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PER : Périodiques |
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Regulation of Ion Channel Distribution at Synapses / Stanley C. FROEHNER in Annual Review of Neuroscience, 16 (1993)
[article]
Titre : Regulation of Ion Channel Distribution at Synapses Type de document : Texte imprimé et/ou numérique Auteurs : Stanley C. FROEHNER, Auteur Année de publication : 1993 Article en page(s) : p.347-368 Langues : Anglais (eng) Mots-clés : Neurotransmitter receptors - Receptor clustering - Postsynaptic cytoskeleton - Extracellular matrix Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=373
in Annual Review of Neuroscience > 16 (1993) . - p.347-368[article] Regulation of Ion Channel Distribution at Synapses [Texte imprimé et/ou numérique] / Stanley C. FROEHNER, Auteur . - 1993 . - p.347-368.
Langues : Anglais (eng)
in Annual Review of Neuroscience > 16 (1993) . - p.347-368
Mots-clés : Neurotransmitter receptors - Receptor clustering - Postsynaptic cytoskeleton - Extracellular matrix Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=373 Regulation of molecular pathways in the Fragile X Syndrome: insights into Autism Spectrum Disorders / S. DE RUBEIS in Journal of Neurodevelopmental Disorders, 3-3 (September 2011)
[article]
Titre : Regulation of molecular pathways in the Fragile X Syndrome: insights into Autism Spectrum Disorders Type de document : Texte imprimé et/ou numérique Auteurs : S. DE RUBEIS, Auteur ; C. BAGNI, Auteur Article en page(s) : p.257-69 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : The Fragile X syndrome (FXS) is a leading cause of intellectual disability (ID) and autism. The disease is caused by mutations or loss of the Fragile X Mental Retardation Protein (FMRP), an RNA-binding protein playing multiple functions in RNA metabolism. The expression of a large set of neuronal mRNAs is altered when FMRP is lost, thus causing defects in neuronal morphology and physiology. FMRP regulates mRNA stability, dendritic targeting, and protein synthesis. At synapses, FMRP represses protein synthesis by forming a complex with the Cytoplasmic FMRP Interacting Protein 1 (CYFIP1) and the cap-binding protein eIF4E. Here, we review the clinical, genetic, and molecular aspects of FXS with a special focus on the receptor signaling that regulates FMRP-dependent protein synthesis. We further discuss the FMRP-CYFIP1 complex and its potential relevance for ID and autism. En ligne : http://dx.doi.org/10.1007/s11689-011-9087-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=343
in Journal of Neurodevelopmental Disorders > 3-3 (September 2011) . - p.257-69[article] Regulation of molecular pathways in the Fragile X Syndrome: insights into Autism Spectrum Disorders [Texte imprimé et/ou numérique] / S. DE RUBEIS, Auteur ; C. BAGNI, Auteur . - p.257-69.
Langues : Anglais (eng)
in Journal of Neurodevelopmental Disorders > 3-3 (September 2011) . - p.257-69
Index. décimale : PER Périodiques Résumé : The Fragile X syndrome (FXS) is a leading cause of intellectual disability (ID) and autism. The disease is caused by mutations or loss of the Fragile X Mental Retardation Protein (FMRP), an RNA-binding protein playing multiple functions in RNA metabolism. The expression of a large set of neuronal mRNAs is altered when FMRP is lost, thus causing defects in neuronal morphology and physiology. FMRP regulates mRNA stability, dendritic targeting, and protein synthesis. At synapses, FMRP represses protein synthesis by forming a complex with the Cytoplasmic FMRP Interacting Protein 1 (CYFIP1) and the cap-binding protein eIF4E. Here, we review the clinical, genetic, and molecular aspects of FXS with a special focus on the receptor signaling that regulates FMRP-dependent protein synthesis. We further discuss the FMRP-CYFIP1 complex and its potential relevance for ID and autism. En ligne : http://dx.doi.org/10.1007/s11689-011-9087-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=343 Regulation of Neuropeptide Gene Expression / Richard H. GOODMAN in Annual Review of Neuroscience, 13 (1990)
[article]
Titre : Regulation of Neuropeptide Gene Expression Type de document : Texte imprimé et/ou numérique Auteurs : Richard H. GOODMAN, Auteur Année de publication : 1990 Article en page(s) : p.111-127 Langues : Anglais (eng) Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=366
in Annual Review of Neuroscience > 13 (1990) . - p.111-127[article] Regulation of Neuropeptide Gene Expression [Texte imprimé et/ou numérique] / Richard H. GOODMAN, Auteur . - 1990 . - p.111-127.
Langues : Anglais (eng)
in Annual Review of Neuroscience > 13 (1990) . - p.111-127
Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=366 La régulation temporelle chez les enfants tout-venant et les enfants ayant un trouble du spectre de l’autisme : caractéristiques des enfants ajustant leur comportement à une contrainte de temps / Mélissa GAUCHER in Bulletin Scientifique de l'arapi (Le), 33 (Juillet 2014)
[article]
Titre : La régulation temporelle chez les enfants tout-venant et les enfants ayant un trouble du spectre de l’autisme : caractéristiques des enfants ajustant leur comportement à une contrainte de temps Type de document : Texte imprimé et/ou numérique Auteurs : Mélissa GAUCHER, Auteur ; Jacques FORGET, Auteur ; Céline CLEMENT, Auteur Article en page(s) : p.39-42 Langues : Français (fre) Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=237
in Bulletin Scientifique de l'arapi (Le) > 33 (Juillet 2014) . - p.39-42[article] La régulation temporelle chez les enfants tout-venant et les enfants ayant un trouble du spectre de l’autisme : caractéristiques des enfants ajustant leur comportement à une contrainte de temps [Texte imprimé et/ou numérique] / Mélissa GAUCHER, Auteur ; Jacques FORGET, Auteur ; Céline CLEMENT, Auteur . - p.39-42.
Langues : Français (fre)
in Bulletin Scientifique de l'arapi (Le) > 33 (Juillet 2014) . - p.39-42
Index. décimale : PER Périodiques Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=237 Regulatory T lymphocytes/Th17 lymphocytes imbalance in autism spectrum disorders: evidence from a meta-analysis / P. ELLUL in Molecular Autism, 12 (2021)
[article]
Titre : Regulatory T lymphocytes/Th17 lymphocytes imbalance in autism spectrum disorders: evidence from a meta-analysis Type de document : Texte imprimé et/ou numérique Auteurs : P. ELLUL, Auteur ; M. ROSENZWAJG, Auteur ; H. PEYRE, Auteur ; G. FOURCADE, Auteur ; E. MARIOTTI-FERRANDIZ, Auteur ; V. TREBOSSEN, Auteur ; D. KLATZMANN, Auteur ; R. DELORME, Auteur Article en page(s) : 68 p. Langues : Anglais (eng) Mots-clés : Asd Immunology Peripheral blood Regulatory T lymphocyte Th17 lymphocytes Index. décimale : PER Périodiques Résumé : BACKGROUND: Immune system dysfunction has been proposed to play a critical role in the pathophysiology of autism spectrum disorders (ASD). Conflicting reports of lymphocyte subpopulation abnormalities have been described in numerous studies of patients with ASD. To better define lymphocytes abnormalities in ASD, we performed a meta-analysis of the lymphocyte profiles from subjects with ASD. METHODS: We used the PRISMA recommendations to query PubMed, Embase, PsychoINFO, BIOSIS, Science Direct, Cochrane CENTRAL, and Clinicaltrials.gov for terms related to clinical diagnosis of ASD and to lymphocytes' populations. We selected studies exploring lymphocyte subpopulations in children with ASD. The search protocol has been registered in the international Prospective Register of Systematic Reviews (CRD42019121473). RESULTS: We selected 13 studies gathering 388 ASD patients and 326 healthy controls. A significant decrease in the CD4+?lymphocyte was found in ASD patients compared to controls [-?1.51 (95% CI?-?2.99;?-?0.04) p?=?0.04] (I(2)?=?96% [95% CI 94.6, 97.7], p?0.01). No significant difference was found for the CD8+?T, B and natural killer lymphocytes. Considering the CD4+?subpopulation, there was a significant decrease in regulatory T lymphocytes (Tregs) in ASD patients (n?=?114) compared to controls (n?=?107) [-?3.09 (95% CI?-?4.41;?-?1.76) p?=?0.0001]; (I(2)?=?90.9%, [95% CI 76.2, 96.5], p?0.0001) associated with an increase oin the Th17 lymphocytes (ASD; n?=?147 controls; n?=?128) [2.23 (95% CI 0.79; 3.66) p?=?0,002] (I(2)?=?95.1% [95% CI 90.4, 97.5], p?0.0001). LIMITATIONS: Several factors inducing heterogeneity should be considered. First, differences in the staining method may be responsible for a part in the heterogeneity of results. Second, ASD population is also by itself heterogeneous, underlying the need of studying sub-groups that are more homogeneous. CONCLUSION: Our meta-analysis indicates defects in CD4+?lymphocytes, specifically decrease oin Tregs and increase in Th17 in ASD patients and supports the development of targeted immunotherapies in the field of ASD. En ligne : http://dx.doi.org/10.1186/s13229-021-00472-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459
in Molecular Autism > 12 (2021) . - 68 p.[article] Regulatory T lymphocytes/Th17 lymphocytes imbalance in autism spectrum disorders: evidence from a meta-analysis [Texte imprimé et/ou numérique] / P. ELLUL, Auteur ; M. ROSENZWAJG, Auteur ; H. PEYRE, Auteur ; G. FOURCADE, Auteur ; E. MARIOTTI-FERRANDIZ, Auteur ; V. TREBOSSEN, Auteur ; D. KLATZMANN, Auteur ; R. DELORME, Auteur . - 68 p.
Langues : Anglais (eng)
in Molecular Autism > 12 (2021) . - 68 p.
Mots-clés : Asd Immunology Peripheral blood Regulatory T lymphocyte Th17 lymphocytes Index. décimale : PER Périodiques Résumé : BACKGROUND: Immune system dysfunction has been proposed to play a critical role in the pathophysiology of autism spectrum disorders (ASD). Conflicting reports of lymphocyte subpopulation abnormalities have been described in numerous studies of patients with ASD. To better define lymphocytes abnormalities in ASD, we performed a meta-analysis of the lymphocyte profiles from subjects with ASD. METHODS: We used the PRISMA recommendations to query PubMed, Embase, PsychoINFO, BIOSIS, Science Direct, Cochrane CENTRAL, and Clinicaltrials.gov for terms related to clinical diagnosis of ASD and to lymphocytes' populations. We selected studies exploring lymphocyte subpopulations in children with ASD. The search protocol has been registered in the international Prospective Register of Systematic Reviews (CRD42019121473). RESULTS: We selected 13 studies gathering 388 ASD patients and 326 healthy controls. A significant decrease in the CD4+?lymphocyte was found in ASD patients compared to controls [-?1.51 (95% CI?-?2.99;?-?0.04) p?=?0.04] (I(2)?=?96% [95% CI 94.6, 97.7], p?0.01). No significant difference was found for the CD8+?T, B and natural killer lymphocytes. Considering the CD4+?subpopulation, there was a significant decrease in regulatory T lymphocytes (Tregs) in ASD patients (n?=?114) compared to controls (n?=?107) [-?3.09 (95% CI?-?4.41;?-?1.76) p?=?0.0001]; (I(2)?=?90.9%, [95% CI 76.2, 96.5], p?0.0001) associated with an increase oin the Th17 lymphocytes (ASD; n?=?147 controls; n?=?128) [2.23 (95% CI 0.79; 3.66) p?=?0,002] (I(2)?=?95.1% [95% CI 90.4, 97.5], p?0.0001). LIMITATIONS: Several factors inducing heterogeneity should be considered. First, differences in the staining method may be responsible for a part in the heterogeneity of results. Second, ASD population is also by itself heterogeneous, underlying the need of studying sub-groups that are more homogeneous. CONCLUSION: Our meta-analysis indicates defects in CD4+?lymphocytes, specifically decrease oin Tregs and increase in Th17 in ASD patients and supports the development of targeted immunotherapies in the field of ASD. En ligne : http://dx.doi.org/10.1186/s13229-021-00472-4 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=459 Regulatory variant of the TPH2 gene and early life stress are associated with heightened attention to social signals of fear in infants / Linda FORSSMAN in Journal of Child Psychology and Psychiatry, 55-7 (July 2014)
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PermalinkReimagining the environment in developmental psychopathology: from molecules to effective interventions / P. FEARON in Journal of Child Psychology and Psychiatry, 59-4 (April 2018)
PermalinkRejected and Dejected-Accepted and Elated / J. K. BROWN in Developmental Medicine & Child Neurology, 33-8 (August 1991)
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