26 recherche sur le mot-clé 'heritability'

Heritability of autism spectrum disorders: a meta-analysis of twin studies / Beata TICK in Journal of Child Psychology and Psychiatry, 57-5 (May 2016)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 57-5 (May 2016) . - p.585-595 Titre : Heritability of autism spectrum disorders: a meta-analysis of twin studies Type de document : Texte imprimé et/ou numérique Auteurs : Beata TICK, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur ; Michael RUTTER, Auteur ; Frühling V. RIJSDIJK, Auteur Article en page(s) : p.585-595 Langues : Anglais (eng) Mots-clés : Autism spectrum disorders  meta-analysis  heritability  twin studies  DF extremes analysis Index. décimale : PER Périodiques Résumé : Background The etiology of Autism Spectrum Disorder (ASD) has been recently debated due to emerging findings on the importance of shared environmental influences. However, two recent twin studies do not support this and instead re-affirm strong genetic effects on the liability to ASD, a finding consistent with previous reports. This study conducts a systematic review and meta-analysis of all twin studies of ASD published to date and explores the etiology along the continuum of a quantitative measure of ASD. Methods A PubMed Central, Science Direct, Google Scholar, Web of Knowledge structured search conducted online, to identify all twin studies on ASD published to date. Thirteen primary twin studies were identified, seven were included in the meta-analysis by meeting Systematic Recruitment criterion; correction for selection and ascertainment strategies, and applied prevalences were assessed for these studies. In addition, a quantile DF extremes analysis was carried out on Childhood Autism Spectrum Test scores measured in a population sample of 6,413 twin pairs including affected twins. Results The meta-analysis correlations for monozygotic twins (MZ) were almost perfect at .98 (95% Confidence Interval, .96–.99). The dizygotic (DZ) correlation, however, was .53 (95% CI .44–.60) when ASD prevalence rate was set at 5% (in line with the Broad Phenotype of ASD) and increased to .67 (95% CI .61–.72) when applying a prevalence rate of 1%. The meta-analytic heritability estimates were substantial: 64–91%. Shared environmental effects became significant as the prevalence rate decreased from 5–1%: 07–35%. The DF analyses show that for the most part, there is no departure from linearity in heritability. Conclusions We demonstrate that: (a) ASD is due to strong genetic effects; (b) shared environmental effects become significant as a function of lower prevalence rate; (c) previously reported significant shared environmental influences are likely a statistical artefact of overinclusion of concordant DZ twins. En ligne : http://dx.doi.org/10.1111/jcpp.12499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=288 [article] Heritability of autism spectrum disorders: a meta-analysis of twin studies [Texte imprimé et/ou numérique] / Beata TICK, Auteur ; Patrick BOLTON, Auteur ; Francesca HAPPE, Auteur ; Michael RUTTER, Auteur ; Frühling V. RIJSDIJK, Auteur . - p.585-595. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 57-5 (May 2016) . - p.585-595 Mots-clés : Autism spectrum disorders  meta-analysis  heritability  twin studies  DF extremes analysis Index. décimale : PER Périodiques Résumé : Background The etiology of Autism Spectrum Disorder (ASD) has been recently debated due to emerging findings on the importance of shared environmental influences. However, two recent twin studies do not support this and instead re-affirm strong genetic effects on the liability to ASD, a finding consistent with previous reports. This study conducts a systematic review and meta-analysis of all twin studies of ASD published to date and explores the etiology along the continuum of a quantitative measure of ASD. Methods A PubMed Central, Science Direct, Google Scholar, Web of Knowledge structured search conducted online, to identify all twin studies on ASD published to date. Thirteen primary twin studies were identified, seven were included in the meta-analysis by meeting Systematic Recruitment criterion; correction for selection and ascertainment strategies, and applied prevalences were assessed for these studies. In addition, a quantile DF extremes analysis was carried out on Childhood Autism Spectrum Test scores measured in a population sample of 6,413 twin pairs including affected twins. Results The meta-analysis correlations for monozygotic twins (MZ) were almost perfect at .98 (95% Confidence Interval, .96–.99). The dizygotic (DZ) correlation, however, was .53 (95% CI .44–.60) when ASD prevalence rate was set at 5% (in line with the Broad Phenotype of ASD) and increased to .67 (95% CI .61–.72) when applying a prevalence rate of 1%. The meta-analytic heritability estimates were substantial: 64–91%. Shared environmental effects became significant as the prevalence rate decreased from 5–1%: 07–35%. The DF analyses show that for the most part, there is no departure from linearity in heritability. Conclusions We demonstrate that: (a) ASD is due to strong genetic effects; (b) shared environmental effects become significant as a function of lower prevalence rate; (c) previously reported significant shared environmental influences are likely a statistical artefact of overinclusion of concordant DZ twins. En ligne : http://dx.doi.org/10.1111/jcpp.12499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=288

Heritability of children's Secure Base Script Knowledge in middle childhood: a twin study with the Attachment Script Assessment / Annemieke M. WITTE ; Marinus H. VAN IJZENDOORN ; Marian J. BAKERMANS-KRANENBURG in Journal of Child Psychology and Psychiatry, 66-6 (June 2025)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 66-6 (June 2025) . - p.796-804 Titre : Heritability of children's Secure Base Script Knowledge in middle childhood: a twin study with the Attachment Script Assessment Type de document : Texte imprimé et/ou numérique Auteurs : Annemieke M. WITTE, Auteur ; Marinus H. VAN IJZENDOORN, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur Article en page(s) : p.796-804 Langues : Anglais (eng) Mots-clés : Attachment  heritability  ACE models  twins  Secure Base Script Knowledge Index. décimale : PER Périodiques Résumé : Background Are individual differences in attachment security inborn or shaped by the social environment? In infancy and early childhood, the evidence points to a substantial role of the environment, but a large twin study in early adolescence showed considerable heritability. Here we examined the twin heritability of attachment in middle childhood. We hypothesized that in middle childhood some heritability would emerge. Furthermore, we expected a role for cognitive and language abilities in explaining variance in attachment in middle childhood, partly related to the measurement of attachment, and we therefore examined associations with IQ. Methods This pre-registered study included 415 same-sex twin pairs (52% girls, 58% monozygotic) between 8 and 11?years old (M?=?9.59, SD?=?0.79). Participants were recruited from an experimental cohort-sequential study including two age-overlapping longitudinal cohorts. Secure Base Script Knowledge was assessed with the Middle Childhood Attachment Script Assessment . Zygosity of the twins was determined using DNA samples. In the younger cohort, cognitive development was assessed with the Dutch version of the Wechsler Preschool and Primary Scale of Intelligence . In the older cohort, the Dutch version of the Wechsler Intelligence Scale for Children was used . Results Significant additive heritability (38%) was found in the absence of a common environment component. This result diverges from findings in infancy and early childhood and aligns with the results in early adolescence. Conclusions The gene?environment correlation hypothesis suggesting that older children more actively shape their experiences in social contexts may offer a plausible explanation for the heritability of attachment in middle childhood. In middle childhood this mechanism might tip the balance toward a larger role for additive genetics. Larger longitudinal twin studies are needed to replicate the heritability of attachment after preschool age. En ligne : https://doi.org/10.1111/jcpp.14089 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=556 [article] Heritability of children's Secure Base Script Knowledge in middle childhood: a twin study with the Attachment Script Assessment [Texte imprimé et/ou numérique] / Annemieke M. WITTE, Auteur ; Marinus H. VAN IJZENDOORN, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur . - p.796-804. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 66-6 (June 2025) . - p.796-804 Mots-clés : Attachment  heritability  ACE models  twins  Secure Base Script Knowledge Index. décimale : PER Périodiques Résumé : Background Are individual differences in attachment security inborn or shaped by the social environment? In infancy and early childhood, the evidence points to a substantial role of the environment, but a large twin study in early adolescence showed considerable heritability. Here we examined the twin heritability of attachment in middle childhood. We hypothesized that in middle childhood some heritability would emerge. Furthermore, we expected a role for cognitive and language abilities in explaining variance in attachment in middle childhood, partly related to the measurement of attachment, and we therefore examined associations with IQ. Methods This pre-registered study included 415 same-sex twin pairs (52% girls, 58% monozygotic) between 8 and 11?years old (M?=?9.59, SD?=?0.79). Participants were recruited from an experimental cohort-sequential study including two age-overlapping longitudinal cohorts. Secure Base Script Knowledge was assessed with the Middle Childhood Attachment Script Assessment . Zygosity of the twins was determined using DNA samples. In the younger cohort, cognitive development was assessed with the Dutch version of the Wechsler Preschool and Primary Scale of Intelligence . In the older cohort, the Dutch version of the Wechsler Intelligence Scale for Children was used . Results Significant additive heritability (38%) was found in the absence of a common environment component. This result diverges from findings in infancy and early childhood and aligns with the results in early adolescence. Conclusions The gene?environment correlation hypothesis suggesting that older children more actively shape their experiences in social contexts may offer a plausible explanation for the heritability of attachment in middle childhood. In middle childhood this mechanism might tip the balance toward a larger role for additive genetics. Larger longitudinal twin studies are needed to replicate the heritability of attachment after preschool age. En ligne : https://doi.org/10.1111/jcpp.14089 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=556

Heritability of quantitative autism spectrum traits in adults: A family-based study / S. C. TAYLOR in Autism Research, 14-8 (August 2021)  

Public ISBD [article]  inAutism Research > 14-8 (August 2021) . - p.1543-1553 Titre : Heritability of quantitative autism spectrum traits in adults: A family-based study Type de document : Texte imprimé et/ou numérique Auteurs : S. C. TAYLOR, Auteur ; S. STEEMAN, Auteur ; Brielle N. GEHRINGER, Auteur ; H. C. DOW, Auteur ; A. LANGER, Auteur ; E. RAWOT, Auteur ; L. PEREZ, Auteur ; M. GOODMAN, Auteur ; Z. SMERNOFF, Auteur ; M. GREWAL, Auteur ; O. ESHRAGHI, Auteur ; Ashley A. PALLATHRA, Auteur ; C. OKSAS, Auteur ; M. MENDEZ, Auteur ; Ruben C. GUR, Auteur ; D. J. RADER, Auteur ; M. BUCAN, Auteur ; Laura ALMASY, Auteur ; Edward S. BRODKIN, Auteur Article en page(s) : p.1543-1553 Langues : Anglais (eng) Mots-clés : Adult  Autism Spectrum Disorder/genetics  Autistic Disorder  Executive Function  Humans  Phenotype  Surveys and Questionnaires  adult  autism spectrum disorder  family studies  heritability  phenotype  quantitative trait Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) comprises a multi-dimensional set of quantitative behavioral traits expressed along a continuum in autistic and neurotypical individuals. ASD diagnosis-a dichotomous trait-is known to be highly heritable and has been used as the phenotype for most ASD genetic studies. But less is known about the heritability of autism spectrum quantitative traits, especially in adults, an important prerequisite for gene discovery. We sought to measure the heritability of many autism-relevant quantitative traits in adults high in autism spectrum traits and their extended family members. Among adults high in autism spectrum traits (n = 158) and their extended family members (n = 245), we calculated univariate and bivariate heritability estimates for 19 autism spectrum traits across several behavioral domains. We found nearly all tested autism spectrum quantitative traits to be significantly heritable (h(2)  = 0.24-0.79), including overall ASD traits, restricted repetitive behaviors, broader autism phenotype traits, social anxiety, and executive functioning. The degree of shared heritability varied based on method and specificity of the assessment measure. We found high shared heritability for the self-report measures and for most of the informant-report measures, with little shared heritability among performance-based cognition tasks. These findings suggest that many autism spectrum quantitative traits would be good, feasible candidates for future genetics studies, allowing for an increase in the power of autism gene discovery. Our findings suggest that the degree of shared heritability between traits depends on the assessment method (self-report vs. informant-report vs. performance-based tasks), as well as trait-specificity. LAY SUMMARY: We found that the scores from questionnaires and tasks measuring different types of behaviors and abilities related to autism spectrum disorder (ASD) were heritable (strongly influenced by gene variants passed down through a family) among autistic adults and their family members. These findings mean that these scores can be used in future studies interested in identifying specific genes and gene variants that are associated with different behaviors and abilities related with ASD. En ligne : http://dx.doi.org/10.1002/aur.2571 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=449 [article] Heritability of quantitative autism spectrum traits in adults: A family-based study [Texte imprimé et/ou numérique] / S. C. TAYLOR, Auteur ; S. STEEMAN, Auteur ; Brielle N. GEHRINGER, Auteur ; H. C. DOW, Auteur ; A. LANGER, Auteur ; E. RAWOT, Auteur ; L. PEREZ, Auteur ; M. GOODMAN, Auteur ; Z. SMERNOFF, Auteur ; M. GREWAL, Auteur ; O. ESHRAGHI, Auteur ; Ashley A. PALLATHRA, Auteur ; C. OKSAS, Auteur ; M. MENDEZ, Auteur ; Ruben C. GUR, Auteur ; D. J. RADER, Auteur ; M. BUCAN, Auteur ; Laura ALMASY, Auteur ; Edward S. BRODKIN, Auteur . - p.1543-1553. Langues : Anglais (eng) in Autism Research > 14-8 (August 2021) . - p.1543-1553 Mots-clés : Adult  Autism Spectrum Disorder/genetics  Autistic Disorder  Executive Function  Humans  Phenotype  Surveys and Questionnaires  adult  autism spectrum disorder  family studies  heritability  phenotype  quantitative trait Index. décimale : PER Périodiques Résumé : Autism spectrum disorder (ASD) comprises a multi-dimensional set of quantitative behavioral traits expressed along a continuum in autistic and neurotypical individuals. ASD diagnosis-a dichotomous trait-is known to be highly heritable and has been used as the phenotype for most ASD genetic studies. But less is known about the heritability of autism spectrum quantitative traits, especially in adults, an important prerequisite for gene discovery. We sought to measure the heritability of many autism-relevant quantitative traits in adults high in autism spectrum traits and their extended family members. Among adults high in autism spectrum traits (n = 158) and their extended family members (n = 245), we calculated univariate and bivariate heritability estimates for 19 autism spectrum traits across several behavioral domains. We found nearly all tested autism spectrum quantitative traits to be significantly heritable (h(2)  = 0.24-0.79), including overall ASD traits, restricted repetitive behaviors, broader autism phenotype traits, social anxiety, and executive functioning. The degree of shared heritability varied based on method and specificity of the assessment measure. We found high shared heritability for the self-report measures and for most of the informant-report measures, with little shared heritability among performance-based cognition tasks. These findings suggest that many autism spectrum quantitative traits would be good, feasible candidates for future genetics studies, allowing for an increase in the power of autism gene discovery. Our findings suggest that the degree of shared heritability between traits depends on the assessment method (self-report vs. informant-report vs. performance-based tasks), as well as trait-specificity. LAY SUMMARY: We found that the scores from questionnaires and tasks measuring different types of behaviors and abilities related to autism spectrum disorder (ASD) were heritable (strongly influenced by gene variants passed down through a family) among autistic adults and their family members. These findings mean that these scores can be used in future studies interested in identifying specific genes and gene variants that are associated with different behaviors and abilities related with ASD. En ligne : http://dx.doi.org/10.1002/aur.2571 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=449

Heritability, stability, and prevalence of tonic and phasic irritability as indicators of disruptive mood dysregulation disorder / A. A. MOORE in Journal of Child Psychology and Psychiatry, 60-9 (September 2019)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 60-9 (September 2019) . - p.1032-1041 Titre : Heritability, stability, and prevalence of tonic and phasic irritability as indicators of disruptive mood dysregulation disorder Type de document : Texte imprimé et/ou numérique Auteurs : A. A. MOORE, Auteur ; D. M. LAPATO, Auteur ; Melissa A. BROTMAN, Auteur ; E. LEIBENLUFT, Auteur ; S. H. AGGEN, Auteur ; J. M. HETTEMA, Auteur ; T. P. YORK, Auteur ; J. L. SILBERG, Auteur ; R. ROBERSON-NAY, Auteur Article en page(s) : p.1032-1041 Langues : Anglais (eng) Mots-clés : Disruptive behavior  emotional dysregulation  heritability  mood disorder  twins Index. décimale : PER Périodiques Résumé : BACKGROUND: Little is known about genetic and environmental influences on the components of disruptive mood dysregulation disorder (DMDD), tonic irritability (i.e., irritable mood) and phasic irritability (i.e., temper outbursts). This study examined prevalence, stability, and heritability of tonic irritability, phasic irritability, and a DMDD proxy (pDMDD) based on DSM-5 criteria. METHODS: pDMDD was derived using data from clinical interviews of parents and their twins (N = 1,431 twin pairs), ages 8-17, participating in Waves 1 and 2 of the Virginia Twin Study of Adolescent Behavioral Development. Biometrical modeling was used to compare a common pathway model (CPM) and an independent pathway model (IPM), and heritability estimates were obtained for pDMDD using the symptoms of irritable mood (tonic irritability; DMDD Criterion D), intense temper outbursts (phasic irritability; DMDD Criterion A), and frequent temper outbursts (phasic irritability; DMDD Criterion C). RESULTS: Lifetime prevalence of pDMDD was 7.46%. The stability of DMDD symptoms and the pDMDD phenotype across approximately one year were moderate (.30-.69). A CPM was a better fit to the data than an IPM. Phasic irritability loaded strongly onto the pDMDD latent factor (.89-.96) whereas tonic irritability did not (.28). Genetic influences accounted for approximately 59% of the variance in the latent pDMDD phenotype, with the remaining 41% of the variance due to unique environmental effects. The heritability of tonic irritability (54%) was slightly lower than that of frequent and intense temper (components of phasic irritability; 61% and 63%, respectively). CONCLUSIONS: Compared to tonic irritability, phasic irritability appears to be slightly more stable and heritable, as well as a stronger indicator of the latent factor. Furthermore, environmental experiences appear to play a substantial role in the development of irritability and DMDD, and researchers should seek to elucidate these mechanisms in future work. En ligne : http://dx.doi.org/10.1111/jcpp.13062 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=405 [article] Heritability, stability, and prevalence of tonic and phasic irritability as indicators of disruptive mood dysregulation disorder [Texte imprimé et/ou numérique] / A. A. MOORE, Auteur ; D. M. LAPATO, Auteur ; Melissa A. BROTMAN, Auteur ; E. LEIBENLUFT, Auteur ; S. H. AGGEN, Auteur ; J. M. HETTEMA, Auteur ; T. P. YORK, Auteur ; J. L. SILBERG, Auteur ; R. ROBERSON-NAY, Auteur . - p.1032-1041. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 60-9 (September 2019) . - p.1032-1041 Mots-clés : Disruptive behavior  emotional dysregulation  heritability  mood disorder  twins Index. décimale : PER Périodiques Résumé : BACKGROUND: Little is known about genetic and environmental influences on the components of disruptive mood dysregulation disorder (DMDD), tonic irritability (i.e., irritable mood) and phasic irritability (i.e., temper outbursts). This study examined prevalence, stability, and heritability of tonic irritability, phasic irritability, and a DMDD proxy (pDMDD) based on DSM-5 criteria. METHODS: pDMDD was derived using data from clinical interviews of parents and their twins (N = 1,431 twin pairs), ages 8-17, participating in Waves 1 and 2 of the Virginia Twin Study of Adolescent Behavioral Development. Biometrical modeling was used to compare a common pathway model (CPM) and an independent pathway model (IPM), and heritability estimates were obtained for pDMDD using the symptoms of irritable mood (tonic irritability; DMDD Criterion D), intense temper outbursts (phasic irritability; DMDD Criterion A), and frequent temper outbursts (phasic irritability; DMDD Criterion C). RESULTS: Lifetime prevalence of pDMDD was 7.46%. The stability of DMDD symptoms and the pDMDD phenotype across approximately one year were moderate (.30-.69). A CPM was a better fit to the data than an IPM. Phasic irritability loaded strongly onto the pDMDD latent factor (.89-.96) whereas tonic irritability did not (.28). Genetic influences accounted for approximately 59% of the variance in the latent pDMDD phenotype, with the remaining 41% of the variance due to unique environmental effects. The heritability of tonic irritability (54%) was slightly lower than that of frequent and intense temper (components of phasic irritability; 61% and 63%, respectively). CONCLUSIONS: Compared to tonic irritability, phasic irritability appears to be slightly more stable and heritable, as well as a stronger indicator of the latent factor. Furthermore, environmental experiences appear to play a substantial role in the development of irritability and DMDD, and researchers should seek to elucidate these mechanisms in future work. En ligne : http://dx.doi.org/10.1111/jcpp.13062 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=405

Editorial Perspective: Why is there such a mismatch between traditional heritability estimates and molecular genetic findings for behavioural traits? / Anita THAPAR in Journal of Child Psychology and Psychiatry, 55-10 (October 2014)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 55-10 (October 2014) . - p.1088-1091 Titre : Editorial Perspective: Why is there such a mismatch between traditional heritability estimates and molecular genetic findings for behavioural traits? Type de document : Texte imprimé et/ou numérique Auteurs : Anita THAPAR, Auteur ; Gordon HAROLD, Auteur Article en page(s) : p.1088-1091 Langues : Anglais (eng) Mots-clés : Molecular genetic studies  behaviour genetic studies  psychiatric disorders  behavioural traits  heritability  developmental psychopathology Index. décimale : PER Périodiques Résumé : The puzzle of the disparity between molecular- and traditional behaviour genetic study findings has prompted widespread discussion. Fundamental questions have been raised across the whole field of complex genetic traits as well as for behavioural traits. We consider explanations for recent findings and discuss what they mean for the field of developmental psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.12294 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=238 [article] Editorial Perspective: Why is there such a mismatch between traditional heritability estimates and molecular genetic findings for behavioural traits? [Texte imprimé et/ou numérique] / Anita THAPAR, Auteur ; Gordon HAROLD, Auteur . - p.1088-1091. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 55-10 (October 2014) . - p.1088-1091 Mots-clés : Molecular genetic studies  behaviour genetic studies  psychiatric disorders  behavioural traits  heritability  developmental psychopathology Index. décimale : PER Périodiques Résumé : The puzzle of the disparity between molecular- and traditional behaviour genetic study findings has prompted widespread discussion. Fundamental questions have been raised across the whole field of complex genetic traits as well as for behavioural traits. We consider explanations for recent findings and discuss what they mean for the field of developmental psychopathology. En ligne : http://dx.doi.org/10.1111/jcpp.12294 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=238

Single nucleotide polymorphism heritability and differential patterns of genetic overlap between inattention and four neurocognitive factors in youth / Lauren MICALIZZI in Development and Psychopathology, 33-1 (February 2021)  

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Childhood peer network characteristics: genetic influences and links with early mental health trajectories / Eszter SZEKELY in Journal of Child Psychology and Psychiatry, 57-6 (June 2016)  

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Development of ADHD symptoms in preschool children: Genetic and environmental contributions / E. M. EILERTSEN in Development and Psychopathology, 31-4 (October 2019)  

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Discriminant value of repetitive behaviors in families with autism spectrum disorder and obsessional compulsive disorder probands / A. LEFEBVRE in Autism Research, 14-11 (November 2021)  

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Editorial: Translational genetics of child psychopathology: a distant dream? / Barbara MAUGHAN in Journal of Child Psychology and Psychiatry, 55-10 (October 2014)  

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