Advancing paternal age and offspring violent offending: A sibling-comparison study / Ralf KUJA-HALKOLA in Development and Psychopathology, 24-3 (August 2012)  

Public ISBD [article]  inDevelopment and Psychopathology > 24-3 (August 2012) . - p.739-53 Titre : Advancing paternal age and offspring violent offending: A sibling-comparison study Type de document : Texte imprimé et/ou numérique Auteurs : Ralf KUJA-HALKOLA, Auteur ; Yudi PAWITAN, Auteur ; Brian M. D'ONOFRIO, Auteur ; Niklas LANGSTROM, Auteur ; Paul LICHTENSTEIN, Auteur Année de publication : 2012 Article en page(s) : p.739-53 Langues : Anglais (eng) Index. décimale : PER Périodiques Résumé : Children born to older fathers are at higher risk to develop severe psychopathology (e.g., schizophrenia and bipolar disorder), possibly because of increased de novo mutations during spermatogenesis with older paternal age. Because severe psychopathology is correlated with antisocial behavior, we examined possible associations between advancing paternal age and offspring violent offending. Interlinked Swedish national registers provided information on fathers' age at childbirth and violent criminal convictions in all offspring born from 1958 to 1979 (N = 2,359,921). We used ever committing a violent crime and number of violent crimes as indices of violent offending. The data included information on multiple levels; we compared differentially exposed siblings in within-family analyses to rigorously test causal influences. In the entire population, advancing paternal age predicted offspring violent crime according to both indices. Congruent with a causal effect, this association remained for rates of violent crime in within-family analyses. However, in within-family analyses, we found no association with ever committing a violent crime, suggesting that factors shared by siblings (genes and environment) confounded this association. Life-course persistent criminality has been proposed to have a partly biological etiology; our results agree with a stronger biological effect (i.e., de novo mutations) on persistent violent offending. En ligne : http://dx.doi.org/10.1017/S095457941200034X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=177 [article] Advancing paternal age and offspring violent offending: A sibling-comparison study [Texte imprimé et/ou numérique] / Ralf KUJA-HALKOLA, Auteur ; Yudi PAWITAN, Auteur ; Brian M. D'ONOFRIO, Auteur ; Niklas LANGSTROM, Auteur ; Paul LICHTENSTEIN, Auteur . - 2012 . - p.739-53. Langues : Anglais (eng) in Development and Psychopathology > 24-3 (August 2012) . - p.739-53 Index. décimale : PER Périodiques Résumé : Children born to older fathers are at higher risk to develop severe psychopathology (e.g., schizophrenia and bipolar disorder), possibly because of increased de novo mutations during spermatogenesis with older paternal age. Because severe psychopathology is correlated with antisocial behavior, we examined possible associations between advancing paternal age and offspring violent offending. Interlinked Swedish national registers provided information on fathers' age at childbirth and violent criminal convictions in all offspring born from 1958 to 1979 (N = 2,359,921). We used ever committing a violent crime and number of violent crimes as indices of violent offending. The data included information on multiple levels; we compared differentially exposed siblings in within-family analyses to rigorously test causal influences. In the entire population, advancing paternal age predicted offspring violent crime according to both indices. Congruent with a causal effect, this association remained for rates of violent crime in within-family analyses. However, in within-family analyses, we found no association with ever committing a violent crime, suggesting that factors shared by siblings (genes and environment) confounded this association. Life-course persistent criminality has been proposed to have a partly biological etiology; our results agree with a stronger biological effect (i.e., de novo mutations) on persistent violent offending. En ligne : http://dx.doi.org/10.1017/S095457941200034X Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=177

Childhood neurodevelopmental disorders and risk of coercive sexual victimization in childhood and adolescence – a population-based prospective twin study / Vide OHLSSON GOTBY in Journal of Child Psychology and Psychiatry, 59-9 (September 2018)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 59-9 (September 2018) . - p.957-965 Titre : Childhood neurodevelopmental disorders and risk of coercive sexual victimization in childhood and adolescence – a population-based prospective twin study Type de document : Texte imprimé et/ou numérique Auteurs : Vide OHLSSON GOTBY, Auteur ; Paul LICHTENSTEIN, Auteur ; Niklas LANGSTROM, Auteur ; Erik PETTERSSON, Auteur Article en page(s) : p.957-965 Langues : Anglais (eng) Mots-clés : Attention-deficit/Hyperactivity disorder  autism spectrum disorder  neuropsychiatric disorders  sexual abuse  twins Index. décimale : PER Périodiques Résumé : Background Autism spectrum disorder (ASD), Attention-deficit/Hyperactivity disorder (ADHD), and other related neurodevelopmental disorders (NDDs) have, in some previous studies, been shown to increase the risk of being sexually victimized. However, no studies have examined whether the association is driven by a general NDD phenotype versus specific diagnoses, nor the etiology of the association. Method Using a genetically informative, prospective design, we examined the association between ASD and ADHD in childhood and coercive sexual victimization up to age 18. A total of 4,500 children participating in the Child and Adolescent Twin Study in Sweden (CATSS) were rated by their parents on NDDs at age 9 or 12 years, and self-reported at age 18 on lifetime experiences of coercive sexual touching and/or coercive sex. First, we regressed sexual victimization on the NDDs. Second, we regressed sexual victimization on general and specific NDD symptoms identified via a bifactor model. Third, we decomposed the observed associations into genetic and environmental parts. Results In females, ASD was associated with an almost threefolded increased risk of coercive sexual victimization, and ADHD with a doubled risk. In males, the risk associated with ASD and ADHD was of the same magnitude but not significant. When controlling for overall NDD symptom load ASD or ADHD, no longer uniquely predicted coercive sexual victimization. The association between the NDD general factor and coercive sexual victimization was due to shared genetics. Conclusions General NDD symptom load, rather than specific ASD or ADHD symptoms, seems to be a moderate vulnerability factor for coercive sexual victimization. We speculate that an evocative gene?environment correlation might account for this observation, such that sexual perpetrators actively target NDD individuals. En ligne : https://doi.org/10.1111/jcpp.12884 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368 [article] Childhood neurodevelopmental disorders and risk of coercive sexual victimization in childhood and adolescence – a population-based prospective twin study [Texte imprimé et/ou numérique] / Vide OHLSSON GOTBY, Auteur ; Paul LICHTENSTEIN, Auteur ; Niklas LANGSTROM, Auteur ; Erik PETTERSSON, Auteur . - p.957-965. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 59-9 (September 2018) . - p.957-965 Mots-clés : Attention-deficit/Hyperactivity disorder  autism spectrum disorder  neuropsychiatric disorders  sexual abuse  twins Index. décimale : PER Périodiques Résumé : Background Autism spectrum disorder (ASD), Attention-deficit/Hyperactivity disorder (ADHD), and other related neurodevelopmental disorders (NDDs) have, in some previous studies, been shown to increase the risk of being sexually victimized. However, no studies have examined whether the association is driven by a general NDD phenotype versus specific diagnoses, nor the etiology of the association. Method Using a genetically informative, prospective design, we examined the association between ASD and ADHD in childhood and coercive sexual victimization up to age 18. A total of 4,500 children participating in the Child and Adolescent Twin Study in Sweden (CATSS) were rated by their parents on NDDs at age 9 or 12 years, and self-reported at age 18 on lifetime experiences of coercive sexual touching and/or coercive sex. First, we regressed sexual victimization on the NDDs. Second, we regressed sexual victimization on general and specific NDD symptoms identified via a bifactor model. Third, we decomposed the observed associations into genetic and environmental parts. Results In females, ASD was associated with an almost threefolded increased risk of coercive sexual victimization, and ADHD with a doubled risk. In males, the risk associated with ASD and ADHD was of the same magnitude but not significant. When controlling for overall NDD symptom load ASD or ADHD, no longer uniquely predicted coercive sexual victimization. The association between the NDD general factor and coercive sexual victimization was due to shared genetics. Conclusions General NDD symptom load, rather than specific ASD or ADHD symptoms, seems to be a moderate vulnerability factor for coercive sexual victimization. We speculate that an evocative gene?environment correlation might account for this observation, such that sexual perpetrators actively target NDD individuals. En ligne : https://doi.org/10.1111/jcpp.12884 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=368

Childhood Neurodevelopmental Disorders and Violent Criminality: A Sibling Control Study / Sebastian LUNDSTROM in Journal of Autism and Developmental Disorders, 44-11 (November 2014)  

Public ISBD [article]  inJournal of Autism and Developmental Disorders > 44-11 (November 2014) . - p.2707-2716 Titre : Childhood Neurodevelopmental Disorders and Violent Criminality: A Sibling Control Study Type de document : Texte imprimé et/ou numérique Auteurs : Sebastian LUNDSTROM, Auteur ; Mats FORSMAN, Auteur ; Henrik LARSSON, Auteur ; Nora KEREKES, Auteur ; Eva SERLACHIUS, Auteur ; Niklas LANGSTROM, Auteur ; Paul LICHTENSTEIN, Auteur Article en page(s) : p.2707-2716 Langues : Anglais (eng) Mots-clés : Autism spectrum disorders  Attention deficit hyperactivity disorder  Neurodevelopmental disorders  Criminality  Familial confounding Index. décimale : PER Périodiques Résumé : The longitudinal relationship between attention deficit hyperactivity disorder (ADHD) and violent criminality has been extensively documented, while long-term effects of autism spectrum disorders (ASDs), tic disorders (TDs), and obsessive compulsive disorder (OCD) on criminality have been scarcely studied. Using population-based registers of all child and adolescent mental health services in Stockholm, we identified 3,391 children, born 1984–1994, with neurodevelopmental disorders, and compared their risk for subsequent violent criminality with matched controls. Individuals with ADHD or TDs were at elevated risk of committing violent crimes, no such association could be seen for ASDs or OCD. ADHD and TDs are risk factors for subsequent violent criminality, while ASDs and OCD are not associated with violent criminality. En ligne : http://dx.doi.org/10.1007/s10803-013-1873-0 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=241 [article] Childhood Neurodevelopmental Disorders and Violent Criminality: A Sibling Control Study [Texte imprimé et/ou numérique] / Sebastian LUNDSTROM, Auteur ; Mats FORSMAN, Auteur ; Henrik LARSSON, Auteur ; Nora KEREKES, Auteur ; Eva SERLACHIUS, Auteur ; Niklas LANGSTROM, Auteur ; Paul LICHTENSTEIN, Auteur . - p.2707-2716. Langues : Anglais (eng) in Journal of Autism and Developmental Disorders > 44-11 (November 2014) . - p.2707-2716 Mots-clés : Autism spectrum disorders  Attention deficit hyperactivity disorder  Neurodevelopmental disorders  Criminality  Familial confounding Index. décimale : PER Périodiques Résumé : The longitudinal relationship between attention deficit hyperactivity disorder (ADHD) and violent criminality has been extensively documented, while long-term effects of autism spectrum disorders (ASDs), tic disorders (TDs), and obsessive compulsive disorder (OCD) on criminality have been scarcely studied. Using population-based registers of all child and adolescent mental health services in Stockholm, we identified 3,391 children, born 1984–1994, with neurodevelopmental disorders, and compared their risk for subsequent violent criminality with matched controls. Individuals with ADHD or TDs were at elevated risk of committing violent crimes, no such association could be seen for ASDs or OCD. ADHD and TDs are risk factors for subsequent violent criminality, while ASDs and OCD are not associated with violent criminality. En ligne : http://dx.doi.org/10.1007/s10803-013-1873-0 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=241

Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study / Henrik LARSSON in Journal of Child Psychology and Psychiatry, 55-5 (May 2014)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 55-5 (May 2014) . - p.428-435 Titre : Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study Type de document : Texte imprimé et/ou numérique Auteurs : Henrik LARSSON, Auteur ; Amir SARIASLAN, Auteur ; Niklas LANGSTROM, Auteur ; Brian M. D'ONOFRIO, Auteur ; Paul LICHTENSTEIN, Auteur Article en page(s) : p.428-435 Mots-clés : ADHD  family income  childhood  causality  quasi-experimental approaches Index. décimale : PER Périodiques Résumé : Background Studies have found negative associations between socioeconomic position and attention deficit/hyperactivity disorder (ADHD), but it remains unclear if this association is causal. The aim of this study was to determine the extent to which the association between family income in early childhood and subsequent ADHD depends on measured and unmeasured selection factors. Methods A total of 811,803 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Diagnosis of ADHD was assessed via the Swedish national Patient Register and the Swedish Prescribed Drug Register. Annual family income during offspring's first 5 years in life was collected prospectively from the Swedish Integrated Database for Labour Market Research and divided into quartiles by (lower) family disposable income. We predicted ADHD from family income while controlling for covariates and also comparing differently exposed cousins and siblings to control for unmeasured familial confounding. Results The crude analyses suggested that children exposed to lower income levels were at increased risk for ADHD (HRQuartile1 = 2.52; 95% CI, 2.42–2.63; HRQuartile2 = 1.52; 95% CI, 1.45–1.58; HRQuartile3 = 1.20; 95% CI, 1.14–1.15). This dose-dependent association decreased after adjustment for measured covariates (HRQuartile1 = 2.09; 95% CI, 2.00–2.19; HRQuartile2 = 1.36; 95% CI, 1.30–1.42; HRQuartile3 = 1.13; 95% CI, 1.08–1.18). Although the association was attenuated in cousin comparisons (HRQuartile1 = 1.61; 95% CI, 1.40–1.84; HRQuartile2 = 1.28; 95% CI, 1.12–1.45; HRQuartile3 = 1.14; 95% CI, 1.01–1.28) and sibling comparison models (HRQuartile1 = 1.37; 95% CI, 1.07–1.75; HRQuartile2 = 1.37; 95% CI, 1.12–1.68; HRQuartile3 = 1.23; 95% CI, 1.04–1.45), it remained statistically significant across all levels of decreased disposable family income. Conclusions Our results indicated that low family income in early childhood was associated with increased likelihood of ADHD. The link remained even after controlling for unmeasured selection factors, highlighting family income in early childhood as a marker of causal factors for ADHD. En ligne : http://dx.doi.org/10.1111/jcpp.12140 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=231 [article] Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study [Texte imprimé et/ou numérique] / Henrik LARSSON, Auteur ; Amir SARIASLAN, Auteur ; Niklas LANGSTROM, Auteur ; Brian M. D'ONOFRIO, Auteur ; Paul LICHTENSTEIN, Auteur . - p.428-435. in Journal of Child Psychology and Psychiatry > 55-5 (May 2014) . - p.428-435 Mots-clés : ADHD  family income  childhood  causality  quasi-experimental approaches Index. décimale : PER Périodiques Résumé : Background Studies have found negative associations between socioeconomic position and attention deficit/hyperactivity disorder (ADHD), but it remains unclear if this association is causal. The aim of this study was to determine the extent to which the association between family income in early childhood and subsequent ADHD depends on measured and unmeasured selection factors. Methods A total of 811,803 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Diagnosis of ADHD was assessed via the Swedish national Patient Register and the Swedish Prescribed Drug Register. Annual family income during offspring's first 5 years in life was collected prospectively from the Swedish Integrated Database for Labour Market Research and divided into quartiles by (lower) family disposable income. We predicted ADHD from family income while controlling for covariates and also comparing differently exposed cousins and siblings to control for unmeasured familial confounding. Results The crude analyses suggested that children exposed to lower income levels were at increased risk for ADHD (HRQuartile1 = 2.52; 95% CI, 2.42–2.63; HRQuartile2 = 1.52; 95% CI, 1.45–1.58; HRQuartile3 = 1.20; 95% CI, 1.14–1.15). This dose-dependent association decreased after adjustment for measured covariates (HRQuartile1 = 2.09; 95% CI, 2.00–2.19; HRQuartile2 = 1.36; 95% CI, 1.30–1.42; HRQuartile3 = 1.13; 95% CI, 1.08–1.18). Although the association was attenuated in cousin comparisons (HRQuartile1 = 1.61; 95% CI, 1.40–1.84; HRQuartile2 = 1.28; 95% CI, 1.12–1.45; HRQuartile3 = 1.14; 95% CI, 1.01–1.28) and sibling comparison models (HRQuartile1 = 1.37; 95% CI, 1.07–1.75; HRQuartile2 = 1.37; 95% CI, 1.12–1.68; HRQuartile3 = 1.23; 95% CI, 1.04–1.45), it remained statistically significant across all levels of decreased disposable family income. Conclusions Our results indicated that low family income in early childhood was associated with increased likelihood of ADHD. The link remained even after controlling for unmeasured selection factors, highlighting family income in early childhood as a marker of causal factors for ADHD. En ligne : http://dx.doi.org/10.1111/jcpp.12140 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=231

Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study / Agnieszka BUTWICKA in Journal of Autism and Developmental Disorders, 47-1 (January 2017)  

Public ISBD [article]  inJournal of Autism and Developmental Disorders > 47-1 (January 2017) . - p.80-89 Titre : Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study Type de document : Texte imprimé et/ou numérique Auteurs : Agnieszka BUTWICKA, Auteur ; Niklas LANGSTROM, Auteur ; Henrik LARSSON, Auteur ; Sebastian LUNDSTROM, Auteur ; Eva SERLACHIUS, Auteur ; Catarina ALMQVIST, Auteur ; Louise FRISÉN, Auteur ; Paul LICHTENSTEIN, Auteur Article en page(s) : p.80-89 Langues : Anglais (eng) Mots-clés : Autism spectrum disorder  Addiction  ADHD  Intellectual disability  ICD Index. décimale : PER Périodiques Résumé : Despite limited and ambiguous empirical data, substance use-related problems have been assumed to be rare among patients with autism spectrum disorders (ASD). Using Swedish population-based registers we identified 26,986 individuals diagnosed with ASD during 1973–2009, and their 96,557 non-ASD relatives. ASD, without diagnosed comorbidity of attention deficit hyperactivity disorder (ADHD) or intellectual disability, was related to a doubled risk of substance use-related problems. The risk of substance use-related problems was the highest among individuals with ASD and ADHD. Further, risks of substance use-related problems were increased among full siblings of ASD probands, half-siblings and parents. We conclude that ASD is a risk factor for substance use-related problems. The elevated risks among relatives of probands with ASD suggest shared familial (genetic and/or shared environmental) liability. En ligne : http://dx.doi.org/10.1007/s10803-016-2914-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298 [article] Increased Risk for Substance Use-Related Problems in Autism Spectrum Disorders: A Population-Based Cohort Study [Texte imprimé et/ou numérique] / Agnieszka BUTWICKA, Auteur ; Niklas LANGSTROM, Auteur ; Henrik LARSSON, Auteur ; Sebastian LUNDSTROM, Auteur ; Eva SERLACHIUS, Auteur ; Catarina ALMQVIST, Auteur ; Louise FRISÉN, Auteur ; Paul LICHTENSTEIN, Auteur . - p.80-89. Langues : Anglais (eng) in Journal of Autism and Developmental Disorders > 47-1 (January 2017) . - p.80-89 Mots-clés : Autism spectrum disorder  Addiction  ADHD  Intellectual disability  ICD Index. décimale : PER Périodiques Résumé : Despite limited and ambiguous empirical data, substance use-related problems have been assumed to be rare among patients with autism spectrum disorders (ASD). Using Swedish population-based registers we identified 26,986 individuals diagnosed with ASD during 1973–2009, and their 96,557 non-ASD relatives. ASD, without diagnosed comorbidity of attention deficit hyperactivity disorder (ADHD) or intellectual disability, was related to a doubled risk of substance use-related problems. The risk of substance use-related problems was the highest among individuals with ASD and ADHD. Further, risks of substance use-related problems were increased among full siblings of ASD probands, half-siblings and parents. We conclude that ASD is a risk factor for substance use-related problems. The elevated risks among relatives of probands with ASD suggest shared familial (genetic and/or shared environmental) liability. En ligne : http://dx.doi.org/10.1007/s10803-016-2914-2 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=298

Maternal age at first birth and offspring criminality: Using the children of twins design to test causal hypotheses / Claire A. COYNE in Development and Psychopathology, 25-1 (February 2013)  

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Relative immaturity and ADHD: findings from nationwide registers, parent- and self-reports / Linda HALLDNER in Journal of Child Psychology and Psychiatry, 55-8 (August 2014)  

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Stimulant ADHD medication and risk for substance abuse / Zheng CHANG in Journal of Child Psychology and Psychiatry, 55-8 (August 2014)  

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