Associations between the parent–child relationship and adolescent self-worth: a genetically informed study of twin parents and their adolescent children / Tom A. MCADAMS in Journal of Child Psychology and Psychiatry, 58-1 (January 2017)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 58-1 (January 2017) . - p.46-54 Titre : Associations between the parent–child relationship and adolescent self-worth: a genetically informed study of twin parents and their adolescent children Type de document : Texte imprimé et/ou numérique Auteurs : Tom A. MCADAMS, Auteur ; Frühling V. RIJSDIJK, Auteur ; Jurgita NARUSYTE, Auteur ; Jody M. GANIBAN, Auteur ; David REISS, Auteur ; Erica SPOTTS, Auteur ; Jenae M. NEIDERHISER, Auteur ; Paul LICHTENSTEIN, Auteur ; Thalia C. ELEY, Auteur Article en page(s) : p.46-54 Langues : Anglais (eng) Mots-clés : Adolescence  parenting  parent–child relationships  children-of-twins  self-esteem Index. décimale : PER Périodiques Résumé : Background Low self-worth during adolescence predicts a range of emotional and behavioural problems. As such, identifying potential sources of influence on self-worth is important. Aspects of the parent–child relationship are often associated with adolescent self-worth but to date it is unclear whether such associations may be attributable to familial confounding (e.g. genetic relatedness). We set out to clarify the nature of relationships between parental expressed affection and adolescent self-worth, and parent–child closeness and adolescent self-worth. Methods We used data from the Twin and Offspring Study in Sweden, a children-of-twins sample comprising 909 adult twin pairs with adolescent children. Using these data we were able to apply structural equation models with which we could examine whether associations remained after accounting for genetic transmission. Results Results demonstrated that parent–child closeness and parental-expressed affection were both phenotypically associated with adolescent self-worth. Associations could not be attributed to genetic relatedness between parent and child. Conclusions Parent–child closeness and parental affection are associated with adolescent self-worth above and beyond effects attributable to genetic relatedness. Data were cross-sectional, so the direction of effects cannot be confirmed but findings support the notion that positive parent–child relationships increase adolescent self-worth. En ligne : http://dx.doi.org/10.1111/jcpp.12600 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298 [article] Associations between the parent–child relationship and adolescent self-worth: a genetically informed study of twin parents and their adolescent children [Texte imprimé et/ou numérique] / Tom A. MCADAMS, Auteur ; Frühling V. RIJSDIJK, Auteur ; Jurgita NARUSYTE, Auteur ; Jody M. GANIBAN, Auteur ; David REISS, Auteur ; Erica SPOTTS, Auteur ; Jenae M. NEIDERHISER, Auteur ; Paul LICHTENSTEIN, Auteur ; Thalia C. ELEY, Auteur . - p.46-54. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 58-1 (January 2017) . - p.46-54 Mots-clés : Adolescence  parenting  parent–child relationships  children-of-twins  self-esteem Index. décimale : PER Périodiques Résumé : Background Low self-worth during adolescence predicts a range of emotional and behavioural problems. As such, identifying potential sources of influence on self-worth is important. Aspects of the parent–child relationship are often associated with adolescent self-worth but to date it is unclear whether such associations may be attributable to familial confounding (e.g. genetic relatedness). We set out to clarify the nature of relationships between parental expressed affection and adolescent self-worth, and parent–child closeness and adolescent self-worth. Methods We used data from the Twin and Offspring Study in Sweden, a children-of-twins sample comprising 909 adult twin pairs with adolescent children. Using these data we were able to apply structural equation models with which we could examine whether associations remained after accounting for genetic transmission. Results Results demonstrated that parent–child closeness and parental-expressed affection were both phenotypically associated with adolescent self-worth. Associations could not be attributed to genetic relatedness between parent and child. Conclusions Parent–child closeness and parental affection are associated with adolescent self-worth above and beyond effects attributable to genetic relatedness. Data were cross-sectional, so the direction of effects cannot be confirmed but findings support the notion that positive parent–child relationships increase adolescent self-worth. En ligne : http://dx.doi.org/10.1111/jcpp.12600 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298

Exploring anxiety symptoms in a large-scale twin study of children with autism spectrum disorders, their co-twins and controls / Victoria HALLETT in Journal of Child Psychology and Psychiatry, 54-11 (November 2013)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 54-11 (November 2013) . - p.1176-1185 Titre : Exploring anxiety symptoms in a large-scale twin study of children with autism spectrum disorders, their co-twins and controls Type de document : Texte imprimé et/ou numérique Auteurs : Victoria HALLETT, Auteur ; Angelica RONALD, Auteur ; Emma COLVERT, Auteur ; Catherine S. AMES, Auteur ; Emma WOODHOUSE, Auteur ; Stephanie LIETZ, Auteur ; Tracy GARNETT, Auteur ; Nicola GILLAN, Auteur ; Frühling V. RIJSDIJK, Auteur ; Lawrence SCAHILL, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur Article en page(s) : p.1176-1185 Langues : Anglais (eng) Mots-clés : Autism spectrum disorders  anxiety  twin  siblings  comorbidity Index. décimale : PER Périodiques Résumé : Background Although many children with autism spectrum disorders (ASDs) experience difficulties with anxiety, the manifestation of these difficulties remains unresolved. The current study assessed anxiety in a large population-based twin sample, aged 10–15 years. Phenotypic analyses were used to explore anxiety symptoms in children with ASDs, their unaffected co-twins and a control sample. Methods Participants included 146 families from the Twins Early Development Study (TEDS) where one or both children had a suspected ASD. Eighty control families were also included. The Revised Child Anxiety and Depression scale (Chorpita, Yim, Moffitt, Umemoto Francis, 2000) was completed (self- and parent-report), along with diagnostic and cognitive tests. Children were categorized into four groups (a) ASD (b) Broader Autism Phenotype (BAP: mainly co-twins of children with ASDs, with high subclinical autistic traits) (c) unaffected co-twins (with neither ASDs nor BAP) (d) controls. Results Children in the ASD and BAP groups scored significantly higher than controls for all parent-rated (although not child-rated) anxiety subscales. There were no significant differences between the ASD and BAP groups for any of the parent-rated anxiety subscales. Compared with controls, unaffected co-twins showed significantly heightened Social Anxiety, Generalized Anxiety, and Panic symptoms. Significant associations were observed between certain anxiety subscales and both IQ and ASD symptoms. For example, greater parent-rated Social Anxiety was associated with higher IQ and increased social and communicative impairments. Significant interrater correlations were observed for anxiety reports in children with ASDs (r = .27–.54; p .01), their unaffected co-twins (r = .32–.63; p .01) and controls (r = .23–.43; p .01) suggesting that children in this sample with and without ASD symptoms were able to report on their anxiety symptoms with some accuracy. Conclusions These findings support previous reports of heightened anxiety in children with ASDs, at least on parent-reported measures. Unaffected co-twins of children with ASDs also showed increased anxiety, generating questions about the potential etiological overlap between ASDs and anxiety. Progress in this area now depends on more refined anxiety measurement in ASDs and continued investigation of interrater differences. En ligne : http://dx.doi.org/10.1111/jcpp.12068 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=217 [article] Exploring anxiety symptoms in a large-scale twin study of children with autism spectrum disorders, their co-twins and controls [Texte imprimé et/ou numérique] / Victoria HALLETT, Auteur ; Angelica RONALD, Auteur ; Emma COLVERT, Auteur ; Catherine S. AMES, Auteur ; Emma WOODHOUSE, Auteur ; Stephanie LIETZ, Auteur ; Tracy GARNETT, Auteur ; Nicola GILLAN, Auteur ; Frühling V. RIJSDIJK, Auteur ; Lawrence SCAHILL, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur . - p.1176-1185. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 54-11 (November 2013) . - p.1176-1185 Mots-clés : Autism spectrum disorders  anxiety  twin  siblings  comorbidity Index. décimale : PER Périodiques Résumé : Background Although many children with autism spectrum disorders (ASDs) experience difficulties with anxiety, the manifestation of these difficulties remains unresolved. The current study assessed anxiety in a large population-based twin sample, aged 10–15 years. Phenotypic analyses were used to explore anxiety symptoms in children with ASDs, their unaffected co-twins and a control sample. Methods Participants included 146 families from the Twins Early Development Study (TEDS) where one or both children had a suspected ASD. Eighty control families were also included. The Revised Child Anxiety and Depression scale (Chorpita, Yim, Moffitt, Umemoto Francis, 2000) was completed (self- and parent-report), along with diagnostic and cognitive tests. Children were categorized into four groups (a) ASD (b) Broader Autism Phenotype (BAP: mainly co-twins of children with ASDs, with high subclinical autistic traits) (c) unaffected co-twins (with neither ASDs nor BAP) (d) controls. Results Children in the ASD and BAP groups scored significantly higher than controls for all parent-rated (although not child-rated) anxiety subscales. There were no significant differences between the ASD and BAP groups for any of the parent-rated anxiety subscales. Compared with controls, unaffected co-twins showed significantly heightened Social Anxiety, Generalized Anxiety, and Panic symptoms. Significant associations were observed between certain anxiety subscales and both IQ and ASD symptoms. For example, greater parent-rated Social Anxiety was associated with higher IQ and increased social and communicative impairments. Significant interrater correlations were observed for anxiety reports in children with ASDs (r = .27–.54; p .01), their unaffected co-twins (r = .32–.63; p .01) and controls (r = .23–.43; p .01) suggesting that children in this sample with and without ASD symptoms were able to report on their anxiety symptoms with some accuracy. Conclusions These findings support previous reports of heightened anxiety in children with ASDs, at least on parent-reported measures. Unaffected co-twins of children with ASDs also showed increased anxiety, generating questions about the potential etiological overlap between ASDs and anxiety. Progress in this area now depends on more refined anxiety measurement in ASDs and continued investigation of interrater differences. En ligne : http://dx.doi.org/10.1111/jcpp.12068 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=217

Genetic origin of the relationship between parental negativity and behavior problems from early childhood to adolescence: A longitudinal genetically sensitive study / Silvia ALEMANY in Development and Psychopathology, 25-2 (May 2013)  

Public ISBD [article]  inDevelopment and Psychopathology > 25-2 (May 2013) . - p.487-500 Titre : Genetic origin of the relationship between parental negativity and behavior problems from early childhood to adolescence: A longitudinal genetically sensitive study Type de document : Texte imprimé et/ou numérique Auteurs : Silvia ALEMANY, Auteur ; Frühling V. RIJSDIJK, Auteur ; Claire Margaret Alison HAWORTH, Auteur ; Lourdes FAÑANÁS, Auteur ; Robert PLOMIN, Auteur Article en page(s) : p.487-500 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Little is known about how genetic and environmental factors contribute to the association between parental negativity and behavior problems from early childhood to adolescence. The current study fitted a cross-lagged model in a sample consisting of 4,075 twin pairs to explore (a) the role of genetic and environmental factors in the relationship between parental negativity and behavior problems from age 4 to age 12, (b) whether parent-driven and child-driven processes independently explain the association, and (c) whether there are sex differences in this relationship. Both phenotypes showed substantial genetic influence at both ages. The concurrent overlap between them was mainly accounted for by genetic factors. Causal pathways representing stability of the phenotypes and parent-driven and child-driven effects significantly and independently account for the association. Significant but slight differences were found between males and females for parent-driven effects. These results were highly similar when general cognitive ability was added as a covariate. In summary, the longitudinal association between parental negativity and behavior problems seems to be bidirectional and mainly accounted for by genetic factors. Furthermore, child-driven effects were mainly genetically mediated, and parent-driven effects were a function of both genetic and shared-environmental factors. En ligne : http://dx.doi.org/10.1017/S0954579412001198 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=199 [article] Genetic origin of the relationship between parental negativity and behavior problems from early childhood to adolescence: A longitudinal genetically sensitive study [Texte imprimé et/ou numérique] / Silvia ALEMANY, Auteur ; Frühling V. RIJSDIJK, Auteur ; Claire Margaret Alison HAWORTH, Auteur ; Lourdes FAÑANÁS, Auteur ; Robert PLOMIN, Auteur . - p.487-500. Langues : Anglais (eng) in Development and Psychopathology > 25-2 (May 2013) . - p.487-500 Index. décimale : PER Périodiques Résumé : Little is known about how genetic and environmental factors contribute to the association between parental negativity and behavior problems from early childhood to adolescence. The current study fitted a cross-lagged model in a sample consisting of 4,075 twin pairs to explore (a) the role of genetic and environmental factors in the relationship between parental negativity and behavior problems from age 4 to age 12, (b) whether parent-driven and child-driven processes independently explain the association, and (c) whether there are sex differences in this relationship. Both phenotypes showed substantial genetic influence at both ages. The concurrent overlap between them was mainly accounted for by genetic factors. Causal pathways representing stability of the phenotypes and parent-driven and child-driven effects significantly and independently account for the association. Significant but slight differences were found between males and females for parent-driven effects. These results were highly similar when general cognitive ability was added as a covariate. In summary, the longitudinal association between parental negativity and behavior problems seems to be bidirectional and mainly accounted for by genetic factors. Furthermore, child-driven effects were mainly genetically mediated, and parent-driven effects were a function of both genetic and shared-environmental factors. En ligne : http://dx.doi.org/10.1017/S0954579412001198 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=199

Heritability of autism spectrum disorders: a meta-analysis of twin studies / Beata TICK in Journal of Child Psychology and Psychiatry, 57-5 (May 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-5 (May 2016) . - p.585-595 Titre : Heritability of autism spectrum disorders: a meta-analysis of twin studies Type de document : Texte imprimé et/ou numérique Auteurs : Beata TICK, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur ; Michael RUTTER, Auteur ; Frühling V. RIJSDIJK, Auteur Article en page(s) : p.585-595 Langues : Anglais (eng) Mots-clés : Autism spectrum disorders  meta-analysis  heritability  twin studies  DF extremes analysis Index. décimale : PER Périodiques Résumé : Background The etiology of Autism Spectrum Disorder (ASD) has been recently debated due to emerging findings on the importance of shared environmental influences. However, two recent twin studies do not support this and instead re-affirm strong genetic effects on the liability to ASD, a finding consistent with previous reports. This study conducts a systematic review and meta-analysis of all twin studies of ASD published to date and explores the etiology along the continuum of a quantitative measure of ASD. Methods A PubMed Central, Science Direct, Google Scholar, Web of Knowledge structured search conducted online, to identify all twin studies on ASD published to date. Thirteen primary twin studies were identified, seven were included in the meta-analysis by meeting Systematic Recruitment criterion; correction for selection and ascertainment strategies, and applied prevalences were assessed for these studies. In addition, a quantile DF extremes analysis was carried out on Childhood Autism Spectrum Test scores measured in a population sample of 6,413 twin pairs including affected twins. Results The meta-analysis correlations for monozygotic twins (MZ) were almost perfect at .98 (95% Confidence Interval, .96–.99). The dizygotic (DZ) correlation, however, was .53 (95% CI .44–.60) when ASD prevalence rate was set at 5% (in line with the Broad Phenotype of ASD) and increased to .67 (95% CI .61–.72) when applying a prevalence rate of 1%. The meta-analytic heritability estimates were substantial: 64–91%. Shared environmental effects became significant as the prevalence rate decreased from 5–1%: 07–35%. The DF analyses show that for the most part, there is no departure from linearity in heritability. Conclusions We demonstrate that: (a) ASD is due to strong genetic effects; (b) shared environmental effects become significant as a function of lower prevalence rate; (c) previously reported significant shared environmental influences are likely a statistical artefact of overinclusion of concordant DZ twins. En ligne : http://dx.doi.org/10.1111/jcpp.12499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=288 [article] Heritability of autism spectrum disorders: a meta-analysis of twin studies [Texte imprimé et/ou numérique] / Beata TICK, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur ; Michael RUTTER, Auteur ; Frühling V. RIJSDIJK, Auteur . - p.585-595. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-5 (May 2016) . - p.585-595 Mots-clés : Autism spectrum disorders  meta-analysis  heritability  twin studies  DF extremes analysis Index. décimale : PER Périodiques Résumé : Background The etiology of Autism Spectrum Disorder (ASD) has been recently debated due to emerging findings on the importance of shared environmental influences. However, two recent twin studies do not support this and instead re-affirm strong genetic effects on the liability to ASD, a finding consistent with previous reports. This study conducts a systematic review and meta-analysis of all twin studies of ASD published to date and explores the etiology along the continuum of a quantitative measure of ASD. Methods A PubMed Central, Science Direct, Google Scholar, Web of Knowledge structured search conducted online, to identify all twin studies on ASD published to date. Thirteen primary twin studies were identified, seven were included in the meta-analysis by meeting Systematic Recruitment criterion; correction for selection and ascertainment strategies, and applied prevalences were assessed for these studies. In addition, a quantile DF extremes analysis was carried out on Childhood Autism Spectrum Test scores measured in a population sample of 6,413 twin pairs including affected twins. Results The meta-analysis correlations for monozygotic twins (MZ) were almost perfect at .98 (95% Confidence Interval, .96–.99). The dizygotic (DZ) correlation, however, was .53 (95% CI .44–.60) when ASD prevalence rate was set at 5% (in line with the Broad Phenotype of ASD) and increased to .67 (95% CI .61–.72) when applying a prevalence rate of 1%. The meta-analytic heritability estimates were substantial: 64–91%. Shared environmental effects became significant as the prevalence rate decreased from 5–1%: 07–35%. The DF analyses show that for the most part, there is no departure from linearity in heritability. Conclusions We demonstrate that: (a) ASD is due to strong genetic effects; (b) shared environmental effects become significant as a function of lower prevalence rate; (c) previously reported significant shared environmental influences are likely a statistical artefact of overinclusion of concordant DZ twins. En ligne : http://dx.doi.org/10.1111/jcpp.12499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=288

Heterogeneity in antisocial behaviours and comorbidity with depressed mood: a behavioural genetic approach / Richard ROWE in Journal of Child Psychology and Psychiatry, 49-5 (May 2008)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 49-5 (May 2008) . - p.526-534 Titre : Heterogeneity in antisocial behaviours and comorbidity with depressed mood: a behavioural genetic approach Type de document : Texte imprimé et/ou numérique Auteurs : Richard ROWE, Auteur ; Barbara MAUGHAN, Auteur ; Thalia C. ELEY, Auteur ; Georgina M. HOSANG, Auteur ; Frühling V. RIJSDIJK, Auteur Année de publication : 2008 Article en page(s) : p.526-534 Langues : Anglais (eng) Mots-clés : Conduct-disorder aggression delinquency oppositionality depression twin Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01834.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=386 [article] Heterogeneity in antisocial behaviours and comorbidity with depressed mood: a behavioural genetic approach [Texte imprimé et/ou numérique] / Richard ROWE, Auteur ; Barbara MAUGHAN, Auteur ; Thalia C. ELEY, Auteur ; Georgina M. HOSANG, Auteur ; Frühling V. RIJSDIJK, Auteur . - 2008 . - p.526-534. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 49-5 (May 2008) . - p.526-534 Mots-clés : Conduct-disorder aggression delinquency oppositionality depression twin Index. décimale : PER Périodiques En ligne : http://dx.doi.org/10.1111/j.1469-7610.2008.01834.x Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=386

I think, therefore I am: a twin study of attributional style in adolescents / Jennifer Y.F. LAU in Journal of Child Psychology and Psychiatry, 47-7 (July 2006)  

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A longitudinal twin study on the association between ADHD symptoms and reading / Corina U. GREVEN in Journal of Child Psychology and Psychiatry, 53-3 (March 2012)  

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Normative childhood repetitive routines and obsessive compulsive symptomatology in 6-year-old twins / Derek BOLTON in Journal of Child Psychology and Psychiatry, 50-9 (September 2009)  

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Parental criticism and adolescent internalising symptoms: using a Children-of-Twins design with power calculations to account for genetic influence / Yasmin I. AHMADZADEH in Journal of Child Psychology and Psychiatry, 63-5 (May 2022)  

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Reciprocal links between anxiety sensitivity and obsessive-compulsive symptoms in youth: a longitudinal twin study / G. KREBS in Journal of Child Psychology and Psychiatry, 61-9 (September 2020)  

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Research Review: How to interpret associations between polygenic scores, environmental risks, and phenotypes / Jean-Baptiste PINGAULT in Journal of Child Psychology and Psychiatry, 63-10 (October 2022)  

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Shared genetic influences on ADHD symptoms and very low-frequency EEG activity: a twin study / Charlotte TYE in Journal of Child Psychology and Psychiatry, 53-6 (June 2012)  

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