Anxiety in the family: a genetically informed analysis of transactional associations between mother, father and child anxiety symptoms / Yasmin I. AHMADZADEH in Journal of Child Psychology and Psychiatry, 60-12 (December 2019)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 60-12 (December 2019) . - p.1269-1277 Titre : Anxiety in the family: a genetically informed analysis of transactional associations between mother, father and child anxiety symptoms Type de document : Texte imprimé et/ou numérique Auteurs : Yasmin I. AHMADZADEH, Auteur ; T. C. ELEY, Auteur ; L. D. LEVE, Auteur ; D. S. SHAW, Auteur ; M. N. NATSUAKI, Auteur ; D. REISS, Auteur ; Jenae M. NEIDERHISER, Auteur ; T. A. MCADAMS, Auteur Article en page(s) : p.1269-1277 Langues : Anglais (eng) Mots-clés : Anxiety  genetics  longitudinal  parent-child relationships  structural equation modelling Index. décimale : PER Périodiques Résumé : BACKGROUND: Anxiety in parents is associated with anxiety in offspring, although little is known about the mechanisms underpinning these intergenerational associations. We conducted the first genetically sensitive study to simultaneously examine the effects of mother, father and child anxiety symptoms on each other over time. METHOD: Adoptive parent and child symptoms were measured at child ages 6, 7 and 8 years from 305 families involved in the Early Growth and Development Study, using a prospective adoption design. Children were adopted at birth to nonrelatives, and composite data on internalising problems within birth families were used as a proxy measure of offspring inherited risk for anxiety. Structural equation models were fitted to the data to examine prospective associations between adoptive mother, father and child symptoms, whilst accounting for individuals' symptom stability over time. RESULTS: Child anxiety symptoms at age 7 predicted adoptive mothers' anxiety symptoms at age 8. No mother-to-child or child-to-father effects were observed. These results were consistent in sensitivity analyses using only paternal offspring reports and using a second measure of child anxiety symptoms. Fathers' anxiety symptoms at child age 6 prospectively predicted child symptoms, but only when paternal offspring reports were included in the model. Composite data on birth family internalising problems were not associated with child anxiety symptoms. CONCLUSIONS: Results show environmentally mediated associations between parent and child anxiety symptoms. Results support developmental theories suggesting that child anxiety symptoms can exert influence on caregivers, and mothers and fathers may play unique roles during the development of child symptoms. Further research is needed on the role of genetic transmission associated with anxiety symptoms in biologically related families. In the meantime, researchers and clinicians should strive to include fathers in assessments and consider the effects of child symptoms on caregivers. En ligne : http://dx.doi.org/10.1111/jcpp.13068 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412 [article] Anxiety in the family: a genetically informed analysis of transactional associations between mother, father and child anxiety symptoms [Texte imprimé et/ou numérique] / Yasmin I. AHMADZADEH, Auteur ; T. C. ELEY, Auteur ; L. D. LEVE, Auteur ; D. S. SHAW, Auteur ; M. N. NATSUAKI, Auteur ; D. REISS, Auteur ; Jenae M. NEIDERHISER, Auteur ; T. A. MCADAMS, Auteur . - p.1269-1277. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 60-12 (December 2019) . - p.1269-1277 Mots-clés : Anxiety  genetics  longitudinal  parent-child relationships  structural equation modelling Index. décimale : PER Périodiques Résumé : BACKGROUND: Anxiety in parents is associated with anxiety in offspring, although little is known about the mechanisms underpinning these intergenerational associations. We conducted the first genetically sensitive study to simultaneously examine the effects of mother, father and child anxiety symptoms on each other over time. METHOD: Adoptive parent and child symptoms were measured at child ages 6, 7 and 8 years from 305 families involved in the Early Growth and Development Study, using a prospective adoption design. Children were adopted at birth to nonrelatives, and composite data on internalising problems within birth families were used as a proxy measure of offspring inherited risk for anxiety. Structural equation models were fitted to the data to examine prospective associations between adoptive mother, father and child symptoms, whilst accounting for individuals' symptom stability over time. RESULTS: Child anxiety symptoms at age 7 predicted adoptive mothers' anxiety symptoms at age 8. No mother-to-child or child-to-father effects were observed. These results were consistent in sensitivity analyses using only paternal offspring reports and using a second measure of child anxiety symptoms. Fathers' anxiety symptoms at child age 6 prospectively predicted child symptoms, but only when paternal offspring reports were included in the model. Composite data on birth family internalising problems were not associated with child anxiety symptoms. CONCLUSIONS: Results show environmentally mediated associations between parent and child anxiety symptoms. Results support developmental theories suggesting that child anxiety symptoms can exert influence on caregivers, and mothers and fathers may play unique roles during the development of child symptoms. Further research is needed on the role of genetic transmission associated with anxiety symptoms in biologically related families. In the meantime, researchers and clinicians should strive to include fathers in assessments and consider the effects of child symptoms on caregivers. En ligne : http://dx.doi.org/10.1111/jcpp.13068 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412

Commentary: Childhood conduct problems are a public health crisis and require resources: a commentary on Rivenbark et al. () / S. Alexandra BURT in Journal of Child Psychology and Psychiatry, 59-6 (June 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-6 (June 2018) . - p.711-713 Titre : Commentary: Childhood conduct problems are a public health crisis and require resources: a commentary on Rivenbark et al. () Type de document : Texte imprimé et/ou numérique Auteurs : S. Alexandra BURT, Auteur ; L. W. HYDE, Auteur ; P. J. FRICK, Auteur ; Sara R. JAFFEE, Auteur ; D. S. SHAW, Auteur ; R. TREMBLAY, Auteur Article en page(s) : p.711-713 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Conduct problems (CP) are actions that violate societal norms and/or the personal/property rights of others, and include behaviors such as vandalism, theft, bullying, and assault. Roughly 8%-10% of children engage in the more severe childhood-onset form of CP, while another 25% initiate clinically-significant levels of CP during adolescence. As deftly observed in Rivenbark et al. (), however, the high prevalence of CP belies its severity: Youth with CP are at increased risk for a number of deleterious individual outcomes, including academic delay/dropout, low professional achievement, psychopathology, addiction, and family instability. En ligne : http://dx.doi.org/10.1111/jcpp.12930 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=363 [article] Commentary: Childhood conduct problems are a public health crisis and require resources: a commentary on Rivenbark et al. () [Texte imprimé et/ou numérique] / S. Alexandra BURT, Auteur ; L. W. HYDE, Auteur ; P. J. FRICK, Auteur ; Sara R. JAFFEE, Auteur ; D. S. SHAW, Auteur ; R. TREMBLAY, Auteur . - p.711-713. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-6 (June 2018) . - p.711-713 Index. décimale : PER Périodiques Résumé : Conduct problems (CP) are actions that violate societal norms and/or the personal/property rights of others, and include behaviors such as vandalism, theft, bullying, and assault. Roughly 8%-10% of children engage in the more severe childhood-onset form of CP, while another 25% initiate clinically-significant levels of CP during adolescence. As deftly observed in Rivenbark et al. (), however, the high prevalence of CP belies its severity: Youth with CP are at increased risk for a number of deleterious individual outcomes, including academic delay/dropout, low professional achievement, psychopathology, addiction, and family instability. En ligne : http://dx.doi.org/10.1111/jcpp.12930 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=363

Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood / L. W. HYDE in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Titre : Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood Type de document : Texte imprimé et/ou numérique Auteurs : L. W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; L. MURRAY, Auteur ; E. E. FORBES, Auteur Article en page(s) : p.1073-1082 Langues : Anglais (eng) Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood [Texte imprimé et/ou numérique] / L. W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; L. MURRAY, Auteur ; E. E. FORBES, Auteur . - p.1073-1082. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Dysregulated Irritability as a Window on Young Children's Psychiatric Risk: Transdiagnostic Effects via the Family Check-Up / J. D. SMITH in Development and Psychopathology, 31-5 (December 2019)  

Public ISBD [article]  inDevelopment and Psychopathology > 31-5 (December 2019) . - p.1887-1899 Titre : Dysregulated Irritability as a Window on Young Children's Psychiatric Risk: Transdiagnostic Effects via the Family Check-Up Type de document : Texte imprimé et/ou numérique Auteurs : J. D. SMITH, Auteur ; Lauren S. WAKSCHLAG, Auteur ; S. KROGH-JESPERSEN, Auteur ; John T. WALKUP, Auteur ; M. N. WILSON, Auteur ; T. J. DISHION, Auteur ; D. S. SHAW, Auteur Année de publication : 2019 Article en page(s) : p.1887-1899 Langues : Anglais (eng) Mots-clés : Family Check-Up  early childhood  irritability  mental health  parent training  prevention  transdiagnostic Index. décimale : PER Périodiques Résumé : Building on prior work using Tom Dishion's Family Check-Up, the current article examined intervention effects on dysregulated irritability in early childhood. Dysregulated irritability, defined as reactive and intense response to frustration, and prolonged angry mood, is an ideal marker of neurodevelopmental vulnerability to later psychopathology because it is a transdiagnostic indicator of decrements in self-regulation that are measurable in the first years of life that have lifelong implications for health and disease. This study is perhaps the first randomized trial to examine the direct effects of an evidence- and family-based intervention, the Family Check-Up (FCU), on irritability in early childhood and the effects of reductions in irritability on later risk of child internalizing and externalizing symptomatology. Data from the geographically and sociodemographically diverse multisite Early Steps randomized prevention trial were used. Path modeling revealed intervention effects on irritability at age 4, which predicted lower externalizing and internalizing symptoms at age 10.5. Results indicate that family-based programs initiated in early childhood can reduce early childhood irritability and later risk for psychopathology. This holds promise for earlier identification and prevention approaches that target transdiagnostic pathways. Implications for future basic and prevention research are discussed. En ligne : http://dx.doi.org/10.1017/s0954579419000816 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412 [article] Dysregulated Irritability as a Window on Young Children's Psychiatric Risk: Transdiagnostic Effects via the Family Check-Up [Texte imprimé et/ou numérique] / J. D. SMITH, Auteur ; Lauren S. WAKSCHLAG, Auteur ; S. KROGH-JESPERSEN, Auteur ; John T. WALKUP, Auteur ; M. N. WILSON, Auteur ; T. J. DISHION, Auteur ; D. S. SHAW, Auteur . - 2019 . - p.1887-1899. Langues : Anglais (eng) in Development and Psychopathology > 31-5 (December 2019) . - p.1887-1899 Mots-clés : Family Check-Up  early childhood  irritability  mental health  parent training  prevention  transdiagnostic Index. décimale : PER Périodiques Résumé : Building on prior work using Tom Dishion's Family Check-Up, the current article examined intervention effects on dysregulated irritability in early childhood. Dysregulated irritability, defined as reactive and intense response to frustration, and prolonged angry mood, is an ideal marker of neurodevelopmental vulnerability to later psychopathology because it is a transdiagnostic indicator of decrements in self-regulation that are measurable in the first years of life that have lifelong implications for health and disease. This study is perhaps the first randomized trial to examine the direct effects of an evidence- and family-based intervention, the Family Check-Up (FCU), on irritability in early childhood and the effects of reductions in irritability on later risk of child internalizing and externalizing symptomatology. Data from the geographically and sociodemographically diverse multisite Early Steps randomized prevention trial were used. Path modeling revealed intervention effects on irritability at age 4, which predicted lower externalizing and internalizing symptoms at age 10.5. Results indicate that family-based programs initiated in early childhood can reduce early childhood irritability and later risk for psychopathology. This holds promise for earlier identification and prevention approaches that target transdiagnostic pathways. Implications for future basic and prevention research are discussed. En ligne : http://dx.doi.org/10.1017/s0954579419000816 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412

Longitudinal examination of pathways to peer problems in middle childhood: A siblings-reared-apart design / L. D. LEVE in Development and Psychopathology, 31-5 (December 2019)  

Public ISBD [article]  inDevelopment and Psychopathology > 31-5 (December 2019) . - p.1633-1647 Titre : Longitudinal examination of pathways to peer problems in middle childhood: A siblings-reared-apart design Type de document : Texte imprimé et/ou numérique Auteurs : L. D. LEVE, Auteur ; A. M. GRIFFIN, Auteur ; M. N. NATSUAKI, Auteur ; G. T. HAROLD, Auteur ; Jenae M. NEIDERHISER, Auteur ; J. M. GANIBAN, Auteur ; D. S. SHAW, Auteur ; D. REISS, Auteur Année de publication : 2019 Article en page(s) : p.1633-1647 Langues : Anglais (eng) Mots-clés : adoption design  inhibitory control  parental hostility  peer problems  siblings Index. décimale : PER Périodiques Résumé : To advance research from Dishion and others on associations between parenting and peer problems across childhood, we used a sample of 177 sibling pairs reared apart since birth (because of adoption of one of the siblings) to examine associations between parental hostility and children's peer problems when children were ages 7 and 9.5 years (n = 329 children). We extended conventional cross-lagged parent-peer models by incorporating child inhibitory control as an additional predictor and examining genetic contributions via birth mother psychopathology. Path models indicated a cross-lagged association from parental hostility to later peer problems. When child inhibitory control was included, birth mother internalizing symptoms were associated with poorer child inhibitory control, which was associated with more parental hostility and peer problems. The cross-lagged paths from parental hostility to peer problems were no longer significant in the full model. Multigroup analyses revealed that the path from birth mother internalizing symptoms to child inhibitory control was significantly higher for birth parent-reared children, indicating the possible contribution of passive gene-environment correlation to this association. Exploratory analyses suggested that each child's unique rearing context contributed to his or her inhibitory control and peer behavior. Implications for the development of evidence-based interventions are discussed. En ligne : http://dx.doi.org/10.1017/s0954579419000890 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412 [article] Longitudinal examination of pathways to peer problems in middle childhood: A siblings-reared-apart design [Texte imprimé et/ou numérique] / L. D. LEVE, Auteur ; A. M. GRIFFIN, Auteur ; M. N. NATSUAKI, Auteur ; G. T. HAROLD, Auteur ; Jenae M. NEIDERHISER, Auteur ; J. M. GANIBAN, Auteur ; D. S. SHAW, Auteur ; D. REISS, Auteur . - 2019 . - p.1633-1647. Langues : Anglais (eng) in Development and Psychopathology > 31-5 (December 2019) . - p.1633-1647 Mots-clés : adoption design  inhibitory control  parental hostility  peer problems  siblings Index. décimale : PER Périodiques Résumé : To advance research from Dishion and others on associations between parenting and peer problems across childhood, we used a sample of 177 sibling pairs reared apart since birth (because of adoption of one of the siblings) to examine associations between parental hostility and children's peer problems when children were ages 7 and 9.5 years (n = 329 children). We extended conventional cross-lagged parent-peer models by incorporating child inhibitory control as an additional predictor and examining genetic contributions via birth mother psychopathology. Path models indicated a cross-lagged association from parental hostility to later peer problems. When child inhibitory control was included, birth mother internalizing symptoms were associated with poorer child inhibitory control, which was associated with more parental hostility and peer problems. The cross-lagged paths from parental hostility to peer problems were no longer significant in the full model. Multigroup analyses revealed that the path from birth mother internalizing symptoms to child inhibitory control was significantly higher for birth parent-reared children, indicating the possible contribution of passive gene-environment correlation to this association. Exploratory analyses suggested that each child's unique rearing context contributed to his or her inhibitory control and peer behavior. Implications for the development of evidence-based interventions are discussed. En ligne : http://dx.doi.org/10.1017/s0954579419000890 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=412

The role of child negative emotionality in parenting and child adjustment: Gene-environment interplay / E. A. SHEWARK in Journal of Child Psychology and Psychiatry, 62-12 (December 2021)  

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Trajectories and Predictors of Children's Early-Starting Conduct Problems: Child, Family, Genetic, and Intervention Effects / D. S. SHAW in Development and Psychopathology, 31-5 (December 2019)  

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