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Editorial: The passage of time / E. FOMBONNE in Journal of Child Psychology and Psychiatry, 60-7 (July 2019)
[article]
Titre : Editorial: The passage of time Type de document : Texte imprimé et/ou numérique Auteurs : E. FOMBONNE, Auteur Article en page(s) : p.713-715 Langues : Anglais (eng) Mots-clés : Longitudinal causality methodology secular changes Index. décimale : PER Périodiques Résumé : In this editorial, the author reflects on changes that occurred in the quality of research on developmental psychopathology over the last 35 years. This is illustrated in the increased quality of nine longitudinal studies that are included in the current issue of JCPP. Using approaches that capitalize on the passage of time, ranging from 28 days to 40 years across investigations, these studies employed multiple levels of analysis, used sophisticated statistical methods to control for confounding factors, included measurement at both the biological, cognitive, and behavioral levels, and collectively provided results that allow improved assessment of causality. En ligne : http://dx.doi.org/10.1111/jcpp.13090 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401
in Journal of Child Psychology and Psychiatry > 60-7 (July 2019) . - p.713-715[article] Editorial: The passage of time [Texte imprimé et/ou numérique] / E. FOMBONNE, Auteur . - p.713-715.
Langues : Anglais (eng)
in Journal of Child Psychology and Psychiatry > 60-7 (July 2019) . - p.713-715
Mots-clés : Longitudinal causality methodology secular changes Index. décimale : PER Périodiques Résumé : In this editorial, the author reflects on changes that occurred in the quality of research on developmental psychopathology over the last 35 years. This is illustrated in the increased quality of nine longitudinal studies that are included in the current issue of JCPP. Using approaches that capitalize on the passage of time, ranging from 28 days to 40 years across investigations, these studies employed multiple levels of analysis, used sophisticated statistical methods to control for confounding factors, included measurement at both the biological, cognitive, and behavioral levels, and collectively provided results that allow improved assessment of causality. En ligne : http://dx.doi.org/10.1111/jcpp.13090 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=401 Prenatal maternal infection and risk for autism in offspring: A meta-analysis / N. TIOLECO in Autism Research, 14-6 (June 2021)
[article]
Titre : Prenatal maternal infection and risk for autism in offspring: A meta-analysis Type de document : Texte imprimé et/ou numérique Auteurs : N. TIOLECO, Auteur ; A. E. SILBERMAN, Auteur ; K. STRATIGOS, Auteur ; Sharmila BANERJEE-BASU, Auteur ; M. N. SPANN, Auteur ; A. H. WHITAKER, Auteur ; J. Blake TURNER, Auteur Article en page(s) : p.1296-1316 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder Autistic Disorder/epidemiology Causality Female Humans Pregnancy Pregnancy Complications Prenatal Exposure Delayed Effects/epidemiology Risk Factors autism infections pregnancy prenatal risk factors Index. décimale : PER Périodiques Résumé : While prenatal maternal infection has received attention as a preventable and treatable risk factor for autism, findings have been inconsistent. This paper presents the results of a meta-analysis to determine whether the weight of the evidence supports such an association. Studies with a categorical diagnosis of autism as the outcome and an assessment of its association with prenatal maternal infection or fever (or the data necessary to compute this association) were included. A total of 36 studies met these criteria. Two independent reviewers extracted data on study design, methods of assessment, type of infectious agent, site of infection, trimester of exposure, definition of autism, and effect size. Analyses demonstrated a statistically significant association of maternal infection/fever with autism in offspring (OR = 1.32; 95% CI = 1.20-1.46). Adjustment for evident publication bias slightly weakened this association. There was little variation in effect sizes across agent or site of infection. Small differences across trimester of exposure were not statistically significant. There was some evidence that recall bias associated with status on the outcome variable leads to differential misclassification of exposure status. Nonetheless, the overall association is only modestly reduced when studies potentially contaminated by such bias are removed. Although causality has not been firmly established, these findings suggest maternal infection during pregnancy confers an increase in risk for autism in offspring. Given the prevalence of this risk factor, it is possible that the incidence of autism would be reduced by 12%-17% if maternal infections could be prevented or safely treated in a timely manner. LAY SUMMARY: This study is a meta-analysis of the association of maternal infection during pregnancy and subsequent autism in offspring. In combining the results from 36 studies of this association we find that a significant relationship is present. The association does not vary much across the types of infections or when they occur during pregnancy. We conclude that the incidence of autism could be substantially reduced if maternal infections could be prevented or safely treated in a timely manner. En ligne : http://dx.doi.org/10.1002/aur.2499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=449
in Autism Research > 14-6 (June 2021) . - p.1296-1316[article] Prenatal maternal infection and risk for autism in offspring: A meta-analysis [Texte imprimé et/ou numérique] / N. TIOLECO, Auteur ; A. E. SILBERMAN, Auteur ; K. STRATIGOS, Auteur ; Sharmila BANERJEE-BASU, Auteur ; M. N. SPANN, Auteur ; A. H. WHITAKER, Auteur ; J. Blake TURNER, Auteur . - p.1296-1316.
Langues : Anglais (eng)
in Autism Research > 14-6 (June 2021) . - p.1296-1316
Mots-clés : Autism Spectrum Disorder Autistic Disorder/epidemiology Causality Female Humans Pregnancy Pregnancy Complications Prenatal Exposure Delayed Effects/epidemiology Risk Factors autism infections pregnancy prenatal risk factors Index. décimale : PER Périodiques Résumé : While prenatal maternal infection has received attention as a preventable and treatable risk factor for autism, findings have been inconsistent. This paper presents the results of a meta-analysis to determine whether the weight of the evidence supports such an association. Studies with a categorical diagnosis of autism as the outcome and an assessment of its association with prenatal maternal infection or fever (or the data necessary to compute this association) were included. A total of 36 studies met these criteria. Two independent reviewers extracted data on study design, methods of assessment, type of infectious agent, site of infection, trimester of exposure, definition of autism, and effect size. Analyses demonstrated a statistically significant association of maternal infection/fever with autism in offspring (OR = 1.32; 95% CI = 1.20-1.46). Adjustment for evident publication bias slightly weakened this association. There was little variation in effect sizes across agent or site of infection. Small differences across trimester of exposure were not statistically significant. There was some evidence that recall bias associated with status on the outcome variable leads to differential misclassification of exposure status. Nonetheless, the overall association is only modestly reduced when studies potentially contaminated by such bias are removed. Although causality has not been firmly established, these findings suggest maternal infection during pregnancy confers an increase in risk for autism in offspring. Given the prevalence of this risk factor, it is possible that the incidence of autism would be reduced by 12%-17% if maternal infections could be prevented or safely treated in a timely manner. LAY SUMMARY: This study is a meta-analysis of the association of maternal infection during pregnancy and subsequent autism in offspring. In combining the results from 36 studies of this association we find that a significant relationship is present. The association does not vary much across the types of infections or when they occur during pregnancy. We conclude that the incidence of autism could be substantially reduced if maternal infections could be prevented or safely treated in a timely manner. En ligne : http://dx.doi.org/10.1002/aur.2499 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=449 Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study / Henrik LARSSON in Journal of Child Psychology and Psychiatry, 55-5 (May 2014)
[article]
Titre : Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study Type de document : Texte imprimé et/ou numérique Auteurs : Henrik LARSSON, Auteur ; Amir SARIASLAN, Auteur ; Niklas LANGSTROM, Auteur ; Brian M. D'ONOFRIO, Auteur ; Paul LICHTENSTEIN, Auteur Article en page(s) : p.428-435 Mots-clés : ADHD family income childhood causality quasi-experimental approaches Index. décimale : PER Périodiques Résumé : Background Studies have found negative associations between socioeconomic position and attention deficit/hyperactivity disorder (ADHD), but it remains unclear if this association is causal. The aim of this study was to determine the extent to which the association between family income in early childhood and subsequent ADHD depends on measured and unmeasured selection factors. Methods A total of 811,803 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Diagnosis of ADHD was assessed via the Swedish national Patient Register and the Swedish Prescribed Drug Register. Annual family income during offspring's first 5 years in life was collected prospectively from the Swedish Integrated Database for Labour Market Research and divided into quartiles by (lower) family disposable income. We predicted ADHD from family income while controlling for covariates and also comparing differently exposed cousins and siblings to control for unmeasured familial confounding. Results The crude analyses suggested that children exposed to lower income levels were at increased risk for ADHD (HRQuartile1 = 2.52; 95% CI, 2.42–2.63; HRQuartile2 = 1.52; 95% CI, 1.45–1.58; HRQuartile3 = 1.20; 95% CI, 1.14–1.15). This dose-dependent association decreased after adjustment for measured covariates (HRQuartile1 = 2.09; 95% CI, 2.00–2.19; HRQuartile2 = 1.36; 95% CI, 1.30–1.42; HRQuartile3 = 1.13; 95% CI, 1.08–1.18). Although the association was attenuated in cousin comparisons (HRQuartile1 = 1.61; 95% CI, 1.40–1.84; HRQuartile2 = 1.28; 95% CI, 1.12–1.45; HRQuartile3 = 1.14; 95% CI, 1.01–1.28) and sibling comparison models (HRQuartile1 = 1.37; 95% CI, 1.07–1.75; HRQuartile2 = 1.37; 95% CI, 1.12–1.68; HRQuartile3 = 1.23; 95% CI, 1.04–1.45), it remained statistically significant across all levels of decreased disposable family income. Conclusions Our results indicated that low family income in early childhood was associated with increased likelihood of ADHD. The link remained even after controlling for unmeasured selection factors, highlighting family income in early childhood as a marker of causal factors for ADHD. En ligne : http://dx.doi.org/10.1111/jcpp.12140 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=231
in Journal of Child Psychology and Psychiatry > 55-5 (May 2014) . - p.428-435[article] Family income in early childhood and subsequent attention deficit/hyperactivity disorder: a quasi-experimental study [Texte imprimé et/ou numérique] / Henrik LARSSON, Auteur ; Amir SARIASLAN, Auteur ; Niklas LANGSTROM, Auteur ; Brian M. D'ONOFRIO, Auteur ; Paul LICHTENSTEIN, Auteur . - p.428-435.
in Journal of Child Psychology and Psychiatry > 55-5 (May 2014) . - p.428-435
Mots-clés : ADHD family income childhood causality quasi-experimental approaches Index. décimale : PER Périodiques Résumé : Background Studies have found negative associations between socioeconomic position and attention deficit/hyperactivity disorder (ADHD), but it remains unclear if this association is causal. The aim of this study was to determine the extent to which the association between family income in early childhood and subsequent ADHD depends on measured and unmeasured selection factors. Methods A total of 811,803 individuals born in Sweden between 1992 and 2000 were included in this nationwide population-based cohort study. Diagnosis of ADHD was assessed via the Swedish national Patient Register and the Swedish Prescribed Drug Register. Annual family income during offspring's first 5 years in life was collected prospectively from the Swedish Integrated Database for Labour Market Research and divided into quartiles by (lower) family disposable income. We predicted ADHD from family income while controlling for covariates and also comparing differently exposed cousins and siblings to control for unmeasured familial confounding. Results The crude analyses suggested that children exposed to lower income levels were at increased risk for ADHD (HRQuartile1 = 2.52; 95% CI, 2.42–2.63; HRQuartile2 = 1.52; 95% CI, 1.45–1.58; HRQuartile3 = 1.20; 95% CI, 1.14–1.15). This dose-dependent association decreased after adjustment for measured covariates (HRQuartile1 = 2.09; 95% CI, 2.00–2.19; HRQuartile2 = 1.36; 95% CI, 1.30–1.42; HRQuartile3 = 1.13; 95% CI, 1.08–1.18). Although the association was attenuated in cousin comparisons (HRQuartile1 = 1.61; 95% CI, 1.40–1.84; HRQuartile2 = 1.28; 95% CI, 1.12–1.45; HRQuartile3 = 1.14; 95% CI, 1.01–1.28) and sibling comparison models (HRQuartile1 = 1.37; 95% CI, 1.07–1.75; HRQuartile2 = 1.37; 95% CI, 1.12–1.68; HRQuartile3 = 1.23; 95% CI, 1.04–1.45), it remained statistically significant across all levels of decreased disposable family income. Conclusions Our results indicated that low family income in early childhood was associated with increased likelihood of ADHD. The link remained even after controlling for unmeasured selection factors, highlighting family income in early childhood as a marker of causal factors for ADHD. En ligne : http://dx.doi.org/10.1111/jcpp.12140 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=231 Improving parenting, child attachment, and externalizing behaviors: Meta-analysis of the first 25 randomized controlled trials on the effects of Video-feedback Intervention to promote Positive Parenting and Sensitive Discipline / Marinus H. VAN IJZENDOORN in Development and Psychopathology, 35-1 (February 2023)
[article]
Titre : Improving parenting, child attachment, and externalizing behaviors: Meta-analysis of the first 25 randomized controlled trials on the effects of Video-feedback Intervention to promote Positive Parenting and Sensitive Discipline Type de document : Texte imprimé et/ou numérique Auteurs : Marinus H. VAN IJZENDOORN, Auteur ; Carlo SCHUENGEL, Auteur ; Qiang WANG, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur Article en page(s) : p.241-256 Langues : Anglais (eng) Mots-clés : causality individual participant data meta-analysis parenting attitudes risk of bias strange situation procedure Index. décimale : PER Périodiques Résumé : Improving parenting, child attachment, and externalizing behaviors: Meta-analysis of the first 25 randomized controlled trials on the effects of Video-feedback Intervention to promote Positive Parenting and Sensitive Discipline (VIPP-SD). VIPP-SD combines support of parental sensitive responsiveness with coaching parents in sensitive limit setting. Here, we present meta-analyses of 25 RCTs conducted with more than 2,000 parents and caregivers. Parents or children had various risks. We examined its effectiveness in promoting parental cognitions and behavior regarding sensitive parenting and limit setting, in promoting secure child-parent attachment, and reducing externalizing child behavior. Web of Science, MEDLINE, PubMed, and recent reviews were searched for relevant trials (until May 10, 2021). Multilevel meta-analysis with META, METAFOR, and DMETAR in R took account of the 3-level structure of the datasets (studies, participants, measures). The meta-analyses showed substantial combined effect sizes for parenting behavior (r = .18) and attitudes (r = .16), and for child attachment security (r = .23), but not for child externalizing behavior (r = .07). In the subset of studies examining effects on both parenting and attachment, the association between effect sizes for parenting and for attachment amounted to r = .48. We consider the way in which VIPP-SD uses video-feedback an active intervention component. Whether VIPP-SD indeed stimulates secure attachment through enhanced positive parenting remains an outstanding question for further experimental study and individual participant data meta-analysis. En ligne : https://doi.org/10.1017/S0954579421001462 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499
in Development and Psychopathology > 35-1 (February 2023) . - p.241-256[article] Improving parenting, child attachment, and externalizing behaviors: Meta-analysis of the first 25 randomized controlled trials on the effects of Video-feedback Intervention to promote Positive Parenting and Sensitive Discipline [Texte imprimé et/ou numérique] / Marinus H. VAN IJZENDOORN, Auteur ; Carlo SCHUENGEL, Auteur ; Qiang WANG, Auteur ; Marian J. BAKERMANS-KRANENBURG, Auteur . - p.241-256.
Langues : Anglais (eng)
in Development and Psychopathology > 35-1 (February 2023) . - p.241-256
Mots-clés : causality individual participant data meta-analysis parenting attitudes risk of bias strange situation procedure Index. décimale : PER Périodiques Résumé : Improving parenting, child attachment, and externalizing behaviors: Meta-analysis of the first 25 randomized controlled trials on the effects of Video-feedback Intervention to promote Positive Parenting and Sensitive Discipline (VIPP-SD). VIPP-SD combines support of parental sensitive responsiveness with coaching parents in sensitive limit setting. Here, we present meta-analyses of 25 RCTs conducted with more than 2,000 parents and caregivers. Parents or children had various risks. We examined its effectiveness in promoting parental cognitions and behavior regarding sensitive parenting and limit setting, in promoting secure child-parent attachment, and reducing externalizing child behavior. Web of Science, MEDLINE, PubMed, and recent reviews were searched for relevant trials (until May 10, 2021). Multilevel meta-analysis with META, METAFOR, and DMETAR in R took account of the 3-level structure of the datasets (studies, participants, measures). The meta-analyses showed substantial combined effect sizes for parenting behavior (r = .18) and attitudes (r = .16), and for child attachment security (r = .23), but not for child externalizing behavior (r = .07). In the subset of studies examining effects on both parenting and attachment, the association between effect sizes for parenting and for attachment amounted to r = .48. We consider the way in which VIPP-SD uses video-feedback an active intervention component. Whether VIPP-SD indeed stimulates secure attachment through enhanced positive parenting remains an outstanding question for further experimental study and individual participant data meta-analysis. En ligne : https://doi.org/10.1017/S0954579421001462 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=499 Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder in Autism, 25-7 (October 2021)
[article]
Titre : Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder Type de document : Texte imprimé et/ou numérique Article en page(s) : p.1946-1959 Langues : Anglais (eng) Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=451
in Autism > 25-7 (October 2021) . - p.1946-1959[article] Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder [Texte imprimé et/ou numérique] . - p.1946-1959.
Langues : Anglais (eng)
in Autism > 25-7 (October 2021) . - p.1946-1959
Mots-clés : Autism Spectrum Disorder/epidemiology/etiology Causality Family Female Humans Pregnancy Prenatal Exposure Delayed Effects/epidemiology/etiology Risk Factors Smoking/adverse effects autism spectrum disorders environmental factors risk factor epidemiology to the research presented in this manuscript. Index. décimale : PER Périodiques Résumé : What is Already Known about This Subject: Genetics, (including de novo mutations), environmental factors (including toxic exposures), and their interactions impact autism spectrum disorder etiology. Paternal smoking is a candidate risk for autism spectrum disorder due to biological plausibility, high prevalence, and potential intervention.What This Study Adds: This original study and its replication confirms that paternal factors can substantially contribute to autism spectrum disorder risk for their offspring. It specifically indicates that paternal smoking both before and during pregnancy contributes significantly to autism spectrum disorder risk.Implications for practice, research, or policy: Smoking prevention, especially in pregnancy planning, may decrease autism spectrum disorder risk in offspring. En ligne : http://dx.doi.org/10.1177/13623613211007319 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=451 Prenatal exposure to paternal smoking and likelihood for autism spectrum disorder / Bora KIM in Autism, 26-7 (October 2022)
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