Adolescent girls’ neural response to reward mediates the relation between childhood financial disadvantage and depression / Sarah E. ROMENS in Journal of Child Psychology and Psychiatry, 56-11 (November 2015)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 56-11 (November 2015) . - p.1177-1184 Titre : Adolescent girls’ neural response to reward mediates the relation between childhood financial disadvantage and depression Type de document : texte imprimé Auteurs : Sarah E. ROMENS, Auteur ; Melynda D. CASEMENT, Auteur ; Rose MCALOON, Auteur ; Kate KEENAN, Auteur ; Alison E. HIPWELL, Auteur ; Amanda E. GUYER, Auteur ; Erika E. FORBES, Auteur Article en page(s) : p.1177-1184 Langues : Anglais (eng) Mots-clés : Socioeconomic status  reward  depression  neural  medial prefrontal cortex Index. décimale : PER Périodiques Résumé : Background Children who experience socioeconomic disadvantage are at heightened risk for developing depression; however, little is known about neurobiological mechanisms underlying this association. Low socioeconomic status (SES) during childhood may confer risk for depression through its stress-related effects on the neural circuitry associated with processing monetary rewards. Methods In a prospective study, we examined the relationships among the number of years of household receipt of public assistance from age 5–16 years, neural activation during monetary reward anticipation and receipt at age 16, and depression symptoms at age 16 in 123 girls. Results Number of years of household receipt of public assistance was positively associated with heightened response in the medial prefrontal cortex during reward anticipation, and this heightened neural response mediated the relationship between socioeconomic disadvantage and current depression symptoms, controlling for past depression. Conclusions Chronic exposure to socioeconomic disadvantage in childhood may alter neural circuitry involved in reward anticipation in adolescence, which in turn may confer risk for depression. En ligne : http://dx.doi.org/10.1111/jcpp.12410 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=270 [article] Adolescent girls’ neural response to reward mediates the relation between childhood financial disadvantage and depression [texte imprimé] / Sarah E. ROMENS, Auteur ; Melynda D. CASEMENT, Auteur ; Rose MCALOON, Auteur ; Kate KEENAN, Auteur ; Alison E. HIPWELL, Auteur ; Amanda E. GUYER, Auteur ; Erika E. FORBES, Auteur . - p.1177-1184. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 56-11 (November 2015) . - p.1177-1184 Mots-clés : Socioeconomic status  reward  depression  neural  medial prefrontal cortex Index. décimale : PER Périodiques Résumé : Background Children who experience socioeconomic disadvantage are at heightened risk for developing depression; however, little is known about neurobiological mechanisms underlying this association. Low socioeconomic status (SES) during childhood may confer risk for depression through its stress-related effects on the neural circuitry associated with processing monetary rewards. Methods In a prospective study, we examined the relationships among the number of years of household receipt of public assistance from age 5–16 years, neural activation during monetary reward anticipation and receipt at age 16, and depression symptoms at age 16 in 123 girls. Results Number of years of household receipt of public assistance was positively associated with heightened response in the medial prefrontal cortex during reward anticipation, and this heightened neural response mediated the relationship between socioeconomic disadvantage and current depression symptoms, controlling for past depression. Conclusions Chronic exposure to socioeconomic disadvantage in childhood may alter neural circuitry involved in reward anticipation in adolescence, which in turn may confer risk for depression. En ligne : http://dx.doi.org/10.1111/jcpp.12410 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=270

Annual Research Review: Puberty and the development of anhedonia - considering childhood adversity and inflammation / Tina GUPTA in Journal of Child Psychology and Psychiatry, 65-4 (April 2024)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 65-4 (April 2024) . - p.459-480 Titre : Annual Research Review: Puberty and the development of anhedonia - considering childhood adversity and inflammation Type de document : texte imprimé Auteurs : Tina GUPTA, Auteur ; Kristen L. ECKSTRAND, Auteur ; Erika E. FORBES, Auteur Article en page(s) : p.459-480 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Anhedonia, or diminished pleasure and motivation, is a symptom of severe mental illness (e.g., depressive disorder, bipolar disorder, schizophrenia) that emerges during adolescence. Anhedonia is a pernicious symptom that is related to social impairments, treatment resistance, and suicide. As the mechanisms of anhedonia are postulated to include the frontostriatal circuitry and the dopamine neuromodulatory system, the development and plasticity of these systems during the vulnerable period of adolescence, as well as their sensitivity to pubertal hormones, suggest that pubertal maturation could play a role in the development of anhedonia. This review takes a developmental perspective, considering the possibility that anhedonia emerges in the context of pubertal maturation and adolescent development, with childhood adversity and chronic inflammation influencing neural reward systems to accelerate anhedonia's progression. Here, we review the relevant extant literature on the components of this model and suggest directions for future research. En ligne : https://doi.org/10.1111/jcpp.13955 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523 [article] Annual Research Review: Puberty and the development of anhedonia - considering childhood adversity and inflammation [texte imprimé] / Tina GUPTA, Auteur ; Kristen L. ECKSTRAND, Auteur ; Erika E. FORBES, Auteur . - p.459-480. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 65-4 (April 2024) . - p.459-480 Index. décimale : PER Périodiques Résumé : Anhedonia, or diminished pleasure and motivation, is a symptom of severe mental illness (e.g., depressive disorder, bipolar disorder, schizophrenia) that emerges during adolescence. Anhedonia is a pernicious symptom that is related to social impairments, treatment resistance, and suicide. As the mechanisms of anhedonia are postulated to include the frontostriatal circuitry and the dopamine neuromodulatory system, the development and plasticity of these systems during the vulnerable period of adolescence, as well as their sensitivity to pubertal hormones, suggest that pubertal maturation could play a role in the development of anhedonia. This review takes a developmental perspective, considering the possibility that anhedonia emerges in the context of pubertal maturation and adolescent development, with childhood adversity and chronic inflammation influencing neural reward systems to accelerate anhedonia's progression. Here, we review the relevant extant literature on the components of this model and suggest directions for future research. En ligne : https://doi.org/10.1111/jcpp.13955 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=523

Brain activity and infant attachment history in young men during loss and reward processing / Karina QUEVEDO in Development and Psychopathology, 29-2 (May 2017)  

Public ISBD [article]  inDevelopment and Psychopathology > 29-2 (May 2017) . - p.465-476 Titre : Brain activity and infant attachment history in young men during loss and reward processing Type de document : texte imprimé Auteurs : Karina QUEVEDO, Auteur ; Theodore E. A. WATERS, Auteur ; Hannah SCOTT, Auteur ; Glenn I. ROISMAN, Auteur ; Daniel S. SHAW, Auteur ; Erika E. FORBES, Auteur Article en page(s) : p.465-476 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : There is now ample evidence that the quality of early attachment experiences shapes expectations for supportive and responsive care and ultimately serves to scaffold adaptation to the salient tasks of development. Nonetheless, few studies have identified neural mechanisms that might give rise to these associations. Using a moderately large sample of low-income male participants recruited during infancy (N = 171), we studied the predictive significance of attachment insecurity and disorganization at age 18 months (as measured in the Strange Situation Procedure) for patterns of neural activation to reward and loss at age 20 years (assessed during a reward-based task as part of a functional magnetic resonance imaging scan). Results indicated that individuals with a history of insecure attachment showed hyperactivity in (a) reward- and emotion-related (e.g., basal ganglia and amygdala) structures and (b) emotion regulation and self-referential processing (cortical midline structures) in response to positive and negative outcomes (and anticipation of those outcomes). Further, the neural activation of individuals with a history of disorganized attachment suggested that they had greater emotional reactivity in anticipation of reward and employed greater cognitive control when negative outcomes were encountered. Overall, results suggest that the quality of early attachments has lasting impacts on brain function and reward processing. En ligne : http://dx.doi.org/10.1017/s0954579417000116 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=305 [article] Brain activity and infant attachment history in young men during loss and reward processing [texte imprimé] / Karina QUEVEDO, Auteur ; Theodore E. A. WATERS, Auteur ; Hannah SCOTT, Auteur ; Glenn I. ROISMAN, Auteur ; Daniel S. SHAW, Auteur ; Erika E. FORBES, Auteur . - p.465-476. Langues : Anglais (eng) in Development and Psychopathology > 29-2 (May 2017) . - p.465-476 Index. décimale : PER Périodiques Résumé : There is now ample evidence that the quality of early attachment experiences shapes expectations for supportive and responsive care and ultimately serves to scaffold adaptation to the salient tasks of development. Nonetheless, few studies have identified neural mechanisms that might give rise to these associations. Using a moderately large sample of low-income male participants recruited during infancy (N = 171), we studied the predictive significance of attachment insecurity and disorganization at age 18 months (as measured in the Strange Situation Procedure) for patterns of neural activation to reward and loss at age 20 years (assessed during a reward-based task as part of a functional magnetic resonance imaging scan). Results indicated that individuals with a history of insecure attachment showed hyperactivity in (a) reward- and emotion-related (e.g., basal ganglia and amygdala) structures and (b) emotion regulation and self-referential processing (cortical midline structures) in response to positive and negative outcomes (and anticipation of those outcomes). Further, the neural activation of individuals with a history of disorganized attachment suggested that they had greater emotional reactivity in anticipation of reward and employed greater cognitive control when negative outcomes were encountered. Overall, results suggest that the quality of early attachments has lasting impacts on brain function and reward processing. En ligne : http://dx.doi.org/10.1017/s0954579417000116 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=305

Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood / Luke W. HYDE in Journal of Child Psychology and Psychiatry, 59-10 (October 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Titre : Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood Type de document : texte imprimé Auteurs : Luke W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; Lynne MURRAY, Auteur ; Erika E. FORBES, Auteur Article en page(s) : p.1073-1082 Langues : Anglais (eng) Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369 [article] Deflections from adolescent trajectories of antisocial behavior: contextual and neural moderators of antisocial behavior stability into emerging adulthood [texte imprimé] / Luke W. HYDE, Auteur ; R. WALLER, Auteur ; D. S. SHAW, Auteur ; Lynne MURRAY, Auteur ; Erika E. FORBES, Auteur . - p.1073-1082. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-10 (October 2018) . - p.1073-1082 Mots-clés : Antisocial behavior amygdala conduct disorder desistance ventral striatum Index. décimale : PER Périodiques Résumé : BACKGROUND: Early adulthood is a critical period when young men involved in antisocial behavior (AB) may desist. Factors including marriage and employment have been shown to predict desistance, but little work has examined whether biological factors (e.g. neural reactivity) predict deflections from lifelong AB trajectories. METHODS: We examined the continuity of, or desistance from, AB in early adulthood using group-based trajectories of AB across adolescence in a sample of 242 men from low-income, urban families. We examined contextual factors (romantic relationship quality, employment, neighborhood danger) and neural factors (amygdala reactivity to fearful faces, ventral striatum reactivity to reward) as moderators of the continuity of AB from adolescence (age 10-17) into early adulthood (age 22-23), and whether these pathways differed by race. RESULTS: High relationship satisfaction and employment at age 20 predicted decreased AB at age 22-23, but only among men with adolescent-onset/moderate AB trajectories. Ventral striatum reactivity predicted continued AB, but only among African-American men with early-starting AB. Amygdala reactivity to fearful faces was related to later AB for those in the early-starting group, but in divergent directions depending on race: amygdala reactivity to fearful faces was positively related to AB in European-Americans and negatively related to AB among African-Americans. CONCLUSIONS: Contextual factors only predicted deflections of AB in those engaged in late-starting, moderate levels of AB, whereas neural factors predicted continued AB only in those with early-starting, severe AB, and in divergent ways based on participant race. Though there is limited power to infer causality from this observational design, research on desistance broadly can contribute to informing personalized interventions for those engaged in serious adolescence AB. En ligne : http://dx.doi.org/10.1111/jcpp.12931 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=369

Determining the key childhood and adolescent risk factors for future BPD symptoms using regularized regression: comparison to depression and conduct disorder / Joseph E. BEENEY in Journal of Child Psychology and Psychiatry, 62-2 (February 2021)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 62-2 (February 2021) . - p.223-231 Titre : Determining the key childhood and adolescent risk factors for future BPD symptoms using regularized regression: comparison to depression and conduct disorder Type de document : texte imprimé Auteurs : Joseph E. BEENEY, Auteur ; Erika E. FORBES, Auteur ; Alison E. HIPWELL, Auteur ; Melissa NANCE, Auteur ; Alexis MATTIA, Auteur ; Joely M. LAWLESS, Auteur ; Layla BANIHASHEMI, Auteur ; Stephanie D. STEPP, Auteur Année de publication : 2021 Article en page(s) : p.223-231 Langues : Anglais (eng) Mots-clés : Risk factors  borderline personality disorder  comorbidity  longitudinal studies  machine learning Index. décimale : PER Périodiques Résumé : OBJECTIVE: Research has yielded factors considered critical to risk for borderline personality disorder (BPD). Yet, these factors overlap and are relevant to other disorders, like depression and conduct disorder (CD). Regularized regression, a machine learning approach, was developed to allow identification of the most important variables in large datasets with correlated predictors. We aimed to identify critical predictors of BPD symptoms in late adolescence (ages 16-18) and determine the specificity of factors to BPD versus disorders with putatively similar etiology. METHOD: We used a prospective longitudinal dataset (n = 2,450) of adolescent girls assessed on a range of clinical, psychosocial, and demographic factors, highlighted by previous research on BPD. Predictors were grouped by developmental periods: late childhood (8-10) and early (11-13) and mid-adolescence (14-15), yielding 128 variables from 41 constructs. The same variables were used in models predicting depression and CD symptoms. RESULTS: The best-fitting model for BPD symptoms included 19 predictors and explained 33.2% of the variance. Five constructs - depressive and anxiety symptoms, self-control, harsh punishment, and poor social and school functioning - accounted for most of the variance explained. BPD was differentiated from CD by greater problems with mood and anxiety in BPD and differences in parenting risk factors. Whereas the biggest parenting risk for BPD was a punitive style of parenting, CD was predicted by both punitive and disengaged styles. BPD was differentiated from MDD by greater social problems and poor behavioral control in BPD. CONCLUSIONS: The best predictors of BPD symptoms in adolescence are features suggesting complex comorbidity, affective activation, and problems with self-control. Though some risk factors were non-specific (e.g., inattention), the disorders were distinguished in clinically significant ways. En ligne : http://dx.doi.org/10.1111/jcpp.13269 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=440 [article] Determining the key childhood and adolescent risk factors for future BPD symptoms using regularized regression: comparison to depression and conduct disorder [texte imprimé] / Joseph E. BEENEY, Auteur ; Erika E. FORBES, Auteur ; Alison E. HIPWELL, Auteur ; Melissa NANCE, Auteur ; Alexis MATTIA, Auteur ; Joely M. LAWLESS, Auteur ; Layla BANIHASHEMI, Auteur ; Stephanie D. STEPP, Auteur . - 2021 . - p.223-231. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 62-2 (February 2021) . - p.223-231 Mots-clés : Risk factors  borderline personality disorder  comorbidity  longitudinal studies  machine learning Index. décimale : PER Périodiques Résumé : OBJECTIVE: Research has yielded factors considered critical to risk for borderline personality disorder (BPD). Yet, these factors overlap and are relevant to other disorders, like depression and conduct disorder (CD). Regularized regression, a machine learning approach, was developed to allow identification of the most important variables in large datasets with correlated predictors. We aimed to identify critical predictors of BPD symptoms in late adolescence (ages 16-18) and determine the specificity of factors to BPD versus disorders with putatively similar etiology. METHOD: We used a prospective longitudinal dataset (n = 2,450) of adolescent girls assessed on a range of clinical, psychosocial, and demographic factors, highlighted by previous research on BPD. Predictors were grouped by developmental periods: late childhood (8-10) and early (11-13) and mid-adolescence (14-15), yielding 128 variables from 41 constructs. The same variables were used in models predicting depression and CD symptoms. RESULTS: The best-fitting model for BPD symptoms included 19 predictors and explained 33.2% of the variance. Five constructs - depressive and anxiety symptoms, self-control, harsh punishment, and poor social and school functioning - accounted for most of the variance explained. BPD was differentiated from CD by greater problems with mood and anxiety in BPD and differences in parenting risk factors. Whereas the biggest parenting risk for BPD was a punitive style of parenting, CD was predicted by both punitive and disengaged styles. BPD was differentiated from MDD by greater social problems and poor behavioral control in BPD. CONCLUSIONS: The best predictors of BPD symptoms in adolescence are features suggesting complex comorbidity, affective activation, and problems with self-control. Though some risk factors were non-specific (e.g., inattention), the disorders were distinguished in clinically significant ways. En ligne : http://dx.doi.org/10.1111/jcpp.13269 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=440

Emotional reactivity and regulation in anxious and nonanxious youth: a cell-phone ecological momentary assessment study / Patricia Z. TAN in Journal of Child Psychology and Psychiatry, 53-2 (February 2012)  

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Error-related brain activity in pediatric anxiety disorders remains elevated following individual therapy: a randomized clinical trial / Cecile D. LADOUCEUR in Journal of Child Psychology and Psychiatry, 59-11 (November 2018)  

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Girls' brain structural connectivity in late adolescence relates to history of depression symptoms / Rajpreet CHAHAL in Journal of Child Psychology and Psychiatry, 61-11 (November 2020)  

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Interactions between empathy and resting heart rate in early adolescence predict violent behavior in late adolescence and early adulthood / Chardée A. GALÁN in Journal of Child Psychology and Psychiatry, 58-12 (December 2017)  

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Maladaptive social information processing in childhood predicts young men's atypical amygdala reactivity to threat / Daniel Ewon CHOE in Journal of Child Psychology and Psychiatry, 56-5 (May 2015)  

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Maternal depression, child frontal asymmetry, and child affective behavior as factors in child behavior problems / Erika E. FORBES in Journal of Child Psychology and Psychiatry, 47-1 (January 2006)  

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Maternal depression in childhood and aggression in young adulthood: evidence for mediation by offspring amygdala–hippocampal volume ratio / Mary GILLIAM in Journal of Child Psychology and Psychiatry, 56-10 (October 2015)  

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More time awake after sleep onset is linked to reduced ventral striatum response to rewards in youth with anxiety / Nathan A. SOLLENBERGER in Journal of Child Psychology and Psychiatry, 64-1 (January 2023)  

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Parental autonomy granting and child perceived control: effects on the everyday emotional experience of anxious youth / Kristy BENOIT ALLEN in Journal of Child Psychology and Psychiatry, 57-7 (July 2016)  

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Parents still matter! Parental warmth predicts adolescent brain function and anxiety and depressive symptoms 2 years later / Rosalind D. BUTTERFIELD in Development and Psychopathology, 33-1 (February 2021)  

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