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Auteur Paul LICHTENSTEIN |
Documents disponibles écrits par cet auteur (35)
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Heritability for adolescent antisocial behavior differs with socioeconomic status: gene–environment interaction / Catherine TUVBLAD in Journal of Child Psychology and Psychiatry, 47-7 (July 2006)
[article]
Titre : Heritability for adolescent antisocial behavior differs with socioeconomic status: gene–environment interaction Type de document : Texte imprimé et/ou numérique Auteurs : Catherine TUVBLAD, Auteur ; Martin GRANN, Auteur ; Paul LICHTENSTEIN, Auteur Année de publication : 2006 Article en page(s) : p.734–743 Langues : Anglais (eng) Mots-clés : Antisocial-behavior socioeconomic-status twin-study sex-differences gene–environment-interaction Index. décimale : PER Périodiques Résumé : Background: Socioeconomic status is often assumed to be of importance for the development of antisocial behavior, yet it explains only a fraction of the variance. One explanation for this paradox could be that socioeconomic status moderates the influence of genetic and environmental effects on antisocial behavior.
Method: TCHAD is a Swedish longitudinal population-based twin study that contains 1,480 twin pairs born 1985–1986. The present study included 1,133 twin pairs, aged 16–17 years. Antisocial behavior was measured through self-report. Family socioeconomic status was assessed by parental-reported education and occupational status. Neighborhood socioeconomic conditions were assessed using five aggregated level variables: ethnic diversity, basic educational level, unemployment level, buying power, and crime-rate. We used structural equation modeling to test whether socioeconomic status interacted with latent genetic and environmental effects for antisocial behavior.
Results: Genetic influences on antisocial behavior were more important in adolescents in socioeconomically more advantaged environments, whereas the shared environment was higher in adolescents in socioeconomically less advantaged environments. Heritability for antisocial behavior was higher in girls than in boys, irrespective of socioeconomic background.
Conclusions: Our results suggest that different intervention policies should be considered in different socioeconomic areas. In socioeconomically advantaged areas, it might be more fruitful to focus on individually based preventions and treatments. In socioeconomically disadvantaged areas, intervention and prevention policies might be more effective on a community level, to account for shared environmental risk factors.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2005.01552.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=758
in Journal of Child Psychology and Psychiatry > 47-7 (July 2006) . - p.734–743[article] Heritability for adolescent antisocial behavior differs with socioeconomic status: gene–environment interaction [Texte imprimé et/ou numérique] / Catherine TUVBLAD, Auteur ; Martin GRANN, Auteur ; Paul LICHTENSTEIN, Auteur . - 2006 . - p.734–743.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 47-7 (July 2006) . - p.734–743
Mots-clés : Antisocial-behavior socioeconomic-status twin-study sex-differences gene–environment-interaction Index. décimale : PER Périodiques Résumé : Background: Socioeconomic status is often assumed to be of importance for the development of antisocial behavior, yet it explains only a fraction of the variance. One explanation for this paradox could be that socioeconomic status moderates the influence of genetic and environmental effects on antisocial behavior.
Method: TCHAD is a Swedish longitudinal population-based twin study that contains 1,480 twin pairs born 1985–1986. The present study included 1,133 twin pairs, aged 16–17 years. Antisocial behavior was measured through self-report. Family socioeconomic status was assessed by parental-reported education and occupational status. Neighborhood socioeconomic conditions were assessed using five aggregated level variables: ethnic diversity, basic educational level, unemployment level, buying power, and crime-rate. We used structural equation modeling to test whether socioeconomic status interacted with latent genetic and environmental effects for antisocial behavior.
Results: Genetic influences on antisocial behavior were more important in adolescents in socioeconomically more advantaged environments, whereas the shared environment was higher in adolescents in socioeconomically less advantaged environments. Heritability for antisocial behavior was higher in girls than in boys, irrespective of socioeconomic background.
Conclusions: Our results suggest that different intervention policies should be considered in different socioeconomic areas. In socioeconomically advantaged areas, it might be more fruitful to focus on individually based preventions and treatments. In socioeconomically disadvantaged areas, intervention and prevention policies might be more effective on a community level, to account for shared environmental risk factors.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2005.01552.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=758 Higher aggression is related to poorer academic performance in compulsory education / Eero VUOKSIMAA in Journal of Child Psychology and Psychiatry, 62-3 (March 2021)
[article]
Titre : Higher aggression is related to poorer academic performance in compulsory education Type de document : Texte imprimé et/ou numérique Auteurs : Eero VUOKSIMAA, Auteur ; Richard J. ROSE, Auteur ; Lea PULKKINEN, Auteur ; Teemu PALVIAINEN, Auteur ; Kaili RIMFELD, Auteur ; Sebastian LUNDSTROM, Auteur ; Meike BARTELS, Auteur ; Catharina VAN BEIJSTERVELDT, Auteur ; Anne HENDRIKS, Auteur ; Eveline L. DE ZEEUW, Auteur ; Robert PLOMIN, Auteur ; Paul LICHTENSTEIN, Auteur ; Dorret I. BOOMSMA, Auteur ; Jaakko KAPRIO, Auteur Article en page(s) : p.327-338 Langues : Anglais (eng) Mots-clés : Aggression cognition development educational attainment school performance Index. décimale : PER Périodiques Résumé : BACKGROUND: To conduct a comprehensive assessment of the association between aggression and academic performance in compulsory education. METHOD: We studied aggression and academic performance in over 27,000 individuals from four European twin cohorts participating in the ACTION consortium (Aggression in Children: Unraveling gene-environment interplay to inform Treatment and InterventiON strategies). Individual level data on aggression at ages 7-16 were assessed by three instruments (Achenbach System of Empirically Based Assessment, Multidimensional Peer Nomination Inventory, Strengths and Difficulties Questionnaire) including parental, teacher and self-reports. Academic performance was measured with teacher-rated grade point averages (ages 12-14) or standardized test scores (ages 12-16). Random effect meta-analytical correlations with academic performance were estimated for parental ratings (in all four cohorts) and self-ratings (in three cohorts). RESULTS: All between-family analyses indicated significant negative aggression-academic performance associations with correlations ranging from -.06 to -.33. Results were similar across different ages, instruments and raters and either with teacher-rated grade point averages or standardized test scores as measures of academic performance. Meta-analytical r's were -.20 and -.23 for parental and self-ratings, respectively. In within-family analyses of all twin pairs, the negative aggression-academic performance associations were statistically significant in 14 out of 17 analyses (r = -.17 for parental- and r = -.16 for self-ratings). Separate analyses in monozygotic (r = -.07 for parental and self-ratings), same-sex dizygotic (r's = -.16 and -.17 for parental and self-ratings) and opposite-sex dizygotic (r's = -.21 and -.19 for parental and self-ratings) twin pairs suggested partial confounding by genetic effects. CONCLUSIONS: There is a robust negative association between aggression and academic performance in compulsory education. Part of these associations were explained by shared genetic effects, but some evidence of a negative association between aggression and academic performance remained even in within-family analyses of monozygotic twin pairs. En ligne : http://dx.doi.org/10.1111/jcpp.13273 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=443
in Journal of Child Psychology and Psychiatry > 62-3 (March 2021) . - p.327-338[article] Higher aggression is related to poorer academic performance in compulsory education [Texte imprimé et/ou numérique] / Eero VUOKSIMAA, Auteur ; Richard J. ROSE, Auteur ; Lea PULKKINEN, Auteur ; Teemu PALVIAINEN, Auteur ; Kaili RIMFELD, Auteur ; Sebastian LUNDSTROM, Auteur ; Meike BARTELS, Auteur ; Catharina VAN BEIJSTERVELDT, Auteur ; Anne HENDRIKS, Auteur ; Eveline L. DE ZEEUW, Auteur ; Robert PLOMIN, Auteur ; Paul LICHTENSTEIN, Auteur ; Dorret I. BOOMSMA, Auteur ; Jaakko KAPRIO, Auteur . - p.327-338.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 62-3 (March 2021) . - p.327-338
Mots-clés : Aggression cognition development educational attainment school performance Index. décimale : PER Périodiques Résumé : BACKGROUND: To conduct a comprehensive assessment of the association between aggression and academic performance in compulsory education. METHOD: We studied aggression and academic performance in over 27,000 individuals from four European twin cohorts participating in the ACTION consortium (Aggression in Children: Unraveling gene-environment interplay to inform Treatment and InterventiON strategies). Individual level data on aggression at ages 7-16 were assessed by three instruments (Achenbach System of Empirically Based Assessment, Multidimensional Peer Nomination Inventory, Strengths and Difficulties Questionnaire) including parental, teacher and self-reports. Academic performance was measured with teacher-rated grade point averages (ages 12-14) or standardized test scores (ages 12-16). Random effect meta-analytical correlations with academic performance were estimated for parental ratings (in all four cohorts) and self-ratings (in three cohorts). RESULTS: All between-family analyses indicated significant negative aggression-academic performance associations with correlations ranging from -.06 to -.33. Results were similar across different ages, instruments and raters and either with teacher-rated grade point averages or standardized test scores as measures of academic performance. Meta-analytical r's were -.20 and -.23 for parental and self-ratings, respectively. In within-family analyses of all twin pairs, the negative aggression-academic performance associations were statistically significant in 14 out of 17 analyses (r = -.17 for parental- and r = -.16 for self-ratings). Separate analyses in monozygotic (r = -.07 for parental and self-ratings), same-sex dizygotic (r's = -.16 and -.17 for parental and self-ratings) and opposite-sex dizygotic (r's = -.21 and -.19 for parental and self-ratings) twin pairs suggested partial confounding by genetic effects. CONCLUSIONS: There is a robust negative association between aggression and academic performance in compulsory education. Part of these associations were explained by shared genetic effects, but some evidence of a negative association between aggression and academic performance remained even in within-family analyses of monozygotic twin pairs. En ligne : http://dx.doi.org/10.1111/jcpp.13273 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=443 Hypospadias and increased risk for neurodevelopmental disorders / Agnieszka BUTWICKA in Journal of Child Psychology and Psychiatry, 56-2 (February 2015)
[article]
Titre : Hypospadias and increased risk for neurodevelopmental disorders Type de document : Texte imprimé et/ou numérique Auteurs : Agnieszka BUTWICKA, Auteur ; Paul LICHTENSTEIN, Auteur ; Mikael LANDÉN, Auteur ; Anna S. NORDENVALL, Auteur ; Anna NORDENSTRÖM, Auteur ; Agneta NORDENSKJÖLD, Auteur ; Louise FRISÉN, Auteur Article en page(s) : p.155-161 Langues : Anglais (eng) Mots-clés : Hormones ADHD autism spectrum disorder ICD intellectual disability Index. décimale : PER Périodiques Résumé : Background Hypospadias (aberrant opening of the urethra on the underside of the penis) occurs in 1 per 300 newborn boys. It has been previously unknown whether this common malformation is associated with increased psychiatric morbidity later in life. Studies of individuals with hypospadias also provide an opportunity to examine whether difference in androgen signaling is related to neurodevelopmental disorders. To elucidate the mechanisms behind a possible association, we also studied psychiatric outcomes among brothers of the hypospadias patients. Methods Registry study within a national cohort of all 9,262 males with hypospadias and their 4,936 healthy brothers born in Sweden between 1973 and 2009. Patients with hypospadias and their brothers were matched with controls by year of birth and county. The following outcomes were evaluated (1) any psychiatric (2) psychotic, (3) mood, (4) anxiety, (5) eating, and (6) personality disorders, (7) substance misuse, (8) attention-deficit hyperactivity disorder (ADHD), (9) autism spectrum disorders (ASD), (10) intellectual disability, and (11) other behavioral/emotional disorders with onset in childhood. Results Patients with hypospadias were more likely to be diagnosed with intellectual disability (OR 3.2; 95% CI 2.8–3.8), ASD (1.4; 1.2–1.7), ADHD (1.5; 1.3–1.9), and behavioral/emotional disorders (1.4; 1.2–1.6) compared with the controls. Brothers of patients with hypospadias had an increased risk of ASD (1.6; 1.3–2.1) and other behavioral/emotional disorders with onset in childhood (1.2; 0.9–1.5) in comparison to siblings of healthy individuals. A slightly higher, although not statistically significant, risk was found for intellectual disability (1.3; 1.0–1.9). No relation between other psychiatric diagnosis and hypospadias was found. Conclusions This is the first study to identify an increased risk for neurodevelopmental disorders in patients with hypospadias, as well as an increased risk for ASD in their brothers, suggesting a common familial (genetic and/or environmental) liability. En ligne : http://dx.doi.org/10.1111/jcpp.12290 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.155-161[article] Hypospadias and increased risk for neurodevelopmental disorders [Texte imprimé et/ou numérique] / Agnieszka BUTWICKA, Auteur ; Paul LICHTENSTEIN, Auteur ; Mikael LANDÉN, Auteur ; Anna S. NORDENVALL, Auteur ; Anna NORDENSTRÖM, Auteur ; Agneta NORDENSKJÖLD, Auteur ; Louise FRISÉN, Auteur . - p.155-161.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 56-2 (February 2015) . - p.155-161
Mots-clés : Hormones ADHD autism spectrum disorder ICD intellectual disability Index. décimale : PER Périodiques Résumé : Background Hypospadias (aberrant opening of the urethra on the underside of the penis) occurs in 1 per 300 newborn boys. It has been previously unknown whether this common malformation is associated with increased psychiatric morbidity later in life. Studies of individuals with hypospadias also provide an opportunity to examine whether difference in androgen signaling is related to neurodevelopmental disorders. To elucidate the mechanisms behind a possible association, we also studied psychiatric outcomes among brothers of the hypospadias patients. Methods Registry study within a national cohort of all 9,262 males with hypospadias and their 4,936 healthy brothers born in Sweden between 1973 and 2009. Patients with hypospadias and their brothers were matched with controls by year of birth and county. The following outcomes were evaluated (1) any psychiatric (2) psychotic, (3) mood, (4) anxiety, (5) eating, and (6) personality disorders, (7) substance misuse, (8) attention-deficit hyperactivity disorder (ADHD), (9) autism spectrum disorders (ASD), (10) intellectual disability, and (11) other behavioral/emotional disorders with onset in childhood. Results Patients with hypospadias were more likely to be diagnosed with intellectual disability (OR 3.2; 95% CI 2.8–3.8), ASD (1.4; 1.2–1.7), ADHD (1.5; 1.3–1.9), and behavioral/emotional disorders (1.4; 1.2–1.6) compared with the controls. Brothers of patients with hypospadias had an increased risk of ASD (1.6; 1.3–2.1) and other behavioral/emotional disorders with onset in childhood (1.2; 0.9–1.5) in comparison to siblings of healthy individuals. A slightly higher, although not statistically significant, risk was found for intellectual disability (1.3; 1.0–1.9). No relation between other psychiatric diagnosis and hypospadias was found. Conclusions This is the first study to identify an increased risk for neurodevelopmental disorders in patients with hypospadias, as well as an increased risk for ASD in their brothers, suggesting a common familial (genetic and/or environmental) liability. En ligne : http://dx.doi.org/10.1111/jcpp.12290 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=259 Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study / Agnieszka BUTWICKA in Journal of Autism and Developmental Disorders, 47-1 (January 2017)
[article]
Titre : Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study Type de document : Texte imprimé et/ou numérique Auteurs : Agnieszka BUTWICKA, Auteur ; Niklas LANGSTROM, Auteur ; Henrik LARSSON, Auteur ; Sebastian LUNDSTROM, Auteur ; Eva SERLACHIUS, Auteur ; Catarina ALMQVIST, Auteur ; Louise FRISÉN, Auteur ; Paul LICHTENSTEIN, Auteur Article en page(s) : p.80-89 Langues : Anglais (eng) Mots-clés : Autism spectrum disorder Addiction ADHD Intellectual disability ICD Index. décimale : PER Périodiques Résumé : Despite limited and ambiguous empirical data, substance use-related problems have been assumed to be rare among patients with autism spectrum disorders (ASD). Using Swedish population-based registers we identified 26,986 individuals diagnosed with ASD during 1973–2009, and their 96,557 non-ASD relatives. ASD, without diagnosed comorbidity of attention deficit hyperactivity disorder (ADHD) or intellectual disability, was related to a doubled risk of substance use-related problems. The risk of substance use-related problems was the highest among individuals with ASD and ADHD. Further, risks of substance use-related problems were increased among full siblings of ASD probands, half-siblings and parents. We conclude that ASD is a risk factor for substance use-related problems. The elevated risks among relatives of probands with ASD suggest shared familial (genetic and/or shared environmental) liability. En ligne : http://dx.doi.org/10.1007/s10803-016-2914-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298
in Journal of Autism and Developmental Disorders > 47-1 (January 2017) . - p.80-89[article] Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study [Texte imprimé et/ou numérique] / Agnieszka BUTWICKA, Auteur ; Niklas LANGSTROM, Auteur ; Henrik LARSSON, Auteur ; Sebastian LUNDSTROM, Auteur ; Eva SERLACHIUS, Auteur ; Catarina ALMQVIST, Auteur ; Louise FRISÉN, Auteur ; Paul LICHTENSTEIN, Auteur . - p.80-89.
Langues : Anglais (eng)
in Journal of Autism and Developmental Disorders > 47-1 (January 2017) . - p.80-89
Mots-clés : Autism spectrum disorder Addiction ADHD Intellectual disability ICD Index. décimale : PER Périodiques Résumé : Despite limited and ambiguous empirical data, substance use-related problems have been assumed to be rare among patients with autism spectrum disorders (ASD). Using Swedish population-based registers we identified 26,986 individuals diagnosed with ASD during 1973–2009, and their 96,557 non-ASD relatives. ASD, without diagnosed comorbidity of attention deficit hyperactivity disorder (ADHD) or intellectual disability, was related to a doubled risk of substance use-related problems. The risk of substance use-related problems was the highest among individuals with ASD and ADHD. Further, risks of substance use-related problems were increased among full siblings of ASD probands, half-siblings and parents. We conclude that ASD is a risk factor for substance use-related problems. The elevated risks among relatives of probands with ASD suggest shared familial (genetic and/or shared environmental) liability. En ligne : http://dx.doi.org/10.1007/s10803-016-2914-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298 A longitudinal twin study of the direction of effects between psychopathic personality and antisocial behaviour / Mats FORSMAN in Journal of Child Psychology and Psychiatry, 51-1 (January 2010)
[article]
Titre : A longitudinal twin study of the direction of effects between psychopathic personality and antisocial behaviour Type de document : Texte imprimé et/ou numérique Auteurs : Mats FORSMAN, Auteur ; Paul LICHTENSTEIN, Auteur ; Henrik ANDERSHED, Auteur ; Henrik LARSSON, Auteur Année de publication : 2010 Article en page(s) : p.39-47 Langues : Anglais (eng) Mots-clés : Antisocial-behaviour psychopathic-personality longitudinal twins Index. décimale : PER Périodiques Résumé : Background: Antisocial behaviour may partly develop as a consequence of psychopathic personality. However, neither the direction of effects nor the aetiology of the association has previously been clarified. The aim in this study was to investigate the direction of effects between psychopathic personality and antisocial behaviour, and to investigate the genetic and environmental contribution to this association.
Method: Twins (n = 2,255) in the Swedish Twin Study of Child and Adolescent Development were prospectively followed from adolescence to adulthood. We used a longitudinal cross-lagged twin model to study the associations between psychopathic personality and antisocial behaviour.
Results: Psychopathic personality in mid-adolescence predicted antisocial behaviour in adulthood (p < .001), but not the other way around. However, bidirectional effects were found when a measure of persistent antisocial behaviour (from age 8–9 to age 16–17) was used. Psychopathic personality predicted both rule-breaking behaviour (p < .001) and aggressive behaviour (p < .01). Genetic factors were of importance in mediating the longitudinal associations between psychopathic personality and antisocial behaviour.
Conclusions: This study provides evidence that genetically influenced psychopathic personality is a robust predictor of adult antisocial behaviour, but also that persistent antisocial behaviour has an impact on adult psychopathic personality via genetic effects.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02141.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=939
in Journal of Child Psychology and Psychiatry > 51-1 (January 2010) . - p.39-47[article] A longitudinal twin study of the direction of effects between psychopathic personality and antisocial behaviour [Texte imprimé et/ou numérique] / Mats FORSMAN, Auteur ; Paul LICHTENSTEIN, Auteur ; Henrik ANDERSHED, Auteur ; Henrik LARSSON, Auteur . - 2010 . - p.39-47.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 51-1 (January 2010) . - p.39-47
Mots-clés : Antisocial-behaviour psychopathic-personality longitudinal twins Index. décimale : PER Périodiques Résumé : Background: Antisocial behaviour may partly develop as a consequence of psychopathic personality. However, neither the direction of effects nor the aetiology of the association has previously been clarified. The aim in this study was to investigate the direction of effects between psychopathic personality and antisocial behaviour, and to investigate the genetic and environmental contribution to this association.
Method: Twins (n = 2,255) in the Swedish Twin Study of Child and Adolescent Development were prospectively followed from adolescence to adulthood. We used a longitudinal cross-lagged twin model to study the associations between psychopathic personality and antisocial behaviour.
Results: Psychopathic personality in mid-adolescence predicted antisocial behaviour in adulthood (p < .001), but not the other way around. However, bidirectional effects were found when a measure of persistent antisocial behaviour (from age 8–9 to age 16–17) was used. Psychopathic personality predicted both rule-breaking behaviour (p < .001) and aggressive behaviour (p < .01). Genetic factors were of importance in mediating the longitudinal associations between psychopathic personality and antisocial behaviour.
Conclusions: This study provides evidence that genetically influenced psychopathic personality is a robust predictor of adult antisocial behaviour, but also that persistent antisocial behaviour has an impact on adult psychopathic personality via genetic effects.En ligne : http://dx.doi.org/10.1111/j.1469-7610.2009.02141.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=939 Machine-Learning prediction of comorbid substance use disorders in ADHD youth using Swedish registry data / Yanli ZHANG-JAMES in Journal of Child Psychology and Psychiatry, 61-12 (December 2020)
PermalinkMaltreatment-associated neurodevelopmental disorders: a co-twin control analysis / Lisa DINKLER in Journal of Child Psychology and Psychiatry, 58-6 (June 2017)
PermalinkMaternal age at first birth and offspring criminality: Using the children of twins design to test causal hypotheses / Claire A. COYNE in Development and Psychopathology, 25-1 (February 2013)
PermalinkParental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence / Yasmin I. AHMADZADEH in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)
PermalinkParental knowledge is an environmental influence on adolescent externalizing / Kristine MARCEAU in Journal of Child Psychology and Psychiatry, 56-2 (February 2015)
PermalinkPerceived child impairment and the 'autism epidemic' / Sebastian LUNDSTROM in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)
PermalinkRelative immaturity and ADHD: findings from nationwide registers, parent- and self-reports / Linda HALLDNER in Journal of Child Psychology and Psychiatry, 55-8 (August 2014)
PermalinkStimulant ADHD medication and risk for substance abuse / Zheng CHANG in Journal of Child Psychology and Psychiatry, 55-8 (August 2014)
PermalinkTrajectories leading to autism spectrum disorders are affected by paternal age: findings from two nationally representative twin studies / Sebastian LUNDSTROM in Journal of Child Psychology and Psychiatry, 51-7 (July 2010)
PermalinkA twin study of genetic and environmental contributions to attention-deficit/hyperactivity disorder over time / Mark J. TAYLOR in Journal of Child Psychology and Psychiatry, 64-11 (November 2023)
PermalinkVisual Disengagement: Genetic Architecture and Relation to Autistic Traits in the General Population / Monica SIQUEIROS SANCHEZ in Journal of Autism and Developmental Disorders, 50-6 (June 2020)
PermalinkVolitional eye movement control and ADHD traits: a twin study / Monica SIQUEIROS SANCHEZ in Journal of Child Psychology and Psychiatry, 61-12 (December 2020)
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