How nonshared environmental factors come to correlate with heredity / Christopher R. BEAM in Development and Psychopathology, 34-1 (February 2022)  

Public ISBD [article]  inDevelopment and Psychopathology > 34-1 (February 2022) . - p.321-333 Titre : How nonshared environmental factors come to correlate with heredity Type de document : texte imprimé Auteurs : Christopher R. BEAM, Auteur ; Patrizia PEZZOLI, Auteur ; Jane MENDLE, Auteur ; S. Alexandra BURT, Auteur ; Michael C. NEALE, Auteur ; Steven M. BOKER, Auteur ; Pamela K. KEEL, Auteur ; Kelly L. KLUMP, Auteur Article en page(s) : p.321-333 Langues : Anglais (eng) Mots-clés : affect  developmental behavioral genetics  gene?environment interplay  longitudinal modeling  mood Index. décimale : PER Périodiques Résumé : Conventional longitudinal behavioral genetic models estimate the relative contribution of genetic and environmental factors to stability and change of traits and behaviors. Longitudinal models rarely explain the processes that generate observed differences between genetically and socially related individuals. We propose that exchanges between individuals and their environments (i.e., phenotype?environment effects) can explain the emergence of observed differences over time. Phenotype?environment models, however, would require violation of the independence assumption of standard behavioral genetic models; that is, uncorrelated genetic and environmental factors. We review how specification of phenotype?environment effects contributes to understanding observed changes in genetic variability over time and longitudinal correlations among nonshared environmental factors. We then provide an example using 30 days of positive and negative affect scores from an all-female sample of twins. Results demonstrate that the phenotype?environment effects explain how heritability estimates fluctuate as well as how nonshared environmental factors persist over time. We discuss possible mechanisms underlying change in gene?environment correlation over time, the advantages and challenges of including gene?environment correlation in longitudinal twin models, and recommendations for future research. En ligne : http://dx.doi.org/10.1017/s0954579420001017 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474 [article] How nonshared environmental factors come to correlate with heredity [texte imprimé] / Christopher R. BEAM, Auteur ; Patrizia PEZZOLI, Auteur ; Jane MENDLE, Auteur ; S. Alexandra BURT, Auteur ; Michael C. NEALE, Auteur ; Steven M. BOKER, Auteur ; Pamela K. KEEL, Auteur ; Kelly L. KLUMP, Auteur . - p.321-333. Langues : Anglais (eng) in Development and Psychopathology > 34-1 (February 2022) . - p.321-333 Mots-clés : affect  developmental behavioral genetics  gene?environment interplay  longitudinal modeling  mood Index. décimale : PER Périodiques Résumé : Conventional longitudinal behavioral genetic models estimate the relative contribution of genetic and environmental factors to stability and change of traits and behaviors. Longitudinal models rarely explain the processes that generate observed differences between genetically and socially related individuals. We propose that exchanges between individuals and their environments (i.e., phenotype?environment effects) can explain the emergence of observed differences over time. Phenotype?environment models, however, would require violation of the independence assumption of standard behavioral genetic models; that is, uncorrelated genetic and environmental factors. We review how specification of phenotype?environment effects contributes to understanding observed changes in genetic variability over time and longitudinal correlations among nonshared environmental factors. We then provide an example using 30 days of positive and negative affect scores from an all-female sample of twins. Results demonstrate that the phenotype?environment effects explain how heritability estimates fluctuate as well as how nonshared environmental factors persist over time. We discuss possible mechanisms underlying change in gene?environment correlation over time, the advantages and challenges of including gene?environment correlation in longitudinal twin models, and recommendations for future research. En ligne : http://dx.doi.org/10.1017/s0954579420001017 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474

The nature and nurture of primary and secondary callous–unemotional traits: evidence from two independent twin samples / Rachel C. TOMLINSON in Journal of Child Psychology and Psychiatry, 67-6 (June 2026)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 67-6 (June 2026) . - p.988-997 Titre : The nature and nurture of primary and secondary callous–unemotional traits: evidence from two independent twin samples Type de document : texte imprimé Auteurs : Rachel C. TOMLINSON, Auteur ; Patrizia PEZZOLI, Auteur ; Essi VIDING, Auteur ; Stephane A. DE BRITO, Auteur ; Kelly L. KLUMP, Auteur ; S. Alexandra BURT, Auteur ; Luke W. HYDE, Auteur Article en page(s) : p.988-997 Langues : Anglais (eng) Mots-clés : Callous–unemotional traits  anxiety  behavior genetics  twin study  genotype-environment interaction Index. décimale : PER Périodiques Résumé : Background Callous?unemotional (CU) traits identify youth with more severe and chronic trajectories of conduct problems. However, the etiology of CU traits may be heterogeneous, undermining the search for effective treatments. The level of co-occurring anxiety has been used to identify ?primary? (lower anxiety) versus ?secondary? (higher anxiety) variants of CU traits. The primary variant has been hypothesized to emerge from strong genetic influence and secondary variants as an adaptation to adversity, such as exposure to childhood maltreatment. However, little research has tested this hypothesis directly. Methods We examined whether anxiety moderates the etiology of CU traits to determine whether this phenotypic feature can help distinguish CU traits with stronger genetic or environmental risk. In two population-based twin cohorts (initial sample: N?=?1,196, aged 6?11, oversampled for exposure to neighborhood disadvantage; follow-up sample: N?=?13,486, age 7), we used genotype-by-environment interaction twin modeling to examine if parent-reported child anxiety moderated the etiology of concurrent parent-reported child CU traits. Results Anxiety moderated the etiology of CU traits across both samples, such that nonshared environmental influences increased as anxiety increased. Additionally, in the larger sample, genetic influences decreased with increasing anxiety. Conclusions These findings support theories suggesting that co-occurring anxiety may distinguish CU traits with different origins: CU traits with higher anxiety appear more influenced by nonshared environmental factors?potentially including adversity?and may show weaker genetic influence. Assessing for co-occurring child anxiety is likely important for diagnosing and personalizing treatments among children with CU traits. En ligne : https://doi.org/10.1111/jcpp.70107 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=587 [article] The nature and nurture of primary and secondary callous–unemotional traits: evidence from two independent twin samples [texte imprimé] / Rachel C. TOMLINSON, Auteur ; Patrizia PEZZOLI, Auteur ; Essi VIDING, Auteur ; Stephane A. DE BRITO, Auteur ; Kelly L. KLUMP, Auteur ; S. Alexandra BURT, Auteur ; Luke W. HYDE, Auteur . - p.988-997. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 67-6 (June 2026) . - p.988-997 Mots-clés : Callous–unemotional traits  anxiety  behavior genetics  twin study  genotype-environment interaction Index. décimale : PER Périodiques Résumé : Background Callous?unemotional (CU) traits identify youth with more severe and chronic trajectories of conduct problems. However, the etiology of CU traits may be heterogeneous, undermining the search for effective treatments. The level of co-occurring anxiety has been used to identify ?primary? (lower anxiety) versus ?secondary? (higher anxiety) variants of CU traits. The primary variant has been hypothesized to emerge from strong genetic influence and secondary variants as an adaptation to adversity, such as exposure to childhood maltreatment. However, little research has tested this hypothesis directly. Methods We examined whether anxiety moderates the etiology of CU traits to determine whether this phenotypic feature can help distinguish CU traits with stronger genetic or environmental risk. In two population-based twin cohorts (initial sample: N?=?1,196, aged 6?11, oversampled for exposure to neighborhood disadvantage; follow-up sample: N?=?13,486, age 7), we used genotype-by-environment interaction twin modeling to examine if parent-reported child anxiety moderated the etiology of concurrent parent-reported child CU traits. Results Anxiety moderated the etiology of CU traits across both samples, such that nonshared environmental influences increased as anxiety increased. Additionally, in the larger sample, genetic influences decreased with increasing anxiety. Conclusions These findings support theories suggesting that co-occurring anxiety may distinguish CU traits with different origins: CU traits with higher anxiety appear more influenced by nonshared environmental factors?potentially including adversity?and may show weaker genetic influence. Assessing for co-occurring child anxiety is likely important for diagnosing and personalizing treatments among children with CU traits. En ligne : https://doi.org/10.1111/jcpp.70107 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=587

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