17 recherche sur le mot-clé 'twin study'

Effect of co-twin gender on neurodevelopmental symptoms: a twin register study / J. M. ERIKSSON in Molecular Autism, 7 (2016)  

Public ISBD [article]  inMolecular Autism > 7 (2016) . - 8p. Titre : Effect of co-twin gender on neurodevelopmental symptoms: a twin register study Type de document : Texte imprimé et/ou numérique Auteurs : J. M. ERIKSSON, Auteur ; S. LUNDSTRÖM, Auteur ; P. LICHTENSTEIN, Auteur ; Susanne BEJEROT, Auteur ; E. ERIKSSON, Auteur Article en page(s) : 8p. Langues : Anglais (eng) Mots-clés : Attention Deficit Disorder with Hyperactivity/genetics/physiopathology  Autism Spectrum Disorder/genetics/physiopathology  Child  Comorbidity  Diseases in Twins  Female  Follow-Up Studies  Gender Identity  Humans  Interview, Psychological  Male  Neurodevelopmental Disorders/genetics/physiopathology  Parents  Pregnancy  Prenatal Exposure Delayed Effects  Sex Characteristics  Stereotyped Behavior  Sweden/epidemiology  Testosterone/physiology  Tic Disorders/genetics/physiopathology  Twins, Dizygotic/psychology  Asperger syndrome  Attention-deficit hyperactivity disorders  Autistic disorder  Symptom assessment  Twin study Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) are neurodevelopmental disorders thought to have both genetic and environmental causes. It has been hypothesized that exposure to elevated levels of prenatal testosterone is associated with elevated traits of ASD and ADHD. Assuming that testosterone levels from a dizygotic male twin fetus may lead to enhanced testosterone exposure of its co-twins, we aimed to test the prenatal testosterone hypothesis by comparing same-sex with opposite-sex dizygotic twins with respect to neurodevelopmental symptoms. METHODS: Neuropsychiatric traits were assessed in a population-based twin cohort from the Child and Adolescent Twin Study in Sweden (CATSS). Parental interviews were conducted for 16,312 dizygotic twins, 9 and 12 years old, with the Autism-Tics, ADHD, and other Comorbidities inventory (A-TAC). RESULTS: Girls with a female co-twin had an increased risk of reaching the cut-off score for ADHD compared with girls with a male co-twin. Both boys and girls with a female co-twin displayed a larger number of traits related to attention deficit and repetitive and stereotyped behaviors than those with a male twin. In girls, this also extended to social interaction and the combined measures for ASD and ADHD, however, with small effect sizes. CONCLUSIONS: Our results are reverse to what would have been expected from the prenatal testosterone hypothesis but consistent with a previous study of ASD and ADHD traits in dizygotic twins. The seemingly protective effect for girls of having a twin brother may be an effect of parent report bias, but may also be an unexpected effect of sharing the intrauterine environment with a male co-twin. En ligne : http://dx.doi.org/10.1186/s13229-016-0074-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=328 [article] Effect of co-twin gender on neurodevelopmental symptoms: a twin register study [Texte imprimé et/ou numérique] / J. M. ERIKSSON, Auteur ; S. LUNDSTRÖM, Auteur ; P. LICHTENSTEIN, Auteur ; Susanne BEJEROT, Auteur ; E. ERIKSSON, Auteur . - 8p. Langues : Anglais (eng) in Molecular Autism > 7 (2016) . - 8p. Mots-clés : Attention Deficit Disorder with Hyperactivity/genetics/physiopathology  Autism Spectrum Disorder/genetics/physiopathology  Child  Comorbidity  Diseases in Twins  Female  Follow-Up Studies  Gender Identity  Humans  Interview, Psychological  Male  Neurodevelopmental Disorders/genetics/physiopathology  Parents  Pregnancy  Prenatal Exposure Delayed Effects  Sex Characteristics  Stereotyped Behavior  Sweden/epidemiology  Testosterone/physiology  Tic Disorders/genetics/physiopathology  Twins, Dizygotic/psychology  Asperger syndrome  Attention-deficit hyperactivity disorders  Autistic disorder  Symptom assessment  Twin study Index. décimale : PER Périodiques Résumé : BACKGROUND: Autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) are neurodevelopmental disorders thought to have both genetic and environmental causes. It has been hypothesized that exposure to elevated levels of prenatal testosterone is associated with elevated traits of ASD and ADHD. Assuming that testosterone levels from a dizygotic male twin fetus may lead to enhanced testosterone exposure of its co-twins, we aimed to test the prenatal testosterone hypothesis by comparing same-sex with opposite-sex dizygotic twins with respect to neurodevelopmental symptoms. METHODS: Neuropsychiatric traits were assessed in a population-based twin cohort from the Child and Adolescent Twin Study in Sweden (CATSS). Parental interviews were conducted for 16,312 dizygotic twins, 9 and 12 years old, with the Autism-Tics, ADHD, and other Comorbidities inventory (A-TAC). RESULTS: Girls with a female co-twin had an increased risk of reaching the cut-off score for ADHD compared with girls with a male co-twin. Both boys and girls with a female co-twin displayed a larger number of traits related to attention deficit and repetitive and stereotyped behaviors than those with a male twin. In girls, this also extended to social interaction and the combined measures for ASD and ADHD, however, with small effect sizes. CONCLUSIONS: Our results are reverse to what would have been expected from the prenatal testosterone hypothesis but consistent with a previous study of ASD and ADHD traits in dizygotic twins. The seemingly protective effect for girls of having a twin brother may be an effect of parent report bias, but may also be an unexpected effect of sharing the intrauterine environment with a male co-twin. En ligne : http://dx.doi.org/10.1186/s13229-016-0074-z Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=328

Etiological links between autism and difficulties in initiating and maintaining sleep: a familial co-aggregation and twin study / M. J. TAYLOR in Journal of Child Psychology and Psychiatry, 63-3 (March 2022)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 63-3 (March 2022) . - p.315-323 Titre : Etiological links between autism and difficulties in initiating and maintaining sleep: a familial co-aggregation and twin study Type de document : Texte imprimé et/ou numérique Auteurs : M. J. TAYLOR, Auteur ; H. LARSSON, Auteur ; S. LUNDSTRÖM, Auteur ; P. LICHTENSTEIN, Auteur ; Agnieszka BUTWICKA, Auteur Article en page(s) : p.315-323 Langues : Anglais (eng) Mots-clés : Autism  comorbidity  genetics  sleep  twin study Index. décimale : PER Périodiques Résumé : BACKGROUND: Difficulties initiating and maintaining sleep (DIMS) are frequent features of autism, yet little is known about why these conditions co-occur. One possibility is that they share etiological factors, yet this hypothesis remains to be tested using quantitative genetic designs. We thus investigated etiological links between autism and DIMS using familial co-aggregation and twin methods. METHODS: Twins, siblings, half-siblings, and cousins of 50,097 individuals with autism were identified from Swedish population registries. Their risk of DIMS, defined through diagnoses of insomnia and/or melatonin prescriptions, was then estimated. Twin analyses conducted on 15,279 child and adolescent twin pairs investigated etiological links between DIMS and ASD. RESULTS: 22.8% of autistic individuals had DIMS. Monozygotic co-twins of individuals with autism were most at risk of DIMS compared to the reference group (OR?=?6.6 [2.5-17.4]), followed by dizygotic co-twins (OR?=?2.6 [1.5-4.5]) and full siblings (OR?=?2.5 [2.4-2.6]). Half-siblings and cousins of individuals with autism were least likely to have DIMS relative to the reference group (OR range?=?1.3-1.5). Twin analyses estimated a correlation of 0.57 (0.53-0.61) between autism and DIMS, with a genetic correlation of 0.62 (0.60-0.68). These overlapping genetic factors explained 94% of the covariance between these conditions. Autistic traits also showed genetic overlap with DIMS. CONCLUSIONS: Our results suggest that shared genetic mechanisms underlie autism and DIMS, which may lead them to co-occur. Untangling the etiological overlap between these conditions has potential to assist in understanding the etiology of each condition, as well as their associated outcomes. En ligne : http://dx.doi.org/10.1111/jcpp.13473 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457 [article] Etiological links between autism and difficulties in initiating and maintaining sleep: a familial co-aggregation and twin study [Texte imprimé et/ou numérique] / M. J. TAYLOR, Auteur ; H. LARSSON, Auteur ; S. LUNDSTRÖM, Auteur ; P. LICHTENSTEIN, Auteur ; Agnieszka BUTWICKA, Auteur . - p.315-323. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 63-3 (March 2022) . - p.315-323 Mots-clés : Autism  comorbidity  genetics  sleep  twin study Index. décimale : PER Périodiques Résumé : BACKGROUND: Difficulties initiating and maintaining sleep (DIMS) are frequent features of autism, yet little is known about why these conditions co-occur. One possibility is that they share etiological factors, yet this hypothesis remains to be tested using quantitative genetic designs. We thus investigated etiological links between autism and DIMS using familial co-aggregation and twin methods. METHODS: Twins, siblings, half-siblings, and cousins of 50,097 individuals with autism were identified from Swedish population registries. Their risk of DIMS, defined through diagnoses of insomnia and/or melatonin prescriptions, was then estimated. Twin analyses conducted on 15,279 child and adolescent twin pairs investigated etiological links between DIMS and ASD. RESULTS: 22.8% of autistic individuals had DIMS. Monozygotic co-twins of individuals with autism were most at risk of DIMS compared to the reference group (OR?=?6.6 [2.5-17.4]), followed by dizygotic co-twins (OR?=?2.6 [1.5-4.5]) and full siblings (OR?=?2.5 [2.4-2.6]). Half-siblings and cousins of individuals with autism were least likely to have DIMS relative to the reference group (OR range?=?1.3-1.5). Twin analyses estimated a correlation of 0.57 (0.53-0.61) between autism and DIMS, with a genetic correlation of 0.62 (0.60-0.68). These overlapping genetic factors explained 94% of the covariance between these conditions. Autistic traits also showed genetic overlap with DIMS. CONCLUSIONS: Our results suggest that shared genetic mechanisms underlie autism and DIMS, which may lead them to co-occur. Untangling the etiological overlap between these conditions has potential to assist in understanding the etiology of each condition, as well as their associated outcomes. En ligne : http://dx.doi.org/10.1111/jcpp.13473 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=457

Evidence for shared genetic risk between ADHD symptoms and reduced mathematics ability: a twin study / Corina U. GREVEN in Journal of Child Psychology and Psychiatry, 55-1 (January 2014)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 55-1 (January 2014) . - p.39-48 Titre : Evidence for shared genetic risk between ADHD symptoms and reduced mathematics ability: a twin study Type de document : Texte imprimé et/ou numérique Auteurs : Corina U. GREVEN, Auteur ; Yulia KOVAS, Auteur ; Erik G. WILLCUTT, Auteur ; Stephen A. PETRILL, Auteur ; Robert PLOMIN, Auteur Article en page(s) : p.39-48 Langues : Anglais (eng) Mots-clés : ADHD  mathematics  twin study  genetics  reading  general cognitive ability Index. décimale : PER Périodiques Résumé : Background Attention-deficit/hyperactivity disorder (ADHD) symptoms and mathematics ability are associated, but little is known about the genetic and environmental influences underlying this association. Methods Data came from more than 6,000 twelve-year-old twin pairs from the UK population-representative Twins Early Development Study. Parents rated each twin's behaviour using a DSM-IV-based 18-item questionnaire of inattentive and hyperactive–impulsive ADHD symptoms. Mathematics tests based on the UK National Curriculum were completed by each twin. The twins also completed standardised tests of reading and general cognitive ability. Multivariate twin model fitting was applied. Results Inattentive and hyperactive–impulsive ADHD symptoms were highly heritable (67% and 73% respectively). Mathematics ability was moderately heritable (46%). Mathematics ability and inattentiveness showed a significantly greater phenotypic correlation (rp = ?.26) and genetic correlation (rA = ?.41) than mathematics ability and hyperactivity–impulsivity (rp = ?.18; rA = ?.22). The genetic correlation between inattentiveness and mathematics ability was largely independent from hyperactivity-impulsivity, and was only partially accounted for by genetic influences related to reading and general cognitive ability. Conclusions Results revealed the novel finding that mathematics ability shows significantly stronger phenotypic and genetic associations with inattentiveness than with hyperactivity–impulsivity. Genetic associations between inattentiveness and mathematics ability could only partially be accounted for by hyperactivity–impulsivity, reading and general cognitive ability. Results suggest that mathematics ability is associated with ADHD symptoms largely because it shares genetic risk factors with inattentiveness, and provide further evidence for considering inattentiveness and hyperactivity–impulsivity separately. DNA markers for ADHD symptoms (especially inattentiveness) may also be candidate risk factors for mathematics ability and vice versa. En ligne : http://dx.doi.org/10.1111/jcpp.12090 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=220 [article] Evidence for shared genetic risk between ADHD symptoms and reduced mathematics ability: a twin study [Texte imprimé et/ou numérique] / Corina U. GREVEN, Auteur ; Yulia KOVAS, Auteur ; Erik G. WILLCUTT, Auteur ; Stephen A. PETRILL, Auteur ; Robert PLOMIN, Auteur . - p.39-48. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 55-1 (January 2014) . - p.39-48 Mots-clés : ADHD  mathematics  twin study  genetics  reading  general cognitive ability Index. décimale : PER Périodiques Résumé : Background Attention-deficit/hyperactivity disorder (ADHD) symptoms and mathematics ability are associated, but little is known about the genetic and environmental influences underlying this association. Methods Data came from more than 6,000 twelve-year-old twin pairs from the UK population-representative Twins Early Development Study. Parents rated each twin's behaviour using a DSM-IV-based 18-item questionnaire of inattentive and hyperactive–impulsive ADHD symptoms. Mathematics tests based on the UK National Curriculum were completed by each twin. The twins also completed standardised tests of reading and general cognitive ability. Multivariate twin model fitting was applied. Results Inattentive and hyperactive–impulsive ADHD symptoms were highly heritable (67% and 73% respectively). Mathematics ability was moderately heritable (46%). Mathematics ability and inattentiveness showed a significantly greater phenotypic correlation (rp = ?.26) and genetic correlation (rA = ?.41) than mathematics ability and hyperactivity–impulsivity (rp = ?.18; rA = ?.22). The genetic correlation between inattentiveness and mathematics ability was largely independent from hyperactivity-impulsivity, and was only partially accounted for by genetic influences related to reading and general cognitive ability. Conclusions Results revealed the novel finding that mathematics ability shows significantly stronger phenotypic and genetic associations with inattentiveness than with hyperactivity–impulsivity. Genetic associations between inattentiveness and mathematics ability could only partially be accounted for by hyperactivity–impulsivity, reading and general cognitive ability. Results suggest that mathematics ability is associated with ADHD symptoms largely because it shares genetic risk factors with inattentiveness, and provide further evidence for considering inattentiveness and hyperactivity–impulsivity separately. DNA markers for ADHD symptoms (especially inattentiveness) may also be candidate risk factors for mathematics ability and vice versa. En ligne : http://dx.doi.org/10.1111/jcpp.12090 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=220

Genetic and environmental contributions to anxiety among Chinese children and adolescents – a multi-informant twin study / Jie CHEN in Journal of Child Psychology and Psychiatry, 56-5 (May 2015)  

Public ISBD [article]  inJournal of Child Psychology and Psychiatry > 56-5 (May 2015) . - p.586-594 Titre : Genetic and environmental contributions to anxiety among Chinese children and adolescents – a multi-informant twin study Type de document : Texte imprimé et/ou numérique Auteurs : Jie CHEN, Auteur ; Jing YU, Auteur ; Xinying LI, Auteur ; Jianxin ZHANG, Auteur Article en page(s) : p.586-594 Langues : Anglais (eng) Mots-clés : Anxiety  Chinese children and adolescents  genetic and environmental influence  heritability  twin study Index. décimale : PER Périodiques Résumé : Background Child and adolescent anxiety has become a major public health concern in China, but little was known about the etiology of anxiety in Chinese children and adolescents. The present study aimed to investigate genetic and environmental influences on trait anxiety among Chinese children and adolescents. Rater, sex, and age differences on these estimates were also examined. Methods Self-reported and parent-reported child's trait anxiety was collected from 1,104 pairs of same-sex twins aged 9–18 years. Genetic models were fitted to data from each informant to determine the genetic (A), shared (C), and non-shared environmental (E) influences on trait anxiety. Results The parameter estimates and 95% confidence intervals (CI) of A, C, E on self-reported trait anxiety were 50% [30%, 60%], 5% [0%, 24%], 45% [40%, 49%]. For parent-reported data, the corresponding parameter estimates were 63% [47%, 78%], 13% [1%, 28%], and 24% [22%, 27%], respectively. The heritability of anxiety was higher in girls for self-reported data, but higher in boys for parent-reported data. There was no significant age difference in genetic and environmental contributions for self-reported data, but a significant increase of heritability with age for parent-reported data. Conclusions The trait anxiety in Chinese children and adolescents was highly heritable. Non-shared environmental factors also played an important role. The estimates of genetic and environmental effects differed by rater, sex and age. Our findings largely suggest the cross-cultural generalizability of the etiological model of child and adolescent anxiety. En ligne : http://dx.doi.org/10.1111/jcpp.12310 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=260 [article] Genetic and environmental contributions to anxiety among Chinese children and adolescents – a multi-informant twin study [Texte imprimé et/ou numérique] / Jie CHEN, Auteur ; Jing YU, Auteur ; Xinying LI, Auteur ; Jianxin ZHANG, Auteur . - p.586-594. Langues : Anglais (eng) in Journal of Child Psychology and Psychiatry > 56-5 (May 2015) . - p.586-594 Mots-clés : Anxiety  Chinese children and adolescents  genetic and environmental influence  heritability  twin study Index. décimale : PER Périodiques Résumé : Background Child and adolescent anxiety has become a major public health concern in China, but little was known about the etiology of anxiety in Chinese children and adolescents. The present study aimed to investigate genetic and environmental influences on trait anxiety among Chinese children and adolescents. Rater, sex, and age differences on these estimates were also examined. Methods Self-reported and parent-reported child's trait anxiety was collected from 1,104 pairs of same-sex twins aged 9–18 years. Genetic models were fitted to data from each informant to determine the genetic (A), shared (C), and non-shared environmental (E) influences on trait anxiety. Results The parameter estimates and 95% confidence intervals (CI) of A, C, E on self-reported trait anxiety were 50% [30%, 60%], 5% [0%, 24%], 45% [40%, 49%]. For parent-reported data, the corresponding parameter estimates were 63% [47%, 78%], 13% [1%, 28%], and 24% [22%, 27%], respectively. The heritability of anxiety was higher in girls for self-reported data, but higher in boys for parent-reported data. There was no significant age difference in genetic and environmental contributions for self-reported data, but a significant increase of heritability with age for parent-reported data. Conclusions The trait anxiety in Chinese children and adolescents was highly heritable. Non-shared environmental factors also played an important role. The estimates of genetic and environmental effects differed by rater, sex and age. Our findings largely suggest the cross-cultural generalizability of the etiological model of child and adolescent anxiety. En ligne : http://dx.doi.org/10.1111/jcpp.12310 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=260

A longitudinal twin study of victimization and loneliness from childhood to young adulthood / Timothy MATTHEWS in Development and Psychopathology, 34-1 (February 2022)  

Public ISBD [article]  inDevelopment and Psychopathology > 34-1 (February 2022) . - p.367-377 Titre : A longitudinal twin study of victimization and loneliness from childhood to young adulthood Type de document : Texte imprimé et/ou numérique Auteurs : Timothy MATTHEWS, Auteur ; Avshalom CASPI, Auteur ; Andrea DANESE, Auteur ; Helen L. FISHER, Auteur ; Terrie E. MOFFITT, Auteur ; Louise ARSENEAULT, Auteur Article en page(s) : p.367-377 Langues : Anglais (eng) Mots-clés : bullying  cybervictimization  loneliness  twin study  victimization Index. décimale : PER Périodiques Résumé : The present study used a longitudinal and discordant twin design to explore in depth the developmental associations between victimization and loneliness from mid-childhood to young adulthood. The data were drawn from the Environmental Risk (E-Risk) Longitudinal Twin Study, a birth cohort of 2,232 individuals born in England and Wales during 1994?1995. Diverse forms of victimization were considered, differing across context, perpetrator, and timing of exposure. The results indicated that exposure to different forms of victimization was associated with loneliness in a dose?response manner. In childhood, bullying victimization was uniquely associated with loneliness, over and above concurrent psychopathology, social isolation, and genetic risk. Moreover, childhood bullying victimization continued to predict loneliness in young adulthood, even in the absence of ongoing victimization. Within-twin pair analyses further indicated that this longitudinal association was explained by genetic confounds. In adolescence, varied forms of victimization were correlated with young adult loneliness, with maltreatment, neglect, and cybervictimization remaining robust to controls for genetic confounds. These findings indicate that vulnerability to loneliness in victimized young people varies according to the specific form of victimization in question, and also to the developmental period in which it was experienced. En ligne : http://dx.doi.org/10.1017/s0954579420001005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474 [article] A longitudinal twin study of victimization and loneliness from childhood to young adulthood [Texte imprimé et/ou numérique] / Timothy MATTHEWS, Auteur ; Avshalom CASPI, Auteur ; Andrea DANESE, Auteur ; Helen L. FISHER, Auteur ; Terrie E. MOFFITT, Auteur ; Louise ARSENEAULT, Auteur . - p.367-377. Langues : Anglais (eng) in Development and Psychopathology > 34-1 (February 2022) . - p.367-377 Mots-clés : bullying  cybervictimization  loneliness  twin study  victimization Index. décimale : PER Périodiques Résumé : The present study used a longitudinal and discordant twin design to explore in depth the developmental associations between victimization and loneliness from mid-childhood to young adulthood. The data were drawn from the Environmental Risk (E-Risk) Longitudinal Twin Study, a birth cohort of 2,232 individuals born in England and Wales during 1994?1995. Diverse forms of victimization were considered, differing across context, perpetrator, and timing of exposure. The results indicated that exposure to different forms of victimization was associated with loneliness in a dose?response manner. In childhood, bullying victimization was uniquely associated with loneliness, over and above concurrent psychopathology, social isolation, and genetic risk. Moreover, childhood bullying victimization continued to predict loneliness in young adulthood, even in the absence of ongoing victimization. Within-twin pair analyses further indicated that this longitudinal association was explained by genetic confounds. In adolescence, varied forms of victimization were correlated with young adult loneliness, with maltreatment, neglect, and cybervictimization remaining robust to controls for genetic confounds. These findings indicate that vulnerability to loneliness in victimized young people varies according to the specific form of victimization in question, and also to the developmental period in which it was experienced. En ligne : http://dx.doi.org/10.1017/s0954579420001005 Permalink : https://www.cra-rhone-alpes.org/cid/opac_css/index.php?lvl=notice_display&id=474

The Nature of Covariation Between Autistic Traits and Clumsiness: A Twin Study in a General Population Sample / Sara MORUZZI in Journal of Autism and Developmental Disorders, 41-12 (December 2011)  

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A twin study exploring the association between childhood emotional and behaviour problems and specific psychotic experiences in a community sample of adolescents / S. SHAKOOR in Journal of Child Psychology and Psychiatry, 59-5 (May 2018)  

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Contribution of genes and environment to the longitudinal association between childhood impulsive-aggression and suicidality in adolescence / Massimiliano ORRI in Journal of Child Psychology and Psychiatry, 61-6 (June 2020)  

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Development of ADHD symptoms in preschool children: Genetic and environmental contributions / E. M. EILERTSEN in Development and Psychopathology, 31-4 (October 2019)  

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Exploring the possibility of parents’ broad internalizing phenotype acting through passive gene–environment correlations on daughters’ disordered eating / Shannon M. O’CONNOR in Development and Psychopathology, 34-5 (December 2022)  

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