Mirror me: Imitative responses in adults with autism / Odette SCHUNKE in Autism, 20-2 (February 2016)  

Public ISBD [article]  inAutism > 20-2 (February 2016) . - p.134-144 Titre : Mirror me: Imitative responses in adults with autism Type de document : texte imprimé Auteurs : Odette SCHUNKE, Auteur ; Daniel SCHOTTLE, Auteur ; Eik VETTORAZZI, Auteur ; Valerie C. BRANDT, Auteur ; Ursula KAHL, Auteur ; Tobias BÄUMER, Auteur ; Christos GANOS, Auteur ; Nicole DAVID, Auteur ; Ina PEIKER, Auteur ; Andreas K. ENGEL, Auteur ; Marcel BRASS, Auteur ; Alexander MUNCHAU, Auteur Article en page(s) : p.134-144 Langues : Anglais (eng) Mots-clés : autism  imitation  interference  mirror neurons  reaction times Index. décimale : PER Périodiques Résumé : Dysfunctions of the human mirror neuron system have been postulated to underlie some deficits in autism spectrum disorders including poor imitative performance and impaired social skills. Using three reaction time experiments addressing mirror neuron system functions under simple and complex conditions, we examined 20 adult autism spectrum disorder participants and 20 healthy controls matched for age, gender and education. Participants performed simple finger-lifting movements in response to (1) biological finger and non-biological dot movement stimuli, (2) acoustic stimuli and (3) combined visual-acoustic stimuli with different contextual (compatible/incompatible) and temporal (simultaneous/asynchronous) relation. Mixed model analyses revealed slower reaction times in autism spectrum disorder. Both groups responded faster to biological compared to non-biological stimuli (Experiment 1) implying intact processing advantage for biological stimuli in autism spectrum disorder. In Experiment 3, both groups had similar ‘interference effects’ when stimuli were presented simultaneously. However, autism spectrum disorder participants had abnormally slow responses particularly when incompatible stimuli were presented consecutively. Our results suggest imitative control deficits rather than global imitative system impairments. En ligne : http://dx.doi.org/10.1177/1362361315571757 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=278 [article] Mirror me: Imitative responses in adults with autism [texte imprimé] / Odette SCHUNKE, Auteur ; Daniel SCHOTTLE, Auteur ; Eik VETTORAZZI, Auteur ; Valerie C. BRANDT, Auteur ; Ursula KAHL, Auteur ; Tobias BÄUMER, Auteur ; Christos GANOS, Auteur ; Nicole DAVID, Auteur ; Ina PEIKER, Auteur ; Andreas K. ENGEL, Auteur ; Marcel BRASS, Auteur ; Alexander MUNCHAU, Auteur . - p.134-144. Langues : Anglais (eng) in Autism > 20-2 (February 2016) . - p.134-144 Mots-clés : autism  imitation  interference  mirror neurons  reaction times Index. décimale : PER Périodiques Résumé : Dysfunctions of the human mirror neuron system have been postulated to underlie some deficits in autism spectrum disorders including poor imitative performance and impaired social skills. Using three reaction time experiments addressing mirror neuron system functions under simple and complex conditions, we examined 20 adult autism spectrum disorder participants and 20 healthy controls matched for age, gender and education. Participants performed simple finger-lifting movements in response to (1) biological finger and non-biological dot movement stimuli, (2) acoustic stimuli and (3) combined visual-acoustic stimuli with different contextual (compatible/incompatible) and temporal (simultaneous/asynchronous) relation. Mixed model analyses revealed slower reaction times in autism spectrum disorder. Both groups responded faster to biological compared to non-biological stimuli (Experiment 1) implying intact processing advantage for biological stimuli in autism spectrum disorder. In Experiment 3, both groups had similar ‘interference effects’ when stimuli were presented simultaneously. However, autism spectrum disorder participants had abnormally slow responses particularly when incompatible stimuli were presented consecutively. Our results suggest imitative control deficits rather than global imitative system impairments. En ligne : http://dx.doi.org/10.1177/1362361315571757 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=278

White Matter Microstructure of the Human Mirror Neuron System is Related to Symptom Severity in Adults with Autism / O. FRUNDT in Journal of Autism and Developmental Disorders, 48-2 (February 2018)  

Public ISBD [article]  inJournal of Autism and Developmental Disorders > 48-2 (February 2018) . - p.417-429 Titre : White Matter Microstructure of the Human Mirror Neuron System is Related to Symptom Severity in Adults with Autism Type de document : texte imprimé Auteurs : O. FRUNDT, Auteur ; Robert SCHULZ, Auteur ; D. SCHOTTLE, Auteur ; Bastian CHENG, Auteur ; Götz THOMALLA, Auteur ; Hanna BRAASS, Auteur ; Christos GANOS, Auteur ; Nicole DAVID, Auteur ; Ina PEIKER, Auteur ; Andreas K. ENGEL, Auteur ; T. BAUMER, Auteur ; A. MUNCHAU, Auteur Article en page(s) : p.417-429 Langues : Anglais (eng) Mots-clés : Autism spectrum disorders  Diffusion tensor imaging  Fiber tracking  Imitation  Mirror neuron system Index. décimale : PER Périodiques Résumé : Mirror neuron system (MNS) dysfunctions might underlie deficits in autism spectrum disorders (ASD). Diffusion tensor imaging based probabilistic tractography was conducted in 15 adult ASD patients and 13 matched, healthy controls. Fractional anisotropy (FA) was quantified to assess group differences in tract-related white matter microstructure of both the classical MNS route (mediating "emulation") and the alternative temporo-frontal route (mediating "mimicry"). Multiple linear regression was used to investigate structure-function relationships between MNS connections and ASD symptom severity. There were no significant group differences in tract-related FA indicating an intact classical MNS in ASD. Direct temporo-frontal connections could not be reconstructed challengeing the concept of multiple routes for imitation. Tract-related FA of right-hemispheric parieto-frontal connections was negatively related to autism symptom severity. En ligne : https://doi.org/10.1007/s10803-017-3332-9 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=337 [article] White Matter Microstructure of the Human Mirror Neuron System is Related to Symptom Severity in Adults with Autism [texte imprimé] / O. FRUNDT, Auteur ; Robert SCHULZ, Auteur ; D. SCHOTTLE, Auteur ; Bastian CHENG, Auteur ; Götz THOMALLA, Auteur ; Hanna BRAASS, Auteur ; Christos GANOS, Auteur ; Nicole DAVID, Auteur ; Ina PEIKER, Auteur ; Andreas K. ENGEL, Auteur ; T. BAUMER, Auteur ; A. MUNCHAU, Auteur . - p.417-429. Langues : Anglais (eng) in Journal of Autism and Developmental Disorders > 48-2 (February 2018) . - p.417-429 Mots-clés : Autism spectrum disorders  Diffusion tensor imaging  Fiber tracking  Imitation  Mirror neuron system Index. décimale : PER Périodiques Résumé : Mirror neuron system (MNS) dysfunctions might underlie deficits in autism spectrum disorders (ASD). Diffusion tensor imaging based probabilistic tractography was conducted in 15 adult ASD patients and 13 matched, healthy controls. Fractional anisotropy (FA) was quantified to assess group differences in tract-related white matter microstructure of both the classical MNS route (mediating "emulation") and the alternative temporo-frontal route (mediating "mimicry"). Multiple linear regression was used to investigate structure-function relationships between MNS connections and ASD symptom severity. There were no significant group differences in tract-related FA indicating an intact classical MNS in ASD. Direct temporo-frontal connections could not be reconstructed challengeing the concept of multiple routes for imitation. Tract-related FA of right-hemispheric parieto-frontal connections was negatively related to autism symptom severity. En ligne : https://doi.org/10.1007/s10803-017-3332-9 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=337

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