ISBD [article]  inDevelopment and Psychopathology > 33-4 (October 2021) . - p.1264-1278 Titre : Stress sensitization to depression following childhood adversity: Moderation by HPA axis and serotonergic multilocus profile scores Type de document : Texte imprimé et/ou numérique Auteurs : Lisa R. STARR, Auteur ; Catherine B. STROUD, Auteur ; Zoey A. SHAW, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur Article en page(s) : p.1264-1278 Langues : Anglais (eng) Mots-clés : childhood adversity  depression  gene-by-environment interaction  genetic  stress sensitization  stressful life events Index. décimale : PER Périodiques Résumé : Childhood adversity appears to sensitize youth to stress, increasing depression risk following stressful life events occurring throughout the lifespan. Some evidence suggests hypothalamic–pituitary–adrenal (HPA) axis-related and serotonergic genetic variation moderates this effect, in a “gene-by-environment-by-environment” interaction (G × E × E). However, prior research has tested single genetic variants, limiting power. The current study uses a multilocus genetic profile score (MGPS) approach to capture polygenic risk relevant to HPA axis and serotonergic functioning. Adolescents (N = 241, Mage = 15.90) completed contextual-threat-based interviews assessing childhood adversity and acute life events, and diagnostic interviews assessing depression. Established MGPSs indexed genetic variation linked to HPA axis (10 single nucleotide polymorphisms [SNPs]) and serotonergic (five SNPs) functioning. Results showed significant MGPS × Childhood Adversity × Recent Life Stress interactions predicting depression for both HPA axis and serotonergic MGPSs, with both risk scores predicting stronger Childhood Adversity × Recent Stress interactions. Serotonergic genetic risk specifically predicted sensitization to major interpersonal stressors. The serotonergic MGPS G × E × E was re-tested in an independent replication sample of early adolescent girls, with comparable results. Findings support the notion that genetic variation linked to these two neurobiological symptoms alters stress sensitization, and that gene-by-environment (G × E) interactions may be qualified by environmental exposures occurring at different points in development. En ligne : http://dx.doi.org/10.1017/S0954579420000474 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 [article] Stress sensitization to depression following childhood adversity: Moderation by HPA axis and serotonergic multilocus profile scores [Texte imprimé et/ou numérique] / Lisa R. STARR, Auteur ; Catherine B. STROUD, Auteur ; Zoey A. SHAW, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur . - p.1264-1278. Langues : Anglais (eng) in Development and Psychopathology > 33-4 (October 2021) . - p.1264-1278 Mots-clés : childhood adversity  depression  gene-by-environment interaction  genetic  stress sensitization  stressful life events Index. décimale : PER Périodiques Résumé : Childhood adversity appears to sensitize youth to stress, increasing depression risk following stressful life events occurring throughout the lifespan. Some evidence suggests hypothalamic–pituitary–adrenal (HPA) axis-related and serotonergic genetic variation moderates this effect, in a “gene-by-environment-by-environment” interaction (G × E × E). However, prior research has tested single genetic variants, limiting power. The current study uses a multilocus genetic profile score (MGPS) approach to capture polygenic risk relevant to HPA axis and serotonergic functioning. Adolescents (N = 241, Mage = 15.90) completed contextual-threat-based interviews assessing childhood adversity and acute life events, and diagnostic interviews assessing depression. Established MGPSs indexed genetic variation linked to HPA axis (10 single nucleotide polymorphisms [SNPs]) and serotonergic (five SNPs) functioning. Results showed significant MGPS × Childhood Adversity × Recent Life Stress interactions predicting depression for both HPA axis and serotonergic MGPSs, with both risk scores predicting stronger Childhood Adversity × Recent Stress interactions. Serotonergic genetic risk specifically predicted sensitization to major interpersonal stressors. The serotonergic MGPS G × E × E was re-tested in an independent replication sample of early adolescent girls, with comparable results. Findings support the notion that genetic variation linked to these two neurobiological symptoms alters stress sensitization, and that gene-by-environment (G × E) interactions may be qualified by environmental exposures occurring at different points in development. En ligne : http://dx.doi.org/10.1017/S0954579420000474 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457

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