20 recherche sur le mot-clé 'Early adversity'

Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress / Catherine B. STROUD in Development and Psychopathology, 35-3 (August 2023)  

Public ISBD [article]  inDevelopment and Psychopathology > 35-3 (August 2023) . - p.1272-1287 Titre : Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress Type de document : Texte imprimé et/ou numérique Auteurs : Catherine B. STROUD, Auteur ; Frances R. CHEN, Auteur ; Erin E. DUNNING, Auteur ; Julia CHENG, Auteur ; Carey MARR, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur Article en page(s) : p.1272-1287 Langues : Anglais (eng) Mots-clés : adolescents  depression  early adversity  interpersonal stress  stressful life events Index. décimale : PER Périodiques Résumé : Early adversity confers risk for depression in part through its association with recent (i.e., proximal) acute stress. However, it remains unresolved whether: a) early adversity predicts increases in recent acute stress over time; b) all - or only certain types - of recent events mediate the relationship between early adversity and depression; and c) early adversity places individuals at greater risk for depression via greater exposure to independent (i.e., fateful) interpersonal events or via greater generation of dependent (i.e., partially self-initiated) interpersonal events (i.e., stress generation) or both. These questions were examined in a 3-wave longitudinal study of early adolescent girls (N = 125; M = 12.35 years [SD = .77]) with no history of diagnosable depression using contextual life stress and diagnostic interviews. Path analyses indicated that increases in past-year acute interpersonal, but not non-interpersonal, stress mediated the link between early adversity and depressive symptoms. The mediating role of interpersonal events was limited to independent ones, suggesting increases in interpersonal event exposure, not interpersonal stress generation, acted as a mediator. Finally, findings support prior evidence that early adversity may not directly predict future depressive symptoms. Implications for understanding the role of recent stress in the association between early adversity and adolescent depression are discussed. En ligne : http://dx.doi.org/10.1017/S0954579421001176 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=511 [article] Early adversity and depressive symptoms among early adolescent girls: the mediating role of exposure to recent interpersonal acute stress [Texte imprimé et/ou numérique] / Catherine B. STROUD, Auteur ; Frances R. CHEN, Auteur ; Erin E. DUNNING, Auteur ; Julia CHENG, Auteur ; Carey MARR, Auteur ; Suzanne VRSHEK-SCHALLHORN, Auteur . - p.1272-1287. Langues : Anglais (eng) in Development and Psychopathology > 35-3 (August 2023) . - p.1272-1287 Mots-clés : adolescents  depression  early adversity  interpersonal stress  stressful life events Index. décimale : PER Périodiques Résumé : Early adversity confers risk for depression in part through its association with recent (i.e., proximal) acute stress. However, it remains unresolved whether: a) early adversity predicts increases in recent acute stress over time; b) all - or only certain types - of recent events mediate the relationship between early adversity and depression; and c) early adversity places individuals at greater risk for depression via greater exposure to independent (i.e., fateful) interpersonal events or via greater generation of dependent (i.e., partially self-initiated) interpersonal events (i.e., stress generation) or both. These questions were examined in a 3-wave longitudinal study of early adolescent girls (N = 125; M = 12.35 years [SD = .77]) with no history of diagnosable depression using contextual life stress and diagnostic interviews. Path analyses indicated that increases in past-year acute interpersonal, but not non-interpersonal, stress mediated the link between early adversity and depressive symptoms. The mediating role of interpersonal events was limited to independent ones, suggesting increases in interpersonal event exposure, not interpersonal stress generation, acted as a mediator. Finally, findings support prior evidence that early adversity may not directly predict future depressive symptoms. Implications for understanding the role of recent stress in the association between early adversity and adolescent depression are discussed. En ligne : http://dx.doi.org/10.1017/S0954579421001176 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=511

Early adversity and children's regulatory deficits: Does postadoption parenting facilitate recovery in postinstitutionalized children? / Kalsea J. KOSS in Development and Psychopathology, 32-3 (August 2020)  

Public ISBD [article]  inDevelopment and Psychopathology > 32-3 (August 2020) . - p.879-896 Titre : Early adversity and children's regulatory deficits: Does postadoption parenting facilitate recovery in postinstitutionalized children? Type de document : Texte imprimé et/ou numérique Auteurs : Kalsea J. KOSS, Auteur ; Jamie M. LAWLER, Auteur ; Megan R. GUNNAR, Auteur Article en page(s) : p.879-896 Langues : Anglais (eng) Mots-clés : early adversity  executive function  international adoption  parenting  regulation Index. décimale : PER Périodiques Résumé : Children reared in orphanages typically experience the lack of stable, reliable caregivers and are at increased risk for deficits in regulatory abilities including difficulties in inhibitory control, attention, and emotion regulation. Although adoption results in a radical shift in caregiving quality, there remains variation in postadoption parenting, yet little research has examined postadoption parenting that may promote recovery in children experiencing early life adversity in the form of institutional care. Participants included 93 postinstitutionalized children adopted between 15 and 36 months of age and 52 nonadopted same-aged peers. Parenting was assessed four times during the first 2 years postadoption (at 2, 8, 16, and 24 months postadoption) and children's regulation was assessed at age 5 (M age = 61.68 months) and during kindergarten (M age = 71.55 months). Multiple parenting dimensions including sensitivity/responsiveness, structure/limit setting, and consistency in routines were examined. Both parental sensitivity and structure moderated the effect of preadoption adversity on children's emotion regulation while greater consistency was associated with better inhibitory control and fewer attention problems. Results support the notion that postadoption parenting during toddlerhood and the early preschool years promotes better regulation skills following early adversity. En ligne : http://dx.doi.org/10.1017/s0954579419001226 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=429 [article] Early adversity and children's regulatory deficits: Does postadoption parenting facilitate recovery in postinstitutionalized children? [Texte imprimé et/ou numérique] / Kalsea J. KOSS, Auteur ; Jamie M. LAWLER, Auteur ; Megan R. GUNNAR, Auteur . - p.879-896. Langues : Anglais (eng) in Development and Psychopathology > 32-3 (August 2020) . - p.879-896 Mots-clés : early adversity  executive function  international adoption  parenting  regulation Index. décimale : PER Périodiques Résumé : Children reared in orphanages typically experience the lack of stable, reliable caregivers and are at increased risk for deficits in regulatory abilities including difficulties in inhibitory control, attention, and emotion regulation. Although adoption results in a radical shift in caregiving quality, there remains variation in postadoption parenting, yet little research has examined postadoption parenting that may promote recovery in children experiencing early life adversity in the form of institutional care. Participants included 93 postinstitutionalized children adopted between 15 and 36 months of age and 52 nonadopted same-aged peers. Parenting was assessed four times during the first 2 years postadoption (at 2, 8, 16, and 24 months postadoption) and children's regulation was assessed at age 5 (M age = 61.68 months) and during kindergarten (M age = 71.55 months). Multiple parenting dimensions including sensitivity/responsiveness, structure/limit setting, and consistency in routines were examined. Both parental sensitivity and structure moderated the effect of preadoption adversity on children's emotion regulation while greater consistency was associated with better inhibitory control and fewer attention problems. Results support the notion that postadoption parenting during toddlerhood and the early preschool years promotes better regulation skills following early adversity. En ligne : http://dx.doi.org/10.1017/s0954579419001226 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=429

Annual Research Review: Early adversity, the hypothalamic-pituitary-adrenocortical axis, and child psychopathology / K. J. KOSS in Journal of Child Psychology and Psychiatry, 59-4 (April 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-4 (April 2018) . - p.327-346 Titre : Annual Research Review: Early adversity, the hypothalamic-pituitary-adrenocortical axis, and child psychopathology Type de document : Texte imprimé et/ou numérique Auteurs : K. J. KOSS, Auteur ; M. R. GUNNAR, Auteur Article en page(s) : p.327-346 Langues : Anglais (eng) Mots-clés : Early adversity  hypothalamic-pituitary-adrenocortical axis  psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Research on early adversity, stress biology, and child development has grown exponentially in recent years. FINDINGS: We review the current evidence for the hypothalamic-pituitary-adrenocortical (HPA) axis as a stress-mediating mechanism between various forms of childhood adversity and psychopathology. We begin with a review of the neurobiology of the axis and evidence for relations between early adversity-HPA axis activity and HPA axis activity-psychopathology, as well as discuss the role of regulatory mechanisms and sensitive periods in development. CONCLUSIONS: We call attention to critical gaps in the literature to highlight next steps in this research including focus on developmental timing, sex differences, stress buffering, and epigenetic regulation. A better understanding of individual differences in the adversity-HPA axis-psychopathology associations will require continued work addressing how multiple biological and behavioral systems work in concert to shape development. En ligne : http://dx.doi.org/10.1111/jcpp.12784 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=353 [article] Annual Research Review: Early adversity, the hypothalamic-pituitary-adrenocortical axis, and child psychopathology [Texte imprimé et/ou numérique] / K. J. KOSS, Auteur ; M. R. GUNNAR, Auteur . - p.327-346. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-4 (April 2018) . - p.327-346 Mots-clés : Early adversity  hypothalamic-pituitary-adrenocortical axis  psychopathology Index. décimale : PER Périodiques Résumé : BACKGROUND: Research on early adversity, stress biology, and child development has grown exponentially in recent years. FINDINGS: We review the current evidence for the hypothalamic-pituitary-adrenocortical (HPA) axis as a stress-mediating mechanism between various forms of childhood adversity and psychopathology. We begin with a review of the neurobiology of the axis and evidence for relations between early adversity-HPA axis activity and HPA axis activity-psychopathology, as well as discuss the role of regulatory mechanisms and sensitive periods in development. CONCLUSIONS: We call attention to critical gaps in the literature to highlight next steps in this research including focus on developmental timing, sex differences, stress buffering, and epigenetic regulation. A better understanding of individual differences in the adversity-HPA axis-psychopathology associations will require continued work addressing how multiple biological and behavioral systems work in concert to shape development. En ligne : http://dx.doi.org/10.1111/jcpp.12784 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=353

Biological sensitivity to context: A test of the hypothesized U-shaped relation between early adversity and stress responsivity / Nila SHAKIBA in Development and Psychopathology, 32-2 (May 2020)  

Public ISBD [article]  inDevelopment and Psychopathology > 32-2 (May 2020) . - p.641-660 Titre : Biological sensitivity to context: A test of the hypothesized U-shaped relation between early adversity and stress responsivity Type de document : Texte imprimé et/ou numérique Auteurs : Nila SHAKIBA, Auteur ; Bruce J. ELLIS, Auteur ; Nicole R. BUSH, Auteur ; W. Thomas BOYCE, Auteur Article en page(s) : p.641-660 Langues : Anglais (eng) Mots-clés : autonomic nervous system  biological sensitivity to context  early adversity  hypothalamic–pituitary–adrenal axis  stress reactivity Index. décimale : PER Périodiques Résumé : We conducted signal detection analyses to test for curvilinear, U-shaped relations between early experiences of adversity and heightened physiological responses to challenge, as proposed by biological sensitivity to context theory. Based on analysis of an ethnically diverse sample of 338 kindergarten children (4-6 years old) and their families, we identified levels and types of adversity that, singly and interactively, predicted high (top 25%) and low (bottom 25%) rates of stress reactivity. The results offered support for the hypothesized U-shaped curve and conceptually replicated and extended the work of Ellis, Essex, and Boyce (2005). Across both sympathetic and adrenocortical systems, a disproportionate number of children growing up under conditions characterized by either low or high adversity (as indexed by restrictive parenting, family stress, and family economic condition) displayed heightened stress reactivity, compared with peers growing up under conditions of moderate adversity. Finally, as hypothesized by the adaptive calibration model, a disproportionate number of children who experienced exceptionally stressful family conditions displayed blunted cortisol reactivity to stress. En ligne : http://dx.doi.org/10.1017/s0954579419000518 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=426 [article] Biological sensitivity to context: A test of the hypothesized U-shaped relation between early adversity and stress responsivity [Texte imprimé et/ou numérique] / Nila SHAKIBA, Auteur ; Bruce J. ELLIS, Auteur ; Nicole R. BUSH, Auteur ; W. Thomas BOYCE, Auteur . - p.641-660. Langues : Anglais (eng) in Development and Psychopathology > 32-2 (May 2020) . - p.641-660 Mots-clés : autonomic nervous system  biological sensitivity to context  early adversity  hypothalamic–pituitary–adrenal axis  stress reactivity Index. décimale : PER Périodiques Résumé : We conducted signal detection analyses to test for curvilinear, U-shaped relations between early experiences of adversity and heightened physiological responses to challenge, as proposed by biological sensitivity to context theory. Based on analysis of an ethnically diverse sample of 338 kindergarten children (4-6 years old) and their families, we identified levels and types of adversity that, singly and interactively, predicted high (top 25%) and low (bottom 25%) rates of stress reactivity. The results offered support for the hypothesized U-shaped curve and conceptually replicated and extended the work of Ellis, Essex, and Boyce (2005). Across both sympathetic and adrenocortical systems, a disproportionate number of children growing up under conditions characterized by either low or high adversity (as indexed by restrictive parenting, family stress, and family economic condition) displayed heightened stress reactivity, compared with peers growing up under conditions of moderate adversity. Finally, as hypothesized by the adaptive calibration model, a disproportionate number of children who experienced exceptionally stressful family conditions displayed blunted cortisol reactivity to stress. En ligne : http://dx.doi.org/10.1017/s0954579419000518 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=426

Cumulative early childhood adversity and later antisocial behavior: The mediating role of passive avoidance / Idil YAZGAN in Development and Psychopathology, 33-1 (February 2021)  

Public ISBD [article]  inDevelopment and Psychopathology > 33-1 (February 2021) . - p.340-350 Titre : Cumulative early childhood adversity and later antisocial behavior: The mediating role of passive avoidance Type de document : Texte imprimé et/ou numérique Auteurs : Idil YAZGAN, Auteur ; Jamie L. HANSON, Auteur ; John E. BATES, Auteur ; Jennifer E. LANSFORD, Auteur ; Gregory S. PETTIT, Auteur ; Kenneth A. DODGE, Auteur Article en page(s) : p.340-350 Langues : Anglais (eng) Mots-clés : adverse childhood experiences  antisocial behavior  antisociality  early adversity  learning  passive avoidance Index. décimale : PER Périodiques Résumé : Twenty-six percent of children experience a traumatic event by the age of 4. Negative events during childhood have deleterious correlates later in life, including antisocial behavior. However, the mechanisms that play into this relation are unclear. We explored deficits in neurocognitive functioning, specifically problems in passive avoidance, a construct with elements of inhibitory control and learning as a potential acquired mediator for the pathway between cumulative early childhood adversity from birth to age 7 and later antisocial behavior through age 18, using prospective longitudinal data from 585 participants. Path analyses showed that cumulative early childhood adversity predicted impaired passive avoidance during adolescence and increased antisocial behavior during late adolescence. Furthermore, poor neurocognition, namely, passive avoidance, predicted later antisocial behavior and significantly mediated the relation between cumulative early childhood adversity and later antisocial behavior. This research has implications for understanding the development of later antisocial behavior and points to a potential target for neurocognitive intervention within the pathway from cumulative early childhood adversity to later antisocial behavior. En ligne : http://dx.doi.org/10.1017/s0954579419001809 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=443 [article] Cumulative early childhood adversity and later antisocial behavior: The mediating role of passive avoidance [Texte imprimé et/ou numérique] / Idil YAZGAN, Auteur ; Jamie L. HANSON, Auteur ; John E. BATES, Auteur ; Jennifer E. LANSFORD, Auteur ; Gregory S. PETTIT, Auteur ; Kenneth A. DODGE, Auteur . - p.340-350. Langues : Anglais (eng) in Development and Psychopathology > 33-1 (February 2021) . - p.340-350 Mots-clés : adverse childhood experiences  antisocial behavior  antisociality  early adversity  learning  passive avoidance Index. décimale : PER Périodiques Résumé : Twenty-six percent of children experience a traumatic event by the age of 4. Negative events during childhood have deleterious correlates later in life, including antisocial behavior. However, the mechanisms that play into this relation are unclear. We explored deficits in neurocognitive functioning, specifically problems in passive avoidance, a construct with elements of inhibitory control and learning as a potential acquired mediator for the pathway between cumulative early childhood adversity from birth to age 7 and later antisocial behavior through age 18, using prospective longitudinal data from 585 participants. Path analyses showed that cumulative early childhood adversity predicted impaired passive avoidance during adolescence and increased antisocial behavior during late adolescence. Furthermore, poor neurocognition, namely, passive avoidance, predicted later antisocial behavior and significantly mediated the relation between cumulative early childhood adversity and later antisocial behavior. This research has implications for understanding the development of later antisocial behavior and points to a potential target for neurocognitive intervention within the pathway from cumulative early childhood adversity to later antisocial behavior. En ligne : http://dx.doi.org/10.1017/s0954579419001809 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=443

Cumulative early childhood adversity and later antisocial behavior: The mediating role of passive avoidance – ERRATUM / Idil YAZGAN in Development and Psychopathology, 34-3 (August 2022)  

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The influence of early familial adversity on adolescent risk behaviors and mental health: Stability and transition in family adversity profiles in a cohort sample / Ruth WADMAN in Development and Psychopathology, 32-2 (May 2020)  

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Why and how does early adversity influence development? Toward an integrated model of dimensions of environmental experience / Bruce J. ELLIS in Development and Psychopathology, 34-2 (May 2022)  

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Leveraging the developmental neuroscience of caregiving to promote resilience among youth exposed to adversity / Dylan G. GEE in Development and Psychopathology, 35-5 (December 2023)  

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Developmental consequences of early life stress on risk for psychopathology: Longitudinal associations with children's multisystem physiological regulation and executive functioning / Kristen L. RUDD in Development and Psychopathology, 33-5 (December 2021)  

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